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1 genation to maintain cellular metabolism and visual function.
2 ensity and FAZ area appear to correlate with visual function.
3 liculus (SC) but not significant recovery of visual function.
4 Electroretinogram was used to evaluate visual function.
5 es to the experience-dependent regulation of visual function.
6 ifications can be reversed to restore normal visual function.
7 for synaptic transmission underlying normal visual function.
8 vessels, as well as decreased inner retinal visual function.
9 nment maximally, and regenerates lenses with visual function.
10 nd assessed the cardinal features of BBS and visual function.
11 opment of interventions to retain or restore visual function.
12 mes significantly predicted change in GO-QOL visual function.
13 ave already been lost and thereby to restore visual function.
14 eads to progressive and irreversible loss of visual function.
15 t Development III and tests of executive and visual function.
16 stinct objects and surfaces is a fundamental visual function.
17 toreceptor renewal and recycling to preserve visual function.
18 , retinal morphology correlated tightly with visual function.
19 uired for photoreceptor cell maintenance and visual function.
20 xposure to methamphetamine did not influence visual function.
21 perative visual acuity, and patient-reported visual function.
22 cone photoreceptors, and partially restores visual function.
23 tion is the most relevant imaging marker for visual function.
24 retinal degeneration accompanied by loss of visual function.
25 was independently associated with a reduced visual function.
26 d, preference values declined with worsening visual function.
27 nt, many early-onset cblC patients have poor visual function.
28 is known about its potential consequences on visual function.
29 HDACi can rescue cone photoreceptor-mediated visual function.
30 al postnatal period when mice acquire mature visual function.
31 strocytes play a protective role, preserving visual function.
32 cell loss, overall leading to a reduction of visual function.
33 ith early disease and considerable remaining visual function.
34 lar phenotype that is associated with normal visual function.
35 hy accompanied by significant alterations in visual function.
36 obe than primary regions of sensorimotor and visual function.
37 this reactivity is beneficial or harmful for visual function.
38 ssion and identifying parameters that affect visual function.
39 hosphorylation for its phototransduction and visual function.
40 nocular tests or binocular approximations of visual function.
41 d in those with relatively preserved central visual function.
42 lpful for determining and predicting overall visual function.
43 try, which may be advantageous for restoring visual function.
44 ematic study on both monocular and binocular visual functions.
45 s over different times courses for different visual functions.
46 en retinal signaling and therefore preserved visual functions.
47 enabling factor for generalization to other visual functions.
48 be too slow to support a role in high-level visual functions.
49 ections, which presumably underlie different visual functions.
50 these signals support their contribution to visual functions.
51 een LWS cone topographies suggests differing visual functions.
52 e emerging as increasingly popular models of visual functions.
53 e ocular surface, but on quality of life and visual functioning.
54 means of approximating a patient's reported visual functioning.
55 xyflavone hydrate, significantly rescued dye visual function ( 58 fold increase in OKR, p < 0.001, 3
56 e Local Eye (4.68 [6.62] vs 3.07 [5.60]) and Visual Function (8.44 [11.45] vs 4.42 [8.94]; P < .05) S
57 atistical significance for the evaluation of visual function (91.2% to 96.1%, P < .02) and target int
58 a accommodative IOL effectively restores the visual function, accommodation, and contrast sensitivity
59 tive remodeling, ganglion cell survival, and visual function after experimental glaucoma and nerve cr
60 syndrome type IIa have an earlier decline of visual function and a higher cumulative risk of visual i
64 so contributes to RPE cell death and loss of visual function and could affect the pathology of dry AM
68 subgroup of patients present with reasonable visual function and long-term survival of photoreceptors
72 visual pathways may set important limits on visual function and show greater vulnerability to abnorm
74 orrelated with performance-based measures of visual function and subjective assessment of vision-rela
76 To assess the effect of adalimumab on the visual functioning and quality of life in patients with
77 implant the device in the patient, comparing visual functions and high-resolution oculography before
78 ocular retinoid levels concomitant with low visual function, and a rapid disorganization of RPE cell
79 lar histopathology, accumulation of albumin, visual function, and biochemical and physiological abnor
80 ventions, change in visual acuity, change in visual function, and duration of follow-up are reported.
81 l acuity, refractive error, patient-reported visual function, and early and late complications of sur
82 stigated the relationship among vector dose, visual function, and electroretinography (ERG) findings.
84 in retinal and choroidal anatomic features, visual function, and risk factor profile exist in unilat
86 Robust and sensitive imaging biomarkers for visual function are an unmet medical need in the managem
87 ns provide converging evidence that gains in visual function are minimal and deficits are most severe
88 To determine the association between QOL and visual function as measured by 24-2 and 10-2 VFs in pati
89 ness, and relatedness of adverse events) and visual function, as measured by 3 computer-based, object
90 ness, and relatedness of adverse events) and visual function, as measured by 3 computer-based, object
91 retinal vascular autoregulation testing and visual function assessment using frequency doubling tech
93 med hallucinations (n=21) had reduced higher visual function at baseline, cortical thinning in pariet
96 producing rapid drainage with restoration of visual function avoiding more invasive procedures and en
100 nks to minimize information loss and improve visual functioning but we have little understanding of h
101 ination and a slower initial rate of loss of visual function by electroretinography, compared with ey
103 prescribed), participants were assigned to a visual function category ranging from bilateral normal t
104 t statistically significant deterioration in visual function compared with both RGPL and spectacle we
105 elationship between anatomic progression and visual function decline, including microperimetry, low-l
106 o be caused by specific cognitive and higher visual function deficits and patients who develop such s
111 for up to 2.2 years for safety outcomes and visual function endpoints including Goldmann visual fiel
113 specific DA receptor pathways could improve visual function (evaluated with optokinetic tracking res
114 of implant system performance tests, subject visual function evaluation, and implant-retina interface
115 nt barriers remain in our ability to restore visual function following traumatic injury or disease-in
116 meaningful improvements in patient-reported visual functioning for patients with noninfectious inter
118 ssessed safety, immune response, retinal and visual function, functional vision, and activation of th
123 on outside native photoreceptors and restore visual function in a mouse model of advanced retinal deg
124 sufficient to rescue retinal cell death and visual function in a vertebrate model of inherited blind
128 ditory field responsible for the supranormal visual function in CDCs, the auditory dorsal zone (DZ).
129 h in adults, little is known about trends in visual function in childhood and its association with so
130 is the most frequently performed measure of visual function in clinical practice and most people wor
132 ross-sectional survey of ocular biometry and visual function in healthy eyes across the life span of
133 eptor death, resulting in life-long improved visual function in IL-1 receptor antagonist-treated OIR
134 because research into the factors that limit visual function in infants has found surprisingly mature
136 tributed to the current social patterning in visual function in older adults in the United Kingdom.
137 tches, is a promising approach for restoring visual function in patients suffering from degenerative
139 y is well tolerated, and can rapidly improve visual function in some patients with Leber congenital a
142 dhesion (VMA) resolution on patient-reported visual function in symptomatic VMA/vitreomacular tractio
145 hemispherectomy in childhood may have better visual function in the eye ipsilateral to the side of th
147 le to undergo chemotherapy and radiotherapy, visual function in the fellow eye can be preserved.
148 To investigate the distribution of childhood visual function in the United Kingdom and associations w
149 harder to detect incremental improvement in visual function in those with early disease and consider
151 ignificantly poorer objective and subjective visual functioning in all metrics examined (P < 0.05), w
152 rostheses are promising tools for recovering visual functions in blind patients but, unfortunately, w
153 a DA precursor, improved overall retinal and visual functions in diabetic mice and acute treatment wi
154 e photopigment melanopsin supports reflexive visual functions in people, such as pupil constriction a
156 cell disease may have direct consequences on visual function, including in children, even when visual
157 n-based signals also influence image-forming visual function, including light adaptation, but the mec
158 nopsin and mediate several non-image-forming visual functions, including circadian photoentrainment a
159 2 (99.3%) who had gradable fundus images and Visual Function Index (VF-11) data available were includ
160 and effect sizes for results of the modified Visual Function Index were calculated for 3 categories o
163 ical considerations led to the proposal that visual function is organized in separate processing stre
164 ration have lasting influence on retinal and visual function, likely through developmental programmin
165 ograde melanopsin-based signaling influences visual function locally within the retina, a notion that
168 eripheral changes in AMD and their impact on visual function may contribute to understanding AMD path
169 ogies and circuitry, suggesting that complex visual functions may also be restored.SIGNIFICANCE STATE
170 the number of RGCs in the retina as well as visual function measured by PERG steadily decreased over
171 , vision satisfaction, and dysphotopsia) and visual function measures (near, intermediate, and distan
172 e performed to evaluate associations between visual function measures and retinal layer thicknesses.
176 than 20/100 reported the largest decline in visual function (median difference, -21.0; 95% CI, -40.5
177 Both methods can be useful to follow the visual function of diabetic patients and should be used
181 There was a significant improvement in the visual function of the patients based on the NEI VFQ-25
183 aque monkey was constructed to reconcile the visual functions of V1 with anatomical data on its LGN i
184 ns of the 2 symptom subscales (Local Eye and Visual Function) of the SHPC-18 with visual field severi
185 abeculectomy did not differ significantly in visual function or local eye symptoms from those that un
186 e of the hospitals reported patient reported visual functioning or vision-related quality of life out
188 s was not detected with respect to the other visual function outcomes and all anatomic outcomes asses
190 Microperimetry detected subtle changes in visual function over a 12-month period in eyes with inte
194 e no placebo-controlled trials have assessed visual function preservation, and the observation period
196 All subjects had National Eye Institute Visual Function Questionnaire (NEI VFQ)-25 performed ann
197 ell as on the National Eye Institute 25-Item Visual Function Questionnaire (NEI VFQ-25) (beta = -3.4,
198 sessed by the 25-item National Eye Institute Visual Function Questionnaire (NEI VFQ-25) at baseline a
200 s were evaluated with National Eye Institute Visual Function Questionnaire (NEI VFQ-25), FDT, and sta
201 on, measured with the National Eye Institute Visual Function Questionnaire (NEI VFQ-25), in patients
204 Quality of Life Instrument (NEI-RQL-42), NEI Visual Function Questionnaire (NEI-VFQ), and the Ocular
205 completed the 25-item National Eye Institute Visual Function Questionnaire (NEI-VFQ-25) during EDIC y
207 severity and 25-item National Eye Institute Visual Function Questionnaire (NEI-VFQ25) subscales.
208 Mean best-corrected visual acuity (BCVA) and Visual Function Questionnaire (VFQ) scores significantly
210 as assessed using the National Eye Institute Visual Function Questionnaire (VFQ-25), mental state wit
211 ormal eye health: the National Eye Institute Visual Function Questionnaire - 25 (NEI VFQ-25) where it
212 L was assessed by the National Eye Institute Visual Function Questionnaire 25 (NEI-VFQ-25) and a modi
214 e, nonsurgical FTMH closure, vitrectomy, and Visual Function Questionnaire 25 (VFQ-25) outcomes.
215 [VAQ] and the 25-item National Eye Institute Visual Function Questionnaire [NEI VFQ-25]) and binocula
216 domain of the 25-item National Eye Institute Visual Function Questionnaire and superior or inferior h
218 The Activity Inventory (AI), an adaptive visual function questionnaire, was administered to measu
219 items from the National Eye Institute (NEI) Visual Function Questionnaire-25 (VFQ-25) at baseline th
220 and an existing vision-related QOL tool, the Visual Function Questionnaire-25 (VFQ-25) was administer
221 asures, including the National Eye Institute Visual Function Questionnaire-25 and the Short Form-36 p
223 tly, 6 items from the National Eye Institute Visual Function Questionnaire-25 were used to develop th
226 on Questionnaire-25 were used to develop the Visual Function Questionnaire-Utility Index health state
228 ht of 15,625 possible health states from the Visual Function Questionnaire-Utility Index were valued
229 A theta severity score was calculated for Visual Function Questionnaire-Utility Index-defined heal
231 responses to the Veterans Affairs Low Vision Visual Functioning Questionnaire (higher scores indicate
232 responses to the Veterans Affairs Low Vision Visual Functioning Questionnaire (higher scores indicate
233 jects had the 25-item National Eye Institute Visual Functioning Questionnaire (NEI VFQ-25) performed
235 atients completed the National Eye Institute Visual Functioning Questionnaire (NEI-VFQ) 30 to 90 days
236 Data from the 25-item National Eye Institute Visual Functioning Questionnaire (NEI-VFQ-25) for the fi
238 quality of life (the National Eye Institute Visual Functioning Questionnaire 25 and the Modified Gla
239 ed in this study: the National Eye Institute Visual Functioning Questionnaire-25 (NEI VFQ-25), Glauco
240 sed using the 25-item National Eye Institute Visual Functioning Questionnaire; best spectacle-correct
242 S Medicare beneficiaries using self-reported visual function, reinforcing the value of identifying pa
243 In the DCCT/EDIC cohort, patient-reported visual function remains high in both treatment groups, c
246 early all traditional subscales (P < 0.001), visual function subscale (-3.85 vs. -2.91 logits, P < 0.
247 ncy were 0.83 and 0.89 for the Local Eye and Visual Function subscales, respectively, and remained st
248 sition is essential to unraveling high-level visual functions such as eye movement planning, coordina
250 The ipRGCs regulate other nonimage-forming visual functions such as the pupillary light reflex, mas
256 ts had logMAR visual acuity measurement, FDT visual function testing, autorefraction, A-scan biometry
257 ly better with the system on than off on all visual function tests and functional vision assessments.
259 To assess the relationship of binocular visual function tests with binocular approximations usin
260 in response to the development of sensitive visual function tests, structural markers such as optica
264 therefore a more comprehensive assessment of visual function than acuity, which only determines the s
265 of RPGR ORF15 mutations tended to have worse visual function than carriers of RPGR exon 1 through 14
266 d a clinically meaningful worsening in their visual function than the placebo group (15.0% vs 24.3%,
267 c deficit in binocular rivalry [11], a basic visual function that is thought to rely on the balance o
268 tients with such damage retain some residual visual function that must rely on an alternative pathway
269 e and clinically significant improvements in visual function, thereby making this program a frontrunn
270 trials demonstrated temporary improvement in visual function, this approach has yet to achieve sustai
272 Products, Inc, Sylmar, CA) in restoring some visual function to subjects completely blind from RP.
274 phic changes as well as associations between visual function, ultra-widefield FA-derived metrics, and
275 retina has the remarkable ability to support visual function under conditions of limited illumination
276 at large opsin repertoires serve to optimize visual function under variable light environments by dif
277 emonstrate the efficacy of HITI in improving visual function using a rat model of the retinal degener
280 ere was no association with visual symptoms, visual functioning, visual acuity, refractive error, vis
281 hy, and fundus autofluorescence imaging, and visual function was assessed by electroretinography.
283 t of increasing severity, all-cause impaired visual function was associated with adverse social outco
291 ocular approximations to represent binocular visual function were assessed with Pearson's correlation
292 The binocular approximations of binocular visual function were better or worse eye, average eye, b
299 mild TBI frequently leads to disruptions in visual functioning, while moderate or severe TBI often c
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