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1 otection against challenge with the parental virulent virus.
2 this pandemic, consistent with an unusually virulent virus.
3 ytogenes into rJ.M(Y135Q) generated a highly virulent virus.
4 gene 71 did not result in the generation of virulent virus.
5 ction after experimental challenge with this virulent virus.
6 nts fully protected mice from challenge with virulent virus.
7 brain and spinal cord as efficiently as the virulent viruses.
8 e of resistance to superinfection with fully virulent viruses.
9 ith two copies of nsP3 from SFV6 resulted in virulent viruses.
12 etely protected against challenge with live, virulent virus after receiving two 0.15-microg doses of
14 constructed from cDNA clones derived from a virulent virus and from two attenuated viruses adapted t
15 al (anterograde and retrograde) transport of virulent virus and unidirectional (retrograde) transport
16 rus-neutralizing antibodies to this uniquely virulent virus, and that humans can sustain circulating
18 ining virulent virus may be safe, as long as virulent viruses are present at levels below a critical
19 duction in the Republic of Georgia, a highly virulent virus, ASFV Georgia 2007 (ASFV-G), has caused a
21 rate) were immune to disease upon homologous virulent virus challenge at postinoculation day (PID) 21
24 d to be highly protective against homologous virulent virus challenges in type I interferon receptor
26 c virus with the HN protein derived from the virulent virus exhibited a tissue predilection similar t
27 e found two phylogenetically related, highly virulent viruses (genus Ranavirus, family Iridoviridae)
28 c CVB3 strain, created from sequences of two virulent viruses, is sufficiently attenuated to act as a
29 a vaccine against virulent ASFV-G, a highly virulent virus isolate detected in the Caucasus region i
30 ssible that the source of some of the recent virulent viruses isolated from poultry and wild birds mi
32 ate that live-attenuated vaccines containing virulent virus may be safe, as long as virulent viruses
34 etic analysis and pathotyping confirmed that virulent viruses of different genotypes are circulating
35 lated to this group since 1986 suggests that virulent viruses of this genotype may have evolved unnot
37 ucosal immunity to reinfection with a highly virulent virus requires the accumulation and persistence
38 neglected until the recent emergence of two virulent viruses, severe fever with thrombocytopenia syn
39 wborns inoculated with either tumorigenic or virulent virus strains, and (iii) dominant or semidomina
41 These results suggest that TR339, the most virulent virus tested, attaches to cells through a low-a
42 P3 of SFV6 with that of A774wt resulted in a virulent virus; the virulence of this recombinant was st
43 nuated viruses may limit the pathogenesis of virulent viruses, thus establishing a virulence threshol
44 required for pathogenicity or whether a more virulent virus variant had evolved, we generated a molec
45 CNS cells survive acute infection with this virulent virus, we developed a recombinant JHMV that exp
46 a series of challenges ending with a highly virulent virus with a primary isolate envelope heterolog
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