ライフサイエンスコーパス: vermis

1 ntal cortex, corpus callosum, and cerebellar vermis.

2 with subsequent loss of Atoh1 expression in vermis.

3 o of these are ongoing within the cerebellar vermis.

4 f8 gene dose, resulted in loss of the entire vermis.

5 ), and paravermis, but with decreases in the vermis.

6 II/EAAT4-negative PC subsets in the anterior vermis.

7 ols had greater activation in the cerebellar vermis.

8 us, insula, frontal operculum and cerebellar vermis.

9 al right cerebellum, as well as the anterior vermis.

10 obules I, IV, V, IX, and X of the cerebellar vermis.

11 operculum bilaterally and in the cerebellar vermis.

12 vascular resistance in the dorsal cerebellar vermis.

13 posterior-inferior lobules of the cerebellar vermis.

14 globus pallidus, cerebellum, and cerebellar vermis.

15 ctivity in both the hippocampus and anterior vermis.

16 creases in p38 MAPK activity in the anterior vermis.

17 RKs) and p38 MAPK in the anterior cerebellar vermis.

18 frontal brain, basal ganglia, and cerebellar vermis.

19 observed, most notably lobes IV and V in the vermis.

20 ol of cells that develop into the cerebellar vermis.

21 ularly in the cerebral cortex and cerebellar vermis.

22 amus and premotor cortex, and the cerebellar vermis.

23 s expressed transiently in the anterior lobe vermis.

24 the paracentral motor cortex and cerebellar vermis.

25 thin the cerebellum, especially the inferior vermis.

26 section of the posterior inferior cerebellar vermis.

27 striatum, olfactory tubercle, and cerebellar vermis.

28 ing to the vestibulocerebellum and posterior vermis.

29 he left prefrontal cortex and the cerebellar vermis.

30 tal lobes but not in the superior cerebellar vermis.

31 A-A/BDZ receptors in the superior cerebellar vermis.

32 nd DV bilaterally in the superior cerebellar vermis.

33 diate and lateral cerebellum, as well as the vermis.

34 ociated with smaller regional volumes in the vermis.

35 g orthodromic identification from the caudal vermis.

36 medial wall of the hemisphere project to the vermis.

37 r/ataxia syndrome was found in a part of the vermis.

38 the transmitter used by many neurons in the vermis.

39 hombic lip and overmigrate into the anterior vermis.

40 antly restricted to the cerebellar posterior vermis.

41 c relatives had lower metabolism in anterior vermis (12%; P = 0.01) and hippocampus (19%; P = 0.002)

42 ellar hemispheres (14%, P < 0.001), anterior vermis (40%, P < 0.001) and fusiform gyrus (20%, P < 0.0

43 uncle and bilateral portions of the superior vermis also showed persistent decrease in FA over time.

44 Lobules 10 and 9 in the caudal posterior vermis [also known as nodulus and uvula (NU)] are though

45 In the posterior vermis, although the zebrin II+ bands are wider and mult

46 ased regional brain volumes in the posterior vermis, amygdala, and hippocampus.

47 head movements in the rat caudal cerebellar vermis, an area essential for graviceptive functions.

48 genital brain malformation of the cerebellar vermis and brainstem with abnormalities of axonal decuss

49 ed by hypoplasia/dysplasia of the cerebellar vermis and by ataxia, hypotonia, oculomotor apraxia, and

50 (vestibular nuclear area, cerebellar ventral vermis and floccular lobe), cardiorespiratory control (m

51 kinje cells recorded from the posterior lobe vermis and hemisphere have high simple spike firing freq

52 ion in anterior subregions of the cerebellar vermis and hemisphere in the asymptomatic premutation gr

53 proliferation was similar in the cerebellar vermis and hemispheres in all patients with ciliopathy a

54 /dysfunction was indicated in the cerebellar vermis and hemispheres in both diseases by lower total N

55 d number and size of Purkinje neurons in the vermis and hemispheres, molecular defects, and reduced f

56 observed in multiple lobules throughout the vermis and hemispheres.

57 sterior lobe were profoundly reduced in both vermis and hemispheres.

58 Our finding of decreased metabolism in vermis and hippocampus of asymptomatic relatives suggest

59 results challenge the classical view of the vermis and indicate that it no longer should be consider

60 ing crucial to producing the distinct medial vermis and lateral hemisphere foliation patterns in mamm

61 We found that the right cerebellar vermis and left lobule V of cerebellar anterior lobe wer

62 te nucleus, cerebellar peduncles, cerebellar vermis and lobules V and VI, and corpus callosum.

63 We recorded PC activity in the vermis and lobus simplex of head-fixed mice while monito

64 sence of posterior vermis with some anterior vermis and nodulus present), to severe (the absence of p

65 myo-inositol was higher than controls in the vermis and pons in AOA2 and in the vermis in FRDA.

66 g FGF signaling perturbs the balance between vermis and roof plate development in rhombomere 1.

67 ttributed to the reduction of the cerebellar vermis and some regions of the hemispheres.

68 and ventral striatum, and midline cerebellar vermis and subgenual cingulate cortex.

69 inactivation data showing that the posterior vermis and the caudal fastigial nucleus, to which it pro

70 co-nuclear zone consisting of the cerebellar vermis and the fastigial nucleus.

71 A similar increase in the cerebellar vermis and the left thalamus likewise suggests a role of

72 for total cerebral volume, the volume of the vermis and the midsagittal area and volume of the inferi

73 ed rabies virus into lobules VB-VIIIB of the vermis and used retrograde transneuronal transport of th

74 ecursor cells, resulting in complete loss of vermis and variable hypoplasia of cerebellar hemispheres

75 l processing (sensorimotor cortex, striatum, vermis) and an increased influence of the CbTC circuit.

76 differences were significant in cerebellum (vermis) and thalamus.

77 motor output (anterior and dorsal cerebellar vermis) and the maintenance of equilibrium (vestibular n

78 rin II+ cell mass is absent from the central vermis, and analysis of the anterior lobe reveals severa

79 ment and separately for the whole brainstem, vermis, and cerebellar hemispheres.

80 as well as the caudate, thalamus, cerebellar vermis, and cerebrum in 20 first-episode psychosis patie

81 palsy in the putamen, caudate, thalamus, and vermis, and decreased in the superior cerebellar peduncl

82 ellar network comprising the inferior olive, vermis, and deep cerebellar nuclei including the dentate

83 fossa lesion, including that in the nodulus, vermis, and deep cerebellar structures.

84 the posterior lobe of the cerebellum and the vermis, and in some cases they were the most noticeable

85 x, lingual gyrus, striate cortex, cerebellar vermis, and left thalamus.

86 trophy of the cerebral cortex and cerebellar vermis, and mild atrophy of the cerebellar hemispheres.

87 thalamus, left cerebellar regions, anterior vermis, and right cuneus.

88 us, globus pallidus, hippocampus, cerebellar vermis, and very low expression was detected in the stri

89 ibution, and also in the superior cerebellar vermis; and the non-ACD group had significant reductions

90 arized progression of differentiation in the vermis anlage.

91 cell proliferation in the medial cerebellar (vermis) anlage after E11.5.

92 en syndrome), liver fibrosis, and cerebellar vermis aplasia (Joubert syndrome) in approximately 10% o

93 liopathy giving rise to NPHP with cerebellar vermis aplasia and retinal degeneration.

94 ion with retinal degeneration and cerebellar vermis aplasia in Joubert syndrome are poorly understood

95 Decreased volume of the superior cerebellar vermis appears to represent an important substrate of th

96 n samples, show that the hemispheres and the vermis are affected in JS/MKS and provide evidence of a

97 onal nystagmus revealed that the nodulus and vermis are common areas of injury.

98 pallidus, sensorimotor cortex and cerebellar vermis, as well as increases in the precuneus (BA 7).

99 g the cerebellum, in particular the superior vermis, as well as the medial and inferior frontal corte

100 t these FGFs are not required to pattern the vermis at this stage of development.

101 isorder marked by agenesis of the cerebellar vermis, ataxia, hypotonia, oculomotor apraxia, neonatal

102 gressive gait ataxia with tremor, cerebellar vermis atrophy, and optic-nerve thinning.

103 early infancy and are affected by cerebellar vermis atrophy, ataxia, and peripheral neuropathy.

104 (increased rCBF) were observed in cerebellar vermis, brainstem and right anterior cingulate.

105 to eye movements, suggesting that the caudal vermis can also directly influence vestibulo-ocular path

106 Optical stimulation of the oculomotor vermis caused saccade dysmetria.

107 The vermis classically is thought to be included within the

108 Gray matter volume in the anterior superior vermis correlated with lifetime alcohol consumption in t

109 ole-cell recording from slices of cerebellar vermis derived from juvenile (P18-25) or adult (P60-83)

110 mother-reared monkeys, we found an enlarged vermis, dorsomedial prefrontal cortex, and dorsal anteri

111 tion patterns were present in the cerebellar vermis during bimanual coordination tasks, with greater

112 lus) and IXc,d (ventral uvula) of the caudal vermis during vestibular stimulation.

113 IXc,d (ventral uvula) of the macaque caudal vermis during vestibular stimulation.

114 posterior lobule which echoes the posterior vermis DW 'tail sign' observed in human imaging studies.

115 Therefore, many P-cells in the oculomotor vermis exhibit changes in SS activity specific to adapte

116 ad, responses in ancient cerebellar regions (vermis, fastigal nucleus, archicerebellum) may be more d

117 in one case with epilepsy, which also showed vermis folial malformation.

118 re additionally required to pattern anterior vermis foliation.

119 nd ventral uvula (lobules X and IXc,d of the vermis) for vestibular processing has been strongly sugg

120 In addition, the right cerebellar vermis had enhanced connectivity with motor and cognitiv

121 lic activity and the supramarginal gyrus and vermis had significantly more metabolic activity.

122 Also, in preclinical studies, the vermis has been shown to modulate forebrain dopamine sys

123 These results establish that the oculomotor vermis helps control the characteristics of normal ipsiv

124 Therefore, we show that the caudal vermis hosts a re-encoded, gravitationally polarized rep

125 Unlike the cerebellar vermis, however, MSTd neurons also carry a spatial orien

126 ior fossa malformations including cerebellar vermis hypoplasia (CVH), mega-cisterna magna (MCM) and D

127 rt Syndrome, a ciliopathy causing cerebellar vermis hypoplasia and ataxia.

128 Finally, we report cerebellar vermis hypoplasia in 35% of CHARGE syndrome patients wit

129 nd link reduced FGF signalling to cerebellar vermis hypoplasia in a human syndrome.

130 regression of the rhombic lip and posterior vermis hypoplasia in Lmx1a(-/-) mice.

131 hh signaling defects could contribute to the vermis hypoplasia observed in the human syndromes.

132 n presented congenital ataxia and cerebellar vermis hypoplasia with elongated superior cerebellar ped

133 tisystem disease characterized by cerebellar vermis hypoplasia with prominent superior cerebellar ped

134 thy Joubert syndrome is marked by cerebellar vermis hypoplasia, a phenotype for which the pathogenic

135 l brain disorder characterized by cerebellar vermis hypoplasia, abnormal eye movement, ataxia and men

136 ysgenesis of the corpus callosum, cerebellar vermis hypoplasia, and facial dysmorphism.

137 on axial brain MRI, together with cerebellar vermis hypoplasia, ataxia, and psychomotor delay.

138 al signal), thin corpus callosum, cerebellar vermis hypoplasia, optic nerve hypoplasia and mild ventr

139 disorder featuring absence of the cerebellar vermis (i.e. midline).

140 e image analysis, we measured the cerebellar vermis in 125 normal individuals with a broad age range

141 is preliminary study supports a role for the vermis in ADHD and suggests that further research is nee

142 each lobe of the cerebellar hemispheres and vermis in children with ADHD and comparison subjects and

143 ontal cortex, corpus callosum, and posterior vermis in children with autism and further suggest that

144 ls in the vermis and pons in AOA2 and in the vermis in FRDA.

145 iform gyri and reduced GMV in the cerebellar vermis in FXS at both timepoints, suggesting early, poss

146 endent loss of the most anterior lobe of the vermis in mice lacking Fgf17 and in mice lacking Fgf17 a

147 Lesions of the vermis in particular were associated with dysregulation

148 the pons, the cerebellar hemisphere and the vermis in patients with FRDA and AOA2 to identify potent

149 Injury to the cerebellar vermis in patients with mTBI and anxiety may indicate un

150 after stimulation of the right neocerebellar vermis, in contrast to all other conditions.

151 ent), to complete (the absence of the entire vermis including nodulus).

152 e in AOA2 than controls were observed in the vermis, indicating different mechanisms possibly leading

153 Volumes of the vermis, inferior posterior lobe, fourth ventricle, and t

154 he plasticity at the level of the oculomotor vermis is more fundamentally important for forward adapt

155 Cerebellar fusion and absence of cerebellar vermis is often associated with supratentorial findings.

156 e projection from the cerebral cortex to the vermis is part of the neural substrate for anticipatory

157 c, the cerebellum is underdeveloped, and the vermis is severely reduced.

158 hree groups based on primary tumor location: vermis, left hemisphere, or right hemisphere.

159 ittent theta burst stimulation (iTBS) to the vermis lobule VII or right lateral cerebellar Crus I/II,

160 hippocampal volumes, and smaller cerebellar vermis lobules VI and VII, in comparison with his brothe

161 volumes in the posterior-inferior cerebellar vermis (lobules VIII-X; effect size, 0.54; P =.04), even

162 vity of identified P-cells in the oculomotor vermis, lobules VIc and VII.

163 ggest that cell death is not responsible for vermis loss, but rather that it fails to develop because

164 hese data suggest that an abnormality in the vermis may contribute to the pathophysiology of schizoph

165 These mice show cerebellar hypoplasia with a vermis-midline fusion defect early in development.

166 pmental links between frontal and cerebellar vermis neural abnormalities were supported, in that impa

167 gnificantly, the differentiation of anterior vermis neuroepithelium was shifted rostrally and mediall

168 imilar to lobules 9 and 10 of the cerebellar vermis (nodulus and uvula), MSTd neurons respond selecti

169 ctions of postmortem samples from cerebellar vermis of 10 patients with schizophrenia and 13 control

170 two-photon Ca(2+) imaging in the cerebellar vermis of awake behaving mice.

171 ed Purkinje-cell discharge in the oculomotor vermis of behaving rhesus monkeys (Macaca mulatta) and f

172 HO-1 was induced in Bergmann glia in the vermis of cerebellum.

173 The vermis of lobule VII receives a prominent input from the

174 low doses of kainic acid into the cerebellar vermis of mice elicited reliable and reproducible dyston

175 sitometric units [P=.003]) in the cerebellar vermis of schizophrenic subjects.

176 density of Purkinje cells in the cerebellar vermis of subjects with and without schizophrenia.

177 ateral putamen and insula, anterior lobe and vermis of the cerebellum and superior colliculus).

178 hippocampus (CA1 and dentate), thalamus, and vermis of the cerebellum as early as 3 h post injury.

179 on steady-state blood volume in the midline vermis of the cerebellum in boys with attention deficit

180 The posterior vermis of the cerebellum is considered to be critically

181 fects (Purkinje cell loss) manifested in the vermis of the cerebellum.

182 sked whether Purkinje cells (P-cells) in the vermis of the oculomotor cerebellum, lobules VIc and VII

183 e in the cerebellum (hemispheres and midline vermis) of 10 boys with ADHD who were administered place

184 The oculomotor vermis (OMV) of the cerebellum is necessary for the gene

185 tion of gaze called saccades, the oculomotor vermis (OMV) of the cerebellum must be intact.

186 The influence of the cerebellar vermis on prefrontal and striatal circuitry should be ex

187 each case, the surgery destroyed the midline vermis only (ranging from lobules VI-X).

188 quired to ensure that folia exclusive to the vermis or hemispheres form in the appropriate mediolater

189 resulting from dysfunction in the cerebellar vermis or the basal ganglia.

190 in right amygdala/hippocampus and cerebellar vermis (P < 0.001), relative to global brain.

191 had diminished fractional anisotropy in the vermis (P = .04).

192 which arises predominantly in the cerebellar vermis, preferentially affects children between the ages

193 We found that caudal cerebellar vermis Purkinje cells and cerebellar nuclei neurons sele

194 icant intersubject variability (decreases in vermis ranged from 18% to 60%).

195 cterized by severe defects of the cerebellar vermis, ranging from hypoplasia to aplasia.

196 the surprising result that a portion of the vermis receives dense input from the cerebral cortex.

197 ing mediolateral expansion of the cerebellar vermis, reduced thickness of the granule cell layer and

198 n of Spry2 alone caused loss of the anterior vermis, reducing FGF signaling further, by decreasing Fg

199 ysis of cerebellar sections from the midline vermis revealed that during development, the expression

200 l stress tasks, increased rCBF in cerebellar vermis, right anterior cingulate and right insula covari

201 the CS activity of P-cells in the oculomotor vermis signals the direction but not the magnitude of ey

202 RS had significantly higher MD values in the vermis than did healthy subjects (P < .05) and patients

203 inly in cortical structures, cerebellum, and vermis) that could be attenuated by pretreatment with ha

204 loss of zebrin II/EAAT4-negative PCs in the vermis, the densities of microglia and the Bergmann glia

205 isexpression also affects development of the vermis, the part of the cerebellum that spans the midlin

206 lume was observed in the superior cerebellar vermis; the volume loss persisted regardless of clinical

207 l absence of nodulus, anterior and posterior vermis), to moderate (the absence of posterior vermis wi

208 ion of patients (PwMS and PwCIS), GM loss in vermis VI (R(2)=0.36; p<0.05 when considering age and T2

209 me decrease in posterior lobules (especially vermis VI) was associated with reduced IPS.

210 IIA and right HVI) and portions of posterior vermis (VI and superior VIIA) exhibited increased activa

211 resonance imaging to measure cerebellum and vermis volume in 15 patients with schizophrenia and 15 n

212 They found that 1) vermis volume was greater in patients with schizophrenia

213 l prefrontal cortex, amygdala, and posterior vermis volumes were significantly associated with the se

214 in that impaired neuronal functioning in the vermis was associated with impaired neuronal functioning

215 on; specifically, lobule VII in the anterior vermis was missing.

216 The inferior vermis was significantly smaller in the schizophrenic gr

217 The cerebellar vermis was strongly activated by capsaicin, whereas ligh

218 terior quadrangular lobule, lingula, and the vermis were activated.

219 autistic twins, and volumes of the posterior vermis were altered in both autistic twins and co-twins.

220 hippocampus, corpus callosum, and cerebellar vermis were compared between mother-reared (n = 15) and

221 Blocks of alcohol-fixed cerebellar vermis were dissected at autopsy from the brains of 14 e

222 levels of the MAP kinases in the cerebellar vermis were linked to additional downstream targets of t

223 l cerebellum and the midsagittal area of the vermis were measured manually.

224 The blocks of vermis were sectioned and stained with 1% cresyl violet.

225 In the vermis, which supports a range of behavioral functions,

226 ophrenia than in normal subjects, 2) greater vermis white matter volume in the patients with schizoph

227 ormation characterized by missing cerebellar vermis with apparent fusion of the cerebellar hemisphere

228 p of Purkinje cells in the caudal cerebellar vermis with responses that reflect an estimate of head t

229 rmis), to moderate (the absence of posterior vermis with some anterior vermis and nodulus present), t

230 evere (the absence of posterior and anterior vermis with some nodulus present), to complete (the abse