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1 ice with an AgRP neuron-specific deletion of vesicular GABA transporter.
2 ice with an AgRP neuron-specific deletion of vesicular GABA transporter.
3 icle proteins, such as synaptophysin and the vesicular GABA transporter.
4 teins: the biosynthetic enzyme for GABA, the vesicular GABA transporter, a transcription factor that
5 by approximately 40%, whereas the density of vesicular GABA transporter and bassoon coimmunoreactive
6 tion revealed coexpression of SNAP-25, VGAT (vesicular GABA transporter), and GAD65/67 (glutamic acid
8 through delivery of saporin-conjugated anti-vesicular GABA transporter antibodies (SAVAs) in vitro a
9 sessed vesicular monoamine transporter 2 and vesicular GABA transporter, but they lacked immunostaini
10 suggest that transcriptional control of the vesicular GABA transporter by NO regulates GABA transmis
11 vesicular monoamine transporter 2) and VGAT (vesicular GABA transporter), consistent with vesicular s
12 es in glutamate acid decarboxylase (GAD) and vesicular GABA transporter expression, these findings pu
13 ber of GABAergic interneurons, and GABA(A)-, vesicular GABA transporter-, GAD65-, and GAT3-immunoreac
15 TRN neurons through conditional deletion of vesicular GABA transporter has no effect on spontaneous
17 areas as indicated by the colocalization of vesicular GABA transporter immunoreactivity predominantl
18 decarboxylase) 65 and 67 isoforms, and VGAT (vesicular GABA transporter) in interneurons from the str
19 e vesicular glutamate transporter 3 with the vesicular GABA transporter, indicating that GABA, glycin
20 either by cell type-specific deletion of the vesicular GABA transporter or by expression of botulinum
21 ndicated by elevated GABA synthetic enzymes, vesicular GABA transporter, perineuronal nets, and enhan
22 reduced by blocking action potentials or the vesicular GABA transporter, phasic and tonic currents de
23 -positive presynaptic endings; GAD-positive, vesicular GABA transporter-positive inhibitory endings a
25 analysis identified a selective reduction of vesicular GABA transporter punctae on parvalbumin positi
26 singDyrk1A We also identified a reduction of vesicular GABA transporter punctae specifically on parva
31 We found that CLC-3 colocalized with the vesicular GABA transporter VGAT in the CA1 region of the
32 prefrontal cortex (mPFC), expression of the vesicular GABA transporter VGAT was unchanged; however,
33 amino acid transporter, VIAAT (also known as vesicular GABA transporter VGAT) transports GABA or glyc
34 l organ neurons, marked by expression of the vesicular GABA transporter VGAT, drastically suppresses
37 utons were identified by the presence of the vesicular GABA transporter (VGAT) and NPY was found in 1
40 rthermore, co-staining for synaptophysin and vesicular GABA transporter (VGAT) revealed a group of sm
41 fication of a unique amino acid motif in the vesicular GABA transporter (VGAT) that controls its syna
42 GABA transporters (GAT-1 and GAT-3), and the vesicular GABA transporter (VGAT) was evaluated by using
45 NO) signaling elevates the expression of the vesicular GABA transporter (VGAT) within recurrent colla
46 n of hippocampal interneurons, expression of vesicular GABA transporter (vGAT), and extracellular GAB
47 luR, NMDA receptor (NR) subunits, GAD65, the vesicular GABA transporter (VGAT), and the neuronal glut
48 , as well as glutamic acid decarboxylase and vesicular GABA transporter (VGAT), markers of GABAergic
49 genetic and optogenetic activation of septal vesicular GABA transporter (vGAT)-containing neurons or
54 mmunoreactivity (IR) (approximately 27%) and vesicular GABA transporter (VGAT)/p38 IR (approximately
56 nsities of ChC cartridges immunoreactive for vesicular GABA transporter (vGAT+), which is present in
58 rojecting neurons in the LH that express the vesicular GABA transporter (VGAT; a marker for GABA-rele
59 rters responsible for packaging either GABA (vesicular GABA transporter [vGAT]) or glutamate (vesicul
61 shown that VGLUT3 is found together with the vesicular GABA transporter (VIAAT) on synaptic vesicle m
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