ライフサイエンスコーパス: virus protein

1 AT1 colocalizes with the corresponding Nipah virus protein.

2 nts performed with vectors encoding a single virus protein.

3 re of M156R, the first structure of a myxoma virus protein.

4 rst report of STAT activation by a DNA tumor virus protein.

5 endogenous processing of de novo synthesized virus protein.

6 d chromatin association of NS1, an influenza virus protein.

7 gether can account for nearly half the total virus protein.

8 on detecting natural selection on influenza virus proteins.

9 d localized to rafts in the absence of other virus proteins.

10 cription factor NF-kappaB, responds to Ebola virus proteins.

11 ponse of CD8+ cells to both HIV and vaccinia virus proteins.

12 2D(b)-restricted peptides from two influenza virus proteins.

13 vaccine expressed multiple immunodeficiency virus proteins.

14 opment of the HDV genomic RNA by hepatitis B virus proteins.

15 lk culture PBMC against nonstructural dengue virus proteins.

16 e VSV G protein over either of the influenza virus proteins.

17 nthesized or exogenously delivered influenza virus proteins.

18 nce for the phosphoglycosylation of vaccinia virus proteins.

19 n which RB has been inactivated by DNA tumor virus proteins.

20 of the most abundantly SUMOylated influenza virus proteins.

21 eptide pools representing distinct influenza virus proteins.

22 sequence and biochemical information for the virus proteins.

23 e network perturbations caused by DNA tumour virus proteins.

24 NA) surface glycoprotein and other influenza virus proteins.

25 disordered residues for a set of influenza A virus proteins.

26 included an apparent smaller variant of the virus protein 1 (VP1) and a small proportion of a cleave

27 mposed primarily of the major capsid protein virus protein 1 (VP1), and pentameric arrangement of VP1

28 chimeric protein, formed by fusing vaccinia virus protein 14K (A27) to the CS of Plasmodium yoelii,

29 vant-like effect of the immunogenic vaccinia virus protein 14K.

30 Interestingly, virus protein 24 (VP24) and nucleoprotein (NP) appear to

31 was to understand the possible role of a FMD virus protein 3A, in causing disease in cattle.

32 ell response to rotavirus and is directed to virus protein 6 (VP6).

33 xpanded from single B cells responding to RV virus protein 6 or virus protein 7.

34 B cells responding to RV virus protein 6 or virus protein 7.

35 Using Protein C Epitope -Tobacco Etch Virus-Protein A Epitope (PTP)-tagged Tb11.01.4590, addit

36 utoinhibitory domain and African swine fever virus protein A238L] block the Ca(2+)-dependent reductio

37 The vaccinia virus proteins A30 and G7 are known to play essential ro

38 is also assembled downstream of the Vaccinia virus protein A36 and the phagocytic Fc-gamma receptor F

39 Subsequently, as the virus proteins accumulate, secondary cleavages of eIF4G

40 on of replication products was detected, but virus protein accumulation was reduced two- to threefold

41 studied their roles in plaquing efficiency, virus protein accumulation, infectious-center titer, and

42 In this study, we examined whether Ebola virus proteins affect BST2-mediated induction of NF-kapp

43 SV) vector, to deliver and express influenza virus proteins against which vaccinated animals develop

44 Unlike other DNA virus proteins, AL1 does not contain the pRb binding con

45 Virus protein analysis by various techniques, including

46 phosphorylation mediated by a herpes simplex virus protein and inhibits viral replication.

47 The direct interaction between a respiratory virus protein and the pneumococcus resulting in increase

48 e host factors that associate with influenza virus proteins and affect viral replication.

49 s, numerous interactions between hepatitis C virus proteins and cellular components have been identif

50 controls CD4 T-cell reactivity to influenza virus proteins and how the influenza virus-specific memo

51 he E1- and E4-deleted vector expresses fewer virus proteins and induces less apoptosis, leading to bl

52 ism induced by vaccination against six Ebola virus proteins and provide additional evidence that cyto

53 n target more conserved regions of influenza virus proteins and recognize a broader array of influenz

54 ze the spatio-temporal distribution of Ebola virus proteins and RNA during virus replication.

55 ction of Nature's equivalents (e.g. enzymes, viruses, proteins and DNA).

56 ting the complex interaction between lipids, virus proteins, and buffer conditions in membrane fusion

57 F formation, colocalization with associating virus proteins, and characterization of virus replicatio

58 viruses expressing all individual influenza virus proteins, and so it is unlikely that the stimulati

59 that recognize the other agent at the whole-virus, protein, and peptide levels, consistent with bidi

60 protective antibody development is to direct virus protein antigens specifically to dendritic cells,

61 well as to cytomegalovirus and Epstein-Barr virus protein antigens, were also regulated by either or

62 The chicken anemia virus protein Apoptin induces apoptosis in the absence o

63 The chicken anemia virus protein Apoptin selectively induces apoptosis in t

64 such that only a subset of so-called latent virus proteins are expressed in virus infected tumours a

65 orly understood and it is not known if other virus proteins are required.

66 a 775-residue multifunctional herpes simplex virus protein associated with numerous functions related

67 ctivated NF-kappaB in concert with the Ebola virus proteins at least as effectively as wild-type BST2

68 ince SFs have been defined for all influenza virus proteins based on known structural, functional, an

69 The crystal structure of Epstein-Barr virus protein BCRF1, an analog of cellular interleukin-1

70 dies provided evidence that E10R, a vaccinia virus protein belonging to the ERV1/ALR family, has a re

71 e production and identify a new host protein-virus protein binding interface that could become a usef

72 A method to identify mutations of virus proteins by using protein mass mapping is describe

73 A virus protein called Tat plays a dual role in HIV infect

74 are independently targeted by a single mumps virus protein, called V, that assembles STAT-directed ub

75 Single-amino-acid mutations in Sindbis virus proteins can convert clinically silent encephaliti

76 ese findings provide evidence that mammalian virus proteins can inhibit RNA silencing, implicating th

77 2 protein, in the absence of other influenza virus proteins, can induce neuraminidase-specific antibo

78 Furthermore, we demonstrate that other virus proteins cannot substitute for this lack of functi

79 f a wide variety of analytes, including DNA, viruses, proteins, chemical vapors, and pesticides.

80 particles (VLPs) composed of an icosahedral virus protein coat encapsulating a functionalized spheri

81 acks icosahedral order characteristic of the virus protein coating (capsid).

82 ng known residues at the interface of a host-virus protein complex with a partially solved structure.

83 ecificity; the tomato and bean golden mosaic virus proteins complexed with each other.

84 Mutagenesis of the vaccinia virus protein confirmed that changing the electrostatic

85 The cowpox virus protein CPXV012 deprives the endoplasmic reticulum

86 By contrast, overexpression of the cowpox virus protein CrmA blocked apoptosis induced by engageme

87 The cowpox virus protein CrmA, a known inhibitor of ICE family prot

88 that NS4B regulates the function of host or virus proteins directly involved in HCV production.

89 In many instances, archaeal virus proteins display very low levels of sequence homol

90 ing of the role of VP40 and additional Ebola virus proteins during budding.

91 trating direct interactions between host and virus proteins during infection is a major goal and chal

92 s probably involve conformational changes of virus proteins during their association with the vector.

93 g the functional interactions of three Nipah virus proteins during viral assembly and particularly on

94 tion of pocket proteins with human papilloma virus protein E7 partially, but not completely, restored

95 of the interaction between the Epstein-Barr virus protein EBNA2 with BTD and explore the extent to w

96 parable to that produced by the Epstein-Barr virus protein EBNA2, a well-characterized, potent transa

97 ansferase p300 and an essential Epstein-Barr virus protein, EBNA3C, involved in regulation of viral a

98 Activation of NF-kappaB by the Ebola virus proteins either alone or together with BST2 requir

99 tation has been attributed to the absence of virus proteins either facilitating movement or counterac

100 ous studies indicated that exposure to Ebola virus proteins expressed from packaged Venezuelan equine

101 ly), plays important roles in the control of virus protein expression and that this knowledge could b

102 ight the possibility of increasing influenza virus protein expression and the need for a delicate bal

103 Virus protein expression evaluated by indirect immunoflu

104 ring effects of copyback DIs on nondefective virus protein expression.

105 We now demonstrate that the vaccinia virus protein F11, which localizes to the plasma membran

106 uch viral prosurvival protein is the fowlpox virus protein FPV039, which is a potent apoptosis inhibi

107 The structure of the human virus protein free in solution consists of an eight-stra

108 eoside triphosphatase; 20-kDa protein (p20); virus protein, genome linked (VPg); proteinase (Pro); po

109 hibition of RNA interference (RNAi) by plant virus proteins has been shown to enhance viral replicati

110 the eukaryotic homologue of a herpes simplex virus protein, has the crystallin motif of heat shock pr

111 the past 2 decades, several novel influenza virus proteins have been identified that modulate viral

112 ed similarities to interactions of other DNA virus proteins (human papillomavirus type 16 E6 and E7,

113 za A virus NS1 protein or the herpes simplex virus protein ICP34.5, rescues growth of influenza delNS

114 GADD34 has homology with the Herpes Simplex Virus protein, ICP34.5, which overcomes the protein synt

115 otein processing, we used the herpes simplex virus protein ICP47 to block peptide transport by TAP1/2

116 In contrast to the cytosolic herpes simplex virus protein ICP47, US6 interacts with TAP inside the e

117 ing and disruption of an essential influenza virus protein in the absence of genetic manipulation of

118 this mutant confirmed the presence of mutant virus protein in the transfected BHK cell lysate.

119 r epithelial infection 24 h after challenge, virus protein in the vaginal lumen 3 days after challeng

120 ficantly increased the concentration of shed virus protein in the vaginal lumen after challenge.

121 epithelium infected, concentrations of shed virus protein in the vaginal lumen, and illness scores,

122 ins with the membrane bilayer and with other virus proteins in an attempt to understand the role this

123 CTL, MC57 and JawsII process the same set of virus proteins in quantitatively different ways.

124 The role of individual virus proteins in the induction of these cytokine respon

125 pears to be brokered by additional influenza virus proteins, in this case M1.

126 id rafts without a requirement for any other virus protein, including the SH and G envelope proteins.

127 s allowed one to identify all 10 influenza A virus proteins, including low-abundance proteins like th

128 However, unlike the small DNA tumor virus proteins, individual HCMV IE proteins target diffe

129 this method to probe the release of specific virus proteins initiated by thermal stimulation, mimicki

130 result of loss of VF formation or important virus protein interactions.

131 The stacked disk aggregate of tobacco mosaic virus protein is an intriguing object due to its high de

132 despite the enhanced activity of the variola virus protein, its cofactor activity in the factor I-med

133 xpression the of the endogenous Epstein-Barr virus protein kinase (EBV PK, encoded by the BGLF4 gene)

134 This protein kinase, designated RVPK (rabies virus protein kinase), phosphorylates P protein (36 kDa)

135 ICP0 is a multifunctional herpes simplex virus protein known primarily as a promiscuous transacti

136 L2 is one of several vaccinia virus proteins known to be necessary for formation of cr

137 Recent evidence that a herpes virus protein lacking a classical secretory signal seque

138 of two transgenic rats expressing the simian virus protein large T antigen under the control of the a

139 The Epstein-Barr virus protein LMP1 is essential for transformation of re

140 Here, we used the Epstein-Barr virus protein LMP2A as a constitutively active BCR surro

141 s utilize caspases during replication to aid virus protein maturation, progeny release, or both.

142 Molluscum contagiosum virus proteins MC159 and MC160 and the equine herpesviru

143 th vaccinia virus, as determined by vaccinia virus protein microarray.

144 D4 and CD8 T-cell responses to several Ebola virus proteins, most notably the viral nucleoprotein.

145 Other substrates such as the Rous sarcoma virus protein NC are phosphorylated by gamma-PAK followi

146 of M1.IMPORTANCE The complement of influenza virus proteins necessary for the budding of progeny viri

147 the structural characteristics of influenza virus protein NS2 (NEP), which interacts with the nuclea

148 as ribosomes (proteins and RNA) or enveloped viruses (proteins, nucleic acids and lipids).

149 physical characterization of nanoparticles, viruses, proteins, nucleic acids, and other macromolecul

150 res coordinated binding of multiple host and virus proteins onto specific regions of the virus genome

151 there are no atomic structures for any CCHF virus proteins or for any Nairovirus proteins.

152 erns of molecular level modifications in the virus proteins or genome that lead to the inhibition of

153 propose a model in which certain influenza A virus proteins (or protein domains) exist as highly plas

154 vide evidence that the 2009 H1N1 influenza A virus protein PA-X plays a role in virus replication and

155 iated with the recently identified influenza virus protein PB1-F2 has been largely defined using mode

156 The influenza A virus protein PB1-F2 has been linked to the pathogenesis

157 pothesis that each isoform of herpes simplex virus proteins performs a specific function that may be

158 investigated the functions of two essential virus proteins, pp150 and pUL96, and determined the impa

159 This could restrict virus protein production and growth.

160 We tested virus protein production, virion composition and infecti

161 we identified several well-established host-virus protein-protein interactions, and confirmed that P

162 BRLF1 promoter in the context of the latent virus, protein-protein interactions, or both.

163 Five conserved vaccinia virus proteins, referred to as Viral Membrane Assembly P

164 vity, implying that K4L is the only vaccinia virus protein required for the nicking-joining enzymatic

165 81), in the absence of any other influenza B virus proteins resulted in the inhibition of IRF-3 nucle

166 or PLZF, as well as ZID, GAGA and a vaccinia virus protein, SalF17R, also interact with varying affin

167 types (VTs) identified in defined influenza virus protein sequence features (SFs) are used for genot

168 Hepatitis C virus proteins serve dual functions in replication and i

169 these A protein-dependent differences in the virus protein shell are not seen using crystallography,

170 play a role in the global arrangement of the virus protein shell.

171 unit vaccine composed of protective vaccinia virus proteins should avoid the complications arising fr

172 Four virus proteins similar to two human macrophage inflammat

173 For both primary and TCLA virus proteins, soluble stabilized trimers generated neu

174 Stabilization of virus protein structure and nucleic acid integrity is ch

175 association between some pairs of influenza virus proteins, such as M2 and NP, appears to be brokere

176 4(+) T cell responses to particular vaccinia virus proteins suggesting that CD4(+) T cell help is pre

177 Finally, we showed that influenza virus protein synthesis and viral mRNA levels decrease i

178 uring infection and also delayed and reduced virus protein synthesis from replicating RNA.

179 d is defective in the inhibition of host and virus protein synthesis showed an altered phosphorylatio

180 duced antiviral activity against influenza A virus protein synthesis was reduced 5- to 20-fold by sup

181 xhibits a general delay in the initiation of virus protein synthesis, but this is not due to a glycop

182 induction by measles virus (MV) and inhibits virus protein synthesis.

183 was found to bind to human immunodeficiency virus protein Tat, and this binding required the nucleol

184 ously proposed for the human T-cell leukemia virus protein Tax are discussed.

185 Among several vaccinia virus proteins tested, the H4L subunit, unique to the vi

186 VP39 is a bifunctional vaccinia virus protein that acts as both an mRNA cap-specific RNA

187 intestinal epithelial cells of M3, a herpes virus protein that binds and inhibits multiple chemokine

188 When the gene for ICP47, a herpes simplex virus protein that blocks the translocation of peptides

189 itutively expressed, phosphorylated vaccinia virus protein that has been implicated in viral DNA repl

190 PA-X is a recently identified influenza virus protein that is composed of the PA N-terminal 191

191 CAP activity is inhibited by CrmA, a cowpox virus protein that prevents host cell apoptosis.

192 This study provides the first evidence for a virus protein that targets IL-18BP and further validates

193 The virus proteins that mediate this centripetal transport a

194 evel of production of an essential influenza virus protein, the M2 ion channel protein.

195 that 10% were orthologs of Chilo iridescent virus proteins, the highest correspondence with any viru

196 roteins predicted that altering the vaccinia virus protein to contain the amino acids present in the

197 The ability of DNA tumor virus proteins to trigger apoptosis in mammalian cells i

198 agents comprise broad classes of pathogens (virus, protein toxins, bacterial spores, vegetative cell

199 the channel-forming trans-membrane domain of virus protein "u" (Vpu) of HIV-1 was determined by NMR s

200 The herpes simplex virus protein UL25 attaches to the external vertices of

201 ing the glycoprotein (GP) and matrix protein virus protein (VP)40, administered 1-3 d before Ebola vi

202 infection of human cells, the herpes simplex virus protein VP16 associates with the endogenous cell-p

203 the estrogen receptor or the herpes simplex virus protein VP16 generates transcriptional regulators

204 ith activation domains of the herpes simplex virus protein VP16 or the tomato Myb-like activator THM1

205 The activation domain from the herpes virus protein VP16 restored the ability of the bacteria

206 The herpes simplex virus protein VP22 is a major phosphoprotein of infected

207 We used this system to investigate the Ebola virus protein VP24, showing that, contrary to previous r

208 Vaccinia virus protein VP8 is a 25 kDa product of the L4R gene an

209 ry-like (VFL) structures and colocalize with virus proteins was characterized.

210 re translated sequences of 5 major influenza virus proteins, we assessed the specificity of CD4 T cel

211 ning glycosylation to the study of influenza virus proteins, we can better understand the effect that

212 titutions in this region of the Rous sarcoma virus protein were lethal due to a severe deficiency in

213 murine CD8(+) T-cell responses to six Ebola virus proteins were examined.

214 ain phage gene products and eukaryotic dsDNA virus proteins were noted, in particular, the primase/he

215 Bulk cultures revealed that a number of virus proteins were recognized in CTL assays.

216 When plasmids expressing individual Ebola virus proteins were transfected into Madin Darby canine

217 he S component and one with a herpes simplex virus protein with an apparent Mr of 43,000.

218 smallpox protein would result in a vaccinia virus protein with increased complement regulatory activ

219 by electron microscopy and immunolabeling of virus proteins with antibodies conjugated to gold beads.

220 V) replication depends on the interaction of virus proteins with host factors.

221 s several direct target genes of hepatitis B virus protein X (HBx), a viral co-factor.

222 ious studies have shown that the hepatitis B virus protein, X, activates all three classes of RNA pol

223 membrane cargo protein vesicular stomatitis virus protein-yellow fluorescent protein revealed that v