ライフサイエンスコーパス: vitamin

1 n as the most active form of this lipophilic vitamin.

2 ticularly lipids, amino acids, and cofactors/vitamins.

3 Vitamin A (all-trans retinol) plays critical roles in ma

4 rh(-/-) mice had approximately 2-fold higher vitamin A (all-trans-retinol (all-trans-ROL)) in the neu

5 ere used for the preparation of milk protein-Vitamin A (Vit A) complexes.

6 The vitamin A activity of alpha-carotene-containing foods is

7 itamin A) and alpha-retinol (with negligible vitamin A activity).

8 rich source of beta-carotene, a precursor of vitamin A and a potential tool for fighting vitamin A de

9 ratios for incident tuberculosis disease by vitamin A and carotenoids levels, controlling for other

10 ght to inform the interpretation of iron and vitamin A biomarkers (ferritin, serum transferrin recept

11 ltiple proximal risk factors (e.g., iron and vitamin A deficiencies, inflammation, malaria, and body

12 eficiency (ferritin <15 ng/mL or 32 pmol/L), vitamin A deficiency (retinol-binding protein <14.7 mug/

13 vitamin A and a potential tool for fighting vitamin A deficiency (VAD) in developing countries.

14 Vitamin A deficiency impairs epithelial integrity, incre

15 Vitamin A deficiency strongly predicted the risk of inci

16 nd cardiac edema, phenotypes associated with vitamin A deficiency.

17 nd its relative contribution to postprandial vitamin A in humans after consumption of raw carrots.Hea

18 in a cohort of children who were exposed to vitamin A in utero or at birth.The aim of this study was

19 TB was observed for the highest quartile of vitamin A intake (hazard ratio = 0.71, 95% confidence in

20 Vitamin A is susceptible to light and heat and thus requ

21 GI GVHD was increased in patients with vitamin A levels below the median (38% vs 12.4% at 100 d

22 f antenatal and newborn supplementation with vitamin A on the cognitive function of children at 8 y o

23 e combined influence of different factors on vitamin A retention and the oxidative status of wheat fl

24 nce global knowledge with regard to iron and vitamin A status assessment in women and preschool child

25 Stability can be improved by binding of vitamin A to milk protein.

26 Smoking and vitamin A use was not associated significantly with base

27 A carotenoid, is cleaved to produce retinol (vitamin A) and alpha-retinol (with negligible vitamin A

28 tinoic acid, an active metabolite of dietary vitamin A, acts as a ligand for nuclear receptor transcr

29 etinoic acid (RA), a bioactive derivative of vitamin A, exhibits diverse effects on gene transcriptio

30 ga-6 lipids and, for the first time, that of vitamin A, naturally present in cod liver oil.

31 he CD103(+)CD11b(+) DC subset expressing the vitamin A-converting enzyme retinaldehyde dehydrogenase

32 Mice maintained on a vitamin A-free diet lose HSCs and show a disrupted re-en

33 by antenatal or newborn supplementation with vitamin A.

34 orbed more of one carotenoid and fat-soluble vitamin also tended to absorb more of the others.

35 le interactions of arsenic metabolism with B vitamins and AS3MT variants on diabetes risk.

36 flow for the determination of 14 fat-soluble vitamins and carotenoids in a single run.

37 Serum concentration biomarkers of several vitamins and carotenoids performed similarly to establis

38 cycle one leads to a depletion of essential vitamins and cofactors and decreased de novo synthesis o

39 The combination of probiotics, vitamins, and biological agents with AIT is highlighting

40 Poor vitamin B-12 (cobalamin) status is widespread in South A

41 40 compared with -0.036 +/- 0.544 mumol/L in vitamin B-12 and placebo groups, respectively; P < 0.001

42 Inflammatory processes, including vitamin B-6 catabolism, could explain such findings.We i

43 estigated long-term clinical implications of vitamin B-6 deficiency in stable outpatient RTRs.In a lo

44 s were observed for graft failure (P = 0.18).Vitamin B-6 deficiency is common in RTRs and does not se

45 Vitamin B-6 deficiency was defined as PLP <20 nmol/L, an

46 PLP influences long-term outcome.We compared vitamin B-6 intake and circulating PLP concentrations of

47 uch findings.We investigated 3 biomarkers of vitamin B-6 status in relation to CRC risk.This was a pr

48 n B-6-deficient RTRs have a worse functional vitamin B-6 status than do healthy controls and vitamin

49 :XA (HK:XA), an inverse marker of functional vitamin B-6 status, and PA:(PLP + pyridoxal) (PAr), a ma

50 nd oxidative stress and an inverse marker of vitamin B-6 status.Plasma PLP concentrations were associ

51 ntrols (47% male; age 54 +/- 11 y), baseline vitamin B-6 was measured as plasma PLP by high-performan

52 bumin, calcium, iron, ferritin, cholesterol, vitamin B-6, and vitamin D (data collected from 2009 thr

53 increment: 1.50; 95% CI: 1.21, 1.86) in RTRs.Vitamin B-6-deficient RTRs have a worse functional vitam

54 amin B-6 status than do healthy controls and vitamin B-6-sufficient RTRs.

55 ntrations of biomarkers in choline pathways, vitamins B-12 and A, and essential fatty acids.A randomi

56 onas rostrata requires an external supply of vitamin B12 (cobalamin) for growth, which it can obtain

57 ls for investigating the biological roles of vitamin B12 .

58 ntary experimental approaches, the impact of vitamin B12 availability and methotrexate exposure on Da

59 Vitamin B12 bound to haptocorrin (holoHC) remained highl

60 Clinical vitamin B12 deficiency can result in megaloblastic anemi

61 Given that vitamin B12 deficiency causes an optic neuropathy throug

62 Vitamin B12 depletion decreased de novo dTMP biosynthesi

63 Significant amounts of vitamin B12 in plants were detected in Hippophae rhamnoi

64 hese individuals exhibit reduced circulating vitamin B12 levels.

65 By increasing the adsorption duration, vitamin B12 molecules gradually diffused in between mont

66 causal role in associations between maternal vitamin B12 status and offspring's cognition.

67 de scavenger, we tested whether cobalamin, a vitamin B12 vitamer, would be neuroprotective in vitro a

68 ains of life require the cofactor cobalamin (vitamin B12), which is produced only by a subset of bact

69 not routinely raise their intake of calcium, vitamin B12, or magnesium beyond the Recommended Dietary

70 een proposed for such different processes as Vitamin B12-dependent biodegradation and zerovalent meta

71 ral density, serum creatinine, magnesium, or vitamin B12.

72 T cells that recognize intermediates of the vitamin B2 biosynthetic pathway presented by the monomor

73 A vitamin B2 product (80% feed grade) imported from China

74 ccounted for a significant fraction of total vitamin B3 content.

75 hesized de novo in five enzymatic steps with vitamin B5 as the starting metabolite, phosphorylated by

76 tudy provides robust evidence that increased vitamin B6 catabolism is independently associated with a

77 tablishes an association between an index of vitamin B6 levels with lung cancer risk.

78 a impacts microbiota composition, decreasing vitamin B6 production and promoting sustained colonizati

79 Here, using the example of vitamin B6, we investigate the regulation of biosynthesi

80 some essential nutrients (Inulin, DHA & EPA, vitamins B6, K1, and D3) as enhancers of calcium bioavai

81 ion within a working day for the analysis of vitamin B9 (folate) in infant formula and adult/pediatri

82 xypeptidase activity releases glutamate from vitamin B9 and other glutamated substrates, which activa

83 kgo biloba, folic acid alone or with other B vitamins, beta-carotene, vitamin C, vitamin D plus calci

84 wn to distinguish rats from humans including vitamin C and bile acid synthesis pathways.

85 , titratable acidity, sugars, organic acids, vitamin C and E, carotenoids, polyphenolics and volatile

86 The higher TPC, AOC, vitamin C content and rehydration ability were obtained

87 ith distinct differences in anthocyanins and vitamin C contents, on human intestinal Caco-2 cells exp

88 intrinsic and extrinsic factors that impact vitamin C degradation in a real food matrix.

89 pH influenced strongly vitamin C degradation in citrate-phosphate buffer but no

90 g nutrient recommendations and estimation of vitamin C intake among infants and young children.

91 ly determined local dysregulation of dietary vitamin C or antioxidants transport contributes to IBD d

92 nstrated in a randomized clinical trial that vitamin C supplementation to pregnant smokers can lessen

93 one or with other B vitamins, beta-carotene, vitamin C, vitamin D plus calcium, and multivitamins or

94 owever, the NO to HNO conversion mediated by vitamins C, E, and aromatic alcohols has been recently s

95 loroacetic acid to release the protein-bound vitamin, centrifugation, and mixing of the supernatant w

96 ge absorption of carotenoids and fat-soluble vitamins could be largely predicted by the soybean oil e

97 wk of gestation) consumed a single amount of vitamin D (511 IU/d from diet and a cholecalciferol supp

98 sociation between the intake of calcium with vitamin D (CaD) and fracture risk.Data from 5823 white p

99 ron, ferritin, cholesterol, vitamin B-6, and vitamin D (data collected from 2009 through 2010) did no

100 Vitamin D (VD) supplementation has been shown to reverse

101 al cell proliferation, migration, and on the vitamin D activating enzyme CYP27B1 (produces 1,25(OH)2D

102 Low levels of vitamin D also appear to be a risk factor for IBD.

103 Translational research in trials combining Vitamin D and aspirin have begun as part of such investi

104 ion of high-density lipoprotein cholesterol, vitamin D and C-reactive protein, and less central obesi

105 To investigate whether common variants in 7 vitamin D and calcium pathway genes (VDR, GC, DHCR7, CYP

106 Vitamin D and placebo groups did not differ in change in

107 These findings point to a role of vitamin D and the VDR in modulating autophagy and cell d

108 iations may be the most feasible approach to vitamin D assessment over time.

109 In contrast, vitamin D at 20 nM concentration suppressed the expressi

110 and non-renal cells and has implications for vitamin D biology in multiple sclerosis and perhaps othe

111 irected towards the potential association of vitamin D concentrations and prostate cancer, but little

112 elucidate the underlying determinants of low vitamin D concentrations in AA populations.

113 olism and its impact on maternal circulating vitamin D concentrations in humans.This study sought to

114 was the only category in which all measured vitamin D concentrations were below the declared value (

115 time outdoors and sunlight exposure), serum vitamin D concentrations, and vitamin D pathway genetic

116 Compared to the declared values, the vitamin D content ranged from 50% to 153% for fortified

117 h an approximate 20% increase in the odds of vitamin D deficiency (</=20 ng/mL) [odds ratio (95% CI):

118 ified subgroup analysis in participants with vitamin D deficiency (baseline deseasonalized 25-hydroxy

119 We hypothesized that vitamin D deficiency alters TRAIL protein levels in huma

120 eters, thus suggesting that the link between vitamin D deficiency and cardiovascular disease may be a

121 lts were seen for participants with baseline vitamin D deficiency and for secondary outcomes.

122 Vitamin D deficiency dropped from 64% to 20%.

123 The high prevalence of vitamin D deficiency in African Americans (AAs) may be a

124 These vGWAS results together suggest that vitamin D deficiency is potentially causal of sero-negat

125 gnificant downregulation with UVB.Correcting vitamin D deficiency with either oral vitamin D3 or UVB

126 mozygous p.Ser267Phe in SLC10A1 are prone to vitamin D deficiency, deviated sex hormones and blood li

127 nondiabetic patients with stage 3-4 CKD and vitamin D deficiency, vitamin D supplementation may impr

128 is C (HCV) infection have high prevalence of vitamin D deficiency.

129 phy and progression to heart failure in both vitamin D deficient and normal mice without inducing sig

130 ng/mL, with 1270 participants (24.9%) being vitamin D deficient.

131 To support these studies, a specialized vitamin D food composition dataset, based on EuroFIR sta

132 nding interests in the potential benefits of vitamin D for preventing breast cancer recurrence and mo

133 A systematic vitamin D fortification of fluid milk products and fat s

134 tor efficacy and safety of ongoing strategic vitamin D fortification.

135 hase of an interventional trial of high-dose vitamin D given during pregnancy.

136 in D receptor (VDR), whose activating ligand vitamin D has been proposed as a modifiable factor in mu

137 to advance the understanding of the role of Vitamin D in HL.

138 no convincing evidence for a direct role of vitamin D in myopia risk.

139 e general European population and implicates vitamin D in the etiology of multiple sclerosis.

140 ets (WD) high in fat and scarce in fiber and vitamin D increase risks of colorectal cancer.

141 ow exposure to sunlight increase the risk of vitamin D insufficiency in children.The aim of the study

142 on and lactose tolerance, had higher dietary vitamin D intake and higher measured 25(OH)D concentrati

143 his study was to evaluate the association of vitamin D intake and serum levels with fracture risk in

144 Plasma 25(OH)D, dietary vitamin D intake, and vitamin D-synthesis GRS were not a

145 000 to date, demonstrated high prevalence of vitamin D intakes below the EFSA Adequate Intake (AI) (<

146 kin.Children with fair and dark skin require vitamin D intakes of 20 and 28 mug/d, respectively, to m

147 Vitamin D is an environmental and dietary agent with kno

148 r evidence on the direct association between Vitamin D levels and the clinical course of HL needs to

149 We aimed to test whether genetically lowered vitamin D levels were associated with risk of asthma, at

150 though asymptomatic, all individuals had low vitamin D levels.

151 Here I have developed a hypothesis that vitamin D may act to control the rate of ageing.

152 observed between placental mRNA abundance of vitamin D metabolic components and circulating vitamin D

153 placental messenger RNA (mRNA) abundance of vitamin D metabolic pathway components were quantified.

154 etic variants located near genes involved in vitamin D metabolism and calcium and renal phosphate tra

155 Little is known about placental vitamin D metabolism and its impact on maternal circulat

156 vance the current understanding of placental vitamin D metabolism and its role in modulating maternal

157 ations nor variants in genes associated with vitamin D metabolism were found.

158 Despite its importance in vitamin D metabolism, the molecular mechanisms underlyin

159 tamin D metabolic components and circulating vitamin D metabolites [i.e., LDL-related protein 2 (LRP2

160 Concentrations of placental and blood vitamin D metabolites and placental messenger RNA (mRNA)

161 its role in modulating maternal circulating vitamin D metabolites during pregnancy.Nested within a f

162 levels, despite normal circulating levels of vitamin D metabolites.

163 d here provide epidemiological evidence that Vitamin D might play a protective role in HL.

164 m of the study was to evaluate the amount of vitamin D needed to ascertain that most children >4 y of

165 ants were randomized to receive 2000 IU/d of vitamin D oral supplementation (high-dose group) vs 354

166 supplements (and sun exposure in the case of vitamin D outcomes).

167 hether genetic polymorphisms of genes in the vitamin D pathway are associated with treatment response

168 posure), serum vitamin D concentrations, and vitamin D pathway genetic variants, adjusting for years

169 s of the effects of genetic variation in the vitamin D pathway on response to vitamin D supplementati

170 fy whether monitoring and supplementation of vitamin D play roles in cardiovascular protection.

171 other B vitamins, beta-carotene, vitamin C, vitamin D plus calcium, and multivitamins or multi-ingre

172 Strikingly, dietary vitamin D prevented these calcium-triggered tumorigenic

173 Here, we document that the vitamin D receptor (VDR) acts as a master transcriptiona

174 Recently, we found that vitamin D receptor (VDR) enhanced Claudin-2 expression i

175 Vitamin D receptor (VDR) knockdown partly abolished MART

176 Vitamin D receptor (VDR) mutations in humans and mice ca

177 on in binding of a transcription factor, the vitamin D receptor (VDR), whose activating ligand vitami

178 erted via signaling mechanisms involving the vitamin D receptor (VDR).

179 d the individual and combined effects of the vitamin D receptor activator paricalcitol (PARI) and die

180 n genes encoding PD-L1 and PD-L2 through the vitamin D receptor, a ligand-regulated transcription fac

181 key locus responsible for skin color, with a vitamin D receptor-binding interval.

182 However, whether winter supplementation of vitamin D reduces the risk among children is unknown.

183 We identified and characterized vitamin D response elements (VDREs) located in both gene

184 persisted when adjusting for baseline 25(OH) vitamin D serum level.

185 oading efficiency show promise for improving vitamin D stability during dry storage.

186 e-assembled casein micelles, and to evaluate vitamin D stability of dry formulations during ambient o

187 rt no association between antenatal maternal vitamin D status and childhood fractures.

188 sion of TRAIL in human milk as a function of vitamin D status in mothers remains unknown.

189 the effect of milk fortification on iron and vitamin D status in these children are scarce.

190 is restricted to patients with low baseline vitamin D status is unknown.

191 xes, there are moderate associations between vitamin D status measured in prepuberty, adolescence, an

192 therefore be a preferential form to optimize vitamin D status within the general population.

193 The best biomarker of vitamin D status, 25hydroxyvitamin D3 (25(OH)D3), howeve

194 ds was started in 2003 in Finland to improve vitamin D status.

195 lar disease (CVD) among individuals with low vitamin D status.

196 risk, high serum 25(OH)D levels and regular vitamin D supplement use were associated with lower rate

197 ic assessment), calcium supplementation, and vitamin D supplementation (OR, 0.12 [95% CI, 0.03 to 0.5

198 tion in the vitamin D pathway on response to vitamin D supplementation are lacking.

199 s result does not support the use of monthly vitamin D supplementation for this purpose.

200 do not support the routine use of high-dose vitamin D supplementation in children for the prevention

201 andomized, controlled, double-blind trial of vitamin D supplementation in pregnancy (400, 2000, or 40

202 with stage 3-4 CKD and vitamin D deficiency, vitamin D supplementation may improve vascular function.

203 INTERPRETATION: High-dose bolus vitamin D supplementation of 100 000 IU colecalciferol m

204 ese findings do not support a dose effect of vitamin D supplementation on bone health and suggest tha

205 of randomised controlled trials showed that vitamin D supplementation reduces the rate of asthma exa

206 cebo-controlled trial to investigate whether vitamin D supplementation that is provided in a sufficie

207 in the future that can be used for providing vitamin D supplementation to patients with fat malabsorp

208 However, meta-analyses of vitamin D supplementation trials have failed to show cle

209 ts were genotyped for functional variants on vitamin D synthetic pathway including GC (rs4588, rs7041

210 Currently the majority of vitamin D testing is performed in large-scale commercial

211 roxyvitamin D [25(OH)D] levels, supplemental vitamin D use, and breast cancer incidence over the subs

212 No trials compared calcium or vitamin D versus placebo.

213 ng observational evidence on associations of vitamin D with lower risk of breast cancer morbidity and

214 elow the EFSA Adequate Intake (AI) (<15mug/d vitamin D) in adults across Europe.

215 and mineral bone disease caused by disturbed vitamin D, calcium, and phosphate metabolism.

216 CYP24A1, the primary inactivating enzyme for vitamin D, is often overexpressed in human cancers, pote

217 f species-specific regulation of immunity by vitamin D, the VDREs are present in primate genes, but n

218 been limited by short duration, low doses of vitamin D, variability in participants' vitamin D-defici

219 s).We compared concentrations of 25(OH)D and vitamin D-binding protein (VDBP) in AA and EA women and

220 A women and investigated determinants of the vitamin D-biomarker concentrations in both populations.W

221 s of vitamin D, variability in participants' vitamin D-deficiency status, and the use of surrogate me

222 rovided in a sufficient dose and duration to vitamin D-deficient individuals would improve insulin se

223 Vitamin D-enriched mushrooms extracts exert a synergisti

224 omodulatory effect of oral administration of vitamin D-enriched mushrooms extracts on high-fat diet (

225 lasma 25(OH)D, dietary vitamin D intake, and vitamin D-synthesis GRS were not associated with any cog

226 ntioned genetic associations are modified by vitamin D.

227 or effects of calcitriol, the active form of vitamin D.

228 erived tolDCs modulated by dexamethasone and vitamin D2 from 31 T1D patients with optimal glycemic co

229 Daily oral supplementation with vitamin D3 (1000 IU) or calcium carbonate (1200 mg eleme

230 e also analyzed along with plasma 25-hydroxy vitamin D3 (25D) detection.

231 5110 participants were randomized to receive vitamin D3 (n = 2558) or placebo (n = 2552).

232 a(ep-/-)) or a topical application of active vitamin D3 (VD3) and/or all-trans retinoic acid (RA) on

233 rs was 0.042 (95% CI, 0.032 to 0.056) in the vitamin D3 + calcium group and 0.060 (95% CI, 0.048 to 0

234 irmed in 109 participants, 45 (3.89%) in the vitamin D3 + calcium group and 64 (5.58%) in the placebo

235 D level of 32.8 ng/mL, supplementation with vitamin D3 and calcium compared with placebo did not res

236 To determine if dietary supplementation with vitamin D3 and calcium reduces the risk of cancer among

237 lation of immune pathway signaling with oral vitamin D3 but significant downregulation with UVB.Corre

238 endent associations between myopia and serum vitamin D3 concentrations nor variants in genes associat

239 tuned to different wavelengths for producing vitamin D3 in human skin.

240 Vitamin D3 is generated secondary to exposure to ultravi

241 Cholecalciferol or vitamin D3 is known to isomerise under various condition

242 ls-Alder reaction confirmed the formation of vitamin D3 isomerisation products.

243 participants with significantly higher serum vitamin D3 levels after treatment (P = 0.007) demonstrat

244 In contrast, participants with lower serum vitamin D3 levels had significant expression of proinfla

245 Vitamin D3 may therefore be a preferential form to optim

246 B1, CYP24A1, and CASR) modify the effects of vitamin D3 or calcium supplementation on colorectal aden

247 ecting vitamin D deficiency with either oral vitamin D3 or UVB does not improve the lipid profile.

248 h 4400 IU/d (n = 26) or 400 IU/d (n = 25) of vitamin D3 were analyzed for immune cell composition by

249 dose of 200 000 IU (5.0 mg) colecalciferol (vitamin D3) followed by monthly 100 000 IU (2.5 mg) cole

250 For this study, model drug (vitamin D3, VD3)-loaded PLGA nano- and microparticles (N

251 ed at investigating the behaviour of Trolox, vitamin E analogue, in presence of macromolecule-bound a

252 ants were randomized to vitamin E, selenium, vitamin E and selenium, or placebo.

253 sed prostate cancer risk in the Selenium and Vitamin E Cancer Prevention Trial (SELECT) through unkno

254 e foundation for improving tocochromanol and vitamin E content in seeds of maize and other major cere

255 The values of vitamin E contents were expressed as mass equivalent of

256 ic potential of tocotrienol, a member of the vitamin E family of compounds with potent in vitro anti-

257 Moderate-strength evidence showed that vitamin E had no benefit on cognition.

258 lectroanalytical method for determination of vitamin E in the form of the total content of tocopherol

259 Vitamin E increased prostate cancer risk in the Selenium

260 rehensive assessment of natural variation in vitamin E levels in maize establishes the foundation for

261 al findings from SELECT and demonstrate that Vitamin E promotes tumorigenesis in the early stages of

262 This study evaluated the effect of dietary vitamin E supplementation (1000mg of DL-alpha-tocopheryl

263 Vitamin E supplementation improves liver histology in pa

264 Vitamin E supplementation reduced lipid oxidation (P</=0

265 toring the normal hollow lumen morphology in Vitamin E treated organoids.

266 for the relative proportions of tocopherol (vitamin E) forms in seeds, and the validity of the most

267 ry sensitive method for the determination of vitamin E, being comparable to reversed-phase high perfo

268 Participants were randomized to vitamin E, selenium, vitamin E and selenium, or placebo.

269 proteins, total dietary fiber, minerals and vitamin E.

270 that maternal periconceptional use of the B vitamin folic acid can prevent many NTDs.

271 This vitamin has a reported capacity to protect different pla

272 romoted a renewal of interest in the role of vitamins in governing phytoplankton dynamics, and illumi

273 senic metabolism with HOMA2-IR differed by B-vitamin intake and AS3MT genetics variants.

274 Phase III trials comparing non-vitamin K antagonist oral anticoagulants (NOACs) with wa

275 including 907 patients with AF treated with vitamin K antagonists (3,865 patient-years), to assess C

276 Bleeding risk with vitamin K antagonists (VKAs) is closely related to the q

277 coagulated with a combination of aspirin and vitamin K antagonists.

278 ctions of vIL-6 with the ER membrane protein vitamin K epoxide reductase complex subunit 1 variant 2

279 suggests novel roles for bacterially derived vitamin K forms known as menaquinones in health and dise

280 ials will assess the risk and benefit of non-vitamin K oral anticoagulants among patients at high ris

281 Whether vitamin K supplementation can prevent and/or treat calci

282 Vitamin K supplementation increased in ALGS after PEBD (

283 genital/acquired FX deficiency or after anti-vitamin K treatment) were characterized by concomitantly

284 To examine the association between dietary vitamin K1 intake and the risk of incident cataracts in

285 A novel method of marking isomers of vitamin K2MK-7 in dietary supplements was developed and

286 the content of biologically active all trans vitamin K2MK-7 was below its declared content.

287 surface methodology to optimize fat-soluble vitamin loading in re-assembled casein micelles, and to

288 es in important biochemical pathways such as vitamin metabolism, the citric acid (TCA) cycle, and ami

289 minated amazing versatility in phytoplankton vitamin metabolism.

290 stent across the carotenoids and fat-soluble vitamins (P < 0.0001).Within the linear range, the avera

291 67 67 References 67 B vitamins play essential roles in central metabolism.

292 rowth from the pregnancy-specific ration and vitamin supplements beyond those of the preconception ra

293 7 metabolites and 55 foods, food groups, and vitamin supplements on the basis of the 2015 Dietary Gui

294 impact of the pregnancy-specific ration and vitamin supplements.

295 expected to include mechanisms to sustain B vitamin supply under demanding circumstances.

296 d their solubility improved further within a vitamin-tea beverage matrix (>85%).

297 al variation for carotenoids and fat-soluble vitamins.The objective was to model the dose-response re

298 sture, total phenolics, reducing sugar and B vitamins (thiamine, riboflavin, and niacin) content of s

299 boflavin biosynthesis genes, known/predicted vitamin uptake transporters and the rbkR gene.

300 e following food fortification and increased vitamin use.