ライフサイエンスコーパス: ventricular pacing

1 ing, His bundle pacing, and endocardial left ventricular pacing.

2 nd mechanical asynchrony in any patient with ventricular pacing.

3 y frequent following temporary cessations of ventricular pacing.

4 alters the characteristics of IC neurons to ventricular pacing.

5 Programming minimized ventricular pacing.

6 ormal pump function and in case of part-time ventricular pacing.

7 of TG and NTG rabbits was subjected to rapid ventricular pacing.

8 rimposed heart failure (HF) induced by rapid ventricular pacing.

9 CHF was produced in 2 groups of dogs by ventricular pacing.

10 duced AF were studied one week after ceasing ventricular pacing.

11 troke or improve survival when compared with ventricular pacing.

12 ol, n=5) and with HF (n=8) produced by rapid ventricular pacing.

13 es heart failure symptoms when compared with ventricular pacing.

14 We induced CHF in nine dogs by rapid ventricular pacing.

15 ers significant improvement as compared with ventricular pacing.

16 ase in the quality of life, as compared with ventricular pacing.

17 prove stroke-free survival, as compared with ventricular pacing.

18 ntricular apex were measured on cessation of ventricular pacing.

19 velopment of the dilated myopathy induced by ventricular pacing.

20 x were obtained during all ectopic beats and ventricular pacing.

21 experimental heart failure produced by rapid ventricular pacing.

22 ase of a normal action potential produced by ventricular pacing.

23 ositioned across the valve ring during rapid ventricular pacing.

24 on fraction </=50% to biventricular or right ventricular pacing.

25 al status with dual-chamber pacing than with ventricular pacing.

26 Three devices (0.3%) were replaced for right ventricular pacing.

27 entricle in conditions of AV block and right ventricular pacing.

28 ntrol and AV junction ablation and permanent ventricular pacing.

29 other group underwent a period of rapid left ventricular pacing.

30 vascular resistance than does single-chamber ventricular pacing.

31 tients who required permanent single-chamber ventricular pacing.

32 no systematic reverse remodeling with right ventricular pacing.

33 mal) and after induction of HF by rapid left ventricular pacing.

34 HCC at rest, but in only the HCW group under ventricular pacing.

35 ects exhibited cardiac memory in response to ventricular pacing.

36 0 ms in the absence of a documented need for ventricular pacing.

37 nd during delivery of biventricular and left ventricular pacing.

38 5% confidence interval, 0-3) required rescue ventricular pacing.

39 n, and was likely equivalent to, backup-only ventricular pacing.

40 ents who had guideline-based indications for ventricular pacing.

41 n the group assigned to dual-chamber minimal ventricular pacing.

42 Stroke volume was altered by right ventricular pacing (160, 210, 260, and 305 beats/min).

43 vs. 19%, p < 0.012, OR 1.57) and to require ventricular pacing (18% vs. 11%, p = 0.006, OR 1.73).

44 Rapid ventricular pacing (180 to 230 bpm) was subsequently ini

45 anterior descending artery followed by right ventricular pacing (240 ppm) for 3 weeks to produce hear

46 Multiple extrasystoles were induced by right ventricular pacing (25% of heart beats).

47 However, 53 patients assigned to ventricular pacing (26 percent) were crossed over to dua

48 during NTP (-4.7+/-1.4 U/mm Hg) than during ventricular pacing (-3.4+/-1.1 U/mm Hg).

49 nd the decline was greater with AV than with ventricular pacing (60 beats/min -40 +/- 11% vs. -17 +/-

50 sinus rhythm, and epicardial and endocardial ventricular pacing (65 records in total).

51 After 1 month of left ventricular pacing, 8 mongrel dogs were monitored for he

52 on to dual-chamber pacing (1014 patients) or ventricular pacing (996 patients) and followed them for

53 mal activation sequence resulting from right ventricular pacing accounts for only part of the reducti

54 Asynchronous ventricular pacing, after the Fontan procedure, has acut

55 At 4 and 8 weeks of ventricular pacing, all LA strains were decreased and LA

56 observed in the control dogs that underwent ventricular pacing alone (n = 4) did not change.

57 Long-term right ventricular pacing alone does not appear to be associate

58 e parameters showed little change with right ventricular pacing alone, indicating no systematic rever

59 Right ventricular pacing also impaired hemodynamics and LV fun

60 Right ventricular pacing alters the ventricular activation seq

61 in the pattern of atrial activation between ventricular pacing and AVNRT in only 21 of 46 patients.

62 ned the short-term effects of single-chamber ventricular pacing and dual-chamber atrioventricular (AV

63 -blind, randomized, controlled comparison of ventricular pacing and dual-chamber pacing in 407 patien

64 art failure induced by 3 to 4 weeks of rapid ventricular pacing and from 16 nonpaced control dogs did

65 ctivation was recorded in 17 patients during ventricular pacing and in 26 patients during AVNRT.

66 f the triangle of Koch in 38 patients during ventricular pacing and in 43 patients during AVNRT.

67 During right ventricular pacing and inferior right septal pacing, act

68 SCaE was reproducibly induced by rapid ventricular pacing and inhibited by 3 mumol/L of ryanodi

69 -term memory (STM) was induced by 2 hours of ventricular pacing and long-term memory (LTM) by ventric

70 ct, septum, and apex were mapped during left ventricular pacing and MES recorded.

71 collected using CARTO3v4 in sinus rhythm or ventricular pacing and reviewed for ripple mapping condu

72 f the paced wall during prolonged rapid left ventricular pacing and that regional stunning contribute

73 al has raised concerns of conventional right ventricular pacing and the risk of heart failure in a su

74 isk of developing HF in the setting of right ventricular pacing and to determine whether these patien

75 induced: ventricular fibrillation (by rapid ventricular pacing) and, after successful defibrillation

76 sodes associated with a device intervention (ventricular pacing), and (3) symptomatic episodes associ

77 focal activation initiated by right or left ventricular pacing, and (iv) atrial flutter.

78 tic root angiogram during breath-hold, rapid ventricular pacing, and injection of 32 mL contrast medi

79 orded in each dog during sinus rhythm, right ventricular pacing, and pacing of the right septum throu

80 ed with dual-chamber pacing as compared with ventricular pacing are observed principally in the subgr

81 amber (atrioventricular) and single-chamber (ventricular) pacing are alternative treatment approaches

82 lation in patients with dual-chamber minimal ventricular pacing as compared with those with conventio

83 Dual-chamber minimal ventricular pacing, as compared with conventional dual-c

84 ine if the atrial response upon cessation of ventricular pacing associated with 1:1 ventriculoatrial

85 clusion, an A-A-V response upon cessation of ventricular pacing associated with 1:1 ventriculoatrial

86 The response upon cessation of ventricular pacing associated with 1:1 ventriculoatrial

87 Of those, 988 patients with <20% right ventricular pacing at 1 week were randomized to DDDR AVS

88 ons: (1) twice at baseline; (2) during right ventricular pacing at 110 bpm; (3) during intravenous in

89 Epicardial ventricular pacing at 130 bpm was achieved by use of a p

90 Mild CHF was produced by rapid ventricular pacing at 180 bpm for 10 days and severe CHF

91 sure) was measured at 3 specific conditions: ventricular pacing at 200 and 300 beats per minute, and

92 l of which underwent simultaneous atrial and ventricular pacing at 220 beats per minute for 14 days.

93 in 21 HF dogs (subjected to 3 weeks of rapid ventricular pacing at 240 beats per minute).

94 CHF was induced by continuous ventricular pacing at 320 to 340 bpm for 3 weeks.

95 PVCs and PVCs at 375 ms compared with rapid ventricular pacing at 400 ms (P<0.0001), whereas no diff

96 n 30 patients during simultaneous atrial and ventricular pacing at 500 ms with S(2) coupling interval

97 erquartile range, -2 to -19 ms) during right ventricular pacing at 6 months (P<0.05).

98 for 1 week, with atrioventricular block and ventricular pacing at 80 bpm); (2) congestive heart fail

99 In 6 hearts, measurements during ventricular pacing at a basic cycle length (BCL) of 120

100 activation during AVNRT (337 +/- 43 ms) and ventricular pacing at a similar cycle length (352 +/- 51

101 refractory period extension by shocks during ventricular pacing at fast rates predict that all tissue

102 the novel possibility that continuous right ventricular pacing at least partially suppresses pacemak

103 echocardiography, or atrioventricular versus ventricular pacing at the same rate.

104 All dogs underwent 8 weeks of high-rate ventricular pacing (at 220 beats per minute for the firs

105 rial pacing (at 70 beats/min) versus minimal ventricular pacing (at 40 beats/min) and followed up for

106 have provoked interest in the utilization of ventricular pacing beyond maintenance of heart rate.

107 d MRI was markedly dyssynchronous with right ventricular pacing but synchronous with right atrial pac

108 ociation functional class, and percent right ventricular pacing, but it was independent of gender and

109 ical benefit as compared with single-chamber ventricular pacing, but the supporting evidence is mainl

110 adiofrequency ablation (AVNA) with permanent ventricular pacing can be used to control rate in patien

111 Although right ventricular pacing can contribute to cardiomyopathy, the

112 Ventricular pacing can improve hemodynamics in heart fai

113 ents who demonstrated a </=0.5 V increase in ventricular pacing capture threshold (100% MRI vs. 98.8%

114 The primary efficacy endpoints were ventricular pacing capture threshold and ventricular sen

115 , between tip and ring electrodes of a right ventricular pacing catheter, and unipolar, from tip to a

116 iac pacing and was greater with AV than with ventricular pacing (change in mean blood pressure +/- SE

117 Contact maps during right ventricular pacing correlated closely to inverse solutio

118 tion predictors were VVIR cumulative percent ventricular pacing (Cum%VP) >80 (HR, 3.58; 95% CI, 1.72-

119 utcome, the interaction of QRS duration with ventricular pacing (DDDR-70) independently contributed t

120 mmed in a manner that promoted more frequent ventricular pacing (DDDR-70), there was a significant ad

121 udy was to determine whether the response to ventricular pacing during tachycardia is useful for diff

122 e aim of this research was to evaluate right ventricular pacing effects on left ventricular function.

123 Pacing permitted (S-L-S sequences without ventricular pacing) episodes accounted for 6.4% (DDD/R),

124 Ventricular desynchronization imposed by ventricular pacing even when AV synchrony is preserved i

125 Burst ventricular pacing excluded atrial tachycardia when the

126 us presumed to have a higher burden of right ventricular pacing, experienced an increased risk of new

127 heart failure were induced by chronic right ventricular pacing for 1 to 2 weeks, 3 to 4 weeks, and 7

128 CHF was induced by rapid ventricular pacing for 10 days.

129 tized dogs with experimental CHF produced by ventricular pacing for 10 days.

130 ricular pacing and long-term memory (LTM) by ventricular pacing for 21 days.

131 multiple comorbidities, AVNA with permanent ventricular pacing for rate control seems safe during fo

132 ent leadless pacemakers are limited to right ventricular pacing, future advanced, communicating, mult

133 in 190 of 342 patients (55.6%) in the right-ventricular-pacing group, as compared with 160 of 349 (4

134 The site of ventricular pacing has a major impact on LV mechanical s

135 ed response amplitude (VERA) obtained during ventricular pacing have been correlated with the presenc

136 e over time than did those assigned to right ventricular pacing (hazard ratio, 0.74; 95% credible int

137 on therapy, which coordinates right and left ventricular pacing in a subset of patients with interven

138 The impact of prolonged right ventricular pacing in adults without structural heart di

139 a safe alternative to minimal (backup-only) ventricular pacing in defibrillator recipients with impa

140 Rapid ventricular pacing in dogs is characterized by a dilated

141 (Biventricular Versus Right Ventricular Pacing in Heart Failure Patients With Atriov

142 The BLOCK HF (Biventricular Versus Right Ventricular Pacing in Heart Failure Patients With Atriov

143 The Biventricular versus Right Ventricular Pacing in Heart Failure Patients with Atriov

144 te that electrical remodeling in response to ventricular pacing in human subjects results in action p

145 controlling SNA and arterial pressure during ventricular pacing in humans.

146 This should not imply that right ventricular pacing in NQRS patients is safe but rather t

147 We assessed the effect of right ventricular pacing in patients who underwent pacemaker i

148 ar pacing was superior to conventional right ventricular pacing in patients with atrioventricular blo

149 By univariate analysis, ventricular pacing in patients with AVB appeared to be a

150 Single-site ventricular pacing in patients with heart failure, atria

151 imal model of heart failure induced by rapid ventricular pacing in pigs.

152 ient randomized trial of dual-chamber versus ventricular pacing in sinus node dysfunction.

153 The mean percent right ventricular pacing in the DDDR AVSH arm was 10% (median

154 set (S-L-S sequences actively facilitated by ventricular pacing including the terminal beat after a p

155 rter defibrillator (ICD) therapy with backup ventricular pacing increases survival in patients with l

156 Right ventricular pacing increases the risk of heart failure i

157 ivity and higher arterial pressure than does ventricular pacing, indicating that cardiac pacing mode

158 solated from normal dogs and dogs with rapid ventricular pacing-induced CHF.

159 solated from normal dogs and dogs with rapid ventricular pacing-induced CHF.

160 are associated with regional dysfunction in ventricular pacing-induced heart failure, regional myoca

161 The percentage of ventricular pacing is used as an indicator of adequate b

162 We conclude that chronic epicardial ventricular pacing is well tolerated by the fetus, can b

163 nted with cardiac dimension crystals, a left ventricular pacing lead, and a pacemaker.

164 Ventricular pacing leads to a dilated myopathy in which

165 c and electromechanical consequences of left ventricular pacing (LVP) and biventricular pacing (BiVP)

166 ges in ventricular function induced by right ventricular pacing may account for some of its associate

167 ds no clear advantage or disadvantage over a ventricular pacing mode that minimizes pacing altogether

168 CHF was produced in dogs by use of the rapid ventricular pacing model.

169 ppressed LA fibrosis in the canine high-rate ventricular pacing model.

170 During short AV delays (<300 ms) and right ventricular pacing, MVC occurred significantly later.

171 nd by pacing mode (DDD/R, VVI/R, and Managed Ventricular Pacing [MVP]).

172 ntaneous activity and following cessation of ventricular pacing (n = 5) to give similar features to W

173 as repeated with heart rate held constant by ventricular pacing (n=3).

174 elopment of CHF produced by 3 weeks of rapid ventricular pacing (n=9).

175 differences were found among the systems at ventricular pacing of 200 and 300 beats per minute, unde

176 We evaluated the effects of the site of ventricular pacing on left ventricular (LV) synchrony an

177 ffects of dual-chamber versus single-chamber ventricular pacing on subsequent stroke in patients with

178 ory (CM) refers to T-wave changes induced by ventricular pacing or arrhythmia that accumulate in magn

179 e QRS complex vector during prior periods of ventricular pacing or arrhythmia.

180 s to an altered T-wave morphology induced by ventricular pacing or arrhythmias that persist for varia

181 s with AVB, alternate single-site RV or left ventricular pacing or biventricular pacing may be superi

182 and were randomly assigned to standard right ventricular pacing or biventricular pacing.

183 that had been randomly programmed to either ventricular pacing or dual-chamber pacing.

184 ely abolished by both biventricular and left ventricular pacing (P<0.05).

185 ing post-MI (63% in control vs. 44% in MI to ventricular pacing; P < 0.01).

186 randomization (after 30 to 60 days of right ventricular pacing postimplant) and every 6 months throu

187 ntricular beat of one of the following three ventricular pacing protocols: constant ventricular rates

188 Comparison of NTP doses and ventricular pacing rates that produced comparable hypote

189 AV nodal ablation and bi-ventricular pacing remains another viable option.

190 for patients with a standard indication for ventricular pacing remains controversial.

191 Right ventricular pacing restores an adequate heart rate in pa

192 In all subjects, ventricular pacing resulted in a prolongation of the act

193 CM induced by right ventricular pacing results in a distinctive T-vector pat

194 Twenty dogs underwent rapid ventricular pacing (RVP) for 4 weeks to create a model o

195 Right ventricular pacing (RVP) increases risk of atrial fibril

196 Seventeen dogs underwent rapid ventricular pacing (RVP) to create heart failure.

197 llowing groups (12 per group): chronic rapid ventricular pacing (RVP; 400 bpm, 3 weeks), RVP and conc

198 ent in pacing mode in the very elderly, with ventricular pacing selected for sicker and older patient

199 o intraventricular conduction abnormalities, ventricular pacing should be avoided as much as possible

200 of right ventricular pacing, suggesting that ventricular pacing should be minimized whenever possible

201 imize detection enhancements and to minimize ventricular pacing, significantly decrease inappropriate

202 Left ventricular (LV) and right ventricular pacing site characteristics have been shown

203 cades of technological advances, the optimal ventricular pacing site to mimic normal human ventricula

204 Patient selection, left ventricular pacing site, and optimal device programming

205 high-rate episode is a high burden of right ventricular pacing, suggesting that ventricular pacing s

206 ly, was less evident during continuous right ventricular pacing, suggesting the novel possibility tha

207 were paced was lower in dual-chamber minimal ventricular pacing than in conventional dual-chamber pac

208 -term RV apical pacing, alternative sites of ventricular pacing that simulate normal biventricular el

209 During ventricular pacing, the peak amplitude of fast Fourier t

210 The ventricular pacing threshold (VPT) and ventricular fibri

211 ism for VER, dogs (n=6) underwent 4 weeks of ventricular pacing to induce VER.

212 Sheep underwent rapid right ventricular pacing to obtain moderate to severe function

213 imaging was used before and after 1 month of ventricular pacing to reconstruct epicardial activation

214 presents a new paradigm that aims to tailor ventricular pacing to the individual patient to achieve

215 g using an intracardiac electrogram during a ventricular pacing train.

216 Ang II, and activation of p53 function with ventricular pacing upregulates the myocyte RAS and the g

217 suggest that electric separation during left ventricular pacing varies within the right ventricle (RV

218 under normal sinus rhythm were compared with ventricular pacing (VDD) at varying sites and AV delays

219 tic and arterial pressure responses to VT or ventricular pacing (VP) in dogs with inducible VT.

220 ympathetic gain and BP recovery during rapid ventricular pacing (VP) in patients referred for electro

221 in the group receiving dual-chamber minimal ventricular pacing vs. 5.4% in the group receiving conve

222 Eight mongrel dogs underwent demand ventricular pacing (VVI) at 250 beats/min for 3 weeks to

223 ial comparing dual-chamber pacing (DDDR) and ventricular pacing (VVIR) in sinus node dysfunction, dem

224 beats min(-1)) using ULFS-49, and atrial or ventricular pacing was achieved via an intra-oesophageal

225 Ventricular pacing was performed adjacent to the His bun

226 gh altered ventricular activation from right ventricular pacing was presumed to be the likely cause f

227 The electrogram sequence upon cessation of ventricular pacing was, categorized as "atrial-ventricul

228 ed in which SNA and hemodynamic responses to ventricular pacing were compared with nitroprusside infu

229 ade atrial activation during tachycardia and ventricular pacing were determined by intracardiac recor

230 edation, sodium channel-blocking agents, and ventricular pacing were effective in suppressing acute e

231 of intracardiac electrograms and atrial and ventricular pacing were performed.

232 synchrony but results in high percentages of ventricular pacing, which causes ventricular desynchroni

233 line (atrial antibradycardia pacing or right ventricular pacing with atrial fibrillation) to dual-cha

234 acing (535 patients) or dual-chamber minimal ventricular pacing with the use of new pacemaker feature

235 es were evaluated during sinus rhythm, right ventricular pacing without preceding atrial contraction,

236 (no pacing, no S-L-S) and pacing associated (ventricular pacing without S-L-S) onset accounted for 44

237 Right ventricular pacing worsens LV function in patients with

238 ar activities (LAVAs) during sinus rhythm or ventricular pacing would be a useful and effective end p

239 r), we hypothesized that CM induced by right ventricular pacing would manifest a TWI pattern differen