1 responds to increased phosphorylation of the viral core protein.

2 are immature intermediates that lack DNA and viral core proteins.

3 ation codon of pr65gag, the precursor of the viral core proteins.

4 into cells infected with VACV expressing the viral core protein A4 fused to yellow fluorescent protei

5           HCV infection or the expression of viral core protein alone in hepatocyte culture or transg

6 and characterized an interaction between the viral core protein and host protein within bgcn homolog

7 involve ubiquitination or methylation of the viral core protein and is not mediated by the nitric oxi

8 S-FLU) that can infect cells and express the viral core proteins and neuraminidase but cannot replica

9 ent marker and a fluorescently tagged Vpr (a viral core protein) enables detection of single-virus fu

10 egion of micro1 continued to colocalize with viral core proteins in punctate structures, indicating t

11                                          The viral core protein induces hepatocarcinogenesis inductio

12                   The L1 and M1 genes encode viral core proteins involved in viral RNA synthesis.

13 lular and virus-induced mechanisms, with the viral core protein playing an important role in steatosi

14  products are the leading candidates for the viral core protein proteinase (vCPP).

15       The hepatitis C virus (HCV) produces a viral core protein that targets to mitochondria and incr

16 w a temporal correlation between the loss of viral core protein VII from the adenovirus genome and a

17 hat the sensitization of cells to TNF by the viral core protein was not due to up-regulation of TNFR

18 o steps in DHBV replication that involve the viral core protein were inhibited by DuIFN-alpha.

19 iated with complete dephosphorylation of the viral core protein, which forms the nucleocapsid wherein

20 case of triacsin C, reduced stability of the viral core protein, which forms the virion nucleocapsid