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1 ite (HA), and (3) binding to bacteria (e.g., viridans streptococci).
2 ifferentiating Streptococcus pneumoniae from viridans streptococci.
3 neumoniae DNA probe before identification as viridans streptococci.
4 e revealed the presence of this gene in most viridans streptococci.
5 ous recombination with DNA from PBP genes of viridans streptococci.
6 ccus pyogenes, Streptococcus agalactiae, and viridans streptococci.
7 9 clinical isolates of S. pneumoniae and 101 viridans streptococci and were as follows: Pneumoslide,
11 he presence of periapical abscesses and oral viridans streptococci DNA-positive thrombi was found (od
12 protein concentrations to adherence by oral viridans streptococci in microplates coated with whole s
13 ar coagulation, more than 60% of isolates of viridans streptococci induced platelet aggregation when
14 ichoic acid (LTA) purified from 2 strains of viridans streptococci induced the accumulation of tumor
15 ntal infection and oral bacteria, especially viridans streptococci, may be associated with the develo
16 lure [P =.01], bacterial etiology other than viridans streptococci [P<.001 except Staphylococcus aure
18 t interferon-gamma, LTA from both strains of viridans streptococci provoked the accumulation of induc
20 PMNL ingested greater numbers of SMG and viridans streptococci than S. aureus but killed these or
23 ypical for endodontic infection, mainly oral viridans streptococci, was measured in 78.2% of thrombi,
24 an did Staphylococcus aureus, in contrast to viridans streptococci, which caused greater chemotaxis t
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