ライフサイエンスコーパス: ventricular enlargement

1 , along with moderate widespread atrophy and ventricular enlargement.

2 ovide insight into a mechanism of reversible ventricular enlargement.

3 hemorrhage or (2) parenchymal lesions and/or ventricular enlargement.

4 t with cortical thinning/sulcal widening and ventricular enlargement.

5 greater severity, especially with regard to ventricular enlargement.

6 y could be shown in those who developed left ventricular enlargement.

7 Patients did not demonstrate evidence of ventricular enlargement.

8 matter volume deficits (-0.7 SD) and fourth ventricular enlargement (1.6 SD); these abnormalities we

9 , hippocampal atrophy (30 mm3 per year), and ventricular enlargement (565 mm3 per year).

10 scans were calcifications (99%), followed by ventricular enlargement (94%), cortical hypogyration (81

11 Ventricular enlargement, a common in vivo marker of agin

12 umferential function might serve to restrain ventricular enlargement after MI.

13 s with reduced left ventricular function and ventricular enlargement after myocardial infarction are

14 fibrosis, and lipid accumulation, along with ventricular enlargement and cardiac dysfunction in both

15 aracterizing structural progression, such as ventricular enlargement and cardiac dysfunction, in ARVD

16 ed and confirmed by meta-analysis, including ventricular enlargement and decreased cerebral (cortical

17 nd abnormalities in brain morphology such as ventricular enlargement and differences in gray matter d

18 Worse RA LS was also associated with right ventricular enlargement and dysfunction and higher N-ter

19 tethering-factors often influenced by right ventricular enlargement and dysfunction.

20 ficantly less brain atrophy as quantified by ventricular enlargement and preserved cortical volume in

21 eks, the phenotype progressed to marked left ventricular enlargement and severely depressed systolic

22 in cognitive functioning and have detectable ventricular enlargement and some loss of cortical mass.

23 onal magnetic resonance microscopy indicated ventricular enlargement and striatal reduction in both m

24 Diabetes modified the relationship between ventricular enlargement and the risk of HF (P=0.011).

25 ship between cortical gray matter deficit or ventricular enlargements and age at symptom onset or len

26 itulates the primary periventricular lesion, ventricular enlargement, and the secondary cortical defi

27 Hypoxia also correlated significantly with ventricular enlargement, but only among patients (effect

28 ble and progressive process characterized by ventricular enlargement, chamber geometric alterations,

29 ased lung uptake chi2=9.6, P=0.002; and left ventricular enlargement chi2=8.3, P=0.004.

30 Ventricular enlargement due to right ventricular volume

31 t ventricular disease, as estimated by right ventricular enlargement during detailed echocardiography

32 cause previous data showed progressive brain ventricular enlargement for a group of adolescents with

33 Pathological mechanisms underlying ventricular enlargement, however, are likely specific to

34 Progressive left ventricular enlargement, hypocontractility, left atrial

35 The rate of ventricular enlargement in alcoholic patients who mainta

36 s were abnormal in 3 of 10 patients, showing ventricular enlargement in one, periventricular hyperint

37 combined treatment significantly attenuated ventricular enlargement in vivo.

38 Parenchymal lesions/ventricular enlargement increased risk for current atten

39 he entire sample, parenchymal lesions and/or ventricular enlargement increased risk relative to no ab

40 of normal intelligence, parenchymal lesions/ventricular enlargement independently increased risk for

41 Ventricular enlargement is unique to the clinical phenot

42 HF rabbits had left ventricular enlargement (left ventricular end-diastolic

43 Twenty percent (n = 45) had left ventricular enlargement (LVE), defined as LV end-diastol

44 symptomatic family members was based on left ventricular enlargement (LVE).

45 lsequestrin mice at 7 weeks showed mild left ventricular enlargement, mild decreased fractional short

46 structural alterations, including atrial and ventricular enlargement, myofibril disarray, fibrosis an

47 By contrast, fourth ventricular enlargement occurred in schizophrenia even w

48 Nonetheless, striatal shrinkage and ventricular enlargement occurred, and striatal projectio

49 We investigated the prognostic role of right ventricular enlargement on multidetector-row chest CT in

50 Right ventricular enlargement on the reconstructed CT 4-CH vie

51 ho underwent rescreening, two (one with left ventricular enlargement only, one with a left bundle bra

52 scar is rare, an important minority exhibits ventricular enlargement or dysfunction.

53 For example, ventricular enlargement or reduced caudate volume or blo

54 tients with poor outcome had greater lateral ventricular enlargement over time than patients with goo

55 Relationships between ventricular enlargements potentially contributing to cal

56 zed Enalapril Study Evaluating Regression of Ventricular Enlargement (PRESERVE) study was designed to

57 zed Enalapril Study Evaluating Regression of Ventricular Enlargement (PRESERVE) trial after a screeni

58 nd between increase in MSFC and both rate of ventricular enlargement (r=0.31, P<0.05) and increase in

59 Lateral ventricular enlargement represents a potential morphomet

60 We used data from the Survival and Ventricular Enlargement (SAVE) echocardiographic substud

61 ion [LVEF] < or = 40%) from the Survival And Ventricular Enlargement (SAVE) echocardiographic substud

62 thmias in 263 patients from the Survival and Ventricular Enlargement (SAVE) study, using quantitative

63 The Survival And Ventricular Enlargement (SAVE) trial randomized 2231 pat

64 nction and were enrolled in the Survival and Ventricular Enlargement (SAVE) trial were studied.

65 despread deficit in cortical gray matter and ventricular enlargement similar to that seen in men with

66 n cortical gray and white matter volumes and ventricular enlargement similar to those seen in alcohol

67 e study cohort consisted of 727 Survival and Ventricular Enlargement Study patients who underwent car

68 d that continuous decorin infusion prevented ventricular enlargement, such that ventricle size remain

69 recursors) or (2) parenchymal lesions and/or ventricular enlargement (suggestive of white matter inju

70 en, women had surgery rarely for severe left ventricular enlargement (systolic diameter > or = 55 mm

71 rction who were enrolled in the Survival and Ventricular Enlargement trial.

72 ust to anatomical variability, including the ventricular enlargement typically associated with neurol

73 nt in ejection fraction, but persistent left ventricular enlargement was more frequent in group 1.

74 More consistent progressive ventricular enlargement was seen during adolescence for

75 ant cortical gray matter deficit and lateral ventricular enlargement were found in schizophrenic pati

76 Parenchymal lesions/ventricular enlargement were not related to lifetime att