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1 the lethal effects of honeybee or Russell's viper venom.
2 factor, factors IXa and VIIIa, and Russell's viper venom.
3 ex or by a purified activator from Russell's viper venom.
10 GDFX and was also activated by the Russell's viper venom at similar rate, but it cleaved the chromoge
11 peptide, was isolated from the Azemiops feae viper venom by combination of gel filtration and reverse
17 oresceinated antibody, and enzyme (Russell's viper venom factor X activator)-labeled antibody is allo
19 the ApoE(-/-) (-22.22+/-7.95%) and Russell's viper venom-injected (-10.37+/-17.60%) mice compared wit
20 0.54, R(12 weeks) 3.83+/-0.52) and Russell's viper venom-injected wild-type mice (R(1)=4.57+/-0.86).
21 s, such as cyclic RGD peptides isolated from viper venom, may prove to be useful as anti-inflammatory
23 are small non-enzymatic proteins present in viper venoms reported to modulate hemostasis of victims
24 antigen can enhance defense against Russell viper venom (RVV) and determined whether such responses
28 of coagulation factors V and X by Russell's viper venom (RVV) has been implicated in the development
29 showed that factor X activator of Russell's viper venom (RVV-X) contains six N-linked oligosaccharid
30 coagulable state, we have used the Russell's viper venom test (RVV) to show that red blood cells (RBC
31 ation by the factor X activator from Russell viper venom, the mutants were characterized with respect
32 rogram/mL) did not prolong a modified Russel viper venom time, suggesting no significant inhibition o
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