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1 small neuronal subsets provided a Golgi-like vital stain.
2 k-up time, Schirmer test, and ocular surface vital staining.
4 regions were fully contractile and viable by vital staining and microscopy but demonstrated 25% reduc
7 munofluorescence, cytochemistry, fluorescent vital stains, and fluid-phase markers in conjunction wit
12 }-6-aminocaproyl-d-erythro-sphingos ine as a vital stain for chlamydiae proved to be a sensitive meth
14 n, or beta-COP (a specific golgi protein) or vital stains for endoplasmic reticulum (ER) and golgi.
15 orofluorescein diacetate (a vacuolar luminal vital stain), had a pronounced shift in red/green fluore
18 escape, we followed acidification using the vital stain LysoTracker red and acquisition of the proto
20 endplates (pi-junctions) were identified by vital staining of lateral plantar nerve (LPN) and sural
21 with high synaptic density as ascertained by vital staining of recycling synaptic vesicles, and were
23 , peeling of the internal limiting membrane, vital staining of the internal limiting membrane with in
27 This approach makes use of a lineage-tracing vital stain that is retained through cell generations an
31 nature of this compartment was confirmed by vital staining with a pH sensitive dye, LysoSensor yello
34 ata on new techniques and technology such as vital staining with methylene blue and protoporphyrin fl
38 duces muscle membrane damage, as revealed by vital staining (with Evans blue dye) of the diaphragm an
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