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1 s, to potentially milder conditions, such as vesicoureteral reflux.
2 ebrile urinary tract infection commonly have vesicoureteral reflux.
3  kidney after bladder warming would indicate vesicoureteral reflux.
4 velopment of renal scarring in children with vesicoureteral reflux.
5 reased the risk of urologic complication and vesicoureteral reflux.
6 s are ureteropelvic junction obstruction and vesicoureteral reflux.
7 he two approaches in low-grade (grades I-II) vesicoureteral reflux.
8 tive value for ruling out high-grade (III-V) vesicoureteral reflux.
9 l need to reexamine treatment modalities for vesicoureteral reflux.
10 cystourethrography has been used to rule out vesicoureteral reflux.
11 ant to prevent further damage, infection and vesicoureteral reflux.
12 place in the algorithm for the management of vesicoureteral reflux.
13 III genes result in the phenotype of primary vesicoureteral reflux.
14 hildren (112 of 117) had grade I, II, or III vesicoureteral reflux.
15 ortical scarring, and long term follow-up of vesicoureteral reflux.
16 er of the 23 piglets with surgically created vesicoureteral reflux.
17 , 2.47; 95% CI, 1.19-5.12), and grade 4 to 5 vesicoureteral reflux (0.60 per person-year; HR, 4.38; 9
18 ale (90%), 375 were white (78%), and 375 had vesicoureteral reflux (78%).
19 nderwent cystourethrography (117 of 302) had vesicoureteral reflux; 96 percent of these children (112
20                          Among children with vesicoureteral reflux after urinary tract infection, ant
21 f pathology is lessened, however the risk of vesicoureteral reflux and consequent febrile urinary tra
22  have greatly increased our understanding of vesicoureteral reflux and provide a promise of novel non
23 ients as bulking agents for the treatment of vesicoureteral reflux and urinary incontinence.
24 ing obstruction, may also be associated with vesicoureteral reflux and/or obstruction of the bladder
25  incontinence, decreasing surgical rates for vesicoureteral reflux, and decreasing recurrent urinary
26 phric blastema leads to renal hypodysplasia, vesicoureteral reflux, and ectopic ureters to name a few
27  of urinary leak, stricture, compression, or vesicoureteral reflux, and hospital costs were analyzed.
28 nary tract infections, indwelling catheters, vesicoureteral reflux, and immobilization hypercalcuria
29 ection (UTI) requiring antibiotics, grade of vesicoureteral reflux, and posttransplant bladder capaci
30  circumcision debate, dysfunctional voiding, vesicoureteral reflux, and the diagnosis and follow-up o
31 reteroceles, along with the recognition that vesicoureteral reflux associated with ureteroceles can b
32 bnormal screening renal sonograms often have vesicoureteral reflux, but a normal sonogram does not re
33  demonstrated moderate success in correcting vesicoureteral reflux, but little is known about its imp
34 el noninvasive molecular diagnostic tests of vesicoureteral reflux by proteomics methodology.
35 le nature of bladder imaging in children for vesicoureteral reflux detection makes the search for non
36 chard Wahl reviews urinary tract infections, vesicoureteral reflux, dysfunctional voiding, and approp
37                    Traditional management of vesicoureteral reflux focuses on preventing renal compli
38 s in 298 individuals with confirmed UTIs and vesicoureteral reflux from the Randomized Intervention f
39 phylaxis decreased by 47% when adjusting for vesicoureteral reflux grade and bowel and bladder dysfun
40 trasonography imaging of the bladder to find vesicoureteral reflux has yet to be refined enough to be
41                   Both knockouts experienced vesicoureteral reflux, hydronephrosis, renal dysfunction
42 ement of urinary tract infections (UTIs) and vesicoureteral reflux in children and examine new altern
43                             The treatment of vesicoureteral reflux in children has seen a shift from
44  an increasingly popular method for managing vesicoureteral reflux in children.
45 or detection of genetic mutations leading to vesicoureteral reflux in humans by studying differential
46 ion generates excessive apoptosis leading to vesicoureteral reflux in newborns, which underscores the
47 n may play an etiological role in congenital vesicoureteral reflux in otherwise normal children.
48                     The current diagnosis of vesicoureteral reflux involves voiding cystourethrograms
49                     The incidence of primary vesicoureteral reflux is about 1% to 2% of the general p
50 est that in some infants and older children, vesicoureteral reflux is associated with congenital stru
51                            The management of vesicoureteral reflux is evolving, with advocacy ranging
52  tract infection (UTI) risk in children with vesicoureteral reflux is largely unknown.
53                                              Vesicoureteral reflux is neither necessary nor sufficien
54        It has been shown that the finding of vesicoureteral reflux is variable and that single studie
55 enged the traditional paradigm of aggressive vesicoureteral reflux management with surgery or antibio
56                                    Bilateral vesicoureteral reflux of infected urine was induced in 1
57 thral valves (four), urethral atresia (one), vesicoureteral reflux (one), and megacystis (one).
58 ecent studies have shown that in grades I-IV vesicoureteral reflux, open surgical intervention compar
59 ng UTIs but has not been investigated in the vesicoureteral reflux population.
60 sed to treat stress urinary incontinence and vesicoureteral reflux provide for similar rates of succe
61                                      Primary vesicoureteral reflux (pVUR) is one of the most common c
62  voiding cystourethrogram to investigate for vesicoureteral reflux remains controversial.
63 e of MRU for the assessment of urolithiasis, vesicoureteral reflux, renal trauma, and fetal urinary t
64 eteral injection therapy, for patients whose vesicoureteral reflux requires correction, remains uncle
65 ications and seven (4.8%) patients developed vesicoureteral reflux requiring reoperation.
66 he Randomized Intervention for Children with Vesicoureteral Reflux (RIVUR) Study and 295 controls, an
67 he Randomized Intervention for Children With Vesicoureteral Reflux (RIVUR) study, which will evaluate
68                                Consequently, vesicoureteral reflux screening in siblings and offsprin
69 ve been tried as bulking agents to eliminate vesicoureteral reflux since the technique was introduced
70 he Randomized Intervention for Children With Vesicoureteral Reflux Study, a 2-year randomized clinica
71 he Randomized Intervention for Children with VesicoUreteral Reflux Study.
72 phritis and renal scarring may be related to vesicoureteral reflux that is missed by standard voiding
73 controlled trial involving 607 children with vesicoureteral reflux that was diagnosed after a first o
74 ance of VCUG in a pediatric porcine model of vesicoureteral reflux, total radiation exposure can be r
75 he Randomized Intervention for Children With Vesicoureteral Reflux trial and the Careful Urinary Trac
76 porary data regarding the natural history of vesicoureteral reflux, urinary tract infection and renal
77 nts of irreversible bladder dysfunction with vesicoureteral reflux, urinary tract infection and resul
78                                              Vesicoureteral reflux (VUR) (OMIM %193000), a common cau
79 INDINGS: The causal relationship between UTI-vesicoureteral reflux (VUR) and renal scarring has been
80  (UTI) and urinary tract abnormality such as vesicoureteral reflux (VUR) are given prophylactic antib
81 struction [ureteropelvic junction (UPJ)] and vesicoureteral reflux (VUR) have been identified and hav
82                                              Vesicoureteral reflux (VUR) is a risk factor for acute p
83                                              Vesicoureteral reflux (VUR) is diverse in its natural hi
84                                      Primary vesicoureteral reflux (VUR) is the most common congenita
85  in predicting acute pyelonephritis (APN) or vesicoureteral reflux (VUR) using the data of 288 patien
86 al-specific gene can therefore cause primary vesicoureteral reflux (VUR), a hereditary disease affect
87 d to ureteric bud (UB) induction defects and vesicoureteral reflux (VUR), although the mechanisms wer
88 the kidney and urinary tract (CAKUT) include vesicoureteral reflux (VUR).
89                                              Vesicoureteral reflux was most common (15 patients).
90                                              Vesicoureteral reflux was simulated in four pigs, and 48
91       Although the presence of grade IV or V vesicoureteral reflux was the strongest predictor of ren
92 vel of greater than 40 mg/L, and presence of vesicoureteral reflux were all associated with the devel
93                         Sex and grade 1 to 3 vesicoureteral reflux were not associated with risk of r
94                           Early detection of vesicoureteral reflux will be valuable for prevention of

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