ライフサイエンスコーパス: ventricular function

1 result in symptom resolution and recovery of ventricular function.

2 rtality, imparting a negative impact on left ventricular function.

3 recent-onset heart failure and reduced left ventricular function.

4 terms of clinical events or changes in left ventricular function.

5 ng both improved systolic and diastolic left ventricular function.

6 mine their association with aortic valve and ventricular function.

7 xperimental studies metformin preserves left ventricular function.

8 12]), after adjustment for clinical risk and ventricular function.

9 se remodeling without adverse effect on left ventricular function.

10 and mortality in patients with impaired left ventricular function.

11 predictors of recurrence and impact on left ventricular function.

12 onary arterial compliance, and reduced right ventricular function.

13 reatment should focus on the preservation of ventricular function.

14 aching of cardiac physiology focuses on left ventricular function.

15 mplantation on the basis of symptoms or left ventricular function.

16 the relationship between adverse events and ventricular function.

17 endent of the concurrent improvement in left ventricular function.

18 old-standard technique for the assessment of ventricular function.

19 gnificantly from accepted donors with normal ventricular function.

20 hypothesis that TRPM7 is required for normal ventricular function.

21 mproved in parallel with improvement in left ventricular function.

22 ents with symptoms at rest and impaired left ventricular function.

23 ype 1, but rather an improved diastolic left ventricular function.

24 on reperfusion, myocardial remodelling, and ventricular function.

25 able multivessel CAD, and preserved systolic ventricular function.

26 ive coronary artery disease or impaired left ventricular function.

27 coronary artery disease and normal systolic ventricular function.

28 ge acquisition plane, demographics, and left ventricular function.

29 ry, is quite high and is influenced by right ventricular function.

30 e correlated with sensitive measures of left ventricular function.

31 (AMI) has led to limited improvement in left ventricular function.

32 g pulmonary hemodynamic parameters and right ventricular function.

33 on and are sometimes associated with reduced ventricular function.

34 ular remodeling and regional and global left ventricular function.

35 dequately address the effect of PVCs on left ventricular function.

36 ered, its severity, and its effects on right ventricular function.

37 blood pressure, systolic, or diastolic left ventricular function.

38 ship between its protein levels and systolic ventricular function.

39 ationship with age, sex, body size, and left ventricular function.

40 sudden death, and in patients with poor left ventricular function.

41 e in interstitial fibrosis and a decrease in ventricular function.

42 erstitial fibrosis, leading to improved left ventricular function.

43 gradation pattern differed depending on left ventricular function.

44 asuring annulus diameter and valve and right ventricular function.

45 nty-two (65%) had a preserved left and right ventricular function.

46 nse to post-acute myocardial infarction left ventricular functions.

47 the gold standard in the evaluation of left ventricular functions.

48 software programs were used to compare left ventricular functions.

49 ft survival from accepted donors with normal ventricular function (10.6 years) was similar to surviva

50 weaker in severe CHF compared to normal left ventricular function; 2) increasing HRR using rate-adapt

51 rveillance of cardiomyopathy (45 [10.5%]) or ventricular function (36 [8.4%]).

52 ar size (94% versus 80%; P=0.001), and right ventricular function (87% versus 73%; P=0.006).

53 donors with moderately-to-severely depressed ventricular function (9.1 years; P=0.13).

54 l from accepted donors with mildly depressed ventricular function (9.7 years; P=0.24) and from accept

55 including assessment of regional and global ventricular function, absolute myocardial blood flow qua

56 All had rapid and complete recovery of ventricular function after intravenous corticosteroids.

57 r formation, thereby augmenting cardiac left ventricular function after ischemia/reperfusion.

58 humans that myocardial infarct size and left ventricular function after STEMI have a circadian depend

59 aphic assessment of myocardial mass and left ventricular function also did not suggest myocardial atr

60 (R3K5A) mice leads to an improvement in left ventricular function and a partial rescue of left ventri

61 state of lipotoxicity that compromises left ventricular function and aggravate heart failure.

62 ) apical pacing may lead to deterioration in ventricular function and an increased risk of heart fail

63 myocardial infarction model can improve left ventricular function and attenuate cardiac remodeling.

64 ion were accompanied by improvements in left ventricular function and attenuation of left ventricle r

65 ardiopoietic stem cell therapy improves left ventricular function and blunts pathological remodeling.

66 ith recent-onset heart failure, reduced left ventricular function and clinically suspected myocarditi

67 The findings of reduced ventricular function and conduction system defects in My

68 VB3-infected mice developed an impaired left ventricular function and displayed an increased left ven

69 enital AS frequently have impaired diastolic ventricular function and exercise capacity that may be r

70 (CHD) and investigate its relationship with ventricular function and exercise capacity.

71 ed dopamine pharmacokinetics as well as left ventricular function and global hemodynamics after dopam

72 ited myocardial infarct size, preserved left ventricular function and had no influence on haemodynami

73 , providing a rationale for focused study of ventricular function and heart failure after targeting t

74 rdial infarction complicated by reduced left ventricular function and HF.

75 more adverse cardiac remodeling, poorer left ventricular function and higher mortality by increasing

76 rs vs. 52.3 years), had worse right and left ventricular function and higher pulmonary artery pressur

77 gnificant cardiovascular alterations in left ventricular function and in aortic stiffness occur durin

78 is associated with early impairment of left ventricular function and increased myocardial oxidative

79 horacic epidural anesthesia may affect right ventricular function and interfere with the coupling bet

80 ired congenital heart disease with preserved ventricular function and isthmus-dependent re-entry, VT

81 patients scheduled for lung resection, right ventricular function and its response to increased after

82 Regional left ventricular function and late-gadolinium enhancement wer

83 Parameters of left and right ventricular function and LGE burden were measured in 205

84 ficit in human HCM with normal systolic left ventricular function and may contribute to HCM disease p

85 lot underwent cardiac magnetic resonance for ventricular function and metabolic exercise testing.

86 ing 2 CMR studies with a 2-year interval for ventricular function and MF assessment.

87 c complications; and (5) stable, normal left ventricular function and New York Heart Association func

88 IL-10 administration markedly improved left ventricular function and not only inhibited but also rev

89 In isolated perfused heart experiments, left ventricular function and oxygen consumption were similar

90 Despite comparable right ventricular function and pulmonary regurgitant fraction,

91 with unexplained cardiac arrest (normal left ventricular function and QT interval) and selected famil

92 Echocardiographic parameters of right ventricular function and RA area predict mortality in Ei

93 lure (HF) therapy documented to improve left ventricular function and reduce mortality.

94 We assessed glucose metabolism, ventricular function and remodeling, and cardiac gene ex

95 reduce its hemodynamic repercussions on left ventricular function and remodeling, and improve clinica

96 nd interfere with the coupling between right ventricular function and right ventricular afterload.

97 ed by echocardiographic measurements of left ventricular function and structure.

98 me, whereas other risk factors, such as left ventricular function and surgery on the thoracic aorta,

99 It also improves left ventricular function and survival in mice (n=20-22 per g

100 particular focus on those with improved left ventricular function and those who have not experienced

101 cts of thoracic epidural anesthesia on right ventricular function and ventricular-pulmonary coupling.

102 Secondary end points were left ventricular function and volumes by magnetic resonance i

103 eloping standard methods for assessing right ventricular function and, hopefully, its coupling to pul

104 larization priority, diabetes mellitus, left ventricular function, and 3-vessel disease.

105 cal variables, perfusion defect scores, left ventricular function, and absolute myocardial flow reser

106 rt Association functional class 3, poor left ventricular function, and age (all p < 0.001) along with

107 , and the remaining patients had normal left ventricular function, and all but 1 have New York Heart

108 , sex, comorbidities, insurance status, left ventricular function, and aortic stenosis severity betwe

109 with rapid changes in pulsatile blood flow, ventricular function, and downstream impedance in both i

110 Mdivi-1 improves exercise capacity, right ventricular function, and hemodynamics in experimental P

111 attenuates cardiac fibrosis, preserves left ventricular function, and improves survival in a mouse m

112 ilar improvements in clinical symptoms, left ventricular function, and left ventricular reverse remod

113 diovascular parameters (carotid plaque, left ventricular function, and myocardial late gadolinium enh

114 becomes limiting remodeling, preserving left ventricular function, and preventing heart failure.

115 sibility by high-resolution ultrasound, left ventricular function, and quality of life were measured

116 sment reveals normal coronary arteries, left ventricular function, and resting ECG.

117 olution, thromboembolic risk, preserved left ventricular function, and severely decreased levels of a

118 right ventricular systolic pressures, right ventricular function, and survival.

119 ardiographic parameters to evaluate systolic ventricular function are affected by loading conditions.

120 Potential donor hearts with depressed ventricular function are often declined.

121 ic resonance imaging (MRI) for assessment of ventricular function as a primary endpoint.

122 agnetic resonance imaging and change in left ventricular function as affected by timing of treatment

123 infected patients showed alterations in left ventricular function as demonstrated by a lower ejection

124 imals recovered 85.4% of their baseline left ventricular function as measured by 2-dimensional transt

125 lure and with reduced systolic and diastolic ventricular function as shown by echocardiography, where

126 secondary end points included regional left ventricular function assessed by magnetic resonance imag

127 s 10.9+/-2.3 cm/s; P=0.01) and regional left ventricular function, assessed by peak systolic velocity

128 n at discharge and 1 month with normal right ventricular function at 1 year.

129 1/HGF reduced infarct size and improved left ventricular function at 2 months after AMI.

130 iPSC-EV-treated mice exhibited improved left ventricular function at 35 d after myocardial infarction

131 determine the effects of this system on left ventricular function at 4 months after AMI.

132 which translated into impaired systolic left ventricular function at 6 to 12 months post TAVR (p < 0.

133 treperfusion and significantly improved left ventricular function at 7 days after myocardial ischemia

134 rchitectural regeneration and restoration of ventricular function at day 60 (P < 0.05, n = 6-14).

135 ew LE feature a significant decrease in left ventricular function at discharge.

136 mm Hg/mL/m(2)), and worse longitudinal left ventricular function (basal longitudinal strain=-11.6+/-

137 No difference was found in ventricular function between nonrejection and rejection

138 rdial infarction with severely impaired left ventricular function but no inducible ventricular tachyc

139 hearts had comparable chamber dimensions and ventricular function, but as early as 1 week post-MI, KO

140 ) has been used extensively to quantify left ventricular function, but its value for RV assessment ha

141 as an increase in frequency of impaired left ventricular function, but there was no change in Logisti

142 DM2 is already detectable in preserved left ventricular function by cardiovascular magnetic resonanc

143 Doppler-derived pulse-wave velocity and left ventricular function by echocardiography.

144 on could synchronize contraction and restore ventricular function by electrically connecting isolated

145 toms and objective measures of the valve and ventricular function by transthoracic echocardiography.

146 ive measurement of aortic valve gradient and ventricular function by transthoracic echocardiography.

147 jects underwent assessment of diastolic left ventricular function (cine magnetic resonance imaging vo

148 ted a significantly greater increase in left ventricular function compared with all the other groups

149 ffect on recovery of global or regional left ventricular function compared with placebo.

150 fibroblasts, reduced scar size, and improved ventricular function compared with wild-type mice.

151 nscriptional profile in adulthood, impairing ventricular function, conduction, and repolarization.

152 Measure of right-ventricular function could improve the risk stratificati

153 matic severe aortic stenosis and normal left ventricular function, current practice guidelines empiri

154 Although systolic right and left ventricular function declined, peak exercise capacity re

155 ntation may be associated with improved left ventricular function, decreased N-terminal pro B-type na

156 functional class 3 or 30-d death), and left ventricular function did not differ between two groups (

157 o-B-type natriuretic peptide, left and right ventricular function) differed between both groups.

158 nd invasive hemodynamics were used to assess ventricular function, dimensions and vascular mechanical

159 highlights the interest of monitoring right ventricular function during high-frequency oscillatory v

160 ardiac output (Qc, foreign-gas rebreathing), ventricular function (echocardiography and cardiac MRI)

161 Left ventricular function (echocardiography), clinical status

162 Left ventricular function, edema, vascular permeability, and

163 rate aortic regurgitation and preserved left ventricular function (ejection fraction >/=55%).

164 essure (mean >15 mm Hg; p = 0.04), decreased ventricular function (ejection fraction <55%; p = 0.03),

165 Patients with new LE had a decreased left ventricular function (ejection fraction: pre, 55.5 +/- 1

166 cs, risk factors, coronary anatomy, and left ventricular function, end-systolic volume index and B-ty

167 e acute cor pulmonale determination and left ventricular function estimation.

168 upture and the significant reduction in left ventricular function evident as early as 1 week post-MI,

169 nsplantation may be associated with improved ventricular function, exercise tolerance, and long-term

170 stablishes coronary blood flow, and improves ventricular function following an AMI.

171 es generation, myocardial necrosis, and left ventricular function following ischemia/reperfusion inju

172 ificant differences in change in global left ventricular function for patients treated at day 3 (-0.9

173 Improvements in left ventricular function, functional status, and quality of

174 Ventricular function, geometry, and regional strain were

175 Patients with preserved ventricular function had a higher rate of transplant-fre

176 iming of cell delivery on the effect on left ventricular function has not been analyzed.

177 ratio: 1.655; p < 0.001) and impaired right ventricular function (hazard ratio: 2.360; p = 0.001).

178 vision of volumes, diastolic function, right ventricular function, hemodynamics, and valvular regurgi

179 nts with heart failure (HF) who recover left ventricular function (HF-Recovered) have a distinct clin

180 n was significantly associated with impaired ventricular function, highlighting the importance of thi

181 Left ventricular function, histochemical injury, and differen

182 However, activity reduced left ventricular function (ie, ejection and shortening fracti

183 ischemia and reperfusion, postischemic left ventricular function improved commensurately with an inc

184 sonance imaging was performed to assess left ventricular function in 20 women with signs and symptoms

185 creases apoptosis, and greatly enhances left ventricular function in a model of ischemic cardiomyopat

186 mokine during a 4-week period that preserves ventricular function in a rat model of myocardial infarc

187 dothelial progenitor cells and improves left ventricular function in a rat model of myocardial infarc

188 y managed aortic stenosis and preserved left ventricular function in an era where surgical aortic val

189 ss its effect on pulmonary insufficiency and ventricular function in an ovine model of chronic postop

190 -1 receptor antagonist, on coronary and left ventricular function in coronary artery disease (CAD) pa

191 lobal (LVEF) and regional (wall motion) left ventricular function in infarct and border zones at 6 mo

192 at cardiomyocytes and significantly improves ventricular function in mice with heart failure.

193 Tissue Doppler showed better diastolic left ventricular function in mutation carriers (e', P=0.001;

194 n and CVB3 copy number, and an improved left ventricular function in NOD2(-/-) CVB3 mice compared wit

195 apnoea and arrhythmia incidence and improve ventricular function in pacing-induced CHF rabbits.

196 ivation of DN T cells, as well as with worse ventricular function in patients with Chagas disease.

197 (Evaluating Xience and Left Ventricular Function in Percutaneous Coronary Interventi

198 The EXPLORE (Evaluating Xience and Left Ventricular Function in Percutaneous Coronary Interventi

199 The full scorpion venom increased left ventricular function in sedated mice in vivo, prolonged

200 cardiomyocytes, we found severely depressed ventricular function in the Gata4-ablated mice (mutant)

201 fice area, 38+/-18 mm(2)) and preserved left ventricular function in whom initial management was expe

202 loss of myocardial tissue and depressed left ventricular function in WT mice.

203 fractional shortening and other measures of ventricular function, indicating that miR combo promotes

204 In vivo secretoneurin improved left ventricular function, inhibited remodeling, and reduced

205 Baseline left ventricular function is a marker for prognosis regardles

206 Although right-ventricular function is an important determinant of card

207 We demonstrate that systemic right ventricular function is preserved (and may be improved)

208 feasible in TGA patients, but its effect on ventricular function is unclear.

209 The use of donors with depressed ventricular function is uncommon in pediatric heart tran

210 hy but exhibited significantly improved left ventricular function, less cardiac fibrosis and cardiomy

211 on of NaHS improved survival, preserved left ventricular function, limited infarct size, and improved

212 ment in AF symptoms, o2peak, left atrial and ventricular function, lipid levels, and QoL.

213 vs. 14%; p = 0.0027, respectively), and left ventricular function </=35% (26% vs. 10%; p = 0.0071).

214 Left ventricular function <30% was observed in 44.1% of patie

215 Hearts from donors with depressed ventricular function may be considered in selected patie

216 blation before ICD implantation because left ventricular function may improve, consequently decreasin

217 nonsurgical candidates with compromised left ventricular function, MitraClip therapy could be conside

218 transplantation significantly improved left ventricular function, myocardial metabolism, and arterio

219 erior descending coronary disease and normal ventricular function (n=13) were recruited prospectively

220 l retention rates and greater improvement in ventricular function, N-terminal pro-brain natriuretic p

221 e.g., beta-blockers) partially preserve left ventricular function, novel strategies are needed to lim

222 nsfer may thus underlie the early decline in ventricular function observed in diabetic cardiomyopathy

223 ignificant treatment effect on regional left ventricular function observed in either infarct or borde

224 Whether the improved left ventricular function observed in the Aldo-DHF trial is o

225 Autonomic control of ventricular function occurs through regulation of cAMP-d

226 TR2), were less likely to have improved left ventricular function (odds ratio: 0.40 per minor allele;

227 atus or that the deleterious effects on left ventricular function offset any salutary vascular effect

228 hibitor therapy in patients with normal left ventricular function on the progression of myocardial fi

229 a reported no effect of cell therapy on left ventricular function or clinical outcome.

230 on of autologous BMNCs does not improve left ventricular function or quality of life in patients with

231 mbined clinical scores and measures of right-ventricular function or strain.

232 re denied surgery due to increased age, poor ventricular function, or associated comorbidities, putti

233 rategy does not affect mortality, interstage ventricular function, or interstage AVVR in children wit

234 , cardiogenic shock, severely depressed left ventricular function, or ST segment elevation myocardial

235 , new PPI did have a negative effect on left ventricular function over time.

236 the most important factor in improving left ventricular function (p < 0.001) and 30-day combined end

237 myocardial salvage (P<0.001), impaired left ventricular function (P<0.001), and more pronounced repe

238 ng status (P<0.0001), severely impaired left ventricular function (P<0.01), pulmonary hypertension an

239 placebo pigs had persistently depressed left ventricular function (P<0.05).

240 n independent predictor of recovery of right ventricular function (p=0.02).

241 ular hypertrophy, but systolic and diastolic ventricular function parameters were similar in diabetic

242 th CA results in significant improvements in ventricular function, particularly in the absence of ven

243 arkedly limits remodeling and preserves left ventricular function post-MI.

244 Left ventricular function predicts cardiovascular mortality b

245 t had no significant effect on regional left ventricular function recovery.

246 ociated with progressive impairment of right ventricular function, reduced exercise capacity and a po

247 Ventricular function remodeling was similar in Cav3.1(-/

248 myopathy characterized by impaired diastolic ventricular function resulting in a poor clinical progno

249 entilation+10 and +15 further worsened right ventricular function, resulting in about a 40% increase

250 Impaired ventricular function (right or left ventricular ejection

251 patients who presented a worsening of right ventricular function (right ventricular end-diastolic ar

252 Optimal thresholds for ventricular function (RV EF <30%: hazard ratio, 3.90; 95

253 were associated with measures of worse right ventricular function (RV s', r=-0.39, P<0.0001) and pred

254 Right ventricular function (RVF) is an important determinant o

255 AFCA provides long-term benefits on left ventricular function, significantly reducing the number

256 accurately and noninvasively determine right ventricular function, such as cardiac magnetic resonance

257 cluded cine steady-state free precession for ventricular function, T2* for myocardial iron quantifica

258 cantly compromise left ventricular and right ventricular function through different mechanisms that a

259 linking fibrosis, inflammation, and impaired ventricular function to DPP4 inhibition in preclinical s

260 y artery disease and severely depressed left ventricular function to intra-aortic balloon pump (IABP)

261 2 weeks at multivariable analysis: (1) right ventricular function (tricuspid annular plane systolic e

262 activation in CHF patients with reduced left-ventricular function using SPECT to precisely quantify s

263 ntriculography is a method of measuring left ventricular function usually performed at the discretion

264 r cardiac repair in vivo with regard to left ventricular function, vascularization, and amelioration

265 re laboratory-masked analyses for quantified ventricular function, volumes, infarct size, microvascul

266 Ventricular functions, volumes, and dimensions were meas

267 ollow-up of 36 months, normalization of left ventricular function was achieved in >90% of patients wi

268 Longitudinal left ventricular function was assessed by 2D-strain and globa

269 Left ventricular function was assessed by conventional and de

270 Right ventricular function was assessed using transesophageal

271 In patients +CAN, left ventricular function was decreased in both diastole and

272 as noted in 40 subjects (74.1%), while right ventricular function was depressed in 32 (59.3%).

273 Left ventricular function was evaluated using echocardiograph

274 The decline in left ventricular function was further supported by significan

275 Left ventricular function was impaired (ejection fraction <50

276 quently, infarct size was increased and left ventricular function was impaired 28 days after myocardi

277 ewer hearts were functional during EVHP, and ventricular function was increasingly impaired.

278 Right ventricular function was maintained in MGCD0103-treated

279 Ventricular function was mildly depressed in 245 (7%) an

280 e (71%); left atrium was 43+/-6 mm; and left ventricular function was normal (59+/-9%).

281 Left ventricular function was normal in >50% of patients with

282 On propensity-matched analysis, donor ventricular function was not associated with graft survi

283 Ventricular function was preserved in all 5.

284 essure (PAP) and AF in these patients, right ventricular function was reduced in AF, indicating more

285 Myocardial perfusion and left ventricular function were also investigated using a newl

286 Cardiac fibrosis and ventricular function were assessed by histology and echo

287 nd-diastolic pressure, mean PA pressure, and ventricular function were equivalent at pre-Fontan asses

288 , and echocardiographic measurements of left ventricular function were greater in the AMD3100-treated

289 positive correlations between BNP and right ventricular function were seen.

290 Left ventricular functions were consistently depressed in mic

291 ncy oscillatory ventilation can worsen right ventricular function when compared with protective conve

292 c MRI assessed myocardial viability and left ventricular function, whereas bioluminescence imaging as

293 nds on the actual stenosis severity and left ventricular function, which is of prognostic importance.

294 A8 on protein level, showed an improved left ventricular function, which was associated with a reduce

295 ouse model showed significant improvement in ventricular functions, which was histologically supporte

296 ficacy end points were functional status and ventricular function while temporarily weaned from LVAD

297 nt arrhythmia can result in rapid decline in ventricular function with development of heart failure,

298 mice fed a high-fat diet exhibited depressed ventricular function with reduced fractional shortening

299 hemodynamics showing normalization of right ventricular function with right atrial pressure <8 mm Hg

300 ansplant recipients of grafts with depressed ventricular function would have comparable survival with