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1 attributed to a genotype; however, in simple virilizing CAH, we observe wide phenotypic variability,
5 the salt-wasting form and 13 with the simple virilizing form), 39 age-matched normal subjects, and 20
9 ed either with severe salt-wasting or simple virilizing phenotypes or with milder nonclassical phenot
10 potent hAR binding and agonist activity, low virilizing potential, and good pharmacokinetic profiles
11 lly, affected female newborns are profoundly virilized (Prader score of 4/5), and both genders displa
13 the patient with cortisol-secreting tumor), virilizing symptoms (in the patient with testosterone-se
14 antiandrogen for inoperable prostate cancer, virilizing syndromes in women, and the inhibition of sex
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