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1 ed exclusively in mild defects in the caudal vertebral column.
2 , controls segmentation of precursors of the vertebral column.
3 discs (IVD) are essential components of the vertebral column.
4 notochord to constrain these cells along the vertebral column.
5 contributes to neuromuscular control of the vertebral column.
6 cute effects on neuromuscular control of the vertebral column.
7 by the repetition of vertebrae that form the vertebral column.
8 rcoma cases, 198 (4%) were PVOS arising from vertebral column.
9 resegmentation in development from somite to vertebral column.
10 e first two somites do not contribute to the vertebral column.
11 rtebrates is evident in the character of the vertebral column.
12 carring and tissue resorption, have a closed vertebral column.
13 ature at the cervicothoracic boundary of the vertebral column.
14 t of the dome referenced horizontally to the vertebral column.
15 s both in the craniofacial region and in the vertebral column.
16 morphic) life cycle had an increased rate of vertebral column and body form diversification compared
17 ormation or absence of specific bones of the vertebral column and cranial bones of mesodermal origin,
18 nsformations along the cervical and thoracic vertebral column and defects in sternum morphogenesis.
23 malformations are localized along the entire vertebral column and rib cage and are linked to defectiv
24 6R) mutants manifest skeletal defects in the vertebral column and ribcage, revealing a hitherto undef
25 a morphogen mode, whereas development of the vertebral column and the hind limbs has threshold signal
26 rmal development of the caudal aspect of the vertebral column and the spinal cord., It results in neu
28 differentiation in the developing skull and vertebral column and understanding how perturbations in
29 e vertebrates are defined by their segmented vertebral column, and vertebral periodicity is thought t
30 mented precursors of the skeletal muscle and vertebral column are generated during vertebrate embryog
32 m (PSM) and differentiate into the segmented vertebral column as well as other unsegmented tissues.
34 dentity are patterned requires understanding vertebral column cellular and developmental biology.
37 had kidney defects, whereas chimeras without vertebral column defects had highly chimeric kidneys tha
43 entally revises our current understanding of vertebral column evolution in the earliest tetrapods and
54 vidual vertebrae and distinct regions of the vertebral column is accomplished by Polycomb and Hox pro
55 The periodic or segmental pattern of the vertebral column is established early in development whe
57 he close proximity of the spinal cord to the vertebral column limits many conventional therapeutic op
58 and differentiate after birth along with the vertebral column, little is known about the mechanism of
60 posterior-to-anterior transformations of the vertebral column midsection, similar to mice deficient f
61 soft tissues and structures surrounding the vertebral column, neurological deficits, and spinal inst
65 ntiated from the teratoma by the presence of vertebral column often with an appropriate arrangement o
68 ovo cell and tissue interactions pattern the vertebral column remains a fundamental, unresolved issue
70 the 1960s, show features of the shoulder and vertebral column that are significantly similar to those
71 ation, including re-patterning of the caudal vertebral column that is otherwise only seen in salamand
72 d signals are essential for formation of the vertebral column the phenotypes suggested that the lacZ
73 f the mid-RPT from the lateral margin of the vertebral column was measured and calculated as a fracti
76 shifts in vertebral types along most of the vertebral column, with transformations being most obviou
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