ライフサイエンスコーパス: viral coat protein

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1 ommon second-site suppressors located in the viral coat protein.

2 g particular positions atop pentamers of the viral coat protein.

3 Rather, AtNSI specifically acetylates the viral coat protein.

4 ch cell death is activated in the absence of viral coat protein.

5 eath is elicited only in the presence of the viral coat protein.

6 ted via collisional activation of the intact viral coat protein.

7 ein B mediate conformational switches in the viral coat protein.

8 indicate that DRB4 has a negative effect on viral coat protein accumulation.

9 pre- and postfusion states of the HIV-1 gp41 viral coat protein, although very different from one ano

10 ncident as revealed by the distribution of a viral coat protein and an HA epitope tag present on the

11 rminus induces conformational changes in the viral coat protein and facilitates minor spike protein i

12 HSV glycoprotein D (gD) is an immunodominant viral coat protein and is considered an excellent vaccin

13 is unlike those of previously characterized viral coat proteins and is composed of seven alpha helic

14 opeptide acts as an autotransporter; certain viral coat proteins; and proteins containing inteins.

15 While the viral coat protein AR1 is not essential for systemic inf

16 iral inactivation (CTVI), nucleases fused to viral coat proteins are expressed in infected cells and

17 ng the 'full virus vector strategy' with the viral coat protein as fusion partner for the designed an

18 ational design for a self-assembling minimal viral coat protein based on simple polypeptide domains.

19 us genomic RNAs are infectious only when the viral coat protein binds to the RNA 3' termini.

20 The results show that p37, the viral coat protein, blocks RNA silencing.

21 tions confer amino acid substitutions in the viral coat protein but differ in their relative abilitie

22 Our proteomics studies reveal that VLPs lack viral coat proteins but possess a pharmacopoeia of (1) t

23 In particular, we show that the increasing viral coat protein concentration that occurs in infected

24 V type 1 (HAstV-1) virions, specifically the viral coat protein (CP), suppress the complement system,

25 pPRO10, led to the synthesis of up to three viral coat protein (CPs) in addition to NIa.

26 H]6+ precursor ions of the bacteriophage MS2 viral coat protein following concentration and purificat

27 In the absence of scaffolding protein, the viral coat proteins form aberrantly shaped and incorrect

28 Viral coat proteins function in virion assembly and viru

29 Scytovirin binds to viral coat proteins gp120, gp160, and gp41 but not to ce

30 entrated on neurotoxic properties of the HIV viral coat protein, gp120.

31 The viral coat protein has an essential role in in vitro tra

32 s are infectious only in the presence of the viral coat protein; however, the mechanisms describing c

33 has IRES activity and produces low levels of viral coat protein in vitro and in vivo Our findings may

34 s solubilization and subsequent digestion of viral coat proteins in under 30 s.

35 o and in vitro for the polymerization of the viral coat protein into closed T = 7 icosahedral procaps

36 TGB1 mediated insertion of the viral coat protein into PD, probably by its interaction

37 (TSV)] is that, in addition to genomic RNAs, viral coat protein is required to establish infection in

38 ration of therapeutics against the influenza viral coat protein neuraminidase is a response to the co

39 s are infectious only in the presence of the viral coat protein; therefore, an understanding of coat

40 ion/ionization mass spectrometry has enabled viral coat proteins to be characterized directly from th

41 splay of the BC and HI loops as found in the viral coat protein, VP1, of BKV.

42 Viral coat protein was detected by enzyme-linked immunos

43 esidue helix-turn-helix subdomain of the P22 viral coat protein was investigated using circular dichr

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