ライフサイエンスコーパス: vitamin D

1 or effects of calcitriol, the active form of vitamin D.

2 stimuli was detected, which was inhibited by vitamin D.

3 early life innate immune function and serum vitamin D.

4 ntioned genetic associations are modified by vitamin D.

5 The active form of vitamin D [1,25(OH)2D] regulates B cells in vitro and mi

6 Vitamin D (25(OH)D) deficiency has been implicated as a

7 wk of gestation) consumed a single amount of vitamin D (511 IU/d from diet and a cholecalciferol supp

8 etary intakes below the EARs for iron (97%), vitamin D (96%), and folate (70%).

9 al cell proliferation, migration, and on the vitamin D activating enzyme CYP27B1 (produces 1,25(OH)2D

10 thway analyses in CD4+ T cells, we show that vitamin D affects multiple signaling and metabolic pathw

11 Low levels of vitamin D also appear to be a risk factor for IBD.

12 %) people died during the trial, 65 assigned vitamin D and 58 allocated placebo; the difference betwe

13 retrieve well documented analytical data for vitamin D and arrange the data into two datasets - Europ

14 Translational research in trials combining Vitamin D and aspirin have begun as part of such investi

15 ion of high-density lipoprotein cholesterol, vitamin D and C-reactive protein, and less central obesi

16 may reduce risk.We evaluated how intakes of vitamin D and calcium are associated with the incidence

17 ve Nurses' Health Study II (NHS2).Intakes of vitamin D and calcium from foods and supplements were me

18 cal and preclinical evidence suggesting that vitamin D and calcium inhibit colorectal carcinogenesis,

19 To investigate whether common variants in 7 vitamin D and calcium pathway genes (VDR, GC, DHCR7, CYP

20 association between maternal postsupplement vitamin D and cluster membership (P = 0.0014).

21 Vitamin D and placebo groups did not differ in change in

22 These findings point to a role of vitamin D and the VDR in modulating autophagy and cell d

23 ctor 23, parathyroid hormone, sclerostin, or vitamin D and their relevance to the management of diffe

24 e with fat malabsorption disorders malabsorb vitamin D and thus must rely on cutaneous production of

25 Vitamin D and VDR may have a combined role in IA develop

26 uding pharmaceutical prescriptions, calcium, vitamin D, and estrogen.

27 en aged 3 y with and without asthma from the Vitamin D Antenatal Asthma Reduction Trial (VDAART), in

28 lind, placebo-controlled clinical trial (the Vitamin D Antenatal Asthma Reduction Trial).

29 mammary gland, highlighting the potential of vitamin D as a cancer-preventive agent.

30 sistent.We investigated the relation between vitamin D, assessed in 3 different ways, and the risk of

31 iations may be the most feasible approach to vitamin D assessment over time.

32 The Vitamin D Assessment Study is a randomized, double-blind

33 In contrast, vitamin D at 20 nM concentration suppressed the expressi

34 n the article by Scott et al, a high dose of vitamin D attenuated the inflammatory response to UV rad

35 the presence of the group-specific component/vitamin D binding protein gene (GC/DBP) rs4588 functiona

36 s).We compared concentrations of 25(OH)D and vitamin D-binding protein (VDBP) in AA and EA women and

37 and non-renal cells and has implications for vitamin D biology in multiple sclerosis and perhaps othe

38 A women and investigated determinants of the vitamin D-biomarker concentrations in both populations.W

39 ntake alone and with calcium with or without vitamin D (Ca+/-D) on bone health measures in adults.Sea

40 sociation between the intake of calcium with vitamin D (CaD) and fracture risk.Data from 5823 white p

41 and mineral bone disease caused by disturbed vitamin D, calcium, and phosphate metabolism.

42 showed that several genetic variants within vitamin D cascade affect vitamin D function.

43 OH)D-was 0.99 (95% CI 0.92-1.07; p=0.82) for vitamin D compared with placebo.

44 age and education, and both child asthma and vitamin D concentration at age 3 y did not modify the as

45 o cover losses during shelf life, the actual vitamin D concentration of fortified foods and dietary s

46 irected towards the potential association of vitamin D concentrations and prostate cancer, but little

47 elucidate the underlying determinants of low vitamin D concentrations in AA populations.

48 The results indicate variations in vitamin D concentrations in eggs from different sources,

49 olism and its impact on maternal circulating vitamin D concentrations in humans.This study sought to

50 nfluence of maternal pre- and postsupplement vitamin D concentrations on cluster membership.

51 was the only category in which all measured vitamin D concentrations were below the declared value (

52 time outdoors and sunlight exposure), serum vitamin D concentrations, and vitamin D pathway genetic

53 tiple sclerosis, a disease influenced by low vitamin D concentrations.

54 Compared to the declared values, the vitamin D content ranged from 50% to 153% for fortified

55 hway in PSC disease pathology and found that vitamin D could maintain CD28 expression.

56 ron, ferritin, cholesterol, vitamin B-6, and vitamin D (data collected from 2009 through 2010) did no

57 h an approximate 20% increase in the odds of vitamin D deficiency (</=20 ng/mL) [odds ratio (95% CI):

58 ified subgroup analysis in participants with vitamin D deficiency (baseline deseasonalized 25-hydroxy

59 70 years, with nondiabetic CKD stage 3-4 and vitamin D deficiency (serum 25-hydroxyvitamin D </=20 ng

60 Iron deficiency (ID) and vitamin D deficiency (VDD) are common among young Europe

61 We hypothesized that vitamin D deficiency alters TRAIL protein levels in huma

62 eters, thus suggesting that the link between vitamin D deficiency and cardiovascular disease may be a

63 lts were seen for participants with baseline vitamin D deficiency and for secondary outcomes.

64 Vitamin D deficiency associates with mortality in patien

65 Vitamin D deficiency dropped from 64% to 20%.

66 The high prevalence of vitamin D deficiency in African Americans (AAs) may be a

67 Vitamin D deficiency in murine sepsis was associated wit

68 vant murine models and approaches to correct vitamin D deficiency in patients with sepsis should be d

69 In humans, vitamin D deficiency is associated with the following: v

70 These vGWAS results together suggest that vitamin D deficiency is potentially causal of sero-negat

71 nical and clinical findings and suggest that vitamin D deficiency not only promotes bone diseases but

72 gnificant downregulation with UVB.Correcting vitamin D deficiency with either oral vitamin D3 or UVB

73 lant and posttransplant hyperparathyroidism, vitamin D deficiency, and fracture risk after KT.

74 Vitamin D deficiency, defined as a serum 25-hydroxyvitam

75 mozygous p.Ser267Phe in SLC10A1 are prone to vitamin D deficiency, deviated sex hormones and blood li

76 nondiabetic patients with stage 3-4 CKD and vitamin D deficiency, vitamin D supplementation may impr

77 is C (HCV) infection have high prevalence of vitamin D deficiency.

78 s of vitamin D, variability in participants' vitamin D-deficiency status, and the use of surrogate me

79 phy and progression to heart failure in both vitamin D deficient and normal mice without inducing sig

80 Milk from vitamin D deficient mothers showed high levels of TRAIL

81 ng/mL, with 1270 participants (24.9%) being vitamin D deficient.

82 with placebo.Sixty-five overweight or obese, vitamin D-deficient (25-hydroxyvitamin D [25(OH)D] conce

83 .We performed a randomized clinical trial in vitamin D-deficient adults and compared vitamin D replen

84 UVB) light.We tested the hypothesis that, in vitamin D-deficient adults, the replenishment of vitamin

85 rovided in a sufficient dose and duration to vitamin D-deficient individuals would improve insulin se

86 improve insulin sensitivity or secretion in vitamin D-deficient, overweight or obese adults, despite

87 Maternal presupplement vitamin D did not influence cluster membership, whereas

88 ute to variations in serum 25(OH)D level and vitamin D dose-response.

89 e, ethnic or racial origin, body-mass index, vitamin D dosing regimen, use of inhaled corticosteroids

90 is needed to ascertain the effects of daily vitamin D dosing, with or without calcium.

91 Since the non-mineral functions of vitamin D emerges, 1alpha,25(OH)2D3, the active form of

92 Treatment with vitamin D-enriched LE extracts was associated with signi

93 Vitamin D-enriched mushrooms extracts exert a synergisti

94 omodulatory effect of oral administration of vitamin D-enriched mushrooms extracts on high-fat diet (

95 s a previously unreported mechanism by which vitamin D exerts anti-inflammatory effects in humans.

96 Vitamin D exerts multiple immunomodulatory functions and

97 the metabolic mechanisms related to in utero vitamin D exposure may therefore shed light on complex d

98 to analyze the programming role of in utero vitamin D exposure on children's metabolomics profiles.F

99 To support these studies, a specialized vitamin D food composition dataset, based on EuroFIR sta

100 lls treated with variable doses (0-20 nM) of vitamin D for 24 h demonstrated that low levels (0.5 to

101 nding interests in the potential benefits of vitamin D for preventing breast cancer recurrence and mo

102 A systematic vitamin D fortification of fluid milk products and fat s

103 We investigated the effects of the vitamin D fortification policy on vitamin D status in Fi

104 tor efficacy and safety of ongoing strategic vitamin D fortification.

105 tic variants within vitamin D cascade affect vitamin D function.

106 hase of an interventional trial of high-dose vitamin D given during pregnancy.

107 in 292 individuals, 156 (6%) of 2558 in the vitamin D group and 136 (5%) of 2550 in the placebo grou

108 occurred in 303 participants (11.8%) in the vitamin D group and 293 participants (11.5%) in the plac

109 ted having a fall, 1312 (52%) of 2539 in the vitamin D group compared with 1326 (53%) of 2517 in the

110 in D receptor (VDR), whose activating ligand vitamin D has been proposed as a modifiable factor in mu

111 Vitamin D has various cardiovascular pleiotropic effects

112 merges, 1alpha,25(OH)2D3, the active form of vitamin D, has been applied in anti-cancer researches.

113 is necessary to assess the possible role of vitamin D in cancer prevention.

114 to advance the understanding of the role of Vitamin D in HL.

115 r evaluating its effectiveness for producing vitamin D in human skin due to the shorter exposure time

116 no convincing evidence for a direct role of vitamin D in myopia risk.

117 to mimic the effect of dermally synthesized vitamin D in response to ultraviolet type B (UVB) light.

118 ions for the immunotherapeutic properties of vitamin D in skin homeostasis, and implicate arginase-1

119 e general European population and implicates vitamin D in the etiology of multiple sclerosis.

120 RATIONALE: Existing trials of adjunctive vitamin D in the treatment of pulmonary tuberculosis (PT

121 elow the EFSA Adequate Intake (AI) (<15mug/d vitamin D) in adults across Europe.

122 In addition, vitamin D inadequacy (serum 25-hydroxyvitamin D concentr

123 ets (WD) high in fat and scarce in fiber and vitamin D increase risks of colorectal cancer.

124 -frequency variant have an increased risk of vitamin D insufficiency (odds ratio [OR] = 2.2, 95% conf

125 Ecological evidence suggests vitamin D insufficiency (VDI) due to lower ambient ultra

126 variants that strongly increase the risk of vitamin D insufficiency and tested their effect on risk

127 ow exposure to sunlight increase the risk of vitamin D insufficiency in children.The aim of the study

128 Vitamin D insufficiency is common, correctable, and infl

129 sun exposure, physical activity, and dietary vitamin D intake (P > 0.1).Vitamin D supplementation doe

130 on and lactose tolerance, had higher dietary vitamin D intake and higher measured 25(OH)D concentrati

131 his study was to evaluate the association of vitamin D intake and serum levels with fracture risk in

132 lements may result in invalid estimations of vitamin D intake distributions of infants; both under- a

133 ntified, but emerging data suggest that high vitamin D intake may reduce risk.We evaluated how intake

134 tography-mass spectrometry, assessed dietary vitamin D intake with the use of 7-d dietary records, an

135 Plasma 25(OH)D, dietary vitamin D intake, and vitamin D-synthesis GRS were not a

136 000 to date, demonstrated high prevalence of vitamin D intakes below the EFSA Adequate Intake (AI) (<

137 kin.Children with fair and dark skin require vitamin D intakes of 20 and 28 mug/d, respectively, to m

138 We sought to study the effect of a vitamin D intervention in pregnancy between subjects of

139 Vitamin D is an environmental and dietary agent with kno

140 Through the VDR, vitamin D is an environmental factor that helps to maint

141 Vitamin D is necessary for the healthy growth and develo

142 CYP24A1, the primary inactivating enzyme for vitamin D, is often overexpressed in human cancers, pote

143 were 4% lower for every 1-ng/mL increase in vitamin D level (odds ratio, 0.96; 95% CI, 0.93-0.99; P

144 he evidence for a relationship between serum vitamin D levels and nonskeletal health outcomes is inco

145 r evidence on the direct association between Vitamin D levels and the clinical course of HL needs to

146 tructed to determine the association between vitamin D levels and the presence of uveitis.

147 sistent egg allergy and that increased serum vitamin D levels correlate with changes in innate immune

148 Low circulating vitamin D levels have been associated with risk of asthm

149 phthalates and/or BPA to disrupt circulating vitamin D levels in pregnancy.

150 In this retrospective study, lower vitamin D levels were associated with an increased risk

151 We aimed to test whether genetically lowered vitamin D levels were associated with risk of asthma, at

152 g function, antiinflammatory biomarkers, and vitamin D levels, as well as reductions in airway and sy

153 sk alleles whose effect may be modifiable by vitamin D levels.

154 though asymptomatic, all individuals had low vitamin D levels.

155 Here I have developed a hypothesis that vitamin D may act to control the rate of ageing.

156 Vitamin D may have anticancer activities.

157 plementation, supporting the hypothesis that vitamin D may modulate risk for developing MS.

158 Serum vitamin D may play an immune-modulatory role in the deve

159 observed between placental mRNA abundance of vitamin D metabolic components and circulating vitamin D

160 placental messenger RNA (mRNA) abundance of vitamin D metabolic pathway components were quantified.

161 etic variants located near genes involved in vitamin D metabolism and calcium and renal phosphate tra

162 Little is known about placental vitamin D metabolism and its impact on maternal circulat

163 vance the current understanding of placental vitamin D metabolism and its role in modulating maternal

164 ations nor variants in genes associated with vitamin D metabolism were found.

165 Despite its importance in vitamin D metabolism, the molecular mechanisms underlyin

166 Notably, administration of the active vitamin D metabolite calcitriol reversed all these effec

167 tamin D metabolic components and circulating vitamin D metabolites [i.e., LDL-related protein 2 (LRP2

168 he intracellular generation and secretion of vitamin D metabolites along with the regulation of targe

169 Concentrations of placental and blood vitamin D metabolites and placental messenger RNA (mRNA)

170 its role in modulating maternal circulating vitamin D metabolites during pregnancy.Nested within a f

171 loped Standard Reference Material (SRM) 972a Vitamin D Metabolites in Frozen Human Serum as a replace

172 We investigated the role of vitamin D metabolites in regulating intact FGF23 product

173 nd indirectly enhanced the action of ambient vitamin D metabolites via the vitamin D receptor.

174 calcium, phosphate, parathyroid hormone, and vitamin D metabolites.

175 levels, despite normal circulating levels of vitamin D metabolites.

176 d here provide epidemiological evidence that Vitamin D might play a protective role in HL.

177 m of the study was to evaluate the amount of vitamin D needed to ascertain that most children >4 y of

178 es were done to determine whether effects of vitamin D on risk of asthma exacerbation varied accordin

179 ants were randomized to receive 2000 IU/d of vitamin D oral supplementation (high-dose group) vs 354

180 supplements (and sun exposure in the case of vitamin D outcomes).

181 Maternal supplementation of vitamin D, particularly in mothers with initial 25(OH)D

182 hether genetic polymorphisms of genes in the vitamin D pathway are associated with treatment response

183 posure), serum vitamin D concentrations, and vitamin D pathway genetic variants, adjusting for years

184 s of the effects of genetic variation in the vitamin D pathway on response to vitamin D supplementati

185 fy whether monitoring and supplementation of vitamin D play roles in cardiovascular protection.

186 Vitamin D plays an important role in both the innate and

187 other B vitamins, beta-carotene, vitamin C, vitamin D plus calcium, and multivitamins or multi-ingre

188 fatty acids, soy, ginkgo biloba, B vitamins, vitamin D plus calcium, vitamin C or beta-carotene, mult

189 Strikingly, dietary vitamin D prevented these calcium-triggered tumorigenic

190 ar receptor superfamily, including the human vitamin D receptor (hVDR).

191 ulates B cells in vitro and mice without the vitamin D receptor (VDR knockout [KO]) have high serum I

192 or more >/=4 mm pockets were associated with vitamin D receptor (VDR) (rs2228570, P = 0.002, q = 0.04

193 Here, we document that the vitamin D receptor (VDR) acts as a master transcriptiona

194 Recently, we found that vitamin D receptor (VDR) enhanced Claudin-2 expression i

195 Vitamin D receptor (VDR) knockdown partly abolished MART

196 Vitamin D receptor (VDR) mutations in humans and mice ca

197 l small-molecule compounds that activate the vitamin D receptor (VDR), but are devoid of hypercalcemi

198 on in binding of a transcription factor, the vitamin D receptor (VDR), whose activating ligand vitami

199 1,20S(OH)2D3 interacts with the vitamin D receptor (VDR), with similar potency to its na

200 Absence of vitamin D receptor (VDR)-mediated PPARgamma suppression

201 erted via signaling mechanisms involving the vitamin D receptor (VDR).

202 d the individual and combined effects of the vitamin D receptor activator paricalcitol (PARI) and die

203 ted with IL-23 plus IL-1beta upregulated the vitamin D receptor and responded to 1,25D with downregul

204 disrupting the interaction between SMAD3 and vitamin D receptor by altering SMAD3 ubiquitination.

205 ed colorectal adenomas may vary according to vitamin D receptor genotype.

206 n genes encoding PD-L1 and PD-L2 through the vitamin D receptor, a ligand-regulated transcription fac

207 key locus responsible for skin color, with a vitamin D receptor-binding interval.

208 ion of ambient vitamin D metabolites via the vitamin D receptor.

209 vity of VDR toward specific target genes.The vitamin D receptor/retinoid X receptor-alpha heterodimer

210 Data from in vitro experiments suggest that vitamin D reduces the rate of skin aging, whereas popula

211 However, whether winter supplementation of vitamin D reduces the risk among children is unknown.

212 Consistently, vitamin D regulated TRAIL mRNA expression in HME-1 cells

213 Vitamin D regulates many biological processes, but its c

214 l in vitamin D-deficient adults and compared vitamin D replenishment between subjects who received or

215 pha and beta) proteins compared to milk from vitamin D replete women.

216 We identified and characterized vitamin D response elements (VDREs) located in both gene

217 One of the vitamin D responsive genes, 25(OH)D3-24-hydroxylase (cyt

218 persisted when adjusting for baseline 25(OH) vitamin D serum level.

219 e-edged sword, as they suggest that elevated vitamin D signaling in humans could suppress anti-tumor

220 arch novel chemical probes that activate the vitamin D signaling, we report discovery of novel non-st

221 Rs) of UVB, serum vitamin D3 concentrations, vitamin D single-nucleotide polymorphisms, and myopia es

222 When consuming vitamin D sources based on the nutritional recommendatio

223 oading efficiency show promise for improving vitamin D stability during dry storage.

224 e-assembled casein micelles, and to evaluate vitamin D stability of dry formulations during ambient o

225 ectively, were standardized according to the Vitamin D Standardization Program with the use of liquid

226 nadequate dosing regimens, and high baseline vitamin D status among participants.

227 rt no association between antenatal maternal vitamin D status and childhood fractures.

228 To identify genetic variants responsible for vitamin D status and dose-response, we performed two vit

229 through age 3 years, suggesting that higher vitamin D status beginning in early pregnancy is necessa

230 P = 0.13).This study suggested that neonatal vitamin D status does not influence subsequent fracture

231 Vitamin D status in childhood is a significant predictor

232 cts of the vitamin D fortification policy on vitamin D status in Finland between 2000 and 2011.Serum

233 Our results indicate that vitamin D status in mothers modulates TRAIL expression i

234 sion of TRAIL in human milk as a function of vitamin D status in mothers remains unknown.

235 the effect of milk fortification on iron and vitamin D status in these children are scarce.

236 ternal BMD during pregnancy as a function of vitamin D status in women of diverse racial/ethnic backg

237 es based on the nutritional recommendations, vitamin D status is sufficient [S-25(OH)D >/=50 nmol/L],

238 is restricted to patients with low baseline vitamin D status is unknown.

239 xes, there are moderate associations between vitamin D status measured in prepuberty, adolescence, an

240 therefore be a preferential form to optimize vitamin D status within the general population.

241 To investigate a serum biomarker of vitamin D status, 25-hydroxyvitamin D (25OHD) measured a

242 The best biomarker of vitamin D status, 25hydroxyvitamin D3 (25(OH)D3), howeve

243 lar disease (CVD) among individuals with low vitamin D status.

244 ds was started in 2003 in Finland to improve vitamin D status.

245 groups were lower than those in Europeans), vitamin D supplement use of >/=1,000 IU/day (18.9 nmol/L

246 risk, high serum 25(OH)D levels and regular vitamin D supplement use were associated with lower rate

247 cially of fluid milk products, and augmented vitamin D supplement use.

248 ic assessment), calcium supplementation, and vitamin D supplementation (OR, 0.12 [95% CI, 0.03 to 0.5

249 and non-AA mothers in the effect of maternal vitamin D supplementation and asthma/recurrent wheeze in

250 ght or obese adults, despite using high-dose vitamin D supplementation and robust endpoint measures.

251 tion in the vitamin D pathway on response to vitamin D supplementation are lacking.

252 istration of 2000 IU compared with 400 IU of vitamin D supplementation did not reduce overall wintert

253 vity, and dietary vitamin D intake (P > 0.1).Vitamin D supplementation does not improve insulin sensi

254 However, oral vitamin D supplementation does not lower LDL-cholesterol

255 Monthly high-dose vitamin D supplementation does not prevent CVD.

256 rticipants from a previous study of maternal vitamin D supplementation during lactation.

257 s result does not support the use of monthly vitamin D supplementation for this purpose.

258 Vitamin D supplementation has been proposed as a potenti

259 alls and fractures, but randomised trials of vitamin D supplementation have had inconsistent results.

260 do not support the routine use of high-dose vitamin D supplementation in children for the prevention

261 We observed that dietary vitamin D supplementation in mice increases basal levels

262 andomized, controlled, double-blind trial of vitamin D supplementation in pregnancy (400, 2000, or 40

263 studied the metabolic effects of a 12-month vitamin D supplementation in T2DM patients according to

264 on, improvements in metabolic profile due to vitamin D supplementation is influenced by VDR polymorph

265 n addition, they support the hypothesis that vitamin D supplementation is useful in breast cancer pre

266 with stage 3-4 CKD and vitamin D deficiency, vitamin D supplementation may improve vascular function.

267 tes with mortality in patients with CKD, and vitamin D supplementation might mitigate cardiovascular

268 INTERPRETATION: High-dose bolus vitamin D supplementation of 100 000 IU colecalciferol m

269 ese findings do not support a dose effect of vitamin D supplementation on bone health and suggest tha

270 ich pregnant women were randomly assigned to vitamin D supplementation or placebo.

271 of low dietary intakes and low compliance to vitamin D supplementation policies.

272 y reported that in juvenile/adolescent rats, vitamin D supplementation protects from experimental aut

273 Vitamin D supplementation reduced the rate of asthma exa

274 determine whether high-dose vs standard-dose vitamin D supplementation reduces the incidence of winte

275 of randomised controlled trials showed that vitamin D supplementation reduces the rate of asthma exa

276 cebo-controlled trial to investigate whether vitamin D supplementation that is provided in a sufficie

277 in the future that can be used for providing vitamin D supplementation to patients with fat malabsorp

278 However, meta-analyses of vitamin D supplementation trials have failed to show cle

279 We hypothesized that vitamin D supplementation would improve insulin sensitiv

280 Vitamin D supplementation, but not serum 25-hydroxyvitam

281 aling pathway genes were down-regulated upon vitamin D supplementation.

282 ssociation was observed between child use of vitamin D supplements and decreased odds of fracture (ye

283 population stratification and pleiotropy and vitamin D synthesis and metabolism pathways.

284 001), which was perhaps related to increased vitamin D synthesis by birds having more access to sunli

285 he use of 7-d dietary records, and created a vitamin D-synthesis genetic risk score (GRS) at baseline

286 lasma 25(OH)D, dietary vitamin D intake, and vitamin D-synthesis GRS were not associated with any cog

287 ts were genotyped for functional variants on vitamin D synthetic pathway including GC (rs4588, rs7041

288 It acts in the nucleus of vitamin D target cells to regulate the expression of gen

289 Currently the majority of vitamin D testing is performed in large-scale commercial

290 f species-specific regulation of immunity by vitamin D, the VDREs are present in primate genes, but n

291 causation may result from a failure of oral vitamin D to mimic the effect of dermally synthesized vi

292 roxyvitamin D [25(OH)D] levels, supplemental vitamin D use, and breast cancer incidence over the subs

293 been limited by short duration, low doses of vitamin D, variability in participants' vitamin D-defici

294 Vitamin D (VD) supplementation has been shown to reverse

295 Group II: 98.0 (20.25) pg/mL, P = .01); and vitamin D (VDBP) (Group I: 0.06 (0.13) mug/mL, Group II:

296 No trials compared calcium or vitamin D versus placebo.

297 e time of transplant, cinacalcet, and native vitamin D were used significantly more frequently in gro

298 tudinal studies examining the association of vitamin D with incident dementia and cognitive impairmen

299 ng observational evidence on associations of vitamin D with lower risk of breast cancer morbidity and

300 min D-deficient adults, the replenishment of vitamin D with UVB exposure would lower LDL-cholesterol