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1 survival, represents a novel property for a viral structural protein.
2 block viral DNA replication and synthesis of viral structural proteins.
3 icles (VLVs) that contain VSV G but no other viral structural proteins.
4 patches and extensions contained all of the viral structural proteins.
5 d does not require the coexpression of other viral structural proteins.
6 revealed they lacked gB but contained other viral structural proteins.
7 d macrophages by regulating the stability of viral structural proteins.
8 rus assembly site for interaction with other viral structural proteins.
9 omeric state and spatial relationships among viral structural proteins.
10 substrate, or the molecular interactions of viral structural proteins.
11 d cells, requiring the nuclear import of all viral structural proteins.
12 ng in a brief and limited synthesis of HIV-1 viral structural proteins.
13 ar masses similar to those of phosphorylated viral structural proteins.
14 ber mutants were defective for processing of viral structural proteins.
15 the cellular and humoral immune response to viral structural proteins.
16 ed by dramatic conformational changes within viral structural proteins.
18 lation in the 0 and +1 frames to produce the viral structural proteins and a +1 overlapping open read
19 t cells require dynamic interactions between viral structural proteins and a variety of cellular fact
22 In contrast to vRNA hybridization results, viral structural proteins and glycoproteins, evaluated b
23 gE-null mutants produce wild-type levels of viral structural proteins and infectious virions in the
24 tein that is essential for axonal sorting of viral structural proteins and is highly conserved among
26 rus replication cycle is the assembly of the viral structural proteins and the packaging of the eight
28 D4(+) T cells that recognize epitopes in the viral structural proteins and thus can provide direct he
29 ers the cellular immune responses to a human viral structural protein, and that these effects may con
30 promoter, which regulates expression of the viral structural proteins, and a second internal promote
31 l, these genomes produce together all of the viral structural proteins, and cells release a combinati
32 replicates its genome, expresses all of the viral structural proteins, and releases viral particles
33 ble-membrane vesicles for RNA synthesis, and viral structural proteins assemble virions at the ER-Gol
34 hance the immunogenicity of this nonsecreted viral structural protein at the B and T cell level, we c
35 code the viral genome and the genes encoding viral structural proteins, by binding to and oligomerizi
36 merization of the Gag polyprotein, the major viral structural protein capable of forming virus-like p
37 Virus-like particles (VLPs), comprised of viral structural proteins devoid of genetic material, ar
38 IgG), IgG avidity, and IgG response to the 3 viral structural proteins (E1, E2, and C), reflected by
39 antiviral agents could be applied to certain viral structural proteins, enzymes, and other factors or
41 otein, and contains 60 copies of each of the viral structural proteins F and G, which comprise the sh
45 interact with the nucleocapsid domain of the viral structural protein Gag and is incorporated in sign
47 1 (HIV-1) particle assembly mediated by the viral structural protein Gag occurs predominantly on the
48 been observed as its colocalization with the viral structural protein Gag or its accumulation in viru
49 B sequences for the genes encoding the major viral structural protein (Gag) and two regulatory protei
50 ugh a specific interaction between the major viral structural protein, Gag, and an RNA packaging sign
52 bis virus genomes (replicons) which lack the viral structural protein genes and contain heterologous
55 ng may effect cellular immune responses to a viral structural protein in the context of genetic immun
56 ruits the ribosome and drives translation of viral structural proteins in a factor-independent manner
57 gE is required for wild-type localization of viral structural proteins in axons of infected neurons.
59 e interaction of envelope protein with other viral structural proteins in the virus assembly and matu
60 an lymphocytes, while reducing the yields of viral structural proteins, infectivity, and tumor necros
61 However, it is not understood how Gag (the viral structural protein) interacts with these signals t
63 (ET) domains of gE/gI promote the sorting of viral structural proteins into proximal axons to begin a
64 ts indicate that reduced axonal targeting of viral structural proteins is a compelling explanation fo
66 and by enzyme immunoassays with recombinant viral structural protein (K8.1) and latent protein (late
67 of plasmids expressing all of the remaining viral structural proteins led to a substantial increase
68 likely that the cellular immune responses to viral structural proteins may be important for eradicati
69 ased cellular immune response to hepatitis B viral structural proteins may be important for recovery
70 lar model is that deposition of ubiquitin on viral structural proteins mediates class E machinery rec
71 infection, and neither the synthesis of the viral structural proteins nor the presence of the other
74 cycle, further highlighting the paradigm of viral structural proteins playing additional functional
76 -strand molar ratio of approximately 8:1 and viral structural proteins S, M, and N, and 65% were in a
77 sults provide a rare example of an oncogenic viral structural protein, show that interaction of the v
80 This appears to be the first example of a viral structural protein that is also involved in the tr
82 Forest virus RNA replicon encoding a single viral structural protein, the vesicular stomatitis virus
83 y assays that measure only antibodies to the viral structural proteins, the majority of such patients
84 and to differentially regulate expression of viral structural proteins, their replication was made de
87 avage of the viral outer capsid, the fate of viral structural proteins was assessed by sodium dodecyl
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