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1 ulate a crucial proliferation signal for the ventricular myocardium.
2 c, high level BMP-10 expression in the adult ventricular myocardium.
3 he muscular interventricular septum, and the ventricular myocardium.
4 through a thoracotomy directly into the left ventricular myocardium.
5 the pathological changes of noncompaction of ventricular myocardium.
6  transgenic mice developed tumors within the ventricular myocardium.
7  electrical heterogeneities intrinsic to the ventricular myocardium.
8 mal model with targeted activation of JNK in ventricular myocardium.
9 epicardial space and subsequently invade the ventricular myocardium.
10 of percutaneous myocardial GTx to human left ventricular myocardium.
11  a neurohormone synthesized predominantly in ventricular myocardium.
12 titution of the right and rarely of the left ventricular myocardium.
13  Irx4 expression is found exclusively in the ventricular myocardium.
14 zed by fibro-fatty infiltration of the right-ventricular myocardium.
15 on of ET and Ang II in plasma and atrial and ventricular myocardium.
16 ber of proliferating cardiac myocytes in the ventricular myocardium.
17 racteristics is an important property of the ventricular myocardium.
18 lerosis, with downstream effects on the left ventricular myocardium.
19 ractile proteins is faster in atrial than in ventricular myocardium.
20 enital heart disorder, noncompaction of left ventricular myocardium.
21 es in levels between failing and non-failing ventricular myocardium.
22 sing a porphyrinic sensor placed in the left ventricular myocardium.
23 ated with muscarinic receptor stimulation in ventricular myocardium.
24 ted to cells within the compact layer of the ventricular myocardium.
25 s is feasible without instrumenting the left ventricular myocardium.
26 nol can predictably ablate a large volume of ventricular myocardium.
27 al staining is observed in the failing human ventricular myocardium.
28 vely stunned or, more likely, remodeled left ventricular myocardium.
29  but also on transverse-tubular membranes in ventricular myocardium.
30 at ventricular myocytes and in mouse and rat ventricular myocardium.
31 ived from unipolar electrograms sampling the ventricular myocardium.
32 electrical and contractile activities in the ventricular myocardium.
33 ricular and atrial septal defects and a thin ventricular myocardium.
34 ncorporated it into permeabilized human left ventricular myocardium.
35 modulating collagen deposition in mouse left ventricular myocardium.
36 lular sources and strategies to generate new ventricular myocardium.
37 dimensional (3D) electric propagation within ventricular myocardium.
38 haviour of transmurally intact, viable right-ventricular myocardium.
39 S) genes Tbx5, Cx40, and Cx43 throughout the ventricular myocardium.
40 gnaling and thereby promote expansion of the ventricular myocardium.
41 elerate the kinetics of force development in ventricular myocardium.
42 ive late gadolinium enhancement (LGE) in the ventricular myocardium.
43 omyocyte lateral membranes in failing canine ventricular myocardium.
44 hat activates Ca(2+) homeostatic proteins in ventricular myocardium.
45 on was mapped propagating from PF to working ventricular myocardium.
46 ent for pacemaking when administered to left ventricular myocardium.
47 ardium and myocardium resulting in a thinned ventricular myocardium.
48 nsequence of premature stretch activation of ventricular myocardium.
49 f arrhythmic substrate ablation in the thick ventricular myocardium.
50  conduction velocities across the atrial and ventricular myocardium.
51 heart failure, with reduced proliferation of ventricular myocardium.
52  from the atrioventricular node (AV node) to ventricular myocardium [1].
53  microdialysis probes inserted into the left ventricular myocardium (3-4 probes/dog).
54 reatment of wild-type and cTnI(ala2) skinned ventricular myocardium accelerated stretch activation su
55                  The mean T1 of healthy left ventricular myocardium across all examined subjects was
56 ariation measuring nonuniformity of the left ventricular myocardium activity distribution.
57  tissue, and there was fibrosis of the right ventricular myocardium adjacent to the leads.
58  orthotopically after excision of the entire ventricular myocardium and all four native valves.
59 were performed using the SUVmean of the left ventricular myocardium and blood pool and calculation of
60 e is synthesized primarily in the atrial and ventricular myocardium and constitutes the mature biolog
61 trioventricular canal characteristics in the ventricular myocardium and endocardium, indicating a rol
62  express 50% of the normal amount of Cx43 in ventricular myocardium and exhibit marked slowing of ven
63 and1 expression is restricted to the ventral ventricular myocardium and extends along the entire leng
64  cells were identified in SHF-derived distal ventricular myocardium and in three lineages in the outf
65 day 13.5 and exhibit hypoplasia of the right ventricular myocardium and interventricular septum and d
66 diac failure because of noncompaction of the ventricular myocardium and resultant ventricular dilatat
67 he predominant isoform expressed in both the ventricular myocardium and slow skeletal muscle fibres s
68 haMyHC mRNA is expressed in nonfailing human ventricular myocardium and that alphaMyHC mRNA expressio
69 3) is abundantly expressed in the atrial and ventricular myocardium and the rapid ventricular conduct
70 tion (APD90) in atria, no effect on APD90 in ventricular myocardium, and an abbreviation of APD90 in
71 e outlet right ventricle (DORV), hypoplastic ventricular myocardium, and normal coronary vasculature.
72 voked anomalies in the coronary vessels, the ventricular myocardium, and the AV cushions.
73 yHC), alpha and beta, exist in the mammalian ventricular myocardium, and their relative expression is
74 ic lesions predominantly present in the left ventricular myocardium, and vasculatures in these lesion
75          EPDCs begin to populate the compact ventricular myocardium around ED12.
76  changes parallel the changes found in human ventricular myocardium at the receptor level, suggesting
77  cardiomyopathy and in guinea-pig atrial and ventricular myocardium before and during pharmacological
78 tion from beat to beat, with the majority of ventricular myocardium being activated in a centrifugal
79             Myocytes were isolated from left ventricular myocardium biopsy tissues.
80 myocytes and fibrofatty replacement of right ventricular myocardium; biventricular involvement is oft
81 ddition, CGRP receptors were not observed in ventricular myocardium but were prominent in coronary ar
82 ates cardiogenesis and growth of the compact ventricular myocardium by modulating the cardiomyocyte c
83 n of the sympathetic innervation of the left ventricular myocardium by PET.
84         Enhanced sympathetic activity at the ventricular myocardium can destabilize repolarization, i
85 c those in cells isolated from failing human ventricular myocardium, canine pacing-induced cardiomyop
86     We examined cTnI phosphorylation in left ventricular myocardium collected from failing hearts at
87 ith a cardiac defect characterized by a thin ventricular myocardium, common atrioventricular canal, a
88                                          The ventricular myocardium contains functional beta2-adrener
89 al pole of the heart is the region where the ventricular myocardium continues as the vascular smooth
90                           Gene transfer into ventricular myocardium demonstrated the ability of this
91 nts with isolated non-compaction of the left ventricular myocardium, dilated cardiomyopathy (DCM) and
92 ities by E10.5, with hyperplasia of the left ventricular myocardium, distention of the cardinal veins
93 ntricular septum, atrioventricular cushions, ventricular myocardium, dorsal mesenchymal protrusion, p
94                                     In fixed ventricular myocardium, dual-axis electron tomography wa
95  coronary artery disease by imaging the left ventricular myocardium during a first-pass contrast bolu
96 fronts propagating from PFs into the working ventricular myocardium during VF.
97  Hdac3 overexpression produces thickening of ventricular myocardium, especially the interventricular
98 embryos display pronounced hypoplasia of the ventricular myocardium essentially identical to the "thi
99                                Failing human ventricular myocardium exhibited a reduction in Cx43 and
100 /=5 segments ( approximately 25% of the left ventricular myocardium) exhibited a blood flow/metabolis
101 ) both nonfailing intact and explanted human ventricular myocardium expressed substantial amounts of
102  with [13N]-ammonia for delineating the left ventricular myocardium, followed by imaging the expressi
103  and small nucleolar RNAs (snoRNAs) in right ventricular myocardium from 16 infants with nonsyndromic
104 Clark-type oxygen electrode in isolated left ventricular myocardium from 26 explanted failing human h
105 d the stretch activation response in skinned ventricular myocardium from both wild-type (WT) and cMyB
106  and microelectrode techniques in human left ventricular myocardium from patients with hypertrophic c
107 esults were compared with data from the left ventricular myocardium from similar sized normal (contro
108 hree predominant cell types that make up the ventricular myocardium, giving rise to transmural voltag
109 mages revealed uniform opacification of left ventricular myocardium greater than that of the cavity,
110 hydrogel derived from decellularized porcine ventricular myocardium has been shown to halt the post-i
111 s (AT1 and AT2) in chronically failing human ventricular myocardium has not been previously examined.
112           Abnormalities in repolarization of ventricular myocardium have been implicated in the devel
113  the increase in cardiac lactate to the left ventricular myocardium, implying a direct myocardial eff
114 ness on local conduction in right atrial and ventricular myocardium in 19 patients.
115  ECs migrate from the SV into the atrial and ventricular myocardium in Ang1-dependent manner.
116                               Left and right ventricular myocardium in control rats was shown clearly
117        The hypertrophic response of the left ventricular myocardium in response to aortic constrictio
118                                         Left ventricular myocardium in Tg-Y319F mice developed signif
119 9% outer and a 45% inner infarct of the left ventricular myocardium in the heart failure group.
120 iction of Atoh8 expression to atrial but not ventricular myocardium in the mouse.
121 ent, recipient, ventricular CMs in vitro and ventricular myocardium in vivo.
122 dly decrease the inotropic state of the left ventricular myocardium independent of its bradycardic ef
123  activity in atrial myocardium compared with ventricular myocardium, indicating regional differences
124  without isolated non-compaction of the left ventricular myocardium (INLVM).
125 cribed as isolated noncompaction of the left ventricular myocardium (INVM).
126  of transcription (STAT)5 activation in left ventricular myocardium is associated with RIPC s cardiop
127 s and measurements of conduction velocity in ventricular myocardium is complicated by the fact that t
128    We suggest that the proper development of ventricular myocardium is dependent on the invasion of u
129 erging that gene expression profiles of left ventricular myocardium isolated from failing versus nonf
130  direct cytopathic effects on the atrial and ventricular myocardium, later stages of progressive deco
131 lthough the systolic loading sequence of the ventricular myocardium likely affects its coupling with
132 ricular cardiomyocytes to divide and replace ventricular myocardium lost from ischaemia-induced infar
133 ary performance in health and disease, right ventricular myocardium mechanical behaviour has received
134 Cs was slower when compared with normal left ventricular myocardium (median, 54 [interquartile range,
135                                   Unlike rat ventricular myocardium, mouse cardiac muscle resists sup
136 restimation resulted from inclusion of right ventricular myocardium (n=37; 38.1%), LV trabeculations
137 as cross-sectional percentage of viable left ventricular myocardium, n=9; 0.87%+/-1.4% versus n=6; 14
138 edle was advanced percutaneously to the left ventricular myocardium of 6 patients with chronic myocar
139 of A1-AdoR and A1-AdoR/G protein coupling in ventricular myocardium of 6- to 24-month-old Fischer 344
140  amounts of collagen were determined in left ventricular myocardium of 65 F2-mice and combined with g
141 tious units (IU) of this virus into the left ventricular myocardium of adult CD-1 mice.
142 lactosidase gene were identified in the left ventricular myocardium of adult female nude mice 6 weeks
143 lactosidase gene (Ad beta-gal) into the left ventricular myocardium of athymic nude rats (NDRs) versu
144 10(3) myocytes were in mitosis in the entire ventricular myocardium of control hearts and hearts affe
145 es of cardiocyte apoptosis exist in the left ventricular myocardium of dogs with chronic heart failur
146 hat autonomic conditions directly affect the ventricular myocardium of healthy subjects, causing diff
147 phosphorylation of phospholamban in the left ventricular myocardium of HF patients in atrial fibrilla
148 ntal processes, including development of the ventricular myocardium of the heart.
149 ssion of IL-13 was induced in left and right ventricular myocardium of WT mice within days in respons
150 tricular myocardium versus 20.4+/-10.6% left ventricular myocardium, P<0.0001) and corresponded to th
151 PLB) were determined in explanted human left ventricular myocardium (pediatric n=41; adult n=88).
152 trial fibrillation; however, its function in ventricular myocardium remains unexplored.
153 Western blot analyses were performed on left ventricular myocardium remote from the infarct zone in l
154  techniques, we found that uncoupling of the ventricular myocardium results in ectopic sites of ventr
155     Mechanical experiments with skinned left ventricular myocardium revealed that PKCalpha significan
156 embrane potential (DeltaV(m)) in the bulk of ventricular myocardium (so-called virtual electrodes), b
157                                   To achieve ventricular myocardium-specific gene targeting, and to a
158 aximal standardized uptake value of the left ventricular myocardium (SUV(Myo)) as well as the average
159  analyzed inducible NOS (iNOS) expression in ventricular myocardium taken from 11 control subjects (w
160 2 subtypes of the angiotensin II receptor in ventricular myocardium taken from 9 donor hearts before
161 ctivity was approximately fourfold higher in ventricular myocardium than in atrial tissue.
162  us to derive an immortal cell line from the ventricular myocardium that could be controllably withdr
163         In FGF virus-infected regions of the ventricular myocardium, the capillary density across the
164                         In hypothyroid adult ventricular myocardium, there was a selective 60% increa
165 art disease resulting in large scale loss of ventricular myocardium through both apoptotic and necrot
166     BMP-2 protein expression was absent from ventricular myocardium throughout the stages examined.
167                                   Atrial and ventricular myocardium tissue samples were harvested bef
168 tivation was mapped propagating from working ventricular myocardium to PF.
169 n to retrograde propagation from the working ventricular myocardium to PFs, antegrade propagation occ
170   Cell-specific depletion of over 60% of the ventricular myocardium triggered signs of cardiac failur
171                Trpm7 is dispensable in adult ventricular myocardium under basal conditions but is cri
172 RNA beta 1 species that are expressed in rat ventricular myocardium under basal conditions, and deter
173 of electrical coupling across the developing ventricular myocardium using high-speed optical mapping
174 zed by progressive degeneration of the right ventricular myocardium, ventricular arrhythmias, fibrous
175 linium enhancement extent (33.2+/-16.2% left ventricular myocardium versus 20.4+/-10.6% left ventricu
176 virus containing the human beta(2)AR cDNA to ventricular myocardium via catheter-mediated subselectiv
177                                  Canine left ventricular myocardium was collected under conditions to
178 intracellular compartment of guinea pig left ventricular myocardium was measured at 20 degrees C and
179                                   Human left ventricular myocardium was obtained for biochemical anal
180 splantation, more than one-third of the left ventricular myocardium was replaced by fibrosis, mainly
181 lagen are thought to affect the mechanics of ventricular myocardium, we investigated myocardial colla
182    METHODS AND Paired biopsy samples of left ventricular myocardium were obtained from 9 patients wit
183 esent study, mitoK(ATP) channels from bovine ventricular myocardium were reconstituted using planar l
184 carbonate signal mainly confined to the left ventricular myocardium, whereas the [1-(13)C]lactate sig
185 an is expressed strongly in the trabeculated ventricular myocardium, whereas the compact proliferativ
186 grade propagation occurs from PFs to working ventricular myocardium, which suggests PFs are important
187 lts in ectopic activation of atrial genes in ventricular myocardium with an associated impairment of
188 , 20g, 40g, and 70g: (1) over left and right ventricular myocardium with or without fat, (2) either d
189 uate a technique for mapping and ablation of ventricular myocardium with the use of transcatheter sub
190 roperties compared to native adult rat right ventricular myocardium, with stiffnesses controlled by p
191 d looping, Lbh expression is confined to the ventricular myocardium, with the highest intensity in th

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