ライフサイエンスコーパス: vitamin A

1 espect to those introducing higher intake of vitamin A.

2 se in the prevalence of inadequate intake of vitamin A.

3 and brain, Stra6 expression was regulated by vitamin A.

4 Concurrently, fat and lung rely on dietary vitamin A.

5 ILCs were reduced in the absence of dietary vitamin A.

6 STGD and RP, especially for daily intake of vitamin A.

7 noic acid (atRA) is the active metabolite of vitamin A.

8 and 15/56 (26.8%) showed low daily intake of vitamin A.

9 ary disorders plausibly modulated by dietary vitamin A.

10 by antenatal or newborn supplementation with vitamin A.

11 ts either as antioxidants or as precursor of vitamin A.

12 observed for intakes of other carotenoids or vitamin A.

13 s have led to an increase in deficiencies of vitamin A.

14 patients with measles includes provision of vitamin A.

15 Optimal loading of vitamin A (1.46-1.48mg/100mgcasein) was found at 9.7mM p

16 body mass index (27 +/- 5 vs 24 +/- 5 kg/m), Vitamin A (1.7 +/- 0.6 vs 1.2 +/- 0.4 umol/L), and Vitam

17 atio 1:1) to receive either one oral dose of vitamin A (50,000 IU) or placebo immediately after recru

18 complexes were further evaluated for unbound vitamin A, ability of milk protein to bind vitamin A and

19 Fat is found to be synergistic for vitamin A absorption.

20 critical role for STRA6 in the transport of vitamin A across blood-tissue barriers in the eyes, brai

21 Transport of vitamin A across blood-tissue barriers is facilitated by

22 e fruit was found to contain the potentially vitamin A-active keto-carotenoids sapotexanthin and cryp

23 ta support our hypothesis that low levels of vitamin A actively promote GI GVHD and are not simply a

24 Canthaxanthin is a carotenoid that lacks pro-vitamin A activity but is known to have antioxidant acti

25 The vitamin A activity of alpha-carotene-containing foods is

26 itamin A) and alpha-retinol (with negligible vitamin A activity).

27 tinoic acid, an active metabolite of dietary vitamin A, acts as a ligand for nuclear receptor transcr

28 linked in the presence of A2E (adduct of two vitamin A aldehyde and ethanolamine) photodegradation pr

29 central 15-15' double bond to form retinal (vitamin A aldehyde).

30 ine and converts provitamin A carotenoids to vitamin A-aldehyde.

31 Vitamin A (all-trans retinol) plays critical roles in ma

32 rh(-/-) mice had approximately 2-fold higher vitamin A (all-trans-retinol (all-trans-ROL)) in the neu

33 We show that vitamin A (all-trans-retinol) (VA) is required both for

34 ltransferase (LRAT) is the enzyme that traps vitamin A (all-trans-retinol) from the circulation and p

35 Vitamin A also significantly decreased the uptake of the

36 f the recommended dietary allowance (RDA) of vitamin A amongst children aged 3-10years.

37 ts, with 11,474 randomly assigned to receive vitamin A and 11,481 to receive placebo.

38 Serum concentrations of vitamin A and 25-hydroxyvitamin D were significantly red

39 days after supplementation; 70 (0.6%) in the vitamin A and 52 (0.5%) in the placebo group (risk ratio

40 rich source of beta-carotene, a precursor of vitamin A and a potential tool for fighting vitamin A de

41 kers by ameliorating oxidative stress, while vitamin A and beta-carotene may have additional antimyco

42 ratios for incident tuberculosis disease by vitamin A and carotenoids levels, controlling for other

43 We assessed the impact of baseline levels of vitamin A and carotenoids on tuberculosis disease risk.

44 Most common deficiencies concerned vitamin A and D, iron, and zinc.

45 After 2 months, the vitamin A and D3 losses reached 60-68% and 61-68%, respe

46 The determining factors of vitamin A and D3 losses were (in decreasing order) the s

47 Micronutrient deficiencies such as those of vitamin A and iron affect a third of the world's populat

48 ds into apocarotenoids, including retinoids (vitamin A and its derivatives).

49 characterize better the association between vitamin A and malaria infection in different settings.

50 d vitamin A, ability of milk protein to bind vitamin A and solubility of protein and vitamin A as aff

51 ice to diets sufficient and insufficient for vitamin A and used heterozygous siblings as controls.

52 to determine concentrations of supplementary vitamin A and vitamin E esters and beta-carotene in infa

53 sources contributing to excessive intakes of vitamin A and zinc among infants and toddlers may need f

54 excessive intakes (greater than the ULs) of vitamin A and zinc, respectively.

55 specific differences were observed regarding vitamins A and B12, zinc, and potassium.

56 ments and dietary intake of foods containing vitamins A and C.

57 Food fortification is implemented to address vitamins A and D deficiencies in numerous countries.

58 g these parameters is thus essential to make vitamins A and D fortification in oils more efficient.

59 al fat, cholesterol, fatty acids, vitamin C, vitamins A and D, and 21 mineral elements (including tot

60 a at the University of Wisconsin, working on vitamins A and D, respectively.

61 ted in the serum levels of nutrients, mainly vitamins A and E (p < 0.05).

62 lises selective dual-channel fluoresence for vitamins A and E and visible absorbance for beta-caroten

63 This study evaluated the intake of vitamins A and E of infants from 'ready-to-feed' foods a

64 A carotenoid, is cleaved to produce retinol (vitamin A) and alpha-retinol (with negligible vitamin A

65 e report that retinoic acid (RA) or retinol (vitamin A) and ascorbate (vitamin C) act as modulators o

66 me countries could become newly deficient in vitamin A, and an additional 2.2 billion (1.2-2.5) alrea

67 ion, all infants received sufficient fat and vitamin A, and most consumed enough daily energy (79%),

68 Iron, vitamin A, anemia, malaria, and anthropometric measures

69 Low and deficient levels of vitamin A are common in low- and middle-income countries

70 Most effects of vitamin A are exerted by its metabolite, retinoic acid (

71 bind vitamin A and solubility of protein and vitamin A as affected by complexation.

72 at LAL is required for efficient nutritional vitamin A availability.

73 roperties of encapsulated functional lipids--vitamin A, beta-carotene and omega-3 fish oil--on the st

74 order to increase polyunsaturated lipid and vitamin A bioaccessibility and avoid formation of toxic

75 aim was to estimate iron, zinc, protein and vitamin A bioavailability from individual diets, and inv

76 ect refines approaches to interpret iron and vitamin A biomarker values in settings of inflammation a

77 ght to inform the interpretation of iron and vitamin A biomarkers (ferritin, serum transferrin recept

78 n STH infection intensity and limitations of vitamin A biomarkers.

79 Vitamin A bound to retinol binding protein 4 (RBP4) cons

80 The liver is the main storage organ of vitamin A, but activation of the retinoic acid receptors

81 this process, conversion of beta-carotene to vitamin A by BCO1 induces via retinoid signaling the exp

82 e receptor STRA6 mediates cellular uptake of vitamin A by recognizing RBP-retinol to trigger release

83 -vitamin A) impedes the dimerization rate of vitamin A--by approximately fivefold for the vitamin A d

84 ship between intake of antioxidant vitamins (vitamins A, C, D, and E) and individual carotenoids (alp

85 etermine whether slowing the dimerization of vitamin A can prevent vision loss caused by Stargardt di

86 focus on those derived from the diet such as vitamin A, can have a direct or indirect deterministic i

87 e measured directly to accurately assess the vitamin A capacity of alpha-carotene-containing foods.

88 y retinoic acid 6), is the RBP4 receptor and vitamin A channel; however, the role of STRA6 in vitamin

89 relations between bone health and different vitamin A compounds.

90 In controls, this regulation reduced vitamin A consumption when the dietary supply was limite

91 extendable to whole milk powders where total vitamin A content data can be calculated by summing the

92 acid (RA), an essential active metabolite of vitamin A, controls numerous physiological processes.

93 acids (SCFAs) induced the expression of the vitamin A-converting enzyme RALDH1 in intestinal epithel

94 he CD103(+)CD11b(+) DC subset expressing the vitamin A-converting enzyme retinaldehyde dehydrogenase

95 helial cells correlated with the activity of vitamin A-converting enzymes in mesenteric lymph node de

96 nding that several key micronutrients (e.g., vitamin A, copper, manganese, and zinc) support iron's f

97 the intestinal level between the fat-soluble vitamins A, D, E and K (FSVs) are poorly documented.

98 ltiple proximal risk factors (e.g., iron and vitamin A deficiencies, inflammation, malaria, and body

99 Vitamin A deficiency (A(-)) remains a public health conc

100 eficiency (ferritin <15 ng/mL or 32 pmol/L), vitamin A deficiency (retinol-binding protein <14.7 mug/

101 Vitamin A deficiency (VAD) and soil-transmitted helminth

102 vitamin A and a potential tool for fighting vitamin A deficiency (VAD) in developing countries.

103 The accurate estimation of the prevalence of vitamin A deficiency (VAD) is important in planning and

104 a is facing a double burden of malnutrition: vitamin A deficiency (VAD) prevails, whereas the nutriti

105 tes that 190 million preschool children have vitamin A deficiency (VAD).

106 ta-carotene accumulation that will alleviate vitamin A deficiency among people who rely on sorghum as

107 P flour could be used for the eradication of vitamin A deficiency as they were found to meet 29 and 8

108 imed to estimate trends in the prevalence of vitamin A deficiency between 1991 and 2013 and its morta

109 Vitamin A deficiency continues to be a major public heal

110 Vitamin A deficiency impairs epithelial integrity, incre

111 00) deaths from measles were attributable to vitamin A deficiency in 2013, which accounted for 1.7% (

112 n in improving vitamin A status and reducing vitamin A deficiency in children.

113 Vitamin A deficiency is a risk factor for blindness and

114 the relative risks (RRs) for the effects of vitamin A deficiency on mortality from measles and diarr

115 or use in developing country settings, where vitamin A deficiency remains a major public health probl

116 Vitamin A deficiency remains a nutritional concern in su

117 Vitamin A deficiency remains prevalent in south Asia and

118 e for both prevalence and absolute burden of vitamin A deficiency should be used to reconsider, and p

119 Vitamin A deficiency strongly predicted the risk of inci

120 possible confounders, we found that baseline vitamin A deficiency was associated with a 10-fold incre

121 In comparison, trachoma, onchocerciasis, vitamin A deficiency, and refraction and accommodation d

122 and pathophysiological conditions, including vitamin A deficiency, cardiopulmonary diseases, and hypo

123 archical model to estimate the prevalence of vitamin A deficiency, defined as a serum retinol concent

124 ealth strategies to prevent ocular injuries, vitamin A deficiency, perinatal infections and retinopat

125 with a plant-food-based approach to address vitamin A deficiency, reports the analysis of total caro

126 nd cardiac edema, phenotypes associated with vitamin A deficiency.

127 be used as food-based supplements to reduce vitamin A deficiency.

128 ld deaths to estimate deaths attributable to vitamin A deficiency.

129 low-income and middle-income countries were vitamin A deficient.

130 spermatogonial differentiation identical to vitamin A-deficient (VAD) mice; (2) the blockage of sper

131 s in the small intestine of mice raised on a vitamin A-deficient diet.

132 Vitamin A-deficient mice infected with S. mansoni had di

133 In the brains of vitamin A-deficient mice, both Stra6L and Stra6S levels

134 d development of enteric lymphoid tissues in vitamin A-deficient mice.

135 Vitamin A degradation was high and occurred rapidly: mor

136 vely, the dominant-negative proteins disrupt vitamin A delivery from wild-type proteins within the fe

137 All-trans-retinoic acid (ATRA) is an active vitamin A derivative known to modulate a number of physi

138 e photoreceptors, contains 11-cis-retinal (a vitamin A derivative) and light isomerizes it to all-tra

139 s high, because it assesses translocation of vitamin A derivatives across the retinal pigment epithel

140 rebri syndrome group included Down syndrome, vitamin A derivatives, and growth hormone.

141 eroxy- and 4-hydroxy-2-alkenals, and several vitamin A derived metabolites were generated.

142 er demonstrate that replacing vitamin A with vitamin A deuterated at the carbon 20 position (C20-D3-v

143 lated with vitamin A levels, indicating that vitamin A did not protect against mucosal injury.

144 vitamin A--by approximately fivefold for the vitamin A dimer A2E--and subsequent lipofuscinogenesis a

145 in A is a clinically amiable tool to inhibit vitamin A dimerization, which can be used to determine w

146 lesterol accumulation in the RPE, induced by vitamin A dimers or oxidized LDL, inhibits these defense

147 abolic derivatives, and synthetic analogs of vitamin A embody an effective CTCL therapy with over thr

148 naldehyde is further metabolized to retinol (vitamin A), esterified and packaged into triacylglycerol

149 calculated by summing the innate long-chain vitamin A esters with the added esters.

150 etinoic acid (RA), a bioactive derivative of vitamin A, exhibits diverse effects on gene transcriptio

151 atter would meet nearly a half of the RDA of vitamin A for pregnant and lactating women.

152 beta-Carotene is an important source of vitamin A for the mammalian embryo, which depends on its

153 Mice maintained on a vitamin A-free diet lose HSCs and show a disrupted re-en

154 d the total daily supply of provitamin A and vitamin A from diet and supplements was equivalent to 22

155 ss the efficacy of oral supplementation with vitamin A given to infants in the first 3 days of life t

156 lementation deaths to 6 months of age in the vitamin A group (mortality risk 24.5 in 1000 supplemente

157 have a bulging fontanelle; 32 (0.3%) in the vitamin A group and 21 (0.2%) in the placebo group (RR 1

158 Vitamin A has essential but largely unexplained roles in

159 Retinoic acids, which are metabolites of vitamin A, have been shown to be involved in multiple T

160 Thus, STRA6 is critical for vitamin A homeostasis and the adaption of this process t

161 Vitamin A homeostasis is critical to normal cellular fun

162 between diet and immunity and indicates that vitamin A homeostasis must be tightly controlled by ISX

163 min A channel; however, the role of STRA6 in vitamin A homeostasis remains to be defined in vivo We s

164 ed in the prevalence of inadequate intake of vitamin A if rice biofortified with beta-carotene were c

165 deuterated at the carbon 20 position (C20-D3-vitamin A) impedes the dimerization rate of vitamin A--b

166 the stability of flavonoids, carotenoids and vitamin A in broccoli and cauliflower inflorescences gro

167 ogether, our data show an important role for vitamin A in controlling innate lymphoid cells and, cons

168 nd its relative contribution to postprandial vitamin A in humans after consumption of raw carrots.Hea

169 Estimation of unbound vitamin A in milk protein-Vit A complexes was carried ou

170 testinal homeostasis, are also influenced by vitamin A in the small intestines.

171 in a cohort of children who were exposed to vitamin A in utero or at birth.The aim of this study was

172 0% substitution) decreased the prevalence of vitamin A inadequacy from baseline 78% in women and 71%

173 tinoic acid (atRA), the active metabolite of vitamin A, induces gene transcription via binding to nuc

174 In the multivariate analysis, inflammation, vitamin A insufficiency, socioeconomic status, and age w

175 Interestingly, STGD patients with low vitamin A intake (<600 microg RAE/day) showed significan

176 TB was observed for the highest quartile of vitamin A intake (hazard ratio = 0.71, 95% confidence in

177 Software was used to estimate usual rice and vitamin A intake for the simulation analyses.

178 ndividual level to 1) determine the rice and vitamin A intake in nonpregnant, nonlactating women of r

179 acture rates worldwide and a relatively high vitamin A intake.

180 uacy ratio calculations: calcium, vitamin C, vitamin A, iron, fiber, and protein.

181 Data suggest that C20-D3-vitamin A is a clinically amiable tool to inhibit vitami

182 Vitamin A is a multifunctional vitamin implicated in a w

183 Vitamin A is a potent regulator of adaptive immunity.

184 key at the intestinal border, where dietary vitamin A is first absorbed.

185 Since vitamin A is food-derived, tissue-specific uptake and st

186 play a role in the retention of retinol when vitamin A is low.

187 iviral treatment for measles; treatment with vitamin A is recommended for younger children to decreas

188 Here we show that vitamin A is required for the phenotypic conversion of i

189 Vitamin A is susceptible to light and heat and thus requ

190 hydrophobic retinol, the main form in which vitamin A is transported.

191 -trans retinoic acid (ATRA), a derivative of vitamin A, is a common component in cosmetics and commer

192 , the main biologically active metabolite of vitamin A, is known to promote gut homing of lymphocytes

193 uations where an essential nutrient, such as vitamin A, is limited, ILC2 sustain their function and s

194 Furthermore, the vitamins (A, K and B group) and mineral contents (N, P,

195 isease risk with each decreasing quartile of vitamin A level.

196 well documented association between abnormal vitamin A levels and renal malformations in humans, and

197 m infections were increased in patients with vitamin A levels below the median (24% vs 8% at 1 year,

198 GI GVHD was increased in patients with vitamin A levels below the median (38% vs 12.4% at 100 d

199 er transplant was increased in children with vitamin A levels below the median (r = -0.34, P = .03).

200 We determined vitamin A levels of the eyes, brain, and testis, which h

201 Free vitamin A levels were measured in plasma at day 30 postt

202 We hypothesized that higher vitamin A levels would reduce the risk of graft-versus-h

203 mucosal injury, but was not correlated with vitamin A levels, indicating that vitamin A did not prot

204 Vitamin A loss is accompanied by stellate cell activatio

205 nol dehydrogenase 10 (Rdh10), which perturbs Vitamin A metabolism and retinoid signaling, exhibit ful

206 gests the interesting possibility that local vitamin A metabolism could also be a mediator of stimulu

207 ut also further suggests a potential role of vitamin A metabolism in terminal differentiation of the

208 hanism by which postnatal sensory-stimulated vitamin A metabolism modifies the generation of spatiall

209 We also show that vitamin A metabolism, as measured by ALDH activity, was

210 ne through this important branching point of vitamin A metabolism.

211 The vitamin A metabolite all-trans retinoic acid (ATRA) indu

212 ed at identifying the mechanisms whereby the vitamin A metabolite all-trans retinoic acid (RA) promot

213 In the intestines, the vitamin A metabolite retinoic acid (RA) is produced at s

214 The distributed patterns of vitamin A-metabolizing enzymes in the nervous system sug

215 nic responses were dependent on induction of vitamin A-metabolizing enzymes via the beta-catenin/T-ce

216 ga-6 lipids and, for the first time, that of vitamin A, naturally present in cod liver oil.

217 her maternal or newborn supplementation with vitamin A on intelligence, memory, and motor function.

218 f antenatal and newborn supplementation with vitamin A on the cognitive function of children at 8 y o

219 Our results highlight the impact of dietary vitamin A on the regulation of cell-cycle-mediated stem

220 infants to receive one dose of 50,000 IU of vitamin A or placebo in the first 3 days after birth.

221 Neither dietary nor supplemental intake of vitamin A or vitamin C nor supplemental intake of vitami

222 k)] who participated in a placebo-controlled vitamin A- or beta-carotene-supplementation trial was do

223 tive was to increase the water solubility of Vitamin A Palmitate and its stability against different

224 The stability of Vitamin A Palmitate in the complexes towards temperature

225 All results showed a notably increase of Vitamin A Palmitate water solubility and stability in fr

226 tion of water-soluble inclusion complexes of Vitamin A Palmitate with beta-cyclodextrins, without the

227 otene contributing 12-35% of newly converted vitamin A (predicted contribution = 25.5%).

228 Vitamin A promotes development of mucosal tolerance and

229 found that after a period of depletion, pro-vitamin A (PVA) carotenoids were preferentially diverted

230 ntribution of OFSP-wheat composite breads to vitamin A requirements were evaluated.

231 we report that ATRA, an active metabolite of vitamin A, restores mechanical quiescence in PSCs via a

232 e combined influence of different factors on vitamin A retention and the oxidative status of wheat fl

233 Vitamin A retention was related to the extent of oxidati

234 torage, and the factors that mostly affected vitamin A retention were the storage duration, the type

235 For example, the active metabolite of vitamin A, retinoic acid (RA), has been described to mai

236 s study, the estimated total daily intake of vitamin A (retinol equivalents) and vitamin E (alpha-toc

237 Gestational vitamin A (retinol) deficiency poses a risk for ocular b

238 Cellular uptake of vitamin A (retinol) is essential for many biological fun

239 sociated with minimal increases in preformed vitamin A (retinol).

240 simultaneously quantifying iron (ferritin), vitamin A (retinol-binding protein), and inflammation (C

241 foods, whereas the legumes, dairy, eggs, and vitamin A-rich fruit and vegetable food groups were each

242 urce foods, fruits, and vegetables including vitamin A-rich ones was higher in the adequate than in t

243 ewly absorbed alpha-carotene to postprandial vitamin A should not be estimated but should be measured

244 [ferritin or soluble transferrin receptor or vitamin A status (retinol-binding protein or retinol)] a

245 alaria infection, and biomarkers of iron and vitamin A status and compare adjustment approaches, and

246 validated against biochemical indicators of vitamin A status and hypoxemia.

247 knowledge, the associations between maternal vitamin A status and offspring bone development have not

248 ug beta-carotene/g) consumption in improving vitamin A status and reducing vitamin A deficiency in ch

249 nce global knowledge with regard to iron and vitamin A status assessment in women and preschool child

250 t it must be assessed in surveys of iron and vitamin A status for valid interpretation of micronutrie

251 entrations may have the potential to improve vitamin A status in maize-consuming populations.

252 e evaluated several supporting biomarkers of vitamin A status in Zambian children to determine whethe

253 Improvements in vitamin A status might decrease susceptibility to enteri

254 rkers were investigated to better define the vitamin A status of these children.

255 tion groups were comparable at baseline, and vitamin A status was better than anticipated (12.1% defi

256 often used in population surveys to measure vitamin A status, but its interpretation is challenging

257 ation in Kenyan schoolchildren with marginal vitamin A status.

258 n be an efficacious, new approach to improve vitamin A status.

259 Vitamin A-sufficient (A(+)) mice survived and cleared th

260 Neonatal vitamin A supplementation (NVAS) is currently being cons

261 Vitamin A supplementation among individuals at high risk

262 RP-2)-related antigenemia (n=6121) following vitamin A supplementation and standard vaccination.

263 is trial do not support inclusion of newborn vitamin A supplementation as a child survival strategy i

264 Neonatal vitamin A supplementation did not result in any immediat

265 controlled trials of newborn (age 1-3 days) vitamin A supplementation have been inconclusive.

266 Vitamin A supplementation might improve transplant outco

267 was no evidence of a differential effect for vitamin A supplementation on mortality by sex; risk rati

268 find any evidence for a beneficial effect of vitamin A supplementation on mortality in infants at 6 m

269 The impact of early vitamin A supplementation on neurodevelopmental function

270 the list of priority countries for high-dose vitamin A supplementation such that a country's priority

271 a global policy recommendation for neonatal vitamin A supplementation.

272 oling effect sizes from randomised trials of vitamin A supplementation.

273 e models, suggest childhood immunization and vitamin A supplements may confer protection against mala

274 e requirement would have further declines in vitamin A supplies.

275 tinoic acid, the biologically active form of vitamin A that acts as a transcription factor ligand to

276 otein 1 (CRBP1) is essential for trafficking vitamin A through the cytoplasm.

277 ardt disease, we show that the propensity of vitamin A to dimerize is responsible for triggering the

278 Stability can be improved by binding of vitamin A to milk protein.

279 nvironment acquire the ability to metabolize vitamin A to produce retinoic acid (RA), which drives re

280 hway also governs the metabolism of retinol (vitamin A) to its transcriptionally active metabolite, r

281 e protects the labile retinoid moiety during vitamin A transport.

282 n partly be attributed to alterations in the vitamin A-transport proteins retinol-binding protein 4 (

283 Smoking and vitamin A use was not associated significantly with base

284 (SR) concentration is a common indicator of vitamin A (VA) status, but SR is homeostatically control

285 ttle is known about the impact of obesity on vitamin A (VA)[retinol (ROL)], a nutrient that regulates

286 Vitamin A (VA; retinol) supplementation is used to reduc

287 ere used for the preparation of milk protein-Vitamin A (Vit A) complexes.

288 itate has been found to be the most abundant vitamin A vitamer, but retinyl oleate is the prevalent f

289 The fully adjusted ORs for vitamin A, vitamin B-6, vitamin B-12, folic acid, vitami

290 on estimates, we estimated the production of vitamin A, vitamin B12, folate, iron, zinc, calcium, cal

291 Mean adequacies of vitamin A, vitamin C, folate, calcium, iron, and zinc an

292 y examined the relation between carotenoids, vitamin A, vitamin C, vitamin E, and folate intake and r

293 ts consisting of beta-carotene (precursor to vitamin A), vitamins C and E and the mineral magnesium (

294 t 6 months (26 deaths per 1000 livebirths in vitamin A vs 24 deaths per 1000 livebirths in placebo gr

295 Phenotypic rescue by C20-D3-vitamin A was also observed noninvasively by quantitativ

296 pendence of lymphoid tissue inducer cells on vitamin A was furthermore illustrated by impaired develo

297 Children receiving vitamin A were less likely to present with parasitemia (

298 Data further demonstrate that replacing vitamin A with vitamin A deuterated at the carbon 20 pos

299 of 24 (25%) STGD patients a daily intake of vitamin A within the recommended range while 14/24 (58.3

300 MNPs (containing iron, vitamin A, zinc, and 11 other micronutrients) and other