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1 instem and cerebellum hypoplasia, as well as ventriculomegaly.
2 l hypoplasia, hippocampal malformations, and ventriculomegaly.
3 arly lethal phenotypic variant showed severe ventriculomegaly.
4 pia, consistent with a CMS diagnosis, but no ventriculomegaly.
5 emorrhage, periventricular leukomalacia, and ventriculomegaly.
6 cal volumes and is associated with secondary ventriculomegaly.
7 hypoplasia, optic nerve hypoplasia and mild ventriculomegaly.
8 es, including corpus callosum hypoplasia and ventriculomegaly.
9 lar zone (SVZ), subarachnoid hemorrhage, and ventriculomegaly.
10 hemorrhage, periventricular leukomalacia, or ventriculomegaly (17.5 percent vs. 23.9 percent, P=0.03)
12 enatal steroid tended to have lower risks of ventriculomegaly and an echolucent lesion than their pee
13 evident in newborn cranial ultrasound scans (ventriculomegaly and an echolucent lesion) and of cerebr
14 s who shared a phenotype comprising cerebral ventriculomegaly and echogenic kidneys with histopatholo
15 by a reduction in brain matter and secondary ventriculomegaly and is a major cause of developmental d
18 ormly present, whereas cerebellar dysplasia, ventriculomegaly, and arachnoid cysts are nearly invaria
19 facial dysmorphisms, palatal abnormalities, ventriculomegaly, and hypogonadism as well as additional
20 ne fetuses (56%, P < .05) with isolated mild ventriculomegaly, and in 24 of 31 fetuses (77%, P < .05)
21 which MR imaging has proved useful, such as ventriculomegaly, arachnoid cysts, and abdominal masses,
22 toparietal regions most severely, as well as ventriculomegaly, bilateral white matter signal changes,
23 rate parenchymal rarefaction, severe to mild ventriculomegaly, cerebellar hypoplasia with brainstem d
24 icrocephaly, a reduction in cerebral volume, ventriculomegaly, cerebellar hypoplasia, lissencephaly w
25 opathological study revealed megalencephaly, ventriculomegaly, cytomegaly and extensive vacuolization
26 ctrum of antiquitin deficiency extended from ventriculomegaly detected on foetal ultrasound, through
27 r early identification and quantification of ventriculomegaly due to either posthemorrhagic ventricul
28 ears to mediate hypoxia-induced brain injury ventriculomegaly during early postnatal development.
30 normal cortical formation, cerebral atrophy, ventriculomegaly, hydrocephaly, and cerebellar abnormali
32 tal Zika virus infections were similar, with ventriculomegaly in 16 of 17 (94%) and 27 of 28 (96%) in
35 o association of oxygen delivery (except for ventriculomegaly in the BDG group) or carbon dioxide rea
36 cognized to have neurologic effects, such as ventriculomegaly, intraventricular adhesions, subependym
39 , including intrauterine growth restriction, ventriculomegaly, microcephaly, lissencephaly, and exten
40 n of Bbs1 mutant brain neuroanatomy revealed ventriculomegaly of the lateral and third ventricles, th
42 roencephaly, periventricular calcifications, ventriculomegaly, pachygyria, cerebellar hypoplasia, por
43 and p.Asn800Lys, and one child with cerebral ventriculomegaly, periventricular heterotopias, echogeni
46 pathogenesis of developmental brain injury, ventriculomegaly was also observed in mice lacking the e
49 rapeutic potential for reducing fibrosis and ventriculomegaly was tested using a rat model of juvenil
51 T2-weighted imaging, focal tissue loss, and ventriculomegaly were all more commonly detected in BDG
52 more, VEGF infusions into the rats result in ventriculomegaly with an increase of SVZ neuroblast in r
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