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1 including after adjustment for baseline left ventricular end-diastolic volume.
2 ed as an increase of 15% or more in the left ventricular end-diastolic volume.
3  dysfunction by reducing the unstressed left ventricular end-diastolic volume.
4 ter mean LVMT was associated with lower left ventricular end-diastolic volume (0.01 mm/mL; P<0.01), a
5 sponse (defined as percent reduction in left ventricular end-diastolic volume 1 year after CRT-D impl
6 s had mildly dilated right ventricles (right ventricular end-diastolic volume=101+/-26 mL/m(2)) with
7  stroke volume (SV) without a change in left ventricular end diastolic volume (102+/-16% change from
8         There was a larger increase in right ventricular end-diastolic volume (15 mL; 95% confidence
9 0.4 +/- 2 to 7.7 +/- 4 mL; p < .05) and left ventricular end-diastolic volume (18.5 +/- 3 to 14.2 +/-
10 with placebo, a significant decrease in left ventricular end-diastolic volume (-18 mL; P=0.009) and e
11  shown by increased E-wave velocity and left ventricular end-diastolic volume, 2) exhibit a higher pl
12  ventricular remodeling (>20% change in left ventricular end-diastolic volume; 21.91 [2.75-174.29]; P
13 tolic pressure by 40% to 60% (p < .05), left ventricular end-diastolic volume 25 +/- 8%, and stroke v
14 a was linearly related to reductions in left ventricular end-diastolic volume (-4.1 ml; 95% confidenc
15 ificantly (P<0.05) greater reduction in left ventricular end-diastolic volume (-49+/-16% versus -35+/
16                                         Left ventricular end-diastolic volume (72.5 to 77.4 mL/m2, P=
17 (54 +/- 10 to 87 +/-6 mL; p < .05) and right ventricular end-diastolic volume (90 +/-11 to 128 +/- 18
18                                        Right ventricular end-diastolic volume (99.9 to 89.7 mL/m2, P<
19 with change in RV ejection fraction and left ventricular end-diastolic volume, although correlation c
20                    On echocardiography, left ventricular end diastolic volume and deceleration time i
21  was evaluated as the change in indexed left ventricular end diastolic volume and LVEF.
22 odilation reflected by a 10% decline in left ventricular end-diastolic volume and a 30% fall in atria
23 ndary end points, including structural (left ventricular end-diastolic volume and left ventricular ej
24 on compared with placebo did not change left ventricular end-diastolic volume and left ventricular ma
25                                 Indexed left ventricular end-diastolic volume and mass correlated wit
26 d 10 weeks after infarction showed that left ventricular end-diastolic volume and mass increased and
27 and 24+/-5 g/m(2); P<0.001) and indexed left ventricular end-diastolic volume and right ventricular e
28 -of-life score, 6-minute walk distance, left ventricular end-diastolic volume, and left ventricular e
29 s showed a borderline (16%) increase in left ventricular end-diastolic volume (angiography), whereas
30     The limits of agreement between the left ventricular end-diastolic volume as estimated by bioimpe
31 me unloading results in a reduction in right ventricular end-diastolic volume, as expected, but left
32 ction fraction (beta=-0.02/%; P=0.015), left ventricular end-diastolic volume (beta=0.01/mL; P<0.0001
33 right ventricular end-diastolic volume (left ventricular end-diastolic volume/body surface area, 104+
34 104+/-13 and 69+/-18 mL/m(2); P<0.001; right ventricular end-diastolic volume/body surface area, 110+
35 r end-diastolic pressure by 30% to 40%, left ventricular end-diastolic volumes by 33 +/- 9%, and tran
36 Pulmonary capillary wedge pressures and left ventricular end-diastolic volumes by use of echocardiogr
37  The relation between stroke volume and left ventricular end-diastolic volume, by the Frank-Starting
38 timates of ventricular filling pressures and ventricular end-diastolic volumes/cardiac performance va
39  interval, -4.02 to -0.23; P=0.028) and left ventricular end-diastolic volume (coefficient, 7.85; 95%
40 end-diastolic pressure (LVEDP), but not left ventricular end-diastolic volume, consistent with increa
41                                         Left ventricular end-diastolic volume decreased (from 125.1 +
42             Cardiac index increased and left ventricular end-diastolic volume decreased significantly
43 ipping led to a significant increase in left ventricular end-diastolic volume, demonstrating increase
44                      The mean change in left ventricular end-diastolic volume differed significantly
45 sociation with an expected decrease in right ventricular end-diastolic volume during lower-body sucti
46                  Patients who increased left ventricular end-diastolic volume during lower-body sucti
47 sonance imaging demonstrated increasing left ventricular end-diastolic volumes, end-systolic volumes,
48 rior hospitalization for heart failure, left ventricular end-diastolic volume &gt;/=125 mL/m(2), and lef
49 emodeling was defined as an increase in left ventricular end-diastolic volume &gt;/=20% at 6 months.
50                             CMR-derived left ventricular end-diastolic volume &gt;246 mL had good, altho
51 r pressure increase and dilatation, but left ventricular end-diastolic volume improved because of red
52                                         Left ventricular end-diastolic volume improved from 172 ml to
53 ts, there was a paradoxical increase in left ventricular end-diastolic volume in association with an
54  mm Hg lower-body suction, on right and left ventricular end-diastolic volumes in 21 patients with ch
55  to 43+/-14 mL/m(2), P<0.0001), whereas left ventricular end diastolic volume increased (66+/-12 to 7
56                                        Right ventricular end diastolic volume increased by 49.4% afte
57 After 17 cycles of trastuzumab, indexed left ventricular end diastolic volume increased in patients t
58                                         Left ventricular end-diastolic volume increased in the rapid
59 ing without surgery at 5 years, 90% for left ventricular end-diastolic volume index <100 mL/m(2) vers
60 us-ICD group had greater improvement in left ventricular end-diastolic volume index (-26.2 versus -7.
61 a brain natriuretic peptide (79 pg/mL), left ventricular end-diastolic volume index (110 mL/m2), and
62 not correlate with six-month changes in left ventricular end-diastolic volume index (p = 0.26), LVESV
63 phy showed a small, stable reduction in left ventricular end-diastolic volume index (P<0.001), with a
64 ncomitant small stable increase in the right ventricular end-diastolic volume index (P<0.001).
65  RV stroke volume index (P<0.0001), and left ventricular end-diastolic volume index (P=0.0015).
66  3.2 +/- 2.9 years, LVEF 0.21 +/- 0.07, left ventricular end-diastolic volume index 180 +/- 64 ml/m2.
67                        Patients with a right ventricular end-diastolic volume index above 150 ml/m(2)
68                        Patients with a right ventricular end-diastolic volume index above 150 ml/m(2)
69 .001) increase in change from baseline right ventricular end-diastolic volume index and a 429 ml (P <
70                The relationship between left ventricular end-diastolic volume index and stroke volume
71                                     The left ventricular end-diastolic volume index decreased (90.1 +
72 ulmonary artery occlusion pressure, and left ventricular end-diastolic volume index failed to correla
73                                 Resting left ventricular end-diastolic volume index increased (P=0.00
74                                        Right ventricular end-diastolic volume index measured by means
75 ry outcome was change from baseline in right ventricular end-diastolic volume index versus placebo.
76 f SCA were PET sympathetic denervation, left ventricular end-diastolic volume index, creatinine, and
77 tion, initial central venous pressure, right ventricular end-diastolic volume index, pulmonary artery
78  B-type natriuretic peptide, and larger left ventricular end-diastolic volume index.
79 er left atrial volume index, and larger left ventricular end-diastolic volume index.
80 t ventricular end-diastolic volume and right ventricular end-diastolic volume (left ventricular end-d
81  zone and its percentage of myocardium, left ventricular end-diastolic volume, left ventricular end-s
82 rdial scar volume by CMR is superior to left ventricular end-diastolic volume, left ventricular end-s
83 cant improvements in ejection fraction, left ventricular end-diastolic volume, left ventricular end-s
84 entricular ejection fraction (LVEF) and left ventricular end diastolic volume (LVEDV) on cardiac magn
85 onist tolvaptan (30 mg/day) on reducing left ventricular end-diastolic volume (LVEDV) compared with p
86            Gated SPECT measurements for left ventricular end-diastolic volume (LVEDV) index, left ven
87 ry capillary wedge pressure (PCWP), SV, left ventricular end-diastolic volume (LVEDV), and left ventr
88                                         Left ventricular end-diastolic volume (LVEDV), left ventricul
89  with CRT, defined as percent change in left ventricular end-diastolic volume (LVEDV), was analyzed i
90 eft ventricular end-systolic volume and left ventricular end-diastolic volume (LVEDV).
91 ratio, 1.16; P=0.002), exercise indexed left ventricular end-diastolic volume (odds ratio, 1.04; P=0.
92 ) was the only predictor of increase in left ventricular end-diastolic volume (p < 0.001).
93  end-diastolic volume, as expected, but left ventricular end-diastolic volume paradoxically increases
94 ular end-diastolic pressure (LVEDP) and left ventricular end-diastolic volume (preload) in CHF rats,
95 thod (n=53, r=0.85, P<0.0001), and with left ventricular end-diastolic volume (r=0.81, P<0.0001).
96 -beam computed tomography for measuring left ventricular end-diastolic volume (r=0.96; standard error
97 splayed an additional decline in the RV/left ventricular end-diastolic volume ratio (P=0.05) and tren
98                                        Right ventricular end-diastolic volume (RVEDV) was reduced in
99 ange or change of 15 to 30 mL in 3DTTE right ventricular end-diastolic volume; sample sizes were 2x t
100 ow obstruction are inversely related to left ventricular end-diastolic volume, stroke volume, and car
101 earts increased relaxation velocity and left ventricular end diastolic volume to produce higher left
102 aval occlusion (VCO) was used to reduce left ventricular end-diastolic volume to 70 +/- 5% of baselin
103 s minor, and a decrease in the ratio of left ventricular end-diastolic volume to body weight reflecte
104 ume (angiography), whereas the ratio of left ventricular end-diastolic volume to body weight was redu
105                                The mean left ventricular end-diastolic volume was 108 +/- 47 mL, as e
106 regurgitant fraction was 34+/-17%, and right ventricular end-diastolic volume was 114+/-39 cc/m(2).
107 genic right ventricular dysplasia, but right ventricular end-diastolic volume was decreased in RyR2(R
108                 However, the change in right ventricular end-diastolic volume was similar in the two
109                                By CMR, right ventricular end-diastolic volumes were 118 +/- 31 cc/m(2
110  At 6 months follow-up, left atrial and left ventricular end-diastolic volumes were significantly mor
111 eat stress did not significantly change left ventricular end-diastolic volume, while ventricular end-
112  most recent echocardiogram, the median left ventricular end-diastolic volume z score was +1.7 (range
113 with published normal values, left and right ventricular end-diastolic volume z scores were mildly en
114 nsion z-score (OR=2.2, P=0.02) or lower left ventricular end-diastolic volume z-score (OR=1.9, P=0.03

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