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1 al level of regulation for the CrkII protein via binding to 14-3-3 proteins, which was independent fr

2 repressed luciferase reporter gene activity via binding to 3' untranslated regions of TMEFF2, NTRK2,

3 -phosphotransferase I resistance gene (mphA) via binding to a 35-bp DNA operator upstream of the star

4 licing of a defined subset of cassette exons via binding to a C-rich subset of polypyrimidine tracts

5 ily, is a multifunctional cytokine that acts via binding to a cell surface receptor named Fn14 (fibro

6 TWEAK acts on responsive cells via binding to a cell surface receptor named Fn14.

7 TR2 can regulate several target genes via binding to a consensus response element (AGGTCA) in

8 on, as able to down-regulate CD69 expression via binding to a conserved site in the 3'UTR of CD69 mRN

9 FAK recruitment was via binding to a domain within the virulence factor TarP

10 ntrolling insulin receptor exon 11 inclusion via binding to a downstream intronic enhancer element.

11 human papilloma virus type 16 (HPV-16) genes via binding to a DR4 response element in the long contro

12 They function via binding to a family of structurally related cell sur

13 tor of the fungal virulence factor, laccase, via binding to a GC-rich element within the 5'-UAS in re

14 w that CUGBP1 induces the translation of p21 via binding to a GC-rich sequence located within the 5'

15 s of hypoxia on induction of HEF1 expression via binding to a hypoxia-responsive element of the HEF1

16 specific for activated glycoprotein IIb/IIIa via binding to a Ligand-Induced Binding Site (LIBS-MBs)

17 ing HMGA1 to repression of XPA transcription via binding to a negative regulatory element in the endo

18 expression of the CD44 cell-surface molecule via binding to a noncanonical p53-binding sequence in th

19 ne subunit regulates the activity of another via binding to a noncatalytic site(s) rather than throug

20 liferation, whereas proNGF induces apoptosis via binding to a receptor complex of the common neurotro

21 h transcriptionally activates the CXCR4 gene via binding to a responsive element located in positions

22 catalytic domain to the peptidoglycan layer via binding to a secondary cell wall polymer component.

23 the mu-opioid receptor (MOR) gene regulation via binding to a single-stranded (ss) DNA element.

24 open state block of this channel occurs not via binding to a site directly in the pore but rather by

25 TWEAK acts on responsive cells via binding to a small cell surface receptor named Fn14.

26 TWEAK acts on responsive cells via binding to a small cell-surface receptor named fibro

27 Gammaretroviruses that enter cells via binding to a surface receptor use one of two fundame

28 that conferred an elevated promoter activity via binding to a transcription factor SOX5.

29 CSK is brought in contiguity to LCK via binding to a transmembrane adaptor known as phosphop

30 adhesion of alpha4beta7+ mucosal lymphocytes via binding to aberrantly expressed MAdCAM-1 on liver en

31 n epigenetic regulator that localizes to DNA via binding to acetylated histones and controls the expr

32 nd purified Muc5ac reduced infection, likely via binding to alpha2,3-linked sialic acids, consistent

33 ich the anchoring of the motor to the cortex via binding to an adhesion molecule mediates the tetheri

34 AR activity was mediated via binding to an estrogen receptor half-site 3' to the

35 sensing pathway mediated by calmodulin (CaM) via binding to an IQ motif immediately adjacent to the E

36 n of actin cytoskeleton and gene expression, via binding to and modulating the activity of diverse ef

37 nhanced TLR3/4-triggered IFN-beta production via binding to and phosphorylating IL-1 receptor-associa

38 regulated by the ubiquitin proteasome system via binding to and ubiquitination by the E3 ubiquitin li

39 ular, we show that NOTCH1 transactivates MYC via binding to B-cell-specific regulatory elements, thus

40 APRIL functions via binding to BCMA (B cell maturation antigen) and TACI

41 peptidase N (APN) and p19ARF gene expression via binding to canonical DNA recognition sites in the re

42 factor (SRF) controls SMC gene transcription via binding to CArG box DNA sequences found within genes

43 AFM13 recruits natural killer (NK) cells via binding to CD16A as immune effector cells.

44 ells (pDCs) produced FasL in response to HIV via binding to CD4 and chemokine coreceptors.

45 d promote trans infection by dendritic cells via binding to cell surface lectins.

46 y activate intracellular signaling cascades, via binding to cognate cytoplasmic or membrane-associate

47 ression of the collagen alpha(2)(I) promoter via binding to CREB-binding protein (CBP).

48 ells and translocates to the plasma membrane via binding to cytohesin 2 in epidermal growth factor-st

49 G dampens the activation of human DCs by LPS via binding to DC-SIGN and MMR-1, leading to attenuated

50 experiments indicate that the inhibitor acts via binding to DC-SIGN.

51 toskeleton suggests that Nck/Dock regulates, via binding to distinct effectors, various cell type-spe

52 ), which serve as a reservoir for chemokines via binding to Duffy antigen receptor for chemokines (DA

53 IL-6 signals via binding to either the membrane bound IL-6Ralpha (cla

54 lastic tissues of the gastrointestinal tract via binding to elements in the 5'-flanking region of the

55 assembly and interaction with hemicelluloses via binding to emerging cellulose microfibrils.

56 r endothelin 1, which increases PKC activity via binding to endogenous endothelin(A) receptors.

57 role in the release of proteins from the ER via binding to Ero1-Lalpha.

58 und that ERalpha activates p53 transcription via binding to estrogen response element half-sites with

59 d into the cellular component of whole blood via binding to FKBP.

60 ones and cytokines regulate L channel gating via binding to G-protein-coupled receptors.

61 he ability of macrophages to clear pathogens via binding to galectin 9.

62 igate whether CLEC18 modulates host immunity via binding to glycolipids, and are also involved in gly

63 ochondrial outer membrane either directly or via binding to GM130.

64 t-PA induces SK-N-SH cell proliferation via binding to GRP78 on the cell surface.

65 e conferred the ability to invade host cells via binding to GRP78.

66 n the tubulointerstitium and peri-glomerulus via binding to heparan sulphate (HS) chains of proteogly

67 picomolar concentrations, directly virucidal via binding to HIV envelope glycoproteins, and capable o

68 the 233^416 splicing of HPV18 E6E7 pre-mRNAs via binding to hnRNP A1, a well-characterized, abundantl

69 20 is associated with the virions presumably via binding to Hsp70h.

70 rs to mediate parasite-host cell interaction via binding to human galectin-1.

71 ionally regulate the LncRNA-SARCC expression via binding to hypoxia-responsive elements on the promot

72 induced by shPHD2 to induce IGF-1 secretion via binding to IGF-1 gene promoter.

73 R-185-5p, which suppresses VEGF-C expression via binding to its 3' UTR.

74 nt in RNA binding, stabilizes HuR transcript via binding to its 3'-untranslated region.

75 ion, TDP-43 regulates Ran expression, likely via binding to its 3'-UTR.

76 activation, migration, and tissue retention via binding to its extracellular matrix ligand hyalurona

77 Mxtx2 directly activates expression of ndr2 via binding to its first intron and is required for ndr2

78 hat FIP200 functions as an inhibitor of Pyk2 via binding to its kinase domain.

79 on of MM cells in an in vivo scaffold system via binding to its receptor, CD147, on MM cells.

80 n angiogenesis, exerts its angiogenic effect via binding to its receptor, VEGF receptor-2 tyrosine ki

81 sm to recruit STIM1 into the ER-PM junctions via binding to junctate.

82 duced MKP-1 in the spastic cerebral arteries via binding to L-arginyl-glycyl-L-aspartate-dependent in

83 RMS is controlled in part by beta1 integrins via binding to laminin.

84 metabolites, CRMs) that can lead to toxicity via binding to macromolecular targets (e.g., proteins or

85 nction as a master transcriptional regulator via binding to many cis-acting sites genome-wide.

86 lating cholangiocyte Cl- and fluid secretion via binding to membrane P2 receptors, though the physiol

87 g of polycations and plasmid DNA enter cells via binding to membrane-associated proteoglycans.

88 e compounds likely confer antiviral activity via binding to methyltransferase (MTase).

89 al cyclin-dependent kinase inhibitors (CKIs) via binding to Miz1; whether this interaction is importa

90 1 is able to destabilize the MDM2 transcript via binding to multiple AU-/U-rich elements in MDM2 3'un

91 hat toxins that modify inactivation kinetics via binding to Na(V)1.x site 3 lack the ability to bind

92 w that WRN associates with the Mre11 complex via binding to Nbs1 in vitro and in vivo.

93 s in cell adhesion and synaptic organization via binding to neurexins.

94 ite of vitamin A, induces gene transcription via binding to nuclear retinoic acid receptors (RARs).

95 CD36 modulates platelet function via binding to oxidized LDL (oxLDL), cell-derived microp

96 teins that can identify viral RNA as nonself via binding to pathogen associated molecular patter (PAM

97 des resulting in a prolonged serum half-life via binding to patients' serum albumin in vivo.

98 ear and in photoreceptor cells of the retina via binding to PDZ domains in the scaffold protein harmo

99 drolysis and translocates to ER-PM junctions via binding to phosphatidic acid.

100 PCI can penetrate through cellular membranes via binding to phosphatidylethanolamine.

101 hway and is recruited to endosomal membranes via binding to phosphatidylinositol 3-phosphate (PtdIns[

102 RKIN recruitment and enzymatic amplification via binding to phosphorylated UB chains.

103 choline acquisition from host phospholipids (via binding to plcH and pchP promoters), is required for

104 that mediates protein membrane translocation via binding to pleckstrin homolog (PH) domains within ta

105 cell types, which inhibit T cell activation via binding to programmed death-1 (PD-1) on T cells.

106 ng target genes in early Xenopus development via binding to promoter-proximal CTGCNA sequences as par

107 protein PU.1, and can activate transcription via binding to PU.1/IRF composite sequences.

108 n oxygen delivery and demand (e.g. exercise) via binding to purinergic receptors (P2Y) on the endothe

109 e and hormone action involves direct effects via binding to receptors on the intestinal epithelium as

110 prevented by high density lipoprotein (HDL) via binding to scavenger receptor BI (SR-BI), which is c

111 the expression of functionally related genes via binding to shared regulatory sequences, such as the

112 olded domains mediating protein interactions via binding to short linear peptides in proteins.

113 the binding of M-T5 to cullin 1 is indirect via binding to Skp1 in the host SCF complex.

114 tein that regulates neurotransmitter release via binding to SNARE complexes, is essential for AMPAR e

115 timulated proliferation of human aortic SMCs via binding to somatostatin receptors (sst2 and sst5) an

116 2 plays a significant role in cell migration via binding to specific cytoskeletal regulators such as

117 gulates GABRA2 and GABRA4 subunit expression via binding to specific promoter responsive elements, as

118 shown to associate with the plasma membrane via binding to specific transmembrane proteins.

119 induced loss of cell adhesion on fibronectin via binding to syndecan-4, leading to activation of PKCa

120 tenin/TCF directly regulates MSX2 expression via binding to TCF binding elements in multiple regions

121 ected from a phage-displayed peptide library via binding to tetragonal BaTiO3 powder.

122 ptide sequence associates with co-chaperones via binding to tetratricopeptide repeat domains.

123 r superfamily that modulates gene expression via binding to the AGGTCA direct repeat hormone response

124 rowth of normal prostate and prostate cancer via binding to the androgen receptor (AR).

125 cates that AR enhances miR-185-5p expression via binding to the androgen response elements located on

126 und to and aggregated Gram-positive bacteria via binding to the bacterial cell wall peptidoglycan.

127 s been reported to promote cellular adhesion via binding to the beta2 integrin cytoplasmic domain.

128 and that removal of plasma TPO by platelets via binding to the c-Mpl receptor is involved in the cle

129 ce that platelets regulate plasma TPO levels via binding to the c-mpl receptor on circulating platele

130 -accelerating factor (DAF) promoter activity via binding to the cAMP response element, mutation of wh

131 acids apart, eIF3g binds to eIF3a indirectly via binding to the carboxyl-terminal domain of eIF3b.

132 st protein found in milk to neutralize HIV-1 via binding to the chemokine coreceptor site, potentiall

133 een shown to act as a T-cell chemoattractant via binding to the chemokine receptor and HIV-1 corecept

134 at FOXO3a can induce 5' AR promoter activity via binding to the consensus DNA-binding sequence in the

135 pressed the inclusion of an alternative exon via binding to the conserved UGCAUG element in the upstr

136 ore, data demonstrate that gp120 induces p53 via binding to the CXCR4 co-receptor.

137 ted in the modulation of host cell apoptosis via binding to the death domains of tumor necrosis facto

138 modulator that controls various target genes via binding to the DNA hormone response elements.

139 dor-adapted animal, it increases translation via binding to the egl-4 3' UTR.

140 incorporates affinity capture of human IgGs via binding to the Fab region, followed by on-bead IdeS

141 articipates in acute intestinal inflammation via binding to the G-protein-coupled neurokinin-1 recept

142 e is known to mediate certain of its effects via binding to the gamma aminobutyric acid A (GABA(A)) r

143 that RhoA acts to regulate Trio localization via binding to the immunoglobulin-like domain.

144 its inhibition of cell migration is mediated via binding to the low-density lipoprotein receptor rela

145 osterically activated on the mitotic spindle via binding to the microtubule-associated protein, TPX2.

146 otubules to F-actin in growth cone filopodia via binding to the microtubule-binding +TIP protein EB3

147 criptional regulator of myogenic commitment, via binding to the MyoD mRNA 3' untranslated region.

148 ors, and its orexigenic actions occur mainly via binding to the only known ghrelin receptor, the grow

149 on and differentiation of osteoblastic cells via binding to the parathyroid hormone receptor (PTH-1R)

150 ransition by inactivating Cdc25C phosphatase via binding to the phosphorylated serine residue at posi

151 potential to induce human podocyte apoptosis via binding to the PLA2R.

152 5p also promotes HIF2alpha/VEGF-A expression via binding to the promoter region of HIF2alpha.

153 s Rgg-mediated activation of speB expression via binding to the promoter region.

154 aip-1 acts via binding to the proteosome and enhancing proteosomal

155 boundary effects on Hippo activity, probably via binding to the protocadherin Dachsous.

156 rogesterone activates CatSper of human sperm via binding to the serine hydrolase ABHD2.

157 nection between the viral and cell membranes via binding to the sialic acid-containing receptors.

158 s inhibited by low concentrations of 5'-AMP, via binding to the substrate (i.e. the kinase).

159 is a potent transcription coactivator acting via binding to the TEAD transcription factor, and plays

160 ng and is known to be localized to telomeres via binding to the telomere-binding protein TRF2.

161 ays show that TR3 can induce E2F1 expression via binding to the TR3 response element (TR3RE) in the E

162 atively regulates VEGFR2 receptor activation via binding to the VEGFR2, as well as stabilizes cell-ce

163 of cytokines exert their biological effects via binding to their cognate ligand-binding receptor sub

164 steroids are generally known to have actions via binding to their cognate steroid receptors, it is be

165 eal angiogenesis in vivo, and apparently act via binding to their receptors CXCR1 and CXCR2.

166 ted Foxp2 protein repress gene transcription via binding to this consensus site or to a naturally occ

167 lls and that its internalization could occur via binding to transferrin or caveolin.

168 BMP exert their biologic effects via binding to two types of serine/threonine kinase BMP

169 cardiovascular effects and behavioral traits via binding to various receptors (e.g., beta2-adrenergic

170 totic protein, promoting tumor cell survival via binding to VDAC1.

171 3,7,8-tetrachlorodibenzo-rho-dioxin (dioxin) via binding to xenobiotic-responsive elements (XREs) in

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