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1 n the left internal carotid artery and right vertebral artery.
2 e catheterization of the internal carotid or vertebral arteries.
3 aged 53-86 years; mean, 73 years) had normal vertebral arteries.
4 ressure assessed at the internal carotid and vertebral arteries.
5 ery (52 patients, 15 bilateral) intracranial vertebral artery (40 patients, 12 bilateral), basilar ar
6 commonest occlusive sites were: extracranial vertebral artery (52 patients, 15 bilateral) intracrania
7 ries, including the extracranial carotid and vertebral arteries and intracranial arteries, is increas
8           Imaging data on the patency of the vertebral arteries and posterior communicating arteries,
9 on the left side and dissection of the right vertebral artery and no ischemic changes within the brai
10 d mild to severe ostial stenosis of a single vertebral artery, and eight patients (including four men
11    We conclude that endovascular stenting of vertebral artery atherosclerotic disease is safe and eff
12                 The treatment of carotid and vertebral artery dissections is based on rather incomple
13  many patients being treated for carotid and vertebral artery dissections with percutaneous angioplas
14           The presence of unilateral delayed vertebral artery enhancement was significantly associate
15 ral artery stenosis and eight controls, both vertebral arteries filled simultaneously.
16 lavian stenosis or occlusion with retrograde vertebral artery flow confirmed with time-of-flight MR a
17  vertebral asymmetry influence mixing of the vertebral artery flow contributions.
18                                              Vertebral artery flow reversal is often found among pati
19       In the past 5 years, BCVI (carotid and vertebral arteries) has been recognized with increasing
20 ence of congenital cerebrovascular variants; vertebral artery hypoplasia, and an incomplete posterior
21  Eight studies that examined 5704 carotid or vertebral arteries in 1426 trauma patients met inclusion
22 ranial carotid arteries in 251 patients, and vertebral arteries in 82 patients.
23 sient 20 min occlusion of common carotid and vertebral arteries in rats caused a dramatic (3-fold) in
24   Spontaneous dissections of the carotid and vertebral arteries in the neck are a common cause of str
25 uries (43 bilateral), and 79 patients had 97 vertebral artery injuries (18 bilateral).
26 d artery injuries (CAI) and 43 patients with vertebral artery injuries (VAI) for an overall screening
27                    No patients with isolated vertebral artery injuries had positive transcranial Dopp
28  injuries, but monitoring was not useful for vertebral artery injuries.
29 elayed stroke among patients who sustained a vertebral artery injury with or without additional vesse
30 ion (CeAD), a mural hematoma in a carotid or vertebral artery, is a major cause of ischemic stroke in
31 ak contrast enhancement in the right or left vertebral arteries may, in the appropriate clinical sett
32 ulted from carotid injury and 4 (26.7%) from vertebral artery occlusion (P = .03).
33 chanism of brain infarction in patients with vertebral artery occlusive disease.
34 delay in peak enhancement in the ipsilateral vertebral artery ranged from 2 to 4 seconds (mean, 2.5 s
35  normal flow (P < .01) and those with ostial vertebral artery stenosis (P < .01).
36 ty of catheter-based therapy for symptomatic vertebral artery stenosis (VAS).
37        In eight of nine patients with ostial vertebral artery stenosis and eight controls, both verte
38 e studies of the exceedingly rare rotational vertebral artery syndrome have been added to the literat
39 including vertigo associated with rotational vertebral artery syndrome, as well as whiplash and degen
40  than the 'less-reactive' CA measured at the vertebral artery that was associated with WMH severity.
41 e dogs received glucose via both carotid and vertebral arteries to maintain cerebral euglycemia (H-EU
42                           In the Carotid and Vertebral Artery Transluminal Angioplasty Study (CAVATAS
43 ort the long-term results of the Carotid and Vertebral Artery Transluminal Angioplasty Study (CAVATAS
44 sonography at the internal carotid (ICA) and vertebral arteries (VA).
45 red at the internal carotid artery (ICA) and vertebral artery (VA) and CBF velocity at the middle cer
46 the internal carotid arteries (ICAs) and the vertebral arteries (VAs); 3) atherosclerosis of the larg
47 ae leads to a complete loss of the bilateral vertebral arteries (VTAs) that extend along the ventrola
48  signal intensity between the right and left vertebral arteries was compared among the three groups b
49           Using a volume-rendered angiogram, vertebral arteries were measured along the curvature of
50  hundred fourteen (73%) carotid and 65 (67%) vertebral arteries were restudied with arteriography 7 t
51  inflammation within the aorta, carotid, and vertebral arteries with histologic validation in humans

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