ライフサイエンスコーパス: virus DNA

1 esult that was positive for varicella-zoster virus DNA.

2 than the representation of dGMP in vaccinia virus DNA.

3 tion PCR were performed to screen for herpes virus DNA.

4 patient's lesion tested positive for cowpox virus DNA.

5 nged lowering of serum levels of hepatitis B virus DNA.

6 h conventional measures of serum hepatitis B virus DNA.

7 ed onto unintegrated Moloney murine leukemia virus DNAs.

8 assays that can detect as low as 16 pM Ebola Virus DNA, 0.01ng/mL carcinoembryonic antigen (CEA), and

9 the 328 patients with data for Epstein-Barr virus DNA, a detectable viral DNA titre was an independe

10 polymerase chain reaction (PCR) assay for BK virus DNA, a retrospective analysis was done of sequenti

11 s autointegration of Moloney murine leukemia virus DNA, also plays an indirect role in generating the

12 erived from the terminal regions of vaccinia virus DNA and contained in a superhelical plasmid, into

13 sed on restriction enzyme digest patterns of virus DNA and growth rates both in vitro and in vivo.

14 The peak levels of hepatitis B virus DNA and hepatitis B core-related antigen after ces

15 Rebound of hepatitis B virus DNA and hepatitis B core-related antigen was assoc

16 10(4) cellular genomes, whereas Epstein-Barr virus DNA and HIV-1 DNA were detected in 16 and 22 sampl

17 (v) Despite the reduced recoveries of LAT- virus DNA and productive-cycle transcripts in reactivati

18 d capsid has formed, it is packaged with the virus DNA and transported to the cytoplasm where further

19 mutation was transferred into wild-type (wt) virus DNA, and the UL25 mutant ts1249 was isolated and c

20 wide variety of antigens including bacteria, viruses, DNA, and proteins due to the simplicity of thei

21 sease has been made using cloned hepatitis B virus DNA as a transgene in a severe combined immunodefi

22 In arrested cells no foamy virus DNA band was detected in cells harvested at 1 or 7

23 ransactivator IE62, ORF 29 encodes the major virus DNA binding protein, and ORF 21 encodes a protein

24 iation with sorting nexin 17, to ensure that virus DNA bound to L2 is recycled through the trans-Golg

25 igned specifically for detection of smallpox virus DNA, but all viruses of the genus Orthopoxvirus te

26 Weekly plasma samples were tested for BK virus DNA by polymerase chain reaction (PCR).

27 y testing lesions swabs for varicella zoster virus DNA by polymerase chain reaction).

28 Once "uncoated" in this way, the virus DNA can be transcribed and replicated.

29 herpesvirus 6 and human herpesvirus 7 and BK virus DNA, CMV disease, and acute rejection.

30 There were doses of 01/PEME for which the virus DNA concentration in the tumor increased over time

31 virus replication by >1000-fold increase in virus DNA copies over time.

32 e ability to detect and monitor Epstein-Barr virus DNA copy number in a variety of settings.

33 tem lymphoma, and monitoring of Epstein-Barr virus DNA copy number in spinal fluid may be useful in a

34 The quantity of latent virus DNA correlates with and may be a major determinant

35 infected cell cultures and tissues, smallpox virus DNA could be detected in a background of human DNA

36 The utility of Epstein-Barr virus DNA detection and quantification in the serum or p

37 Urine was collected for JC virus DNA detection.

38 d pretreatment levels of plasma Epstein Barr virus DNA (EBV-DNA), as determined by quantitative real-

39 9 which binds to integrated murine leukaemia virus DNA elements and recruits KAP1 to repress them.

40 A binding through specific contacts with the virus DNA ends in the 3'-processing and ST reactions.

41 ve detection of DNA, including a hepatitis B virus DNA fragment.

42 Since the size range of sheared virus DNA fragments governs the limit of accurate protei

43 Terminal repeat analysis of virus DNA from a KS lesion suggests a monoclonal expansi

44 ability of Rep40 to package adeno-associated virus DNA in a tissue culture-based assay.

45 rase chain reaction [PCR] for herpes simplex virus DNA in cerebrospinal fluid) were randomized to rec

46 chain reaction amplification of Epstein-Barr virus DNA in cerebrospinal fluid, 18F-fluoro-deoxyglucos

47 demonstrated 2.75 times less herpes simplex virus DNA in cyclic-AMP treated BeWo cells, but 2.0 to 7

48 B patients with detectable serum hepatitis B virus DNA in European tertiary referral centers.

49 n plants that did become infected, levels of virus DNA in mutants and wild-type did not differ signif

50 on magnetic resonance imaging regressed; JC virus DNA in plasma, likely originating from the brain b

51 It is concluded that quantitative PCR for BK virus DNA in serum is useful both for identifying transp

52 Epstein-Barr virus DNA in serum or plasma has been found in infectiou

53 association of the presence of Epstein-Barr virus DNA in systemic lupus erythematosus patients compa

54 JC virus DNA in the CSF and peripheral blood was quantified

55 ticle reporting the presence of Epstein-Barr virus DNA in the vitreous of a patient with Vogt-Koyanag

56 full-blown AIDS without evidence of vaccine virus DNA in tissues.

57 , but 2.0 to 7.4 times more adeno-associated virus DNA in treated cells.

58 The virus DNA inserts were amplified, quantitated, and spott

59 s study, we investigated whether hepatitis B virus DNA integration occurs preferentially at sites of

60 enetics and biochemistry of avian and murine virus DNA integration, but the pace of discovery increas

61 By transfecting vaccinia virus DNA into cells infected with ectromelia virus and

62 frequent integration of mouse mammary tumor virus DNA into chromosomes, implicating the gene in the

63 e immunized individuals over age 60, vaccine virus DNA is shed in saliva up to 4 weeks.

64 In this suppressed state the virus DNA is vulnerable to mutagenic events that affect

65 In many viruses, DNA is confined at such high density that its b

66 not catalyze base changes in murine leukemia virus DNA, it can be recovered from these virus particle

67 minotransferase and 37% had >/=1 hepatitis B virus DNA level assessed annually.

68 Elevated hepatitis B virus DNA levels in patients in their 40s with perinatal

69 We therefore compared herpes simplex virus DNA levels in specimens before and after 16 months

70 containing only the 298-amino acid Chlorella virus DNA ligase (a 'minimal' eukaryotic ATP-dependent l

71 Chlorella virus DNA ligase (ChVLig) has pluripotent biological act

72 Chlorella virus DNA ligase (ChVLig) is a minimized eukaryal ATP-de

73 Chlorella virus DNA ligase (ChVLig) is an instructive model for me

74 the conformational dynamics of the Chlorella virus DNA ligase (ChVLig), a minimized eukaryal ATP-depe

75 nt ligation of RNA-splinted DNA by Chlorella virus DNA ligase (PBCV-1 DNA ligase).

76 NA) detection method that utilizes Chlorella virus DNA ligase (SplintR((R)) Ligase).

77 The 552 amino acid vaccinia virus DNA ligase consists of three structural domains de

78 roles of conserved amino acids of Chlorella virus DNA ligase during the third step of the ligation p

79 Vaccinia virus DNA ligase has an intrinsic nick-sensing function.

80 Our findings indicate that Chlorella virus DNA ligase has the potential to affect genome inte

81 ion analysis of the 298 amino acid Chlorella virus DNA ligase indicates that motif VI plays a critica

82 Chlorella virus DNA ligase is the smallest eukaryotic ATP-dependen

83 Chlorella virus DNA ligase is the smallest eukaryotic ATP-dependen

84 Vaccinia virus DNA ligase repairs nicked duplex DNA substrates co

85 )) in the nick joining reaction of Chlorella virus DNA ligase, an exemplary ATP-dependent enzyme.

86 to the mitochondria or expressing Chlorella virus DNA ligase, the minimal eukaryal nick-sealing enzy

87 progressive decrease in the median plasma BK virus-DNA load, and undetectable levels at the last foll

88 CPE, visible intracellular virions, and high virus DNA loads.

89 according to the VR definition (hepatitis B virus DNA <200, < 2000, < 20,000 IU/mL) or duration of o

90 JC virus DNA may harbor in CD34+ cells in bone marrow that

91 ion inhibitor, partially reversed the helper virus DNA methylation and restored a portion of vector p

92 A pan-virus DNA microarray (Virochip) was used to detect a hum

93 each containing >/=105 copies herpes simplex virus DNA/ml collected a median of 5 months apart (IQR:

94 tained within those 322 samples were variola virus DNA, obtained from purified viral preparations, at

95 ic assay for the rapid detection of smallpox virus DNA on both the Smart Cycler and LightCycler platf

96 oligodendrocytes, which were positive for JC virus DNA on in situ hybridization.

97 varicella (by detection of varicella zoster virus DNA or epidemiological link) from 42 days after th

98 in cells transgenomic for EBV (Epstein Barr Virus) DNA or for the telomerase gene, the large number

99 y been described which use inactivated whole virus, DNA or RNA that express the virus' Envelope (E) g

100 Since this assembly step is unique to the virus, DNA packaging is a novel target for the developme

101 f exonuclease activity of the herpes simplex virus DNA polymerase (Pol) on DNA replication fidelity w

102 The catalytic subunit of herpes simplex virus DNA polymerase (Pol), a member of the B family pol

103 to the role of D4 as a co-factor of vaccinia virus DNA polymerase and allows a better understanding o

104 The herpes simplex virus DNA polymerase catalytic subunit, which has intrin

105 Herpes simplex virus DNA polymerase consists of a catalytic subunit, Po

106 The vaccinia virus DNA polymerase E9 requires two viral proteins, A20

107 seen even in the presence of herpes simplex virus DNA polymerase inhibitors, but not in the absence

108 he catalytic subunit, Pol, of herpes simplex virus DNA polymerase interacts via its extreme C terminu

109 Herpes simplex virus DNA polymerase is a heterodimer composed of a cata

110 Herpes simplex virus DNA polymerase is a heterodimer composed of UL30,

111 The vaccinia virus DNA polymerase is inherently distributive but acqu

112 Although the vaccinia virus DNA polymerase is inherently distributive, a highl

113 n of the catalytic subunit of herpes simplex virus DNA polymerase with the processivity subunit, UL42

114 rase beta (Pol beta) and African swine fever virus DNA polymerase X (ASFV Pol X) with one-nucleotide

115 cently demonstrated that African swine fever virus DNA polymerase X (Pol X) is extremely error-prone

116 The African swine fever virus DNA polymerase X (pol X), a member of the X family

117 low-fidelity polymerase, African swine fever virus DNA polymerase X (Pol X), and showed that they fol

118 rase beta (Pol beta) and African swine fever virus DNA polymerase X (Pol X).

119 yields of interleukin-12 p40, herpes simplex virus DNA polymerase, and interferon-gamma PCR products

120 e processivity subunit of the herpes simplex virus DNA polymerase, interacts with DNA and promotes pr

121 e processivity subunit of the herpes simplex virus DNA polymerase, UL42, is a monomer and has an intr

122 e processivity subunit of the herpes simplex virus DNA polymerase, UL42, is a monomer in solution.

123 e processivity subunit of the herpes simplex virus DNA polymerase, UL42, is essential for viral repli

124 The processivity factor of herpes simplex virus DNA polymerase, UL42, unlike "sliding clamp" proce

125 f the processivity subunit of herpes simplex virus DNA polymerase, UL42, which is crucial for interac

126 critical for horizontal transmission of the virus, DNA priming/MVA boosting was evaluated for the ab

127 One vaccination with 0.5 microg of CEE virus DNA provided protective immunity for at least 2 mo

128 containing packaging signals from these two viruses, DNA proviruses containing genetic information f

129 e 43 samples that contained purified variola virus DNA ranging in concentration from 1 fg/ microl to

130 Of the 206 samples that contained variola virus DNA ranging in concentrations from 100 fg/ microl

131 a indicated that a host pathway initiated by virus DNA replication and acting through instability mot

132 s origin-binding domain in the initiation of virus DNA replication by analyzing the biochemical activ

133 Thus, the activity of replicative lefs or virus DNA replication is sufficient to trigger apoptosis

134 The identification of BocaSR and its role in virus DNA replication reveals potential avenues for deve

135 Visualization of virus DNA replication showed that a UL37-minus mutant wa

136 n of host protein synthesis, suggesting that virus DNA replication triggers inhibition of host biosyn

137 TNF-alpha is shown to suppress hepatitis B virus DNA replication without cell killing by disrupting

138 virus uracil DNA glycosylase is required for virus DNA replication, coupled with an inability to isol

139 D4, and D5) with essential roles in vaccinia virus DNA replication.

140 ocked late gene expression without affecting virus DNA replication.

141 Inactivated whole virus, DNA, RNA, and vectored vaccine approaches to prev

142 The hepatitis B virus DNA sequence around the precore region was determi

143 s to interact with naked mouse mammary tumor virus DNA somewhat differently than with chromatin and A

144 substrates typically utilized the end of the virus DNA substrate as the point of integration, whereas

145 anscripts disappeared prior to initiation of virus DNA synthesis (considered early), and 100 transcri

146 enome allowed gamma-H2AX accumulation during virus DNA synthesis and impaired both very late viral ge

147 lef-11, p143, dnapol, and ie-1/ie-0) blocked virus DNA synthesis and late gene expression in permissi

148 ocalization of EAP and ICP4 did not occur if virus DNA synthesis and late gene expression were preven

149 nd 100 transcripts were still detected after virus DNA synthesis begins (considered early/late); (v)

150 ) 133 (36%) of the CDSs were expressed after virus DNA synthesis begins (considered late); and (vi) e

151 227 (62%) of the CDSs were expressed before virus DNA synthesis begins; (iv) these 227 CDSs were gro

152 he effect of the HIV-2IN(IN(2)) mutations on virus DNA synthesis was analyzed by packaging IN(2) muta

153 synthesis is then shut off and replaced with virus DNA synthesis.

154 Hepatitis B virus DNA targets in human serum are simultaneously prec

155 ine aminotransferase and 44% had hepatitis B virus DNA testing; hepatitis B e antigen and hepatitis B

156 Vaccinia virus DNA topoisomerase catalyzes resolution of syntheti

157 We demonstrate that the vaccinia virus DNA topoisomerase, a eukaryotic type I enzyme, cat

158 on resolution reaction catalyzed by vaccinia virus DNA topoisomerase.

159 Information on the extent of virus DNA transcription and translation in infected tiss

160 We developed two Sindbis virus DNA vaccines encoding the measles virus hemaggluti

161 diverse insects through events triggered by virus DNA (vDNA) replication.

162 The Sindbis virus DNA vectors expressed reporter genes in transfecte

163 dent muscle following injection with Sindbis virus DNA vectors.

164 patitis C virus, West Nile virus, and dengue virus), DNA viruses (vaccinia virus and human adenovirus

165 nfection by a range of viruses including RNA viruses, DNA viruses and retroviruses.

166 ution, as well as their determinants, in RNA viruses, DNA viruses and retroviruses.

167 JC virus DNA was detectable within cell compartments of nat

168 Varicella zoster virus DNA was detected 2 months after the outbreak in en

169 HHV-8 virus DNA was detected by polymerase chain reaction (PCR

170 Epstein-Barr virus DNA was detected in a lymph node initially and the

171 revealed a normal cell count, herpes simplex virus DNA was detected in all samples by polymerase chai

172 BK virus DNA was detected in serum samples from all 4 case

173 John Cunningham virus DNA was detected in urine of seronegative individu

174 eim, Germany) PCR identification of smallpox virus DNA was developed and compiled in a kit system und

175 t brain, although selection of rearranged JC virus DNA was favored.

176 John Cunningham virus DNA was found in 75% of genitourinary tissue sampl

177 ents without toxoplasmosis, and Epstein-Barr virus DNA was found in 9 of 14 patients with central ner

178 ys postinfection and remained low even after virus DNA was lost from the cells.

179 reas the size distribution of herpes simplex virus DNA was markedly different.

180 JC virus DNA was not detected in CSF or peripheral blood fr

181 cell depletion, showing that the persisting virus DNA was reactivatable.

182 ght hemispheric and brainstem lesions and JC virus DNA was undetectable in his cerebrospinal fluid.

183 short (<10 min) circulating half-life of the virus DNA, we could monitor virus distribution to the tu

184 Two regions of cauliflower mosaic virus DNA were designed as markers to study pathogen tra

185 mit of detection of 50 aM toward hepatitis B virus DNA with the capability of discriminating a single

186 ends of microtubules and ultimately deposit virus DNA within the host nucleus.