ライフサイエンスコーパス: victorin

1 genic because of the production of the toxin victorin.

2 l death induced by the host-selective toxin, victorin.

3 in mitochondrial permeability in response to victorin.

4 d, untreated mitochondria are impermeable to victorin.

5 Further, MPT-inducing conditions permitted victorin access to the mitochondrial matrix and binding

6 ith the hypothesis that an MPT, which allows victorin access to the mitochondrial matrix and binding

7 fense is recapitulated by interactions among victorin (an effector produced by the necrotrophic fungu

8 ay a role in mediating the plant response to victorin because LaCl3 gave near-complete protection aga

9 Rubisco cleavage, DNA laddering, and victorin binding to the P protein occurred concomitantly

10 In LOV1's presence, victorin binding to TRX-h5 activates LOV1 and elicits a

11 in only in toxin-sensitive genotypes whereas victorin binds equally well to P-protein isolated from t

12 features similar to apoptosis; (ii) in vivo, victorin binds to the mitochondrial P-protein only in to

13 sulting from victorin treatment suggest that victorin causes premature senescence of leaves.

14 red by the dominant gene LOCUS ORCHESTRATING VICTORIN EFFECTS1 (LOV1), which encodes a coiled-coil-nu

15 Leaf slices incubated with victorin for 4 hr in the dark accumulated a form of the

16 ial matrix and binding to the P-protein: (i) victorin-induced cell death displays features similar to

17 Results suggest that the victorin-induced collapse in Deltapsi(m) is a consequenc

18 We show that the victorin-induced collapse in mitochondrial transmembrane

19 Also, the victorin-induced collapse of membrane potential was clea

20 ate also gave significant protection against victorin-induced leaf symptoms and prevented LSU cleavag

21 ylene signaling was implicated upstream of a victorin-induced loss in mitochondrial motility and the

22 equence of an MPT and that the timing of the victorin-induced MPT is poised to influence the cell dea

23 spiration, as the latter event prevented the victorin-induced PCD response and binding to P-protein.

24 ding to the P-protein, occurs in vivo during victorin-induced PCD.

25 Victorin-induced proteolysis of ribulose-1,5-bisphosphat

26 The victorin-induced response in sensitive A. sativa has bee

27 the specific LSU cleavage is suggestive of a victorin-induced, senescence-like response.

28 l shrinkage supports the interpretation that victorin induces an apoptotic-like cell death response.

29 In LOV1's absence, victorin inhibits TRX-h5, resulting in compromised defen

30 We isolated 63 victorin-insensitive mutants, including 59 lov1 mutants

31 Victorin is a host-selective toxin produced by Cochliobo

32 We propose that victorin is, or mimics, a conventional pathogen virulenc

33 oxylase complex, at least one of which binds victorin only in toxin-sensitive genotypes in vivo.

34 lular fluid after induction of PCD by either victorin or heat shock.

35 orin, suggesting that ATTRX5 function in the victorin pathway involves an atypical mechanism of actio

36 LaCl3 gave near-complete protection against victorin, preventing both leaf symptoms and LSU cleavage

37 We found that the victorin response was highly specific to ATTRX5, as the

38 us results suggest that a MPT may facilitate victorin's access to the mitochondrial matrix and bindin

39 Victorin sensitivity has been identified in Arabidopsis

40 es in ATTRX5 is required for the response to victorin, suggesting that ATTRX5 function in the victori

41 Victorin, the host-selective toxin produced by Cochliobo

42 DNA from victorin-treated leaf slices showed a pronounced ladderi

43 In vivo, victorin treatment induced the collapse of mitochondrial

44 Another primary symptom of victorin treatment is chlorophyll loss, which along with

45 The symptoms resulting from victorin treatment suggest that victorin causes prematur

46 y induced in sensitive Arabidopsis following victorin treatment.

47 osphate carboxylase/oxygenase (Rubisco), and victorin was found to induce a specific proteolytic clea

48 Previously, victorin was shown to be bound specifically by two prote

49 ) and is pathogenic due to its production of victorin, which induces programmed cell death in sensiti

50 e photorespiratory cycle and is inhibited by victorin, with an effective concentration for 50% inhibi