ライフサイエンスコーパス: visceromotor

1 Visceromotor and cardiovascular responses to colonic dis

2 ctivity in areas also controlling autonomic, visceromotor and skeletomotor responses.

3 ience makes sense, we propose that implicit (visceromotor and somatomotor) emotional processes are di

4 ed of two main classes, termed branchiomotor/visceromotor and somatomotor.

5 hood (8-10 weeks) at which point behavioral, visceromotor, and great splanchnic nerve responses to gr

6 aviors involving integration of respiratory, visceromotor, and somatomotor activity.

7 ced stimuli and to integrate the sensory and visceromotor aspects of emotional behavior.

8 In contrast, facial visceromotor axons selectively required SEMA3A/NRP1.

9 bx20 is expressed by migrating branchiomotor/visceromotor (BM/VM) neurons within the hindbrain during

10 corticotropin-releasing factor and in other visceromotor cell types of the paraventricular hypothala

11 and organ-specific regulation to descending visceromotor commands, whereas the output from M2 could

12 , and motor neurons of the somatic motor and visceromotor cranial nerve nuclei and the ventral horn o

13 Visceromotor electromyographic responses increased withi

14 If the visceromotor functions of M1 and M2 reflect their skelet

15 medial areas involved in cognitive (PL) and visceromotor (IL) functions.

16 o the paraventricular nucleus, including the visceromotor (infralimbic) cortex, median preoptic nucle

17 as 10, 14, and 25 are heavily connected to a visceromotor network.

18 Visceromotor neurons in the dorsal vagal and ambigual nu

19 mediate acute stress effects on hypothalamic visceromotor neurons, (2) comprise targets for corticost

20 motor neuron populations, branchiomotor and visceromotor neurons.

21 t of cranial nerves II, V, VII, and VIII and visceromotor nuclei of nerves VII, IX, and X as well as

22 nd ventral octavolateral nuclei, vagal lobe, visceromotor nuclei, and reticular formation, including

23 rolateral periaqueductal gray); hypothalamic visceromotor pattern generator network (five of six know

24 tricular and supraoptic nuclei, hypothalamic visceromotor pattern generator network), and thalamocort

25 entricular hypothalamic nuclei, hypothalamic visceromotor pattern generator network), orofaciopharyng

26 al gray, Barrington's nucleus), hypothalamic visceromotor pattern-generator network (five of six know

27 ontrol network, neuroendocrine, hypothalamic visceromotor pattern-generator network, thalamocortical

28 ed to identify and characterize hypothalamic visceromotor populations responsive to acute and chronic

29 colorectal distention (CRD) in the rat: the visceromotor reflex (vmr) and L6-S1 dorsal horn neuron a

30 Pain behavior was assessed by visceromotor reflex activity in response to noxious stim

31 l SR140333 injection diminished the enhanced visceromotor reflex to colorectal distention at day 11 i

32 ion of the NK(1)R antagonist SR140333 on the visceromotor reflex to colorectal distention in both gro

33 les in facilitating the acoustic startle and visceromotor reflexes.

34 Thus, a map of visceromotor representation appears to be embedded withi

35 biological stimulus with its somatomotor and visceromotor representation.

36 recordings of pACC neurons and examined the visceromotor response (VMR) to colorectal distention (CR

37 ral nociceptive behavior was measured as the visceromotor response (VMR) to colorectal distention (CR

38 A visceromotor response (VMR) to graded colorectal distens

39 The nociceptive response (visceromotor response [VMR]) to CRD was recorded in norm

40 effects of fractalkine were assessed on the visceromotor response in rats exposed to minocycline or

41 There was a significant enhancement of the visceromotor response in zymosan-, but not saline-treate

42 y afferents significantly blunted the evoked visceromotor response to bladder distension and led to s

43 The visceromotor response to colon distension (15-60 mm Hg)

44 The visceromotor response to colorectal distension was measu

45 Visceral nociception, modeled by the visceromotor response to colorectal distension, and colo

46 avoidance stress significantly increased the visceromotor response to colorectal distention (20-80 mm

47 The visceromotor response to colorectal distention at baseli

48 nt of visceral hypersensitivity by measuring visceromotor response to colorectal distention in rats.

49 PK signaling is responsible for the enhanced visceromotor response to colorectal distention in STZ-D

50 tive colon as demonstrated by an exaggerated visceromotor response to colorectal distention in the F3

51 produces hyperalgesia (i.e., facilitates the visceromotor response to colorectal distention) mediated

52 r saline was given intracolonically, and the visceromotor response to noxious colorectal distention (

53 An exaggerated visceromotor response, referred pain to mechanical stimu

54 ngs of the abdominal musculature, termed the visceromotor response.

55 Visceromotor responses (VMR) to CRD were recorded in rat

56 There was a highly significant difference in visceromotor responses between the phases of the estrous

57 The visceromotor responses measured during the phase of proe

58 of these cell groups in driving hypothalamic visceromotor responses to a given stressor, and (iii) de

59 Intraluminal Cat-S amplified visceromotor responses to colorectal distension and indu

60 Hypersensitive visceromotor responses to colorectal distention in STZ-D

61 Visceromotor responses to distension were significantly

62 In awake rats, visceromotor responses to intragastric acid are quantifi

63 Gastritis and gastric ulcers enhanced the visceromotor responses to intragastric acid.

64 was measured in guinea pig distal colon, and visceromotor responses were recorded in a rat model of c

65 gions that control reproductive behavior and visceromotor responses, confirming a similar analysis by

66 al area of the BST - cell groups involved in visceromotor responses.

67 areas that comprise this network represent a visceromotor system, distinct from the sensory related "