ライフサイエンスコーパス: viral attachment

1 ks the normal hemagglutinin-mediated mode of viral attachment.

2 nt of the identity of the receptors used for viral attachment.

3 tion by modulating gangliosides required for viral attachment.

4 paran sulfate proteoglycan (HSPG), promoting viral attachment.

5 1-positive cells and occurs within 60 min of viral attachment.

6 nize a variety of host receptors, leading to viral attachment.

7 Drugs that can block viral attachment and cell entry independent of antigenic

8 Finally, we found that viral attachment and disassembly must occur within the s

9 capsid proteins that play important roles in viral attachment and disassembly.

10 e glycoprotein, E, of these viruses mediates viral attachment and entry by membrane fusion.

11 The major envelope glycoprotein, E, mediates viral attachment and entry by membrane fusion.

12 Its major envelope glycoprotein, E, mediates viral attachment and entry by membrane fusion.

13 many of the molecular processes involved in viral attachment and entry have been resolved, the popul

14 strategies such as microbicides that target viral attachment and entry into mucosa-resident target c

15 oprotein of hepatitis C virus (HCV) mediates viral attachment and entry into target hepatocytes and e

16 eins, VP4 and VP7, which are responsible for viral attachment and entry, are targets for protective n

17 A single viral glycoprotein (GP) mediates viral attachment and entry.

18 to cellular receptors, thereby facilitating viral attachment and entry.

19 r of human semen itself, was at the level of viral attachment and entry.

20 rliest stages of viral infection, preventing viral attachment and exerting a mild virucidal action.

21 dase (HN), an envelope protein essential for viral attachment and for fusion mediated by the other en

22 that determine reciprocal specificity of the viral attachment and fusion (F) proteins have not been i

23 oviruses requires an interaction between the viral attachment and fusion (F) proteins to enable recep

24 oV-NL63 to heparan sulfates was required for viral attachment and infection of target cells, showing

25 virus and adenovirus receptor (CAR) mediates viral attachment and infection, but its physiologic func

26 In paramyxoviruses, viral attachment and membrane fusion are governed by the

27 comparable to that of acyclovir by blocking viral attachment and penetration into host cells.

28 otavirus were used to test the importance of viral attachment and replication.

29 extracellular matrix, yet they also promote viral attachment and/or entry.

30 usually requires an interaction between the viral-attachment and -fusion proteins.

31 Retrocyclin 2 blocked viral attachment, and its addition during the binding or

32 One of these loops mediates viral attachment, and the other participates in making t

33 steps in reovirus replication subsequent to viral attachment are required for reovirus-induced apopt

34 mation by competing with cell surface HS for viral attachment, binding studies were also performed.

35 that receptor affinity has little impact on viral attachment but is nevertheless a key determinant o

36 Confocal analysis revealed that viral attachment, cell entry, and intracellular transpor

37 Entry of HIV-1 into target cells involves viral attachment, co-receptor binding, and fusion.

38 entry is a multistep process that comprises viral attachment, co-receptor interactions and fusion.

39 After viral attachment, endocytosis, and fusion mediated by th

40 nfection and suggest that further studies of viral attachment factors in NPCs are needed.

41 ate binding and fusion in the absence of the viral attachment (G) protein.

42 le of fusing membranes in the absence of the viral attachment (G) protein.

43 Previous reports have shown that the viral attachment glycoprotein (G) modulates innate and a

44 receptors ephrin B2 and/or ephrin B3 via the viral attachment glycoprotein G, and the concerted effor

45 in alpha(v)beta3 on the COOH terminus of the viral attachment (H) protein and rescued the replication

46 Successful infection following viral attachment, internalization, and nuclear transport

47 ycans (HSPG), the highly conserved target of viral attachment ligands (VALs).

48 ery of functional virus, indicating that the viral attachment mechanism is a primary distinguishing f

49 ucleopolyhedrovirus (AcMNPV), is involved in viral attachment, mediates membrane fusion during virus

50 inhibits infection primarily at a step after viral attachment, potentially by blocking envelope glyco

51 wo mutations, an amino acid change in the E2 viral attachment protein and a deletion within the 3'-UT

52 Outer-capsid protein sigma1 is the viral attachment protein and binds carbohydrate molecule

53 Binding of viral attachment protein final sigma 1 to both sialic ac

54 plinary approach to study the binding of the viral attachment protein G to its host receptor ephrinB2

55 The viral attachment protein of RSV has many surprising feat

56 hment to cells is mediated by the binding of viral attachment protein sigma 1 to junctional adhesion

57 he S1 gene is bicistronic, encoding both the viral attachment protein sigma-1 and the nonstructural p

58 First, while the S1 gene. which encodes the viral attachment protein sigma1 (as well as a nonstructu

59 The S1 gene is bicistronic, encoding the viral attachment protein sigma1 and the nonstructural pr

60 s infections are initiated by the binding of viral attachment protein sigma1 to receptors on the surf

61 The binding of viral attachment protein sigma1 to unidentified receptor

62 The S1 gene encodes viral attachment protein sigma1, and the S4 gene encodes

63 ed genetically to the S1 gene, which encodes viral attachment protein sigma1.

64 ystem (CNS) attributable to polymorphisms in viral attachment protein sigma1.

65 heir original infectivity due to cleavage of viral attachment protein sigma1.

66 respiratory avian coronaviruses based on the viral attachment protein spike (S1).

67 n AF15561, we identified a residue in the E2 viral attachment protein that is a critical determinant

68 ractions and, along with GP2a, serves as the viral attachment protein that is responsible for mediati

69 tion of an amino acid polymorphism in the E2 viral attachment protein using a mouse model of CHIKV mu

70 , using discrete receptor-binding domains in viral attachment protein, final sigma1.

71 membrane fusion requires the presence of the viral attachment protein, G.

72 E2 glycoprotein, which is thought to be the viral attachment protein, interfered with virus attachme

73 he T3C/96 S1 gene segment, which encodes the viral attachment protein, sigma 1, confirmed the serotyp

74 e globular domain of hemagglutinin (HA), the viral attachment protein.

75 virus (RSV) was identified previously as the viral attachment protein.

76 s using representative crystal structures of viral attachment proteins in complex with glycans.

77 ly high affinity that all available receptor-viral attachment proteins were destined to bind at stead

78 e, both before and after they are ligated by viral attachment proteins.

79 f binding is dictated by the availability of viral attachment proteins.

80 yofibers is not a consequence of the loss of viral attachment sites on the surfaces of mature muscle

81 Three GP1 viral attachment subunits assemble to form a chalice, cr

82 surface of the protein and new insights into viral attachment through binding to membrane protein rec

83 annot infect rhesus macaques by proving that viral attachment through gp350 is not the mechanism for

84 tion of viral particles and/or inhibition of viral attachment, thus reducing viral entry.

85 was due to the ability of b12 to block both viral attachment to and uptake by epithelial cells.

86 lement receptor type 2 (CR2) is critical for viral attachment to B lymphocytes.

87 Reovirus cell entry is initiated by viral attachment to cell surface glycans and junctional

88 gs suggest that HHV-7 gp65 may contribute to viral attachment to cell surface proteoglycans.

89 nated mosPTP-1, recruits mosGCTL-1 to enable viral attachment to cells and to enhance viral entry.

90 HD5 treatment of BKV reduced viral attachment to cells, whereas cellular treatment wi

91 major determinant of cell tropism, mediates viral attachment to cellular receptors.

92 did not, suggesting a direct role for E2 in viral attachment to chromosomes.

93 cells indicating therefore that IgM inhibits viral attachment to core-receptors.

94 C), infected cell binding, and inhibition of viral attachment to epithelial cells.

95 d bound RSV with high affinity and inhibited viral attachment to HEp2 cells.

96 cellular matrix proteins, thereby increasing viral attachment to heparan sulfate proteoglycans and in

97 nhibitor that binds to HIV-1 gp120, blocking viral attachment to host CD4 cells.

98 is the viral envelope protein that mediates viral attachment to host cells and elicits membrane fusi

99 spike (S) glycoprotein is required for both viral attachment to permissive cells and for fusion of t

100 T antigen into ts13 cells, and thus neither viral attachment to receptors, viral tegument proteins,

101 the importance of antibodies that neutralize viral attachment to susceptible cells.

102 ct, aggregation of viruses, or inhibition of viral attachment to target cells and the subsequent endo

103 unodeficiency virus (HIV) as they facilitate viral attachment to the host receptor cell.

104 of HuNoV B cell infection and for measuring viral attachment to the surface of B cells.

105 ell-surface proteoglycans is involved in the viral attachment to these CD4-negative epithelial cells.

106 stems from the compound's potential to block viral attachment, virus-cell fusion, or virus entry into