ライフサイエンスコーパス: vestibular dysfunction

1 tial for using antisense technology to treat vestibular dysfunction.

2 l defects and eventually to hearing loss and vestibular dysfunction.

3 ls only in the genetic background exhibiting vestibular dysfunction.

4 ilure of normal canal development results in vestibular dysfunction.

5 mic, hereditary hearing loss with associated vestibular dysfunction.

6 None of the strains displays signs of vestibular dysfunction.

7 that are useful models of human deafness and vestibular dysfunction.

8 sociated with profound retinal, auditory and vestibular dysfunction.

9 ngenital deafness, retinitis pigmentosa, and vestibular dysfunction.

10 d for designing therapies to treat inner ear vestibular dysfunction.

11 of either central neurological or peripheral vestibular dysfunction.

12 target of mutations that cause deafness and vestibular dysfunction.

13 curring in DFNA9 leading to hearing loss and vestibular dysfunction.

14 t may cause or contribute to deafness and/or vestibular dysfunction.

15 itterbug (jbg), exhibit impaired hearing and vestibular dysfunction.

16 amed hurry-scurry (hscy) causes deafness and vestibular dysfunction.

17 ) mouse carries a recessive mutation causing vestibular dysfunction.

18 re completely deaf and also display signs of vestibular dysfunction.

19 ated in ames waltzer and causes deafness and vestibular dysfunction.

20 causes hair cell degeneration, deafness, and vestibular dysfunction.

21 he inner ear that result in hearing loss and vestibular dysfunction.

22 het) is a recessive mutation in mice causing vestibular dysfunction.

23 ngenital deafness, retinitis pigmentosa, and vestibular dysfunction.

24 ion and navigational deficits observed after vestibular dysfunction.

25 ia was one indicating compensated peripheral vestibular dysfunction.

26 f hearing and vision, and varying degrees of vestibular dysfunction.

27 Non-autonomic neurological causes included vestibular dysfunction (32 [5%]) and epilepsy (11 [1.7%]

28 cargo whirlin are implicated in deafness and vestibular dysfunction and have been shown to localize a

29 Autonomic manifestations of vestibular dysfunction and motion sickness are well esta

30 congenital sensorineural deafness, constant vestibular dysfunction and prepubertal onset of retiniti

31 th Bt2 significantly reduced the severity of vestibular dysfunction and prevented earlier mortality.

32 ice, which is characterized by hearing loss, vestibular dysfunction, and coat color dilution.

33 disorder manifested by congenital deafness, vestibular dysfunction, and progressive retinal degenera

34 d by progressive sensorineural hearing loss, vestibular dysfunction, and retinitis pigmentosa.

35 Deafness and vestibular dysfunction are extremely common sensory diso

36 Subclinical vestibular dysfunction, as identified by clinical tests,

37 They are deaf and have vestibular dysfunction but do not develop photoreceptor

38 elial defects manifested by hearing loss and vestibular dysfunction but no retinal pathology.

39 t prove useful in ameliorating some forms of vestibular dysfunction by modifying ongoing primary vest

40 ty-related symptoms; and (3) the severity of vestibular dysfunction can predict whether hyperactivity

41 has been eliminated in supporting cells have vestibular dysfunction caused by failure of synapse form

42 al, Tmc1(Delta)Tmc2(Delta) mice had profound vestibular dysfunction, deafness, and structurally norma

43 Severe vestibular dysfunction developed in mice infected with B

44 een identified as causative for deafness and vestibular dysfunction (DFNB18B).

45 the severity rather than the age of onset of vestibular dysfunction differentiates whether hyperactiv

46 the waltzer (v) locus result in deafness and vestibular dysfunction due to degeneration of the neuroe

47 ouette (pi) locus result in hearing loss and vestibular dysfunction due to neuroepithelial defects in

48 inner (sr) locus results in hearing loss and vestibular dysfunction due to neuroepithelial defects in

49 ids have been used to treat hearing loss and vestibular dysfunction for many years.

50 ased gene therapy to ameliorate auditory and vestibular dysfunction has been proposed.

51 d overt behavioral defects characteristic of vestibular dysfunction have not been described.

52 Patients with vestibular dysfunction have visual, perceptual, and post

53 nel-like 1 (TMC1) cause hearing loss without vestibular dysfunction in both mice and humans, we inves

54 Hearing impairment or vestibular dysfunction in humans often results from a pe

55 target of mutations that cause deafness and vestibular dysfunction in mice and humans.

56 tal deafness in humans and both deafness and vestibular dysfunction in mice homozygous for the shaker

57 vision symptoms in patients, our findings of vestibular dysfunction in these Dfnb31 mutants raise the

58 zation on hair cells leading to deafness and vestibular dysfunction in waltzer mice.

59 Human vestibular dysfunction is an increasing clinical problem

60 progressive, sensorineural hearing loss and vestibular dysfunction known as DFNA9.

61 here are a gene dose-sensitive cause of the vestibular dysfunction observed in EphB-Efnb2 signaling-

62 ar defect, but that may include mutants with vestibular dysfunction of the inner ear.

63 In the case of vestibular dysfunction or motion sickness, the unpleasan

64 Behavioral recovery from vestibular dysfunction produced by unilateral labyrinthe

65 similar hair bundle defects may underlie the vestibular dysfunction reported in humans with mutations

66 used more severe arthritis, myocarditis, and vestibular dysfunction than serotype A.

67 ess associated with retinitis pigmentosa and vestibular dysfunction (USH1D) were previously mapped to

68 However, a milder form of vestibular dysfunction was apparent from altered vestibu

69 Vestibular dysfunction was associated with space and mot

70 d without agoraphobia and to discern whether vestibular dysfunction was associated with specific symp

71 laboratory investigations, suggest that the vestibular dysfunction was immune mediated.

72 2a2 mutations cause hyperactivity; (2) it is vestibular dysfunction, which frequently co-occurs with