ライフサイエンスコーパス: viral structural protein

1 survival, represents a novel property for a viral structural protein.

2 block viral DNA replication and synthesis of viral structural proteins.

3 icles (VLVs) that contain VSV G but no other viral structural proteins.

4 patches and extensions contained all of the viral structural proteins.

5 d does not require the coexpression of other viral structural proteins.

6 revealed they lacked gB but contained other viral structural proteins.

7 d macrophages by regulating the stability of viral structural proteins.

8 rus assembly site for interaction with other viral structural proteins.

9 omeric state and spatial relationships among viral structural proteins.

10 substrate, or the molecular interactions of viral structural proteins.

11 d cells, requiring the nuclear import of all viral structural proteins.

12 ng in a brief and limited synthesis of HIV-1 viral structural proteins.

13 ar masses similar to those of phosphorylated viral structural proteins.

14 ber mutants were defective for processing of viral structural proteins.

15 the cellular and humoral immune response to viral structural proteins.

16 ed by dramatic conformational changes within viral structural proteins.

17 The expression of viral structural proteins along with replicating S and M

18 lation in the 0 and +1 frames to produce the viral structural proteins and a +1 overlapping open read

19 t cells require dynamic interactions between viral structural proteins and a variety of cellular fact

20 Rather, it blocks processing of viral structural proteins and assembly of mature progeny

21 The persistence of viral structural proteins and glycoproteins in GCs was a

22 In contrast to vRNA hybridization results, viral structural proteins and glycoproteins, evaluated b

23 gE-null mutants produce wild-type levels of viral structural proteins and infectious virions in the

24 tein that is essential for axonal sorting of viral structural proteins and is highly conserved among

25 proteins are essential for the expression of viral structural proteins and productive infection.

26 rus replication cycle is the assembly of the viral structural proteins and the packaging of the eight

27 ons entails concerted interactions among the viral structural proteins and the RNA genome.

28 D4(+) T cells that recognize epitopes in the viral structural proteins and thus can provide direct he

29 ers the cellular immune responses to a human viral structural protein, and that these effects may con

30 promoter, which regulates expression of the viral structural proteins, and a second internal promote

31 l, these genomes produce together all of the viral structural proteins, and cells release a combinati

32 replicates its genome, expresses all of the viral structural proteins, and releases viral particles

33 ble-membrane vesicles for RNA synthesis, and viral structural proteins assemble virions at the ER-Gol

34 hance the immunogenicity of this nonsecreted viral structural protein at the B and T cell level, we c

35 code the viral genome and the genes encoding viral structural proteins, by binding to and oligomerizi

36 merization of the Gag polyprotein, the major viral structural protein capable of forming virus-like p

37 Virus-like particles (VLPs), comprised of viral structural proteins devoid of genetic material, ar

38 IgG), IgG avidity, and IgG response to the 3 viral structural proteins (E1, E2, and C), reflected by

39 antiviral agents could be applied to certain viral structural proteins, enzymes, and other factors or

40 g in truncated ORFs and effectively minimize viral structural protein expression.

41 otein, and contains 60 copies of each of the viral structural proteins F and G, which comprise the sh

42 Viral structural proteins form the critical intermediary

43 Expression of viral structural proteins from the protein expression pl

44 In contrast to enzymes, viral structural protein function can be much more chall

45 interact with the nucleocapsid domain of the viral structural protein Gag and is incorporated in sign

46 ing into virions, and RNA interacts with the viral structural protein Gag in the cytoplasm.

47 1 (HIV-1) particle assembly mediated by the viral structural protein Gag occurs predominantly on the

48 been observed as its colocalization with the viral structural protein Gag or its accumulation in viru

49 B sequences for the genes encoding the major viral structural protein (Gag) and two regulatory protei

50 ugh a specific interaction between the major viral structural protein, Gag, and an RNA packaging sign

51 tion cycle, leading to the expression of the viral structural proteins, Gag, Pol, and Env.

52 bis virus genomes (replicons) which lack the viral structural protein genes and contain heterologous

53 We show that selective expression of viral structural proteins gives rise to virus-like parti

54 null mutant infections, only a subset of the viral structural proteins had entered axons.

55 ng may effect cellular immune responses to a viral structural protein in the context of genetic immun

56 ruits the ribosome and drives translation of viral structural proteins in a factor-independent manner

57 gE is required for wild-type localization of viral structural proteins in axons of infected neurons.

58 The role of viral structural proteins in the initiation of adaptive

59 e interaction of envelope protein with other viral structural proteins in the virus assembly and matu

60 an lymphocytes, while reducing the yields of viral structural proteins, infectivity, and tumor necros

61 However, it is not understood how Gag (the viral structural protein) interacts with these signals t

62 The assembly of the viral structural proteins into infectious virions is oft

63 (ET) domains of gE/gI promote the sorting of viral structural proteins into proximal axons to begin a

64 ts indicate that reduced axonal targeting of viral structural proteins is a compelling explanation fo

65 uirement for direct ubiquitin conjugation to viral structural proteins is less well understood.

66 and by enzyme immunoassays with recombinant viral structural protein (K8.1) and latent protein (late

67 of plasmids expressing all of the remaining viral structural proteins led to a substantial increase

68 likely that the cellular immune responses to viral structural proteins may be important for eradicati

69 ased cellular immune response to hepatitis B viral structural proteins may be important for recovery

70 lar model is that deposition of ubiquitin on viral structural proteins mediates class E machinery rec

71 infection, and neither the synthesis of the viral structural proteins nor the presence of the other

72 Amino acids 80-110 of the viral structural protein ODV-EC27 (-EC27) demonstrate 25

73 otein, and in late regions that code for the viral structural proteins, penton base, and fiber.

74 cycle, further highlighting the paradigm of viral structural proteins playing additional functional

75 Some viral structural proteins require ubiquitin ligase activ

76 -strand molar ratio of approximately 8:1 and viral structural proteins S, M, and N, and 65% were in a

77 sults provide a rare example of an oncogenic viral structural protein, show that interaction of the v

78 HCV core protein represents the first known viral structural protein substrate of tTG.

79 Viral structural protein synthesis was similar in 633- a

80 This appears to be the first example of a viral structural protein that is also involved in the tr

81 The VP40 matrix protein is a key viral structural protein that is critical for virion egr

82 Forest virus RNA replicon encoding a single viral structural protein, the vesicular stomatitis virus

83 y assays that measure only antibodies to the viral structural proteins, the majority of such patients

84 and to differentially regulate expression of viral structural proteins, their replication was made de

85 First, we trained ANNs to classify viral structural proteins using amino acid frequency; th

86 e times, the VHS RNase is neutralized by the viral structural proteins VP16 and VP22.

87 avage of the viral outer capsid, the fate of viral structural proteins was assessed by sodium dodecyl

88 At that time, expression of viral structural proteins was low and infected cells dis

89 One of the tyrosine-phosphorylated viral structural proteins was the tegument protein VP22.