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1 arrest or sustained ventricular tachycardia/ventricular fibrillation).
2 ricular arrhythmias (ventricular tachycardia/ventricular fibrillation).
3 , hyperkalemia, or primary arrhythmia (i.e., ventricular fibrillation).
4 ion MI, and 8.9% had ventricular tachycardia/ventricular fibrillation.
5 ignificant increase in response times to the ventricular fibrillation.
6 nin-angiotensin system (RAS), culminating in ventricular fibrillation.
7 an internal cardiac defibrillator shock for ventricular fibrillation.
8 n has focused on SCD-ventricular tachycardia/ventricular fibrillation.
9 am demonstrates continuous activities during ventricular fibrillation.
10 Late mortality was due to heart failure and ventricular fibrillation.
11 levels of CAR predispose to ischemia-induced ventricular fibrillation.
12 n was initiated after 6 minutes of untreated ventricular fibrillation.
13 surgery, 13.3% respiratory failure, and 8.4% ventricular fibrillation.
14 F hospitalization, HF death, and spontaneous ventricular fibrillation.
15 %) and 6 spontaneous ventricular tachycardia/ventricular fibrillation.
16 ence in dynamic properties between AF and VT/ventricular fibrillation.
17 termination of ventricular tachycardia (VT)/ventricular fibrillation.
18 ccessful appropriate ICD discharges were for ventricular fibrillation.
19 s of polymorphic ventricular tachycardia and ventricular fibrillation.
20 hmias due to Brugada syndrome and idiopathic ventricular fibrillation.
21 e submitted to coronary artery occlusion and ventricular fibrillation.
22 7.6% for all OHCA and 17.7% for OHCA due to ventricular fibrillation.
23 (ER) has recently been linked to idiopathic ventricular fibrillation.
24 h promotes the generation and maintenance of ventricular fibrillation.
25 receding ICD shocks distinguishes AF from VT/ventricular fibrillation.
26 ecular paradigm for some forms of idiopathic ventricular fibrillation.
27 urologic recovery after 15 mins of untreated ventricular fibrillation.
28 pacing at 200 and 300 beats per minute, and ventricular fibrillation.
29 under the most severe hemodynamic condition, ventricular fibrillation.
30 reduces induction of ventricular tachycardia/ventricular fibrillation.
31 after out-of-hospital cardiac arrest due to ventricular fibrillation.
32 tion was initiated after 7 mins of untreated ventricular fibrillation.
33 l effects were greatest in patients found in ventricular fibrillation.
34 d of SCD and 2 adults were resuscitated from ventricular fibrillation.
35 urologic function after 15 mins of untreated ventricular fibrillation.
36 rrest resulting from ventricular tachycardia/ventricular fibrillation.
37 hmic mortality and with an increased risk of ventricular fibrillation.
38 ding polymorphic ventricular tachycardia and ventricular fibrillation.
39 ersion of induced ventricular tachycardia or ventricular fibrillation.
40 icular and papillary muscle VE that triggers ventricular fibrillation.
41 aventricular tachycardias converted to VT or ventricular fibrillation.
42 There were 2 procedural deaths related to ventricular fibrillation.
43 s and in patients with no baseline inducible ventricular fibrillation.
44 cation tools for SCD-ventricular tachycardia/ventricular fibrillation.
45 first recurrence of ventricular tachycardia/ventricular fibrillation.
46 onnectivity that persists until the onset of ventricular fibrillation.
47 d to sudden cardiac arrest due to idiopathic ventricular fibrillation.
48 signals and a simulated patient experiencing ventricular fibrillation.
50 /day], intracranial haemorrhage [20 mg/day], ventricular fibrillation [120 mg/day], septic shock [80
51 ion of anesthesia and 7 minutes of untreated ventricular fibrillation, 16 female 3-month-old swine we
52 Protocol A: After 12 minutes of untreated ventricular fibrillation, 18 pigs were randomized to gro
56 cation revealed shocks were delivered for VT/ventricular fibrillation (62%), AF (23%), and supraventr
57 p < 0.001), had more ventricular tachycardia/ventricular fibrillation (81% vs 46%; p < 0.001), shorte
59 often presented with ventricular tachycardia/ventricular fibrillation, a witnessed arrest, or had bys
60 ied out a study to compare response times to ventricular fibrillation across five patient loads: 16,
61 e, a 21q21 locus is strongly associated with ventricular fibrillation after myocardial infarction.
62 At 6 months, 1 patient in each group had ventricular fibrillation and 1 patient in each group die
63 4 and 17 seconds) for shocks that terminated ventricular fibrillation and 7 seconds (25th and 75th pe
64 ality is predicted by early recurrence of VT/ventricular fibrillation and ablation for electrical sto
65 iatric HCM proband with multiple episodes of ventricular fibrillation and aborted sudden cardiac deat
66 ssociated with lower prevalence of recurrent ventricular fibrillation and better postresuscitation my
67 ral highly dangerous heart diseases, such as ventricular fibrillation and congestive heart failure.
68 most episodes of ventricular tachycardia or ventricular fibrillation and decrease mortality in speci
70 of repetitive APs, leading in some cases to ventricular fibrillation and hemodynamic collapse in viv
71 nomic outflow significantly delayed terminal ventricular fibrillation and lengthened the duration of
72 pigs were subjected to 8.5 mins of untreated ventricular fibrillation and prospectively randomized to
73 ctrical activity in 874 children (84.8%) and ventricular fibrillation and pulseless ventricular tachy
74 ulseless electrical activity) and shockable (ventricular fibrillation and pulseless ventricular tachy
75 quisite for aging-dependent QT prolongation, ventricular fibrillation and SCD immediately after trans
76 t uncalibrated dynamic patterns of Im during ventricular fibrillation and show that Im at singularity
79 lood flow were measured in seven pigs before ventricular fibrillation and then following 6 minutes of
80 val of patients resuscitated from near-fatal ventricular fibrillation and those with sustained ventri
81 ined as aborted cardiac arrest or documented ventricular fibrillation and ventricular tachycardia (la
82 ac alternans has been linked to the onset of ventricular fibrillation and ventricular tachycardia, le
83 thetized pigs underwent 15 mins of untreated ventricular fibrillation and were subsequently randomize
84 th, ischemic stroke, ventricular tachycardia/ventricular fibrillation, and heart failure hospitalizat
87 ce of ventricular tachyarrhythmia, including ventricular fibrillation, and shock treatment was assess
89 for VTA (including ventricular tachycardia, ventricular fibrillation, and ventricular flutter) was c
90 laglutide 0.75 mg group); cardiogenic shock; ventricular fibrillation; and an unknown cause (n=3 in t
91 ntable cardioverter defibrillator shocks for ventricular fibrillation; and n=8 without implantable ca
93 g per se, but may increase susceptibility to ventricular fibrillation) are found to be associated wit
97 of presentation with ventricular tachycardia/ventricular fibrillation as opposed to pulseless electri
100 DE, and right ventricular DE for death or VT/ventricular fibrillation at mean follow-up of 48 months
102 icular beats and prevented fatal episodes of ventricular fibrillation, but did not prevent QT prolong
103 termination of rapid ventricular tachycardia/ventricular fibrillation can reasonably be considered an
104 (eg, induced hypothermia for out-of-hospital ventricular fibrillation cardiac arrest and birth asphyx
107 minutes of cardiopulmonary resuscitation for ventricular fibrillation cardiac arrest improves short-t
108 diopulmonary resuscitation is feasible after ventricular fibrillation cardiac arrest in rats and impr
113 mary effectiveness end point was the induced ventricular fibrillation conversion rate compared with a
114 s 99%, and sensitivity analysis of the acute ventricular fibrillation conversion rate was >90% in the
115 f 88%, with success defined as 2 consecutive ventricular fibrillation conversions of 4 attempts.
116 ominant frequency (13.0 versus 10.0 Hz), and ventricular fibrillation duration (160 versus 80 s) were
117 tion restitution (APDR) curve and can reduce ventricular fibrillation duration in failing ventricles.
118 A recent genome-wide association study of ventricular fibrillation during acute myocardial infarct
121 on and then following 6 minutes of untreated ventricular fibrillation during sequential 5 minutes tre
122 eath, sustained ventricular tachycardia, and ventricular fibrillation either documented on ECG or rec
124 =1 ICD-treated ventricular tachycardias (VT)/ventricular fibrillation episode, or a recorded, sustain
125 A total of 34 ventricular tachyarrhythmia/ventricular fibrillation episodes (20 induced; 14 sponta
126 ents with repetitive ventricular tachycardia/ventricular fibrillation episodes alternating with perio
128 290 eligible patients with no arrhythmia or ventricular fibrillation/flutter (CL<200 ms) induced in
131 up of 16 pigs underwent 10 mins of untreated ventricular fibrillation followed by 3 mins of chest com
133 ge-free survival for ventricular tachycardia/ventricular fibrillation >/= 240 beats per minute was eq
135 lly intact survival of patients with OHCA in ventricular fibrillation improved in 2 rural counties in
136 test RR interval </=250 ms) in 0% to 9%, and ventricular fibrillation in 0% to 2%, most of whom were
138 ge of 65 +/- 12 years, a previous history of ventricular fibrillation in 37% of the cases, and a mean
139 tely to terminate ventricular tachycardia or ventricular fibrillation in 43 of 224 patients (19%) ove
144 s are critical in sustaining both atrial and ventricular fibrillation in the human heart and its impl
147 hed reports of infants <3 months of age with ventricular fibrillation in which a primary diagnosis co
149 trict SCD hereafter to cardiac arrest due to ventricular fibrillation, including rhythms shockable by
150 During ischemia, ventricular tachycardia/ventricular fibrillation inducibility was larger in WT t
151 disappearance and no ventricular tachycardia/ventricular fibrillation inducibility without complicati
155 inidine for atrial fibrillation who develops ventricular fibrillation is diagnosed with "acquired LQT
156 Understanding the mechanisms that drive ventricular fibrillation is essential for developing imp
157 tion testing by induction and termination of ventricular fibrillation is widely done at the time of i
160 e genetic defect in a family with idiopathic ventricular fibrillation (IVF) manifesting in childhood
161 tions included pulmonary hemorrhage (n = 2), ventricular fibrillation (n = 1), and stent migration (n
162 on and included atrial fibrillation (n = 4), ventricular fibrillation (n = 6), supraventricular tachy
163 and Lange-Nielsen syndrome (n=5), idiopathic ventricular fibrillation (n=4), left ventricular noncomp
164 s, such as SCD (n=1), aborted SCD because of ventricular fibrillation (n=6), sustained ventricular ta
165 n = 36; polymorphic ventricular tachycardia/ventricular fibrillation, n = 14; other, n = 11), includ
166 ich a cluster of ventricular tachycardias or ventricular fibrillation, negatively affects short- and
168 the majority of SCD-ventricular tachycardia/ventricular fibrillation occurs in patients without know
172 associated with ventricular tachycardia (VT)/ventricular fibrillation or death in patients with cardi
173 pulseless electrical activity, and 20.7% had ventricular fibrillation or pulseless ventricular tachyc
176 or cardiac arrests at home, the incidence of ventricular fibrillation or pulseless ventricular tachyc
177 of-hospital cardiac arrest, shock-refractory ventricular fibrillation or pulseless ventricular tachyc
178 g and whose initial cardiac-arrest rhythm of ventricular fibrillation or pulseless ventricular tachyc
179 hospital cardiac arrest for shock-refractory ventricular fibrillation or pulseless ventricular tachyc
180 rdiac arrest due to initial shock-refractory ventricular fibrillation or pulseless ventricular tachyc
181 sudden cardiac death or nonfatal episode of ventricular fibrillation or sustained ventricular tachyc
182 ifekalant (NIF) are used in the treatment of ventricular fibrillation or tachycardia; however, only f
183 ned as arrhythmic death or ICD discharge for ventricular fibrillation or ventricular tachycardia >240
184 lantable cardiac defibrillator discharge for ventricular fibrillation or ventricular tachycardia >240
187 th, ischemic stroke, ventricular tachycardia/ventricular fibrillation, or heart failure hospitalizati
188 There were no cases of torsades de pointes, ventricular fibrillation, or polymorphic or sustained ve
189 ths who died due to ventricular tachycardia, ventricular fibrillation, or sudden unexpected death, or
190 aborted SCD (appropriate ICD shock, nonfatal ventricular fibrillation, or sustained ventricular tachy
192 h was associated with early recurrence of VT/ventricular fibrillation (P=0.003) and ablation for elec
197 ardia (hazard ratio [HR]: 1.65, p < 0.0001), ventricular fibrillation/polymorphic ventricular tachyca
198 rm survival in patients with shock-resistant ventricular fibrillation /pulseless ventricular tachycar
199 om out-of-hospital cardiac arrest because of ventricular fibrillation, pulseless ventricular tachycar
200 rm and long-term survival in shock-resistant ventricular fibrillation/pulseless ventricular tachycard
201 ockable rhythms evolve to shockable rhythms (ventricular fibrillation/pulseless ventricular tachycard
202 ntaneous episodes of ventricular tachycardia/ventricular fibrillation recorded in 21 patients (6.7%),
206 icial circulation and defibrillation to halt ventricular fibrillation remain of paramount importance
207 5, PKP2-p.Arg79X, and the Chr7q36 idiopathic ventricular fibrillation risk haplotype) in a cohort of
209 ccess was seen in 17 of 19 procedures, and a ventricular fibrillation storm occurred within 24 hours
210 eant for identifying atrial fibrillation and ventricular fibrillation substrate may allow us to recor
211 nsplantation, survived sudden cardiac death, ventricular fibrillation, sustained ventricular tachycar
212 OHCA survival from shockable arrhythmias (ventricular fibrillation/ tachycardia) improved in sever
214 BCE was defined as either (1) an appropriate ventricular fibrillation-terminating implantable cardiov
215 eizures, aborted cardiac arrest, appropriate ventricular fibrillation-terminating implantable cardiov
216 tion between pauses for all reasons and both ventricular fibrillation termination and patient surviva
219 an IHCA caused by ventricular tachycardia or ventricular fibrillation, the rate of 1-year survival wa
220 acing of 200 and 300 beats per minute, under ventricular fibrillation, the right atrium-aorta system
222 e more prevalent in patients with idiopathic ventricular fibrillation, their ability to predict cardi
223 cetylcholine analogue carbamylcholine raises ventricular fibrillation threshold (VFT) and flattens th
224 stable analogue of acetylcholine could raise ventricular fibrillation threshold (VFT), and whether th
225 Purkinje origin VE was identified as the ventricular fibrillation trigger in 6 of 6 cardiac arres
226 nt was occurrence of ventricular tachycardia/ventricular fibrillation-triggered ICD interventions (sh
227 hs, 230-740 ms, arrhythmic storm n=28) and 2 ventricular fibrillation triggers were targeted (25 tran
228 All but 6 of the 66 patients with inducible ventricular fibrillation underwent electrophysiological
229 ulmonary resuscitation 37%; initial rhythms: Ventricular fibrillation/ventricular tachycardia 24%, pu
230 t failure symptoms, shockable arrest rhythm (ventricular fibrillation/ventricular tachycardia), and S
231 fibrillation (AF) (n = 13), or scar-related ventricular fibrillation (VF) (n = 3) were examined.
232 ving documented ventricular tachycardia (VT)/ventricular fibrillation (VF) and Brugada syndrome-relat
233 erized a family presenting with a history of ventricular fibrillation (VF) and sudden death without e
234 Over the last decades, a gradual decrease in ventricular fibrillation (VF) as initial recorded rhythm
235 ized that dantrolene improves survival after ventricular fibrillation (VF) by rectifying the calcium
236 the hypothesis that PH promotes spontaneous ventricular fibrillation (VF) during a critical post-PH
237 Spontaneous ventricular tachyarrhythmia (VT)/ventricular fibrillation (VF) events (n=111) were treate
238 endocardial activation during long-duration ventricular fibrillation (VF) exhibits organized activit
240 n (APD) shortening and recurrent spontaneous ventricular fibrillation (VF) in failing ventricles.
241 incidence of ventricular tachycardia (VT) or ventricular fibrillation (VF) in patients with heart fai
242 tested the hypothesis that ischemia-induced ventricular fibrillation (VF) is facilitated by platelet
243 defined as a rate faster than 180 beats/min)/ventricular fibrillation (VF) or death (primary endpoint
244 e the percentage of patients who experienced ventricular fibrillation (VF) or potentially malignant a
245 l cardiac arrest has raised the concern that ventricular fibrillation (VF) recurrence may be triggere
248 ess the feasibility of terminating sustained ventricular fibrillation (VF) via light-induced excitati
251 D) or sustained ventricular tachycardia (VT)/ventricular fibrillation (VF) with appropriate ICD thera
253 aimed to study whether AF is associated with ventricular fibrillation (VF), the most common cause of
254 eart can lead to cardiac arrhythmias such as ventricular fibrillation (VF), which in turn compromise
263 atients with refractory out-of-hospital (OH) ventricular fibrillation (VF)/ventricular tachycardia (V
264 A are associated with conduction disease and ventricular fibrillation (VF); however, the mechanisms t
266 ognosis of sustained ventricular tachycardia/ventricular fibrillation (VT/VF) in contemporary non-ST-
269 tricular arrhythmia (ventricular tachycardia/ventricular fibrillation [VT/VF]), stage C heart failure
270 ccuracy of sustained ventricular tachycardia/ventricular fibrillation (VTs/VF) inducibility and to id
271 th initial rhythm of ventricular tachycardia/ventricular fibrillation was 56%, of whom 82 had a compl
272 Inducibility of ventricular tachycardia/ventricular fibrillation was associated with an increase
273 or IHCA caused by ventricular tachycardia or ventricular fibrillation was associated with higher rate
275 stimulation-induced ventricular tachycardia/ventricular fibrillation was equivalent in all groups (P
276 illation in patients who were found to be in ventricular fibrillation was included in the propensity
277 7 mins following endotracheal tube clamping, ventricular fibrillation was induced and remained untrea
286 rate terminating ventricular tachycardia or ventricular fibrillation was the same in patients who un
288 ricular tachycardia (VT) >/=240 beats/min or ventricular fibrillation, was a younger age at enrollmen
289 zed by renin and norepinephrine overflow and ventricular fibrillation, was potentiated ( approximatel
290 r ICD discharges for ventricular tachycardia/ventricular fibrillation were equivalent to an aborted d
292 used by pulseless ventricular tachycardia or ventricular fibrillation were stratified by prompt (</=2
294 efibrillation for ventricular tachycardia or ventricular fibrillation) were analyzed with multivariat
295 illation: A Validation on 1617 Patients With Ventricular Fibrillation," which was published in the Fe
296 ss electrical activity/asystole or resistant ventricular fibrillation who were treated with mild ther
297 ttempted to defibrillate after 10 seconds of ventricular fibrillation with a single approximately 30-
298 After prolonged ventricular tachycardia/ventricular fibrillation without resuscitation, asystole
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