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1 ith a low transvalvular gradient and no left ventricular outflow tract obstruction.
2 ecember 2007 for the surgical relief of left ventricular outflow tract obstruction.
3 patients undergoing surgical relief of left ventricular outflow tract obstruction.
4 ent echocardiograms were evaluated for right ventricular outflow tract obstruction.
5 ects of PPVI over BMS in patients with right ventricular outflow tract obstruction.
6 None of the patients have developed left ventricular outflow tract obstruction.
7 ransposition of the great arteries with left ventricular outflow tract obstruction.
8 ransposition of the great arteries with left ventricular outflow tract obstruction.
9 One patient had left ventricular outflow tract obstruction.
10 before and after medical elimination of left ventricular outflow tract obstruction.
11 ning 25 patients had multiple levels of left ventricular outflow tract obstruction, 12 of whom had at
12 Indications for reoperation included right ventricular outflow tract obstruction (19), branch pulmo
13 cardiomyopathy with severe symptomatic left ventricular outflow tract obstruction (47+/-11 years, 63
14 rly complete repair include removal of right ventricular outflow tract obstruction, alleviation of sy
15 association between paroxetine use and right ventricular outflow tract obstruction and between sertra
17 te-term results indicate good relief of left ventricular outflow tract obstruction and need for condu
18 transposition of the great arteries and left ventricular outflow tract obstruction and results in a m
19 It is frequently accompanied by dynamic left ventricular outflow tract obstruction and symptoms of dy
21 tent implantation for the treatment of right ventricular outflow tract obstruction are often left wit
22 ients with hypertrophic cardiomyopathy, left ventricular outflow tract obstruction at rest is a stron
23 se in the risk of Ebstein's anomaly (a right ventricular outflow tract obstruction defect) in infants
24 and between the use of paroxetine and right ventricular outflow tract obstruction defects (odds rati
27 Syncope after exercise may be due to left ventricular outflow tract obstruction from aortic stenos
29 ts undergoing surgery for the relief of left ventricular outflow tract obstruction have low event rat
30 einterventions in this series were for right ventricular outflow tract obstruction, highlighting the
31 ased and surgical procedures to relieve left ventricular outflow tract obstruction in HCM, but it is
32 espite the association of symptoms with left ventricular outflow tract obstruction in HCM, there exis
33 congenital heart disease, reduction of left ventricular outflow tract obstruction in hypertrophic ca
34 herapy for pulmonary regurgitation and right ventricular outflow tract obstruction in selected patien
35 omy and alcohol septal ablation relieve left ventricular outflow tract obstruction in severely sympto
37 ession wave, whereas proximally, severe left ventricular outflow tract obstruction is associated with
40 a is typically seen in association with left ventricular outflow tract obstruction, itself part of a
42 sition of the great arteries (TGA) with left ventricular outflow tract obstruction (LVOTO) may be tre
44 cise capacity, there was mild residual right ventricular outflow tract obstruction (mean gradient, 24
45 ath (13.02%, 95% CI 3.60-25.91%), while left ventricular outflow tract obstruction/mid-ventricular ob
46 sion (n = 24 [0.8%]) and hypotension or left ventricular outflow tract obstruction (n = 112 [3.8%]).
47 t 11 reoperations, including relief of right ventricular outflow tract obstruction (n=5), pulmonary a
50 h suprasystemic pulmonary hypertension, left ventricular outflow tract obstruction or dilated cardiom
51 nosis, left ventricular wall thickness, left ventricular outflow tract obstruction, or family history
52 tion between the use of paroxetine and right ventricular outflow tract obstruction (relative risk, 1.
53 ransposition of the great arteries with left ventricular outflow tract obstruction represents a diffi
55 and G+ probands were younger with less left ventricular outflow tract obstruction than G- probands,
56 dian age, 12.9 years) with significant right ventricular outflow tract obstruction underwent BMS foll
57 icardial tamponade, pulmonary embolism, left ventricular outflow tract obstruction, unexplained hypox
58 s at study entry, including 249 in whom left ventricular outflow tract obstruction was absent both at
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