ライフサイエンスコーパス: viral encephalitis

1 ms or types of presentations often suggest a viral encephalitis.

2 es a role for pathogenic Th17 cells in fatal viral encephalitis.

3 nhances survival in a murine model of lethal viral encephalitis.

4 ne orchestration of leukocyte recruitment in viral encephalitis.

5 as plausible therapeutic targets in treating viral encephalitis.

6 only recently been appreciated as a cause of viral encephalitis.

7 s virus (JEV) is the leading global cause of viral encephalitis.

8 toimmune and inflammatory diseases including viral encephalitis.

9 y be useful adjunct therapy in some types of viral encephalitis.

10 es in the induction of acute seizures during viral encephalitis.

11 ting pathologic immune responses in nonlytic viral encephalitis.

12 targeting ICAM-1 signaling may help control viral encephalitis.

13 hagy) and increases susceptibility to lethal viral encephalitis.

14 tem neuronal dysfunction seen in fatal Nipah viral encephalitis.

15 a promising and novel treatment strategy for viral encephalitis.

16 mergence of novel mechanisms of transmitting viral encephalitis.

17 hat has become a significant global cause of viral encephalitis.

18 is one of the leading etiologies of sporadic viral encephalitis.

19 itate the development of immunotherapies for viral encephalitis.

20 lity by preventing the development of lethal viral encephalitis.

21 ic early radiographic manifestations of this viral encephalitis.

22 test was positive in 12 of 24 patients with viral encephalitis.

23 provides a well-characterized model to study viral encephalitis.

24 ive approach, we have used a murine model of viral encephalitis and an in vivo imaging system that ca

25 system (CNS) provides a model for studies of viral encephalitis and demyelinating disease.

26 mans and is the leading etiological agent of viral encephalitis and eye infections.

27 s emerged as a significant cause of epidemic viral encephalitis and flaccid limb paralysis, yet the m

28 ented to identify patients hospitalized with viral encephalitis and meningitis.

29 ing viral infections, such as other forms of viral encephalitis and other HSV-1 diseases in particula

30 in the T-cell-mediated IL-10 response during viral encephalitis and persistence.

31 focuses on some of the most common agents of viral encephalitis, as well as important emerging viral

32 titers in the brain after day 6, and died of viral encephalitis between days 7 and 12.

33 e CNS but enhances morbidity associated with viral encephalitis by increasing the ratio of IFN-gamma

34 role for NKG2D in host defense during acute viral encephalitis by selectively enhancing CTL activity

35 Viral encephalitis caused by neuroadapted yellow fever 1

36 Thus, the rapid monocyte response to viral encephalitis constitutes an indirect antiviral pat

37 etal dynamics, the worldwide epidemiology of viral encephalitis continues to evolve in surprising way

38 3 sera from New York patients with suspected viral encephalitis demonstrated concordance with results

39 cohort of 11 adult Italian patients in whom viral encephalitis developed.

40 The fourth had viral encephalitis during infancy.

41 oreover, recent investigations indicate that viral encephalitis (e.g., herpes simplex) can trigger sy

42 ovirus, was attributed to large outbreaks of viral encephalitis; however, compelling evidence suggest

43 se encephalitis (JE) is the leading cause of viral encephalitis in Asia, resulting in 70,000 cases ea

44 of severe hand, foot, and mouth disease and viral encephalitis in children across the Asia-Pacific r

45 e Flaviviridae family, is a leading cause of viral encephalitis in Europe and Asia.

46 roportion of patients once suspected to have viral encephalitis in fact have an autoimmune etiology f

47 WEEV is an important cause of viral encephalitis in humans and horses in the Americas,

48 s emerged globally as a significant cause of viral encephalitis in humans, especially in immunocompro

49 virus (WNV) is responsible for outbreaks of viral encephalitis in humans, horses, and birds, with pa

50 s emerged globally as a significant cause of viral encephalitis in humans.

51 litis (HSE) is the most common form of acute viral encephalitis in industrialized countries.

52 MAV-1 produces viral encephalitis in its natural host, providing a good

53 been used to study the pathogenesis of acute viral encephalitis in mice for many years.

54 RNA afforded robust protection against fatal viral encephalitis in mice.

55 WNV has become the leading cause of epidemic viral encephalitis in North America.

56 aviridae) are the leading cause of pediatric viral encephalitis in North America.

57 le virus has become the most common cause of viral encephalitis in several states.

58 le virus (WNV) has been the leading cause of viral encephalitis in the United States since 1999.

59 WNV is now the most common cause of epidemic viral encephalitis in the United States, and it will lik

60 rus (LACV) is the primary cause of pediatric viral encephalitis in the United States.

61 ic Bunyavirus, is a major cause of pediatric viral encephalitis in the United States.

62 virus (LACV) is the major cause of pediatric viral encephalitis in the United States; however, the me

63 Neurologic complications associated with viral encephalitis, including seizures and cognitive imp

64 sfusion-associated and transplant-associated viral encephalitis, including West Nile virus, rabies vi

65 halitis virus is the most important emerging viral encephalitis; interferon alpha was not effective a

66 Viral encephalitis is a major cause of morbidity and mor

67 Acute viral encephalitis is believed to result from direct vir

68 pid monocyte recruitment into the CNS during viral encephalitis is dispensable for T cell migration a

69 migration of lymphocytes into the CNS during viral encephalitis is hindered by the blood-brain barrie

70 acy of the inhibitor in protecting mice from viral encephalitis, JNK inhibition represents a promisin

71 sfusion-associated and transplant-associated viral encephalitis may be an underrecognized risk of the

72 In a viral encephalitis model in mice, disease depended on CT

73 and serum specimens taken from patients with viral encephalitis or a presumed viral infection involvi

74 Investigation found no evidence of viral encephalitis or other recognized causes of rapid-o

75 cy is a broad mechanism of death in cases of viral encephalitis, plethysmography was evaluated in mic

76 patients with several types of autoimmune or viral encephalitis, PNH, or mutations of the Caspr2-enco

77 Acute viral encephalitis requires rapid pathogen elimination w

78 neurological cells in patients with types of viral encephalitis that have not been treatable in the p

79 he central nervous system (CNS) during acute viral encephalitis was studied in vivo using fluorescent

80 Acute viral encephalitis was thus induced in CCL2-deficient (C

81 ain after HIV-1 infection, a murine model of viral encephalitis was used to study relationships, over

82 ribute to age-dependent resistance to lethal viral encephalitis, we compared central nervous system (

83 E.P. became profoundly amnesic in 1992 after viral encephalitis, which damaged his medial temporal lo

84 HSV-1 is the leading cause of sporadic viral encephalitis, with mortality rates approaching 30%