1  The most common presenting sign (84.2%) was virilization.

2 ential for male reproductive development and virilization.

3  traits that may offset the costs of extreme virilization.

4 n the newborn period or early childhood with virilization and adrenal insufficiency, with or without

5 factor required for male sex development and virilization and contributes to prostate cancer initiati

6 ns pose risks for unwanted effects including virilization and hypertrophy of reproductive organs.

7                             In children with virilization and nonpalpable gonads, only those with tes

8 s responsible for testicular descent through virilization and outgrowth of the embryonic gubernaculum

9 ian inhibiting substance in 65 children with virilization at birth and nonpalpable gonads (age at dia

10  levels are excessive, causing inappropriate virilization but failing to induce skeletal maturity.

11 ormone excess, including Cushing's syndrome, virilization, feminization, or--less frequently--hyperte

12 received previous hormonal therapy to induce virilization, fertility, or both.

13 ctive in preventing bone age advancement and virilization in boys with familial male-limited precocio

14             A common cause of female genital virilization is congenital adrenal hyperplasia (CAH), in

15 ulin-like growth factor 3 (INLS3) that cause virilization of female embryos and ovarian descent.

16 ogens, which are secreted and cause in utero virilization of the affected female fetus genitalia in t

17                                  The extreme virilization of the female spotted hyena raises interest

18 n in male genitourinary tissues, affects the virilization of the reproductive tract and results in ge

19 ilms tumor, and intersex disorders (XY under-virilization or XY female).

20                   Cushing's syndrome without virilization was uncommon (5.5%).