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1 terior elements, with partial involvement of vertebral body.
2 mount of polymethylmethacrylate injected per vertebral body.
3 tion of inflatable bone tamps (IBT) into the vertebral body.
4 rienced relapse, with painful collapse of L3 vertebral body.
5 ance in 1984 due to a hemangioma of cervical vertebral body.
6 d the destruction of the anterior rim of the vertebral body.
7 y fractures and liquefactive necrosis of the vertebral body.
8 n the midline to the posterior aspect of the vertebral body.
9 n the intervertebral disc accumulates in the vertebral body.
10  protein from the intervertebral disc to the vertebral body.
11 iffuse away from its place of synthesis, the vertebral body.
12 ight of or over the spinous processes of the vertebral body.
13 ocytic cells of presumptive inner annuli and vertebral bodies.
14  narrowing, and anterior wedging of involved vertebral bodies.
15 c space and permanent fusion of the adjacent vertebral bodies.
16 n the craniofacial structures but not in the vertebral bodies.
17 f 98.3% for the differentiation of edematous vertebral bodies.
18 ed to well-segmented areas of the developing vertebral bodies.
19 n infusion of donor BM cells isolated from 9 vertebral bodies.
20 gth and stiffness in both the tibiae and the vertebral bodies.
21 facial cartilage, heart, bronchi, kidney and vertebral bodies.
22 5/8 phosphorylation was decreased in CV2(-/-)vertebral bodies.
23 terally within the thoracic and lumbar spine vertebral bodies.
24 V2 protein is normally located in the future vertebral bodies.
25 diameter at the aortic arch, carina, and one vertebral body above the gastroesophageal junction was 1
26  epidural empyemas, abscess between adjacent vertebral bodies, abscesses beneath anterior longitudina
27 rning defects that included fusions of ribs, vertebral bodies and abnormal formation of spinous proce
28              Apart from ordinary features of vertebral bodies and discs, progressive spinal destructi
29 bris, and gadolinium-enhancement patterns of vertebral bodies and disks may help differentiate spinal
30 l skeleton consists of a metameric series of vertebral bodies and intervertebral discs, as well as ad
31  from both somitic mesoderm, which forms the vertebral bodies and ribs, and from lateral plate mesode
32           Chordomas are tumors that arise at vertebral bodies and the base of the skull.
33 f occult vertebral body and facet fractures, vertebral body and facet contusions, intervertebral disk
34 dent readers assessed the presence of occult vertebral body and facet fractures, vertebral body and f
35 f the abdominal aorta at the level of the L3 vertebral body and its associations with multiple variat
36 ed diffuse bony involvement including the T7 vertebral body and left pedicle, ribs, pelvis, and calva
37 Lytic disease involving more than 40% of the vertebral body and location at or below T10 confer a hig
38  on chest radiograph with destruction of the vertebral body and preservation of the disc space.
39 rst from intervertebral spaces and then from vertebral bodies, and it progressively underwent apoptos
40 imarily in the anterosuperior portion of the vertebral body, and cleft margins appeared increasingly
41 roplasty, long-term outcome of cement in the vertebral body, and utility of prophylactic vertebroplas
42                                              Vertebral bodies are segmented along the anteroposterior
43                  In lumbar vertebrae reduced vertebral body area and wall thickness were accompanied
44 usions were more widespread and involved the vertebral bodies as well as the neural arches.
45     The purpose of the IBT is to restore the vertebral body back toward its original height, while cr
46 ecipients infused perioperatively with donor vertebral body bone marrow (DBMC-i) vs. 219 noninfused c
47 ecipients infused postoperatively with donor vertebral body bone marrow cells.
48 leen cells in the presence of specific donor vertebral-body bone marrow cell (DBMC) modulators were t
49 populations were isolated from cadaver donor vertebral-body bone marrow cells (DBMC) by using immuno-
50                                              Vertebral body cartilage from embryonic day 7 and embryo
51 entrate diffusible Tsg/BMP4 complexes in the vertebral body cartilage.
52                   Surprisingly, we find that vertebral bodies (centra) arise by secretion of bone mat
53  vertebral body (P<.01), and continuation of vertebral body changes with posterior pharyngeal wall ul
54 rvening joint change (P<.001), more cases of vertebral body collapse (P<.01), more bilateral symmetri
55              At least one moderate or severe vertebral body compression fracture was identified retro
56 DXA, 39 (48%) of 81 patients with unreported vertebral body compression fractures had a nonosteoporot
57                    Most clinically important vertebral body compression fractures in nontrauma patien
58 iewed for the presence of moderate or severe vertebral body compression fractures of the lower thorac
59             Other findings included adjacent vertebral body destruction with psoas muscle abscess (n
60                           Bone marrow in the vertebral bodies displays somewhat variable behavior at
61 ost notochord cells dispersed throughout the vertebral bodies during embryogenesis.
62 The resulting reinforcement of the fractured vertebral bodies eliminated the pain and the need for na
63  able to depict bone marrow in the collapsed vertebral bodies, especially in those with less than 50%
64  vertebral bodies, severe destruction of the vertebral body, focal/heterogeneous contrast enhancement
65            Bone needle was inserted into the vertebral body, followed by injection of PMMA cement.
66                                OO was in the vertebral body for 18 of 57 patients, the neural arch fo
67 ression of CDMP-1 in the notochord inhibited vertebral body formation by blocking migration of sclero
68 , one person was diagnosed with pathological vertebral body fractures and liquefactive necrosis of th
69 asty, 22 (12.4%) developed a total of 36 new vertebral body fractures following treatment.
70           There were 141 thoracic and lumbar vertebral body fractures in the case set.
71 er system detects and anatomically localizes vertebral body fractures in the thoracic and lumbar spin
72 ith positive findings for fractures (59 with vertebral body fractures) and 10 control examinations (w
73  to therapeutic approaches that help prevent vertebral body fractures.
74                        Mean opacification of vertebral body halves was 83% +/- 19 (SD) and 77% +/- 16
75                        Kyphoplasty increased vertebral body height more than vertebroplasty in this m
76   The statistical significance of changes in vertebral body height, wedge angle, and weight with the
77  and function as well as some restoration of vertebral body height.
78 ry, required a >or=20% (or >or=4-mm) loss of vertebral body height.
79         The anterior, central, and posterior vertebral body heights and wedge angles were measured in
80  focal/heterogeneous contrast enhancement of vertebral bodies, heterogeneous signal from the vertebra
81 f vertebral bodies, low-grade destruction of vertebral bodies, hyperintense/homogeneous signal from t
82                                 Five porcine vertebral bodies in one pig underwent intervention (IRE
83 wth plate chondrocytes and in the developing vertebral bodies in the mouse.
84 ed at or below the level of the third sacral vertebral body in all 49 patients with isolated pelvic f
85                                   As percent vertebral body involvement increased, odds of fracture a
86  CT appearance, lesion location, and percent vertebral body involvement independently predicted fract
87 attenuation of the trabecular bone of the L5 vertebral body (L5HU) was measured.
88 grams, and their relationships to particular vertebral body levels were recorded.
89  diffuse/homogeneous contrast enhancement of vertebral bodies, low-grade destruction of vertebral bod
90 ate recipients of allogeneic male islets and vertebral body marrow (VBM) from the same donor.
91 pients of cadaver donor liver allografts and vertebral body marrow infusions.
92                            Adjacent abnormal vertebral body marrow signal intensity was seen in one (
93 and in a phantom model, visualization of the vertebral body marrow was improved and susceptibility ar
94  safe for the treatment of painful posterior vertebral body metastatic tumors.
95 o independent readers visually evaluated all vertebral bodies (n = 163) for the presence of abnormal
96  measure bone density of thoracic and lumbar vertebral bodies on computed tomographic (CT) images.
97 images, diffuse signal intensity patterns in vertebral bodies on MR images, and rim enhancement of di
98 tebral bodies, heterogeneous signal from the vertebral bodies on T2 TIRM images, well-defined paraspi
99 es, hyperintense/homogeneous signal from the vertebral bodies on T2 TIRM images.
100 e location was related to that of a thoracic vertebral body on frontal and lateral chest radiographs
101 ssiveness, spinal level, location within the vertebral body or posterior elements, involvement of sof
102 e IRE electrode to the posterior wall of the vertebral body or the exiting nerve root were 2.93 mm +/
103 , each consisting of two key components: the vertebral body (or centrum) and the vertebral arches.
104 te cancer cells were either coimplanted with vertebral bodies, or inoculated in the tibiae of immunoc
105  more bilateral symmetric involvement of the vertebral body (P<.01), and continuation of vertebral bo
106 acture (P<.01), bone marrow contusion of the vertebral body (P=.01), muscle strain (P<.01) or tear (P
107 Romanus lesions (RLs) and end-plate, diffuse vertebral body, posterior element, and spinous process b
108                 Anatomical sites such as the vertebral body, proximal femur, and distal radius are lo
109 toff value of -80 to differentiate edematous vertebral bodies resulted in a sensitivity of 96.3%, spe
110 cant changes in the images of bone marrow in vertebral body scans; with a decrease in the intensity o
111 d bone marrow edema on STIR images from each vertebral body, separately identifying central and later
112 cic spine, involvement of 2 or more adjacent vertebral bodies, severe destruction of the vertebral bo
113 .19, 0.34) and between a type 1 endplate and vertebral body spondylolisthesis (PPV, 0.28; 95% CI: 0.2
114              Vacuum disk, facet involvement, vertebral body spondylolisthesis, joint disorganization
115      NAT was measured at the level of the C5 vertebral body, subdivided into posterior (NATpost), sub
116  and the ubiquitous osteochondrodysplasia of vertebral bodies to the occasional sarcoma in older anim
117 rument could be navigated into the posterior vertebral body tumors with a transpedicular approach.
118 47 tumors) with painful metastatic posterior vertebral body tumors, some of which were radiation ther
119                          Failure load of the vertebral bodies was determined from destructive biomech
120 anwhile, the development of inner annuli and vertebral bodies was dramatically impaired.
121 furcation and duodenal papilla from adjacent vertebral bodies was measured on 34 cholangiograms, and
122 ion, and morphology of the tibiae and lumbar vertebral bodies were assessed by micro-computed tomogra
123                                              Vertebral bodies were assessed for incident vertebral fr
124                                              Vertebral bodies were only mildly affected, but the inte
125                                    While the vertebral bodies were only moderately affected, the inte
126                              Twelve thoracic vertebral bodies were removed from three human cadavers
127 s of transgenic mice were tougher, and their vertebral bodies were stiffer and stronger than those of
128                       Lesions located in the vertebral body were excluded.
129                          Lesions confined to vertebral body were less frequent (12 cases).
130  marrow, and skeletal muscle adjacent to the vertebral body were present.
131                          DBMC, obtained from vertebral bodies, were administered in 101 recipients of
132 wer chondrogenic cells within the developing vertebral body, which fail to condense appropriately alo
133 and calculated as a fraction of the adjacent vertebral body width.
134  VNCa images were significantly different in vertebral bodies with and without edema (P < .001).
135                In all cases a destruction of vertebral bodies with end plates loss restriction and co
136 98.2%, and accuracy of 97.6% in the group of vertebral bodies with less than 50% sclerosis and/or air
137                                        In 18 vertebral bodies with titanium fixation screws and in a
138 moderate amounts of PMMA may escape from the vertebral body with no significant effect on therapeutic
139 ose single-section quantitative CT of the L4 vertebral body with use of a calibration phantom.
140  with marked mineralization were confined to vertebral body, with "ivory vertebra" appearance.
141 N, or occurrence of first fracture (eg, hip, vertebral body, wrist).

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