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1  the lethal effects of honeybee or Russell's viper venom.
2 factor, factors IXa and VIIIa, and Russell's viper venom.
3 ex or by a purified activator from Russell's viper venom.
4      The X-activating protein from Russell's viper venom activates fXSt. Louis II completely but at a
5 re and 48 hours after injection of Russell's viper venom, an endothelial toxin.
6  Plaque rupture was triggered with Russell's viper venom and histamine.
7 aque disruption was triggered with Russell's viper venom and histamine.
8 ger) inducing plaque disruption with Russell viper venom and histamine.
9  markedly procoagulant in a modified Russell viper venom assay.
10 GDFX and was also activated by the Russell's viper venom at similar rate, but it cleaved the chromoge
11 peptide, was isolated from the Azemiops feae viper venom by combination of gel filtration and reverse
12                                      Because viper venom CTLs exhibit a high degree of sequence simil
13  degrees of sequence similarity to published viper venom CTLs.
14       In conclusion, we report a new, potent viper venom-derived inhibitor of alpha2beta1 integrin, w
15                              Echistatin is a viper venom disintegrin containing RGD loop maintained b
16                                    Russell's viper venom factor X activator (RVV-X) triggers the casc
17 oresceinated antibody, and enzyme (Russell's viper venom factor X activator)-labeled antibody is allo
18        Factor X was activated with Russell's viper venom factor X activator, and single-chain unactiv
19 the ApoE(-/-) (-22.22+/-7.95%) and Russell's viper venom-injected (-10.37+/-17.60%) mice compared wit
20 0.54, R(12 weeks) 3.83+/-0.52) and Russell's viper venom-injected wild-type mice (R(1)=4.57+/-0.86).
21 s, such as cyclic RGD peptides isolated from viper venom, may prove to be useful as anti-inflammatory
22            We characterized HUVEC GPIb using viper venom proteins: alboaggregins A and B, echicetin,
23  are small non-enzymatic proteins present in viper venoms reported to modulate hemostasis of victims
24  antigen can enhance defense against Russell viper venom (RVV) and determined whether such responses
25 e was then induced with the use of Russell's viper venom (RVV) and histamine.
26                     An enzyme from Russell's viper venom (RVV) cleaves human factor V at Arg1018 and
27                                    Russell's viper venom (RVV) contains protein(s) that destabilize A
28  of coagulation factors V and X by Russell's viper venom (RVV) has been implicated in the development
29  showed that factor X activator of Russell's viper venom (RVV-X) contains six N-linked oligosaccharid
30 coagulable state, we have used the Russell's viper venom test (RVV) to show that red blood cells (RBC
31 ation by the factor X activator from Russell viper venom, the mutants were characterized with respect
32 rogram/mL) did not prolong a modified Russel viper venom time, suggesting no significant inhibition o
33           Disintegrins are peptides found in viper venoms which bind to platelets through the glycopr

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