ライフサイエンスコーパス: vitamin D receptor

1 netic loci, including the VDR gene (encoding vitamin D receptor).

2 ion of ambient vitamin D metabolites via the vitamin D receptor.

3 cording to common genetic differences in the vitamin D receptor.

4 in D intake and genetic polymorphisms in the vitamin D receptor.

5 ced by common variations in the gene for the vitamin D receptor.

6 droxyvitamin-D3 (1,25D) was dependent on the vitamin D receptor.

7 cells revealed the presence of cell surface vitamin D receptors.

8 It seems likely that statins activate vitamin D receptors.

9 n genes encoding PD-L1 and PD-L2 through the vitamin D receptor, a ligand-regulated transcription fac

10 and its type I receptor expression, restored vitamin D receptor abundance, and inhibited cell prolife

11 A recent article in Cell shows that vitamin D receptor activation reprograms reactive stroma

12 d the individual and combined effects of the vitamin D receptor activator paricalcitol (PARI) and die

13 Paricalcitol, a selective vitamin D receptor activator, decreased serum parathyroi

14 Furthermore, vitamin D receptor activators can restore Klotho express

15 storation of suppressed Klotho expression by vitamin D receptor activators conferred human aortic smo

16 y procalcific stressors could be restored by vitamin D receptor activators, in vitro and further conf

17 The physiologic vitamin D receptor agonist, 1,25(OH)2D3 (calcitriol), is

18 Vitamin D receptor agonists are known to suppress parath

19 evant synthetic lethal interactions and that vitamin D receptor agonists may show enhanced efficacy i

20 l induced a physical interaction between the vitamin D receptor and beta-catenin in podocytes, which

21 timicrobial effector pathway mediated by the vitamin D receptor and human beta defensin 2.

22 Increased binding of the vitamin D receptor and increased histone H4 acetylation

23 the miR-498 genome, which is occupied by the vitamin D receptor and its coactivators.

24 receptor DAF-12, a homolog of the mammalian vitamin D receptor and liver X receptor.

25 vation of TLR, that induce expression of the vitamin D receptor and localized synthesis of 1,25(OH)(2

26 aled new insights into the regulation of the vitamin D receptor and new targets for its action.

27 genomic effects; the latter mediated via the vitamin D receptor and other transcription factors.

28 was markedly suppressed in mice lacking the vitamin D receptor and partially suppressed in vitamin D

29 ted with IL-23 plus IL-1beta upregulated the vitamin D receptor and responded to 1,25D with downregul

30 25-hydroxyvitamin D promoted binding of the vitamin D receptor and retinoid X receptor to the promot

31 Induction of the vitamin D receptor and the 1-alpha-hydroxylase genes was

32 n macrophages up-regulated expression of the vitamin D receptor and the vitamin D-1-hydroxylase genes

33 Whereas both the vitamin D receptor and vitamin D regulate iNKT cells, th

34 very that every tissue in the human body has vitamin D receptors and that vitamin D has pleiotropic e

35 Importantly, nuclear levels of CTSL, vitamin D receptor, and 53BP1 emerged as a novel triple

36 typed for TaqI and FokI polymorphisms of the vitamin D receptor, and interaction analyses were done t

37 educed concentrations of calcium-sensing and vitamin D receptors, and altered mRNA-binding protein ac

38 between consensus DNA motifs for binding of vitamin D receptor, AP1 and ELK1.

39 hese results indicate that vitamin D and the vitamin D receptor are required for the development of E

40 ion (SLC20A2, whose promoter has a predicted vitamin D receptor binding site, and XPR1), and one unas

41 o RUNX2, C/EBPbeta, retinoid X receptor, and vitamin D receptor binding sites, whereas adipocyte diff

42 ese lactation-induced increases, and reduced vitamin D receptor binding to promoter regions of Ca(2+)

43 hrough suppression of SOCS3 and induction of vitamin D receptor binding with the vitamin D-response e

44 key locus responsible for skin color, with a vitamin D receptor-binding interval.

45 oteinuric activity and podocytes express the vitamin D receptor, but whether vitamin D signaling in p

46 disrupting the interaction between SMAD3 and vitamin D receptor by altering SMAD3 ubiquitination.

47 ed by 1,25-dihydroxyvitamin D binding to the vitamin D receptor caused the decrease in hepcidin mRNA

48 1,25(OH)2D3 binds the vitamin D receptor complex present in many immune popula

49 25(OH)2D3] and GHS rats have increased renal vitamin D receptor content, the current study was undert

50 but engaged distinct TGF-beta-dependent and vitamin D receptor-dependent (VDR-dependent) pathways, r

51 Vitamin D receptor-dependent changes in the expression o

52 IL-1beta in macrophages, and therefore-in a vitamin D receptor-dependent manner-inhibited the abilit

53 rast, activators of PPAR-alpha, RAR, RXR, or vitamin D receptor did not alter ABCA12 expression.

54 precipitation analysis demonstrated that the vitamin D receptor directly binds to the TRPC6 promoter.

55 1 alpha-hydroxylase, 24-hydroxylase, and the vitamin D receptor, each of which was regulated by 1,25(

56 Combined activation of beta-catenin and the vitamin D receptor enhanced differentiation of sebaceous

57 most, demonstrating the greatest increase in vitamin D receptor expression after cholecalciferol.

58 sis, as well as E(2)-mediated enhancement of vitamin D receptor expression in the inflamed CNS.

59 sed serum 25(OH)D levels four-fold, monocyte vitamin D receptor expression three-fold, and 24-hydroxy

60 In this study, we found that vitamin D receptor expression was induced in a CD4+ effe

61 However, antibodies to the vitamin D receptor failed to block 1,25D(3)-stimulated c

62 Vitamin D receptor FokI and BsmI genotypes and the (I/D)

63 n the MKP5 promoter that associated with the vitamin D receptor following 1,25D treatment.

64 sure, the principal source of vitamin D, and vitamin D receptor gene (VDR) polymorphisms (FokI, TaqI,

65 No single-nucleotide polymorphism at the vitamin D receptor gene met our corrected significance t

66 lated by genetic factors, including specific vitamin D receptor gene polymorphisms.

67 igh-resolution SNP, haplotype and LD maps of vitamin D receptor gene region in large samples from fiv

68 ly correlated with the mRNA level of a known vitamin D receptor gene target, calbindin-D9K.

69 Polymorphisms of the Fok1 vitamin D receptor gene were determined.

70 gging single-nucleotide polymorphisms in the vitamin D receptor gene, requiring P < .002 (0.05 divide

71 elopment and severity and that variations in vitamin D receptor genes are associated with asthma susc

72 addition to the estrogen receptor alpha and vitamin D receptor genes.

73 Because folate-related and vitamin D-receptor genetic variants have been associated

74 ses were done to assess the influence of the vitamin D receptor genotype on response to vitamin D(3).

75 ed colorectal adenomas may vary according to vitamin D receptor genotype.

76 ral lines of evidence, including the role of vitamin D receptor genotypes, malnutrition's effects on

77 Vitamin D receptors have a broad tissue distribution tha

78 Vitamin D receptors have been found in all the major car

79 re associated with retinoic acid receptor or vitamin D receptor heterodimers by any of the agonists.

80 podocin promoter to target Flag-tagged human vitamin D receptor (hVDR) to podocytes in DBA/2J mice.

81 25(OH)(2)-vitamin D(3) (1,25D)/human nuclear vitamin D receptor (hVDR) transcription initiation compl

82 ar receptor superfamily, including the human vitamin D receptor (hVDR).

83 were (i) vitamin D-deficient, (ii) minus the vitamin D receptor, (iii) minus a vitamin D 25-hydroxyla

84 Conversely, deletion of the vitamin D receptor in macrophages from diabetic patients

85 Conversely, deletion of the vitamin D receptor in macrophages from diabetic patients

86 Conditional knockout of the vitamin D receptor in myeloid cells but not the endothel

87 ciated receptor gamma, liver X receptor, and vitamin D receptor in shaping the immune and metabolic f

88 f the ligand binding domain (LBD) of the rat vitamin D receptor in ternary complexes with a synthetic

89 Vitamin D receptor inactivation leads to hyperinflammato

90 Overexpression of a dominant negative vitamin D receptor inhibited 1,25(OH)(2)D(3) stimulation

91 Blocking the vitamin D receptor, inhibiting CYP27B1, or limiting 25D3

92 previously shown that the activation of the vitamin D receptor inhibits IgE production and that B ce

93 Using VDR affinity beads, the vitamin D receptor interacting protein (DRIP)/mediator c

94 roid hormone receptor-associated protein 220/vitamin D receptor-interacting protein 205/mediator 1, a

95 plex from primary keratinocytes (KCs) as the vitamin D receptor-interacting protein complex.

96 se; MTHFR:C677T), and the vitamin D pathway (vitamin D receptor: Intron8G/T;).

97 In the intestine, the vitamin D receptor is activated by 1alpha, 25-dihydroxyv

98 eptor and vitamin D regulate iNKT cells, the vitamin D receptor is required for both iNKT cell functi

99 The nuclear vitamin D receptor is widely expressed in tissues, but h

100 xylase that converts vitamin D3 to an active vitamin D receptor ligand; P=1.4x10(-5)).

101 In addition to being a ligand for the vitamin D receptor, lithocholic acid is also a substrate

102 Egr-1, Egr-2, Vitamin D Receptor, MafB/c: Fos and PU.1:interferon regu

103 The vitamin D receptor-mediated increase in Smad3 expression

104 but not TLR4, stimulation markedly inhibited vitamin D receptor mRNA and protein expression, selectiv

105 Unlike the mineralization defect in Vitamin D receptor-null mice, the mineralization defect

106 d, genotypes in that pathway were important: vitamin D receptor (odds ratio [OR], 6.85 [95% confidenc

107 alcitriol, even though it binds to the human vitamin D receptor only about 1% as well as calcitriol.

108 Small interfering RNA silencing of the vitamin D receptor or SRC3 blocked the induction of cath

109 The vitamin D receptor participates in the control of IgE cl

110 ssing dentin matrix protein 1 (DMP1) via the vitamin D receptor pathway.

111 articipants with the tt genotype of the TaqI vitamin D receptor polymorphism.

112 Studies of vitamin D receptor polymorphisms have found that not all

113 In contrast, vitamin D receptor polymorphisms were not significantly

114 erleukin-6, tumor necrosis factor-alpha, and vitamin D receptor polymorphisms.

115 id activated proteins: farnesoid X receptor, vitamin D receptor, pregnane X receptor, and TGR5.

116 was 300 times more active in binding to the vitamin D receptor protein, 30 times more effective in c

117 The wide distribution of the vitamin D receptor provides a number of clinical targets

118 Expression of epidermal IL1f6, S100a8, vitamin D receptor, repetin, and major histocompatibilit

119 vity of VDR toward specific target genes.The vitamin D receptor/retinoid X receptor-alpha heterodimer

120 The vitamin D receptor/retinoid X receptor-alpha heterodimer

121 minor alleles for SNPs in genes encoding the vitamin D receptor (rs4334089, rs11568820) and 25-hydrox

122 These results identify reduced vitamin D receptor signaling as a potential mechanism un

123 In this article, we report that vitamin D receptor signaling attenuates TLR-mediated inf

124 Although both cell types have an intact vitamin D receptor-signaling axis, this study demonstrat

125 vous system relapse, among the HR group, the vitamin D receptor start site (P = .02) and intron 8 gen

126 , 1alpha,25-dihydroxyvitamin D, binds to the vitamin D receptor that regulates numerous genes involve

127 the development of synthetic ligands of the vitamin D receptor that target the TGF-beta-SMAD signali

128 , bone, and kidney and overexpression of the vitamin D receptor, thereby reproducing the human phenot

129 -hydroxylase, a gene repressed by unliganded vitamin D receptor through its interaction with N-CoR, w

130 ith DAC and TSA increases the recruitment of vitamin D receptor to the CYP24A1 promoter.

131 These data may explain the recruitment of vitamin D receptor to the promoter region in MDEC but no

132 ulates B cells in vitro and mice without the vitamin D receptor (VDR knockout [KO]) have high serum I

133 or more >/=4 mm pockets were associated with vitamin D receptor (VDR) (rs2228570, P = 0.002, q = 0.04

134 P sequence analysis of binding sites for the vitamin D receptor (VDR) across the proximal intestine i

135 5D is competed by ZK159222, an antagonist of vitamin D receptor (VDR) action, and can occur in the pr

136 Vitamin D receptor (VDR) activation in HSCs inhibits liv

137 ween observational studies that suggest that vitamin D receptor (VDR) activators provide a survival a

138 Here, we document that the vitamin D receptor (VDR) acts as a master transcriptiona

139 e of 1,25D in all cell types, while use of a vitamin D receptor (VDR) agonist (EB1089) and antagonist

140 o determined if corneas contain mRNA for the vitamin D receptor (VDR) and 1alpha-hydroxylase, the enz

141 e podocyte injury through down regulation of vitamin D receptor (VDR) and activation of renin angiote

142 The vitamin D receptor (VDR) and its ligand 1,25-OH2-VD3 (ca

143 ome analysis revealed an association between vitamin D receptor (VDR) and lipid metabolism in human t

144 The vitamin D receptor (VDR) and p65 formed a complex in tub

145 )D interacts with breast tumor expression of vitamin D receptor (VDR) and retinoid X receptor-alpha (

146 inhibitory interaction between the inactive vitamin D receptor (VDR) and Stat1, which was released u

147 ility to inhibit the interaction between the vitamin D receptor (VDR) and steroid receptor coactivato

148 ls responded by increasing expression of the vitamin D receptor (VDR) and the cell cycle regulatory m

149 Vitamin D receptor (VDR) and the megalin gene polymorphi

150 ived DNA precipitated with antibodies to the vitamin D receptor (VDR) and the retinoid X receptor (RX

151 cts by inhibiting cell growth and increasing vitamin D receptor (VDR) and VDR-mediated transcription.

152 e idea that 1,25-dihydroxyvitamin D3 and the vitamin D receptor (VDR) are involved in regulating skin

153 Ligand-independent actions of the vitamin D receptor (VDR) are required for normal post-mo

154 is report, we explore the interaction of the vitamin D receptor (VDR) at regulatory sites within both

155 3-E1 cells, and assessed localization of the vitamin D receptor (VDR) at sites of action on a genome-

156 The vitamin D receptor (VDR) belongs to the superfamily of s

157 imed to investigate the relationship between vitamin D receptor (VDR) binding in lymphoblastoid cell

158 in vitro assays, vitamin D treatment led to vitamin D receptor (VDR) binding in the promoter region

159 here was evidence of interaction between the vitamin D receptor (VDR) BsmI genotype and serum 25-hydr

160 evidence has shown that the ligand-activated vitamin D receptor (VDR) can transcriptionally induce th

161 Vitamin D receptor (VDR) deficiency (knockout [KO]) resu

162 Vitamin D and vitamin D receptor (VDR) deficiency results in severe sy

163 Global vitamin D receptor (VDR) deletion exaggerates colitis, b

164 lammatory bowel disease (IBD), whereas IL-10/vitamin D receptor (VDR) double KO mice developed fulmin

165 er, we discovered a marked downregulation of vitamin D receptor (VDR) during OIS, and a role for the

166 Recently, we found that vitamin D receptor (VDR) enhanced Claudin-2 expression i

167 dy was to identify determinants of placental vitamin D receptor (VDR) expression and placental calciu

168 The effect of A. fumigatus on nuclear vitamin D receptor (VDR) expression was investigated usi

169 Suppression of endogenous vitamin D receptor (VDR) expression with siRNAs signific

170 ates breast cancer growth through regulating vitamin D receptor (VDR) expression.

171 The vitamin D receptor (VDR) FokI AA genotype and retinoid X

172 single nucleotide polymorphisms (SNP) in the vitamin D receptor (VDR) gene (Fok1, Bsm1, Cdx2) were as

173 study on bone mineral density (BMD) for the vitamin D receptor (VDR) gene and find that VDR is signi

174 The vitamin D receptor (VDR) gene has been involved in the m

175 Polymorphisms of the vitamin D receptor (VDR) gene have been implicated in su

176 Several polymorphisms in the vitamin D receptor (VDR) gene have been reported to be a

177 x technology we have selectively deleted the vitamin D receptor (VDR) gene in the cardiac myocyte in

178 Genetic inactivation of the vitamin D receptor (VDR) gene leads to hypocalcemia, sec

179 on between 25-OHD level and variation at the vitamin D receptor (VDR) gene locus.

180 xamine sun exposure and its interaction with vitamin D receptor (VDR) gene variants on breast cancer

181 base substitution was found in exon 6 in the vitamin D receptor (VDR) gene.

182 der caused by mutations in hairless (HR) and vitamin D receptor (VDR) genes, respectively.

183 Vitamin D receptor (VDR) has been widely detected in the

184 We report that deletion of the vitamin D receptor (VDR) in hematopoietic cells did not

185 in the characterization of Vitamin D and the Vitamin D receptor (VDR) in immune function.

186 of the mediator complex as a coactivator for vitamin D receptor (VDR) in keratinocytes.

187 Emerging data suggest a role for the vitamin D receptor (VDR) in lipogenesis and adipocyte di

188 We demonstrate a role of the vitamin D receptor (VDR) in reducing cerebral soluble an

189 To address the role of the vitamin D receptor (VDR) in renal fibrogenesis, we subje

190 es, we analyzed the consequences of specific vitamin D receptor (Vdr) inactivation in the intestine a

191 The physiological ligand for the vitamin D receptor (VDR) is 1,25-dihydroxyvitamin D(3).

192 Vitamin D receptor (VDR) is a ligand (vitamin D(3))-depe

193 Vitamin D receptor (VDR) is a ligand-dependent transcrip

194 The vitamin D receptor (VDR) is a ligand-responsive transcri

195 The vitamin D receptor (VDR) is a nuclear hormone receptor t

196 The vitamin D receptor (VDR) is a nuclear hormone receptor t

197 The vitamin D receptor (VDR) is a nuclear receptor that medi

198 The vitamin D receptor (VDR) is a nuclear transcription fact

199 vitamins to the full-length rat recombinant vitamin D receptor (VDR) is either similar to or within

200 Here, we reveal that the vitamin D receptor (VDR) is expressed in stroma from hum

201 ster regulator of the hair follicle, and the vitamin D receptor (Vdr) is linked to coordinated contro

202 The data show that expression of the vitamin D receptor (VDR) is required for normal developm

203 The major physiological role of the vitamin D receptor (VDR) is the maintenance of mineral i

204 The vitamin D receptor (VDR) is the single known regulatory

205 Vitamin D receptor (VDR) knock-out (VDRKO) mice have def

206 Vitamin D receptor (VDR) knockdown partly abolished MART

207 experimental allergic asthma was induced in vitamin D receptor (VDR) knockout and in wild-type (WT)

208 The vitamin D receptor (VDR) ligand, 1,25 dihydroxyvitamin D

209 Vitamin D receptor (VDR) ligands are therapeutic agents

210 Here, we show that vitamin D receptor (VDR) ligands inhibit HSC activation

211 Vitamin D receptor (VDR) mutations in humans and mice ca

212 Little is known about the effects of the vitamin D receptor (VDR) on hepatic activity of human ch

213 nd thyroid hormone receptors (SMRT) from the vitamin D receptor (VDR) on those VDREs.

214 Targeted disruption in mice of the vitamin D receptor (VDR) or Runx2 results in marked inhi

215 in D metabolite uptake and activation of the vitamin D receptor (VDR) pathway in colon cancer cells t

216 Vitamin D receptor (VDR) plays an essential role in gast

217 conducted to assess the association between vitamin D receptor (VDR) polymorphisms and genetic susce

218 emiologic studies of vitamin D status and/or vitamin D receptor (VDR) polymorphisms and prostate canc

219 Vitamin D receptor (VDR) polymorphisms have been associa

220 onal 1,25-dihydroxyvitamin D (1,25D) and the vitamin D receptor (VDR) profoundly alter, through multi

221 1,25D3 alters vitamin D receptor (VDR) recruitment to the Cyp11a1 prom

222 Depletion of the vitamin D receptor (VDR) reduced these genoprotective ef

223 The vitamin D receptor (VDR) regulates a diverse set of gene

224 ihydroxyvitamin D3-bound [1,25(OH)2D3-bound] vitamin D receptor (VDR) specifically inhibits TGF-beta-

225 ting enzyme 1alpha-hydroxylase (CYP27B1) and vitamin D receptor (VDR) support anti-inflammatory respo

226 ydroxyvitamin D [25(OH)D] interacts with the vitamin D receptor (VDR) to decrease proliferation and i

227 D(3) (1,25(OH)(2)D(3)) hormone binds to the vitamin D receptor (VDR) to regulate gene expression.

228 The role of the vitamin D receptor (VDR) was also examined.

229 Vitamin D receptor (VDR) was significantly down-regulate

230 in cytochrome P-450 (CYP2R1)(rs10741657AG), vitamin D receptor (VDR)(rs2228570AG, rs1544410CT), olig

231 Treatment of mice with an active ligand of vitamin D receptor (VDR), 1,25-dihydroxyvitamin D(3) (1,

232 It is the ligand of the vitamin D receptor (VDR), a nuclear receptor with transa

233 The vitamin D receptor (VDR), an endocrine nuclear receptor

234 bly through genomic effects modulated by the vitamin D receptor (VDR), and autocrine/paracrine metabo

235 The enzyme is directly regulated by vitamin D receptor (VDR), and it is expressed mainly in

236 on of vitamin D, through its cognate nuclear vitamin D receptor (VDR), and its contribution to divers

237 xpression of the calcium receptor (CaR), the vitamin D receptor (VDR), and the P450 cytochromes, CYP2

238 cytokines by 1,25(OH)(2)D(3) was mediated by vitamin D receptor (VDR), as 1,25(OH)(2)D(3) had no effe

239 l small-molecule compounds that activate the vitamin D receptor (VDR), but are devoid of hypercalcemi

240 Colonic mucosal expression concentrations of vitamin D receptor (VDR), E-cadherin, zonula occluden 1

241 ition led to the hyperphosphorylation of the vitamin D receptor (VDR), enabling an interaction betwee

242 [1alpha,25(OH)(2)D(3)] and its receptor, the vitamin D receptor (VDR), has resulted in significant co

243 sion induced by BXL0124 was blocked by siRNA vitamin D receptor (VDR), indicating that the regulation

244 ve form of vitamin D that interacts with the vitamin D receptor (VDR), is a coordinate regulator of p

245 Although mast cells express the vitamin D receptor (VDR), it is not clear to what extent

246 n-containing transcription factor 2 (Runx2), vitamin D receptor (Vdr), SRY (sex-determining region Y)

247 It mediates these effects by binding to the vitamin D receptor (VDR), which belongs to the superfami

248 amin D, 1,25(OH)(2)D(3), are mediated by the vitamin D receptor (VDR), which heterodimerizes with ret

249 dihydroxyvitamin D, are mediated through the vitamin D receptor (VDR), which heterodimerizes with ret

250 calcitriol are mediated at least in part by vitamin D receptor (VDR), which is expressed in many tis

251 amin D3 exerts its effects by binding to the vitamin D receptor (VDR), which regulates transcription

252 on in binding of a transcription factor, the vitamin D receptor (VDR), whose activating ligand vitami

253 vitamin D3 (1,25(OH)2D3) are mediated by the vitamin D receptor (VDR), whose expression in bone cells

254 1,20S(OH)2D3 interacts with the vitamin D receptor (VDR), with similar potency to its na

255 Vitamin D receptor (VDR)-dependent mechanisms regulate h

256 Vitamin D receptor (VDR)-knockout mice develop severe hy

257 Absence of vitamin D receptor (VDR)-mediated PPARgamma suppression

258 ever, about the role of SWI/SNF and PRMTs in vitamin D receptor (VDR)-mediated transcription.

259 Vitamin D receptor (VDR)-null mice develop polyuria, but

260 Studies in vitamin D receptor (VDR)-null mice have demonstrated tha

261 a high fat diet-resistant lean phenotype of vitamin D receptor (VDR)-null mutant mice mainly due to

262 ifferentiation was reduced, and beta-catenin/vitamin D receptor (VDR)-regulated gene expression was m

263 Both VDREs bound the vitamin D receptor (VDR)-retinoid X receptor (RXR) compl

264 transcriptional repression, mediated by the vitamin D receptor (VDR).

265 g protein (VDRE-BP) or 1,25(OH)(2)D(3)-bound vitamin D receptor (VDR).

266 t are different from genomic effects via the vitamin D receptor (VDR).

267 The effect of 20(OH)D3 was dependent on the vitamin D receptor (VDR).

268 )) is the biologically active ligand for the vitamin D receptor (VDR).

269 coactivator for nuclear receptors including vitamin D receptor (VDR).

270 ous melanoma (CM) cells mediated through the vitamin D receptor (VDR).

271 dence that p63gamma specifically upregulates vitamin D Receptor (VDR).

272 l keratinocytes through interaction with the vitamin D receptor (VDR).

273 ults from a loss of function mutation in the vitamin D receptor (VDR).

274 atenin can be repressed by activation of the vitamin D receptor (VDR).

275 ) in the CAMP promoter that was bound by the vitamin D receptor (VDR).

276 ers with other nuclear receptors such as the vitamin D receptor (VDR).

277 erted via signaling mechanisms involving the vitamin D receptor (VDR).

278 everal NRs in OPCs and OLGs, one of which is vitamin D receptor (VDR).

279 r-binding protein beta (C/EBPbeta), OSX, and vitamin D receptor (VDR).

280 e in cancer development and acts through the vitamin D receptor (VDR).

281 ihydroxyvitamin D(3) (1,25(OH)(2)D(3)) via a vitamin D receptor (VDR)/retinoid X receptor (RXR) heter

282 cription factor binding sites identified two vitamin D receptor (VDR)/retinoid X receptor binding sit

283 at -312 (a DR4-type VDRE) could be bound by vitamin D receptor (VDR)/retinoid X receptor.

284 al keratinocytes, unliganded heterodimers of vitamin D receptor (VDR)/RXR-alpha and retinoic acid rec

285 lial cells signaling by the nuclear receptor Vitamin D Receptor (VDR, NR1I1) induces cell cycle arres

286 Calcitriol, acting through vitamin D receptors (VDR) in the parathyroid, suppresses

287 Vitamin D receptors (VDRs) are found within multiple tis

288 nociceptors ("pain-sensing" nerves) express vitamin D receptors (VDRs), suggesting responsiveness to

289 d this might be due to genetic variations in vitamin D receptors (VDRs).

290 ophosphate leads to the up-regulation of the vitamin D receptor via a pathway that involves the class

291 pression of vitamin D regulatory enzymes and vitamin D receptor was higher in Ksp-KL(-/-) mice than c

292 STAT3, Raf1, and PKCzeta, were increased and Vitamin D receptor was reduced in cancer cells.

293 STAT3, Raf1, and PKCzeta, were increased and vitamin D receptor was reduced in cancer cells.

294 When the vitamin D receptor was silenced with small interfering R

295 We also found that the vitamin D receptor was transiently expressed during MAC

296 ge in positioning of the 20R molecule in the vitamin D receptor when the 2-methylene group is present

297 tivity of VD3 is primarily attributed to the vitamin D receptor, whereas 5 affects Hh inhibition thro

298 transcription of PTH by associating with the vitamin D receptor, which heterodimerizes with retinoic

299 or hormonal receptors, namely PXR, AHR, and vitamin D receptor, which regulate major xenobiotic-meta

300 rmone transitorily down-regulates the kidney vitamin D receptor, which returns to normal levels with