ライフサイエンスコーパス: vertigo

1 ally required in order to achieve control of vertigo.

2 pathophysiological model of migraine-related vertigo.

3 symptoms in patients with both migraine and vertigo.

4 dge of the pathogenesis of both migraine and vertigo.

5 ith mild tinnitus but is not associated with vertigo.

6 ase, 5 of 9 had improvement or resolution of vertigo.

7 ents with a history of positionally provoked vertigo.

8 recurrent attacks of vertigo, and positional vertigo.

9 ntiation of peripheral and central causes of vertigo.

10 ause or exacerbate symptoms in patients with vertigo.

11 egarding diagnostic methods for cervicogenic vertigo.

12 e over the last year concerning cervicogenic vertigo.

13 positioning for benign paroxysmal positional vertigo.

14 symptoms include diplopia, oscillopsia, and vertigo.

15 eral and can be associated with tinnitus and vertigo.

16 mab: one case of urosepsis and an episode of vertigo.

17 improve diagnosis and treatment of recurrent vertigo.

18 the brainstem and cerebellum that can cause vertigo.

19 reatment options exist for the management of vertigo.

20 through which gentamicin leads to control of vertigo.

21 s included hearing loss (13), tinnitus (12), vertigo (8), and facial paresis (1).

22 Of 16 patients with vertigo, 8 (50%) were improved, 7 (47%) unchanged, and 1

23 Physicians find acute vertigo a diagnostic challenge.

24 gn paroxysmal torticollis, benign paroxysmal vertigo, abdominal migraine, and cyclic vomiting syndrom

25 lantar erythrodysesthesia, nausea, vomiting, vertigo, abdominal pain, diarrhea, and thrombocytopenia.

26 hed criteria for the diagnosis of migrainous vertigo allows the development of a standardized, struct

27 Individuals with vestibular vertigo also had a threefold increased odds of depressio

28 rved an 8.4% 1-year prevalence of vestibular vertigo among US adults.

29 dic ataxia type 2 is a prototypical episodic vertigo and ataxia syndrome that is caused by mutations

30 Two sons have episodes of vertigo and ataxia that are not responsive to acetazolam

31 his review focuses on prospective studies of vertigo and balance therapy in the past 3 years, includi

32 ied the pathophysiology of benign positional vertigo and documented the efficacy of particle repositi

33 ted to the hospital with severe non-systemic vertigo and dysarthria, which had lasted for a couple of

34 overlap between vestibular symptoms, such as vertigo and head-movement intolerance, and migraine symp

35 dissociate vestibular impairments that cause vertigo and imbalance in patients.

36 lapping and interrelated problems of emesis, vertigo and migraine which promises an early solution to

37 n for a correlation between benign recurrent vertigo and migraine, and acceptance for vertigo as a ma

38 s, postoperative nausea and vomiting (PONV), vertigo and morning sickness and observing new associati

39 The effects of head pitch on vertigo and previously reported nystagmus are consistent

40 ng different familial syndromes of recurrent vertigo and strong association with migraine suggest sha

41 tery can cause neurological symptoms such as vertigo and syncope.

42 When present, vertigo and tinnitus were assessed by frequency and seve

43 orineural hearing loss, often accompanied by vertigo and tinnitus.

44 story of dysarthria, progressively worsening vertigo, and difficulty walking.

45 rineural hearing loss, tinnitus and episodic vertigo, and familial MD is observed in 5-15% of sporadi

46 nking, disorientation, balance disturbances, vertigo, and impotence), and 3 ("arthro-myo-neuropathy,"

47 aracterized by spells of ataxia, dysarthria, vertigo, and migraines, associated with mutations in the

48 ed spontaneous vertigo, recurrent attacks of vertigo, and positional vertigo.

49 e severity of hearing loss, age, presence of vertigo, and shape of the audiogram.

50 ts of psychogenic dizziness, phobic postural vertigo, and space-motion phobia.

51 ndings have not made the assessment of acute vertigo any easier for the nonspecialist.

52 ty loci for migraine and migraine-associated vertigo are underway.

53 ent vertigo and migraine, and acceptance for vertigo as a manifestation of migraine; efforts to ident

54 utoimmune disorders include hearing loss and vertigo as part of their constellation of symptoms.

55 re concerning cervicogenic vertigo including vertigo associated with rotational vertebral artery synd

56 cterized by recurrent attacks of spontaneous vertigo associated with sensorineural hearing loss (SNHL

57 She had no nystagmus or vertigo at pneumatic otoscopy.

58 Meniere's disease is characterised by severe vertigo attacks and hearing loss.

59 The vertigo attacks have not occurred for the last 18 months

60 is (ie, all 60 patients), the mean number of vertigo attacks in the final 6 months compared with the

61 ry of acute otitis media, otorrhea, otalgia, vertigo, autophony, or tinnitus since her adoption.

62 h static magnetic fields are known to induce vertigo, believed to be via stimulation of the vestibula

63 The most common cause of vertigo, benign paroxysmal positional vertigo, can be cu

64 ecurrent vertigo (mainly migraine-associated vertigo), bilateral vestibulopathy, and Meniere's diseas

65 pain after a post-treatment liver biopsy and vertigo), both unrelated to study drugs.

66 responsible for benign paroxysmal positional vertigo (BPPV) in humans.

67 loped bilateral benign paroxysmal positional vertigo (BPPV) of the posterior canals, deafness, and ab

68 ypofunction and benign paroxysmal positional vertigo (BPPV).

69 ated balance disorders and Benign Positional Vertigo (BPV).

70 Benign recurrent vertigo (BRV) is a common disorder affecting up to 2% of

71 nt for refractory Meniere's disease, reduces vertigo, but damages vestibular function and can worsen

72 use of vertigo, benign paroxysmal positional vertigo, can be cured with a simple positional manoeuvre

73 positional vertigo, the most common cause of vertigo, can now be cured with a simple bedside maneuver

74 report we describe a patient complaining of vertigo caused by spontaneous rupture of dermoid cyst, p

75 evaluated the association between vestibular vertigo, cognitive impairment (memory loss, difficulty c

76 evaluated the association between vestibular vertigo, cognitive impairment and psychiatric conditions

77 he corticosteroid methylprednisolone reduces vertigo compared with gentamicin.

78 estibular neurectomy has a very high rate of vertigo control and is available for patients with good

79 Patients whose vertigo did not improve after injection (ie, non-respond

80 rally well tolerated, but asthenia, fatigue, vertigo, dizziness, sense of imbalance, and loss of conc

81 descending order of frequency): hemiparesis, vertigo/dizziness, diplopia, dysarthria, nystagmus, naus

82 ing that any 'perceptual noise' added by the vertigo does not disrupt the cognitive decision-making p

83 Vertigo due to cerebrovascular disease can be of periphe

84 nt isolated brainstem symptoms (eg, isolated vertigo, dysarthria, diplopia) are not consistently clas

85 ) on qualitative and quantitative aspects of vertigo experienced in the dark by healthy humans when e

86 ess and dizziness (VUD, also known as visual vertigo), fear of falling (FoF), and chronic subjective

87 e is characterized by spontaneous attacks of vertigo, fluctuating sensorineural hearing loss, aural f

88 Quantitative analysis of vertigo focused on the induced perception of horizontal-

89 The primary outcome was vertigo frequency over the final 6 months (18-24 months

90 y in the past 3 years, including advances in vertigo-habituation exercises for adults, pediatric inte

91 ed for the efficacy of a minimal, home-based vertigo-habituation program for adults with peripheral v

92 justed analyses, individuals with vestibular vertigo had an eightfold increased odds of 'serious diff

93 (low dose) can be used in patients for whom vertigo has not been controlled by medical measures.

94 unction or with benign paroxysmal positional vertigo have been published recently, adding to the smal

95 ugh many patients with positionally provoked vertigo have typical benign paroxysmal positional vertig

96 ange of neurological symptoms from tinnitus, vertigo, headaches, and deafness to blindness and convul

97 tes type 2 and hyperlipidemia presented with vertigo, headaches, mainly during physical activity and

98 The identification of migrainous vertigo, however, is hampered by a lack of standardized

99 Vertigo in and around magnetic resonance imaging (MRI) m

100 Other common causes of vertigo include vestibular neuritis, Meniere's syndrome,

101 he recent literature concerning cervicogenic vertigo including vertigo associated with rotational ver

102 Benign paroxysmal positional vertigo is a common disorder of the inner ear that shoul

103 Vertigo is a subtype of dizziness, which results from an

104 Vertigo is often the most debilitating symptom associate

105 Although benign paroxysmal positional vertigo is usually a self-limited disorder, treatment wi

106 high-frequency sensorineural hearing loss or vertigo, is not uncommon.

107 is characterised by intermittent episodes of vertigo lasting from minutes to hours, with fluctuating

108 verview on episodic ataxia, benign recurrent vertigo (mainly migraine-associated vertigo), bilateral

109 versely benign conditions such as migrainous vertigo may have clinical characteristics of central dis

110 For example, migrainous vertigo may have oculomotor abnormalities suggestive of

111 vidence highlights the difficulty that acute vertigo may sometimes pose to the clinician.

112 igher were neutropenia (n = 2) and dizziness/vertigo (n = 1).

113 The other 54 cases were isolated vertigo (n=23), non-NINDS binocular visual disturbance (

114 On a 1-hour dosing schedule, ataxia, vertigo, nystagmus, and a motor aphasia were the princip

115 He has a 4-year history of episodic vertigo of several hours' duration and fluctuating, prog

116 Persistent vertigo or brainstem and cerebellar episodes can herald

117 ination with variable other symptoms such as vertigo or migraine.

118 EDs in Nueces County, Texas, with dizziness, vertigo, or imbalance were identified, excluding those w

119 e very similar, with patients complaining of vertigo, oscillopsia, and sometimes hearing loss.

120 jection of gentamicin can be beneficial when vertigo persists despite optimal medical management.

121 hromosome 22q12 region to a broader migraine/vertigo phenotype by defining affectation status as eith

122 go have typical benign paroxysmal positional vertigo, physicians should be aware of nonbenign variant

123 , makes magnetic-field-induced nystagmus and vertigo possible.

124 Classification of dizziness by subtype (vertigo, presyncope, disequilibrium, and other) assists

125 nar focuses on three common presentations of vertigo: prolonged spontaneous vertigo, recurrent attack

126 for identifying serious conditions in acute vertigo, recent evidence suggests that early steroid tre

127 sentations of vertigo: prolonged spontaneous vertigo, recurrent attacks of vertigo, and positional ve

128 t establishes that a neck disturbance causes vertigo remains the critical problem that must be solved

129 tem and cerebellar dysfunction or persistent vertigo several months before developing CA.

130 s (Lorentz forces) predicts that the induced vertigo should depend on the orientation of the magnetic

131 and symptoms (headache, numbness, weakness, vertigo, syncope, diplopia, hypotension, floaters, other

132 Although the commonest vertigo syndromes are benign, serious conditions such as

133 erstanding the more common familial episodic vertigo syndromes, particularly those associated with mi

134 Benign positional vertigo, the most common cause of vertigo, can now be cu

135 ent in the AMG 334 70 mg group (migraine and vertigo); these events were judged to be unrelated to AM

136 rineural hearing loss, often associated with vertigo, tinnitus, and aural fullness, and believed to b

137 s or a history of hearing loss, tinnitus, or vertigo underwent additional radiologic and audiologic e

138 ent loss of consciousness, effort-associated vertigo, upper limb weakness and temporary vision proble

139 e classified the brainstem symptoms isolated vertigo, vertigo with non-focal symptoms, isolated doubl

140 Vertigo was most discomforting when head pitch was aroun

141 Hearing loss and vertigo were evaluated at baseline and at completion of

142 imaging, cochlear prostheses and aspects of vertigo which reflect the considerable advances that hav

143 Isolated vertigo with horizontal positional nystagmus as an impen

144 These clinical findings suggest that vertigo with horizontal positional nystagmus, even in th

145 tudy has demonstrated effective treatment of vertigo with migraine therapy.

146 sented to hospital because of sudden, severe vertigo with nausea, impaired balance and disturbed visi

147 ied the brainstem symptoms isolated vertigo, vertigo with non-focal symptoms, isolated double vision,

148 on-NINDS binocular visual disturbance (n=9), vertigo with other non-focal symptoms (n=10), isolated s

149 recent advances in the genetics of recurrent vertigo, with an overview on episodic ataxia, benign rec

150 g constellation of perinfusional aphasia and vertigo, with either ataxia of over 2 weeks' duration or

151 ebellum and lateral medulla can present with vertigo without other localizing symptoms.