ライフサイエンスコーパス: vitamin B

1 Vitamin B(6) and vitamin B(1)(2) intake was not associated with EG/SEG ri

2 ltered after adjustment for vitamin B(6) and vitamin B(1)(2) intake.

3 omicron employs three functional, homologous vitamin B(1)(2) transporters that in at least two cases

4 ncies after gastric bypass include thiamine, vitamin B(1)(2), vitamin D, iron, and copper.

5 also contained 100 mg vitamin B-6 and 100 mg vitamin B-1) on vitamin B-12 status and neurophysiologic

6 c pathway for the biosynthesis of cobalamin (vitamin B(12)) has remained poorly characterized because

7 as well as the absorption of iron, calcium, vitamin B(12), and certain medications (e.g. thyroxin).

8 Women in the highest quartile of serum vitamin B-12 (>701 pg/mL) had 2.04 (95% CI: 1.14, 3.62)

9 th a 177% increase in serum concentration of vitamin B-12 (641 compared with 231 pmol/L), a 331% incr

10 Vitamin B-12 (cobalamin) deficiency may produce severe n

11 Poor vitamin B-12 (cobalamin) status is widespread in South A

12 -income countries frequently have inadequate vitamin B-12 (cobalamin) status.

13 re found for riboflavin, vitamin B-6, active vitamin B-12 (holotranscobalamin), and betaine.

14 vegans received either a placebo (n = 34) or vitamin B-12 (n = 42) toothpaste.

15 .62; 95% CI: 0.97, 4.28; P-trend = 0.02) and vitamin B-12 (quartile 5 compared with quartile 1: 2.08;

16 ich cutoff point should be applied for serum vitamin B-12 (SB-12) concentrations to define vitamin B-

17 Low-normal concentrations of vitamin B-12 (VitB12) may be associated with worse cogni

18 of a single intramuscular injection of 10 mg vitamin B-12 (which also contained 100 mg vitamin B-6 an

19 included showed a decrease in mean or median vitamin B-12 across trimesters.

20 s to measure the effect of folic acid and/or vitamin B-12 administration on the incidence of diarrhea

21 Neither folic acid nor vitamin B-12 administration reduced the incidence of dia

22 p (mean +/- SD change: 81 +/- 135 pmol/L for vitamin B-12 and 26 +/- 34 pmol/L for holotranscobalamin

23 ) in these populations, the relation between vitamin B-12 and birth weight (BW) elsewhere is unknown.

24 iologically active form of vitamin B-6), and vitamin B-12 and breast cancer risk in women with a BRCA

25 om serologic tests) had lower mean levels of vitamin B-12 and folate (data collected from 2009 throug

26 Mean levels of vitamin B-12 and folate are lower in individuals with un

27 Vitamin B-12 and folate are micronutrients essential for

28 These findings suggest that, in addition to vitamin B-12 and folate deficiencies alone, there may be

29 ntly associated with anemia, but malaria and vitamin B-12 and folate deficiencies were not.The contri

30 ent AMD, which suggests a potential role for vitamin B-12 and folate in reducing AMD risk.

31 iate log binomial regression showed that low vitamin B-12 and folate intakes in the first trimester w

32 se birth outcomes associated with unbalanced vitamin B-12 and folate intakes or status during pregnan

33 itamin status or chronic diseases related to vitamin B-12 and folate metabolism.

34 B-12 supplementation significantly improved vitamin B-12 and folate status, respectively.

35 These data show that combined vitamin B-12 and folic acid supplementation had no effec

36 Lowering homocysteine with combined vitamin B-12 and folic acid supplementation may reduce f

37 ctures (B-PROOF)] aimed to determine whether vitamin B-12 and folic acid supplementation reduces oste

38 tion.Both postintervention concentrations of vitamin B-12 and holotranscobalamin and their changes ov

39 for ER- breast cancer, and higher intakes of vitamin B-12 and methionine are marginally associated wi

40 40 compared with -0.036 +/- 0.544 mumol/L in vitamin B-12 and placebo groups, respectively; P < 0.001

41 was to examine the association of unbalanced vitamin B-12 and total folate (folic acid supplement + d

42 that vegetarians have lower mean intakes of vitamin B-12 and zinc and higher intakes of fiber, magne

43 ts (n = 30 in the placebo arm; n = 36 in the vitamin B-12 arm) completed the intervention.

44 Higher serum concentrations of folate and vitamin B-12 before ART treatment were associated with h

45 The proportion of women with a plasma total vitamin B-12 concentration <148 pmol/L (deficient) or 14

46 rginal compared with sufficient plasma total vitamin B-12 concentration (>220 pmol/L).

47 th moderately low vitamin B-12 status (serum vitamin B-12 concentrations >/=107 and <210 pmol/L witho

48 - SD age: 73 +/- 3 y; women: 47%) with serum vitamin B-12 concentrations <120 pmol/L at screening.

49 hat has neither normal nor clearly deficient vitamin B-12 concentrations (ie, unknown vitamin B-12 st

50 oubling of the vitamin B-12 intake increased vitamin B-12 concentrations by 11% (95% CI: 9.4%, 12.5%)

51 on was observed; women with serum folate and vitamin B-12 concentrations greater than the median had

52 lysis showed nonsignificantly lower maternal vitamin B-12 concentrations in LBW than in normal-BW inf

53 ve birth compared with women with folate and vitamin B-12 concentrations less than or equal to the me

54 tively], whereas maternal protein intake and vitamin B-12 concentrations most strongly predicted BMC

55 s associated with significantly higher serum vitamin B-12 concentrations relative to nonconsumers (P

56 protein, calcium, and phosphorus intakes and vitamin B-12 concentrations were associated with higher

57 Plasma folate, PLP, and vitamin B-12 concentrations were categorized dichotomous

58 Plasma PLP and vitamin B-12 concentrations were not associated with bre

59 y community-dwelling Chileans with low serum vitamin B-12 concentrations who were consuming bread for

60 had moderate vitamin B-12 deficiency (serum vitamin B-12 concentrations: 107-210 pmol/L) in the abse

61 arian populations and that concentrations of vitamin B-12 decrease from the first to the third trimes

62 Elevated serum tHcy and folate and vitamin B-12 deficiencies predicted increased risk of in

63 Participants with a serum vitamin B-12 deficiency (<185 pmol/L) had higher risk of

64 ticipants were aged >/=75 y and had moderate vitamin B-12 deficiency (serum vitamin B-12 concentratio

65 Overt vitamin B-12 deficiency causes neurologic disturbances i

66 Vitamin B-12 deficiency increases choline use for homocy

67 Moderate vitamin B-12 deficiency is relatively common in older pe

68 e hypothesis that the correction of moderate vitamin B-12 deficiency, in the absence of anemia and of

69 ss healthy older population with more severe vitamin B-12 deficiency.

70 he blood of vegans who are at higher risk of vitamin B-12 deficiency.

71 Of infants, 11.4% were vitamin B-12 deficient and 31.7% marginally deficient at

72 in infant boys of women classified as total vitamin B-12 deficient compared with sufficient.

73 Folic acid and vitamin B-12 from fortified foods and supplements were e

74 their changes over 12 wk were higher in the vitamin B-12 group (mean +/- SD change: 81 +/- 135 pmol/

75 6 mumol/L, respectively) were greater in the vitamin B-12 group than in the placebo group.

76 with folic acid alone or in combination with vitamin B-12 had a significantly higher risk of persiste

77 Preconceptional folate and vitamin B-12 have been linked to beneficial reproductive

78 y intakes and biomarker status of folate and vitamin B-12 in Irish adults.

79 Low intake of dietary vitamin B-12 in the presence of high total folate intake

80 ng a standardized score for each study (mean vitamin B-12 insufficiency / cutoff value), which intern

81 There is no consistent association between vitamin B-12 insufficiency and LBW.

82 Clinical outcomes associated with vitamin B-12 insufficiency could potentially be worsened

83 Our review indicates that vitamin B-12 insufficiency during pregnancy is common ev

84 iew to assess 1) the worldwide prevalence of vitamin B-12 insufficiency in pregnancy and 2) its assoc

85 Vitamin B-12 insufficiency in pregnancy is high in certa

86 The pooled estimates of vitamin B-12 insufficiency were 21%, 19%, and 29% in the

87 The inadequacy of vitamin B-12 intake (<1.2 mug/d) assessed by a food-freq

88 matically reviewed studies that investigated vitamin B-12 intake and biomarkers of vitamin B-12 statu

89 vided information on the association between vitamin B-12 intake and biomarkers.

90 The dose-response relation between vitamin B-12 intake and MMA concentrations indicated a d

91 ose-response relation of all studies between vitamin B-12 intake and status indicated that a doubling

92 The obtained dose-response estimate between vitamin B-12 intake and status provides complementary ev

93 Participants who reported supplementary vitamin B-12 intake had 47% reduced risk of incident any

94 e regarding niacin, folate, vitamin B-6, and vitamin B-12 intake in relation to cognitive function is

95 and status indicated that a doubling of the vitamin B-12 intake increased vitamin B-12 concentration

96 ability of vitamin B-12 to tissues even when vitamin B-12 intake is adequate.

97 y evidence to underpin recommendations for a vitamin B-12 intake of populations.

98 lic acid supplementation in those with a low vitamin B-12 intake or status may have adverse effects.

99 l folate intake was examined as the ratio of vitamin B-12 intake to total folate intake.

100 95% CI: -10%, -4%) for every doubling of the vitamin B-12 intake.

101 aste enters the circulation and corrects the vitamin B-12 markers in the blood of vegans who are at h

102 Changes in vitamin B-12 markers were more prominent in vegans who r

103 The oral application of vitamin B-12 may prevent its deficiency if the vitamin i

104 n common SNPs in genes related to folate and vitamin B-12 metabolism or associated with B vitamin-rel

105 ence of anemia and received 1 mg crystalline vitamin B-12 or a matching placebo as a daily oral table

106 ended Dietary Allowance of folic acid and/or vitamin B-12 or placebo daily for 6 mo.

107 takes and serum concentrations of folate and vitamin B-12 or serum tHcy and 10-y AMD incidence.

108 We tested vitamin B-12 plasma concentrations by using chemilumines

109 pants were assigned to receive daily 500 mug vitamin B-12 plus 400 mug folic acid or placebo suppleme

110 Therefore, vitamin B-12 plus folic acid supplementation cannot be r

111 12 status, including a combined indicator of vitamin B-12 status (3cB12) and scores on the ASQ-3 and

112 transcobalamin, and a composite indicator of vitamin B-12 status (cB-12).

113 min into one variable [combined indicator of vitamin B-12 status (cB-12)].

114 pmol/L; n = 8569, 67.6%) likely had adequate vitamin B-12 status (median MMA: 120 nmol/L; 95% CI: 119

115 people (mean age: 80 y) with moderately low vitamin B-12 status (serum vitamin B-12 concentrations >

116 Impaired vitamin B-12 status also negatively affects BMD.

117 igated vitamin B-12 intake and biomarkers of vitamin B-12 status and estimated dose-response relation

118 imited by heterogeneity in the definition of vitamin B-12 status and imprecise measures of nerve func

119 00 mg vitamin B-6 and 100 mg vitamin B-1) on vitamin B-12 status and neurophysiologic function in eld

120 ifferentiate between inadequate and adequate vitamin B-12 status by using various statistical models.

121 Asymptomatic Chilean elderly with poor vitamin B-12 status displayed improved conductivity in m

122 Insufficient vitamin B-12 status has been linked to poor neurodevelop

123 hildren.We measured the associations between vitamin B-12 status in infancy (2-12 mo) and the develop

124 and block construction scores, respectively.Vitamin B-12 status in infancy is associated with develo

125 The long-term effects of poor vitamin B-12 status in infancy need further investigatio

126 act for neurologic disease of moderately low vitamin B-12 status in older people is unclear.

127 itamin B-12 (SB-12) concentrations to define vitamin B-12 status in population-based research.

128 We aimed to determine whether vitamin B-12 status is associated with electrophysiologi

129 This study shows that maternal vitamin B-12 status is related to choline status in late

130 We investigated whether poor vitamin B-12 status occurs and contributes to low plasma

131 The vitamin B-12 status of the sizable intermediate group (n

132 ic function in asymptomatic elderly with low vitamin B-12 status or whether folate status affects res

133 e functioning in Nepalese children 5 y later.Vitamin B-12 status was assessed in infancy with the use

134 Vitamin B-12 status was assessed with the use of total v

135 Vitamin B-12 status was defined by combining vitamin B-1

136 re was also no evidence of an association of vitamin B-12 status with clinical markers of neurologic

137 d not show any association of any measure of vitamin B-12 status with either peripheral or central ne

138 addressed the association of maternal plasma vitamin B-12 status with postnatal growth rates in term

139 e ASQ-3 and NEPSY II subtests.All markers of vitamin B-12 status with the exception of plasma cobalam

140 ent vitamin B-12 concentrations (ie, unknown vitamin B-12 status).

141 sly associated with neural tube defects, and vitamin B-12 status, as well as between SNPs in SHMT1, w

142 europathy in the elderly, even with a normal vitamin B-12 status, especially if their folate intake i

143 odels, we estimated the associations between vitamin B-12 status, including a combined indicator of v

144 onal use of one cutoff point for classifying vitamin B-12 status.

145 is associated with a progressive decline in vitamin B-12 status.

146 function in older people with moderately low vitamin B-12 status.

147 symptomatic older people with moderately low vitamin B-12 status.

148 We investigated whether vitamin B-12 supplementation benefits neurologic and cog

149 It is uncertain whether vitamin B-12 supplementation can improve neurophysiologi

150 Folic acid or vitamin B-12 supplementation did not reduce the burden o

151 This trial investigated the effectiveness of vitamin B-12 supplementation on electrophysiologic index

152 e is little robust evidence on the effect of vitamin B-12 supplementation on neurologic and cognitive

153 Folic acid and vitamin B-12 supplementation significantly improved vita

154 r whether folate status affects responses to vitamin B-12 supplementation.

155 vegans who reported that they had not taken vitamin B-12 supplements.Vitamin B-12 that is applied to

156 they had not taken vitamin B-12 supplements.Vitamin B-12 that is applied to the oral cavity via toot

157 This effect may reduce the availability of vitamin B-12 to tissues even when vitamin B-12 intake is

158 The 776C-->G polymorphism of the vitamin B-12 transport protein transcobalamin gene (TCN2

159 tivity in myelinated peripheral nerves after vitamin B-12 treatment and an interaction with folate st

160 BW infants and higher odds of LBW with lower vitamin B-12 values (adjusted OR: 1.70; 95% CI: 1.16, 2.

161 Compared with baseline, allocation to vitamin B-12 was associated with a 177% increase in seru

162 Plasma total vitamin B-12 was positively associated with the increase

163 Total intakes of folate and vitamin B-12 were assessed by using a food-frequency que

164 Serum folate and vitamin B-12 were measured in blood samples collected be

165 ates were positively associated, and MMA and vitamin B-12 were not significantly associated with lumb

166 evaluate the association of serum folate and vitamin B-12 with ART outcomes.

167 valuate the associations of serum folate and vitamin B-12 with ART outcomes.

168 -income countries have inadequate cobalamin (vitamin B-12) status.

169 erum concentration biomarkers: folate, 0.49; vitamin B-12, 0.51; alpha-carotene, 0.53; beta-carotene,

170 holine, methionine, riboflavin, vitamin B-6, vitamin B-12, and alcohol intakes.

171 regnancy body mass index, and plasma folate, vitamin B-12, and choline concentrations.

172 ion between serum total homocysteine (tHcy), vitamin B-12, and folate and age-related macular degener

173 including carotenoids, tocopherols, folate, vitamin B-12, and phospholipid fatty acids, were collect

174 SNPs and red blood cell (RBC) folate, serum vitamin B-12, and plasma total homocysteine were determi

175 Serum folate, vitamin B-12, and tHcy were determined from blood sample

176 locks were associated with RBC folate, serum vitamin B-12, and/or plasma homocysteine concentrations.

177 n, serum iron, erythropoietin, serum folate, vitamin B-12, C-reactive protein, and interleukin-6.

178 ntakes of beta-carotene, riboflavin, folate, vitamin B-12, calcium, and iron (controls: 0-7%).

179 NI) for thiamin, riboflavin, niacin, folate, vitamin B-12, calcium, iron, and zinc (range: 14-91% of

180 onceptional supplementation with folic acid, vitamin B-12, choline, and betaine can induce difference

181 lly adjusted ORs for vitamin A, vitamin B-6, vitamin B-12, folic acid, vitamin C, vitamin D, vitamin

182 12 status was assessed with the use of total vitamin B-12, holotranscobalamin, and a composite indica

183 Treatment increased serum vitamin B-12, holotranscobalamin, and cB-12 (P < 0.001)

184 , there was no evidence of an association of vitamin B-12, holotranscobalamin, or cB-12 with any nerv

185 Serum and plasma concentrations of vitamin B-12, holotranscobalamin, total homocysteine (tH

186 cal and other nutrients including vitamin A, vitamin B-12, iron, and zinc at >/=80% of the recommende

187 Vitamin B-12 status was defined by combining vitamin B-12, plasma total homocysteine (tHcy), methylma

188 contain lower amounts of calcium, vitamin D, vitamin B-12, protein, and n-3 (omega-3) fatty acids, al

189 a higher prevalence of inadequacy for iron, vitamin B-12, protein, and zinc than do nonvegetarians.

190 th elevated tHcy were older with lower serum vitamin B-12, red blood cell folate, and dietary micronu

191 For folate, vitamin B-6, and vitamin B-12, results from the use of FFQ data were cons

192 significant group differences were found for vitamin B-12, retinol, linoleic acid (LA), alpha-linolen

193 lence of below-average intakes of vitamin A, vitamin B-12, vitamin C, calcium, and zinc, irrespective

194 ount, vegetarians should optimize intakes of vitamin B-12, zinc, and protein; and both vegetarians an

195 rologic and cognitive function in moderately vitamin B-12-deficient older people.

196 arrier.We studied the effect of the use of a vitamin B-12-fortified toothpaste on vitamin-status mark

197 elders with normal plasma concentrations of vitamin B-12.

198 thiamin, riboflavin, niacin, pyridoxine, and vitamin B-12.

199 population intakes and status of folate and vitamin B-12.

200 ncentrations of nicotinamide, pyridoxal, and vitamin B-12.

201 in D, 64% of iron, 65% of folate, and 46% of vitamin B-12.

202 min 2 (TCN2), which is referred to as active vitamin B-12.

203 trimester, those with the lowest tertile of vitamin B-12:folate ratio had a higher risk of SGA outco

204 ntrations of biomarkers in choline pathways, vitamins B-12 and A, and essential fatty acids.A randomi

205 orus, potassium, folic acid, riboflavin, and vitamins B-12, C, and E, and by approximately 25% for se

206 Riboflavin (vitamin B(2)) is the precursor of the flavin coenzymes f

207 to nitrogen-15 in pyridine and nicotinamide (vitamin B(3) amide) by conducting "signal amplification

208 Vitamin B(6) (pyridoxal 5'-phosphate) is an essential co

209 Vitamin B(6) and vitamin B(1)(2) intake was not associat

210 not materially altered after adjustment for vitamin B(6) and vitamin B(1)(2) intake.

211 ococcal DeltapdxR mutant also exhibited as a vitamin B(6) auxotroph.

212 eletion of pdxS, but not pdxT, resulted in a vitamin B(6) auxotrophic mutant.

213 In addition, we found that disruption of the vitamin B(6) biosynthesis pathway in S. pneumoniae cause

214 Vitamin B(6) is an essential cofactor for a large number

215 eumonia model, indicating that the impact of vitamin B(6) synthesis on virulence depends upon the bac

216 efective growth of the DeltapdxS mutant in a vitamin B(6)-depleted medium could be chemically restore

217 We also found that higher intakes of vitamin B-6 (quartile 5 compared with quartile 1: 2.62;

218 sm through the kynurenine pathway includes 2 vitamin B-6 [pyridoxal 5'-phosphate (PLP)]-dependent enz

219 mg vitamin B-12 (which also contained 100 mg vitamin B-6 and 100 mg vitamin B-1) on vitamin B-12 stat

220 omarkers showed significant seasonality, and vitamin B-6 and folate had the highest fluctuations.

221 Vitamin B-6 and riboflavin intakes from diet and supplem

222 We evaluated the vitamin B-6 biomarkers PLP, pyridoxal, and pyridoxic aci

223 xal + PLP) ratio (PAr), a proposed marker of vitamin B-6 catabolism during activated cellular immunit

224 Inflammatory processes, including vitamin B-6 catabolism, could explain such findings.We i

225 LP, and PA:(PL + PLP) as possible markers of vitamin B-6 catabolism.

226 Vitamin B-6 comprises a group of 6 interrelated vitamers

227 of follow-up between sampling and diagnosis.Vitamin B-6 deficiency as measured by plasma PLP is asso

228 estigated long-term clinical implications of vitamin B-6 deficiency in stable outpatient RTRs.In a lo

229 s were observed for graft failure (P = 0.18).Vitamin B-6 deficiency is common in RTRs and does not se

230 In RTRs, vitamin B-6 deficiency was associated with considerably

231 Vitamin B-6 deficiency was defined as PLP <20 nmol/L, an

232 was low, which was likely due to underlying vitamin B-6 deficiency, which was confirmed by very low

233 y assays may be compromised with co-existing vitamin B-6 deficiency.

234 bations in metabolism, suggesting functional vitamin B-6 deficiency.

235 stress could explain increased catabolism of vitamin B-6 during inflammation.

236 Dietary and total intakes of vitamin B-6 in quartile 4 compared with quartile 1 (HR:

237 PLP influences long-term outcome.We compared vitamin B-6 intake and circulating PLP concentrations of

238 1.8-12.1 y) after transplantation], the mean vitamin B-6 intakes in RTRs and healthy controls were 1.

239 Higher plasma concentrations of the vitamin B-6 marker pyridoxal 5'-phosphate (PLP) have bee

240 Genetic disorders disrupting vitamin B-6 metabolism have severe clinical consequences

241 atment of conditions associated with altered vitamin B-6 metabolism.

242 PA:(PL + PLP) had the highest ICC of all vitamin B-6 metabolites and ratios tested.

243 he association of plasma PLP with functional vitamin B-6 status and explored the potential associatio

244 our analyses of novel markers of functional vitamin B-6 status and vitamin B-6-associated oxidative

245 Low chronic vitamin B-6 status can occur in a subset of women who us

246 Low vitamin B-6 status has been related to increased risk of

247 uch findings.We investigated 3 biomarkers of vitamin B-6 status in relation to CRC risk.This was a pr

248 Worse functional vitamin B-6 status in RTRs is independently associated w

249 e grains and animal-source foods may enhance vitamin B-6 status in Zambians.

250 n B-6-deficient RTRs have a worse functional vitamin B-6 status than do healthy controls and vitamin

251 XA ratio was used as functional biomarker of vitamin B-6 status with a higher ratio reflecting worse

252 ored the potential association of functional vitamin B-6 status with long-term mortality in RTRs.In a

253 :XA (HK:XA), an inverse marker of functional vitamin B-6 status, and PA:(PLP + pyridoxal) (PAr), a ma

254 PLP was used as direct biomarker for vitamin B-6 status, and the 3-HK:XA ratio was used as fu

255 2.14; 95% CI: 1.48, 3.08) than a sufficient vitamin B-6 status, independent of potential confounders

256 , present as potential markers of functional vitamin B-6 status.

257 h a higher ratio reflecting worse functional vitamin B-6 status.Median PLP, 3-HK, and XA concentratio

258 nd oxidative stress and an inverse marker of vitamin B-6 status.Plasma PLP concentrations were associ

259 le or no change after supplementation in low-vitamin B-6 subjects.

260 We investigated the effects of vitamin B-6 supplementation on the in vivo kinetics of o

261 The relative vitamin B-6 vitamer composition of plasma [pyridoxal pho

262 B-6 vitamers point to a tight regulation of vitamin B-6 vitamer concentrations in blood and CSF.

263 Sex influenced vitamin B-6 vitamer concentrations in plasma and CSF and

264 We provide an extensive reference set of vitamin B-6 vitamer concentrations in plasma and CSF.

265 fore be taken into account when interpreting vitamin B-6 vitamer concentrations.

266 liable CSF:plasma ratios and correlations of vitamin B-6 vitamers could be established.

267 Concentrations of vitamin B-6 vitamers in the plasma and CSF of 533 adult

268 trict ratios and strong correlations between vitamin B-6 vitamers point to a tight regulation of vita

269 ntrols (47% male; age 54 +/- 11 y), baseline vitamin B-6 was measured as plasma PLP by high-performan

270 sphate (PLP; the biologically active form of vitamin B-6), and vitamin B-12 and breast cancer risk in

271 blood biomarkers were found for riboflavin, vitamin B-6, active vitamin B-12 (holotranscobalamin), a

272 emiologic evidence regarding niacin, folate, vitamin B-6, and vitamin B-12 intake in relation to cogn

273 For folate, vitamin B-6, and vitamin B-12, results from the use of F

274 bumin, calcium, iron, ferritin, cholesterol, vitamin B-6, and vitamin D (data collected from 2009 thr

275 (PLP), which is the active coenzyme form of vitamin B-6, are reduced during inflammation.

276 by safe and inexpensive interventions (e.g., vitamin B-6, B-9, and B-12 supplementation).

277 In contrast, thiamin, vitamin B-6, calcium, iron, and zinc had linear or quadr

278 Dietary intakes of vitamin B-6, magnesium, and zinc were below the Estimate

279 osphate (PLP), which is the coenzyme form of vitamin B-6, may impair many metabolic processes includi

280 of folate, choline, methionine, riboflavin, vitamin B-6, vitamin B-12, and alcohol intakes.

281 The fully adjusted ORs for vitamin A, vitamin B-6, vitamin B-12, folic acid, vitamin C, vitami

282 markers of functional vitamin B-6 status and vitamin B-6-associated oxidative stress and inflammation

283 of one-carbon and tryptophan metabolites in vitamin B-6-deficient OC users.

284 increment: 1.50; 95% CI: 1.21, 1.86) in RTRs.Vitamin B-6-deficient RTRs have a worse functional vitam

285 Consistent with previous vitamin B-6-restriction studies, fluxes of one-carbon me

286 amin B-6 status than do healthy controls and vitamin B-6-sufficient RTRs.

287 B vitamins [vitamins B-6, B-9 (folate), and B-12] play important rol

288 promise for natural-mode alleviation of this vitamin (B(9)) deficiency.

289 R1 can present activating and non-activating vitamin-B-based ligands to mucosal-associated invariant

290 Vitamin B deficiencies, which can lead to hyperhomocyste

291 IL-10-deficient mice fed the vitamin B-deficient diet exhibited more severe colonic i

292 -chain, TRAV1-2-TRAJ33, and are activated by vitamin B metabolites bound by the major histocompatibil

293 A recent discovery that MR1 presents vitamin B metabolites, presumably from pathogenic and/or

294 like molecules that bind lipid-based Ags and vitamin B precursors, respectively.

295 The antigen-presenting molecule MR1 presents vitamin B-related antigens (VitB antigens) to mucosal-as

296 c association provides further evidence that vitamin B supplements are not chemopreventive for lung c

297 protein 1 (MR1) presents metabolites of the vitamin B synthesis pathways to mucosal-associated invar

298 cause of the high prevalence of supplemental vitamin B use, any possible increased association warran

299 The total amount of vitamins B was 30% and 50% higher in RF cooked salmon th

300 e-like T-cells, can recognise metabolites of vitamin B, when presented by the MHC-related protein, MR