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1 the diets used to promote the development of vitamin E deficiency.
2 tation which was significantly attenuated by vitamin E deficiency.
3 ulnerable than sympathetic nerves to chronic vitamin E deficiency.
4 pha-TTP gene are associated with ataxia with vitamin E deficiency, a disease characterized by periphe
7 eld stimulation (EFS) were not attenuated by vitamin E deficiency; at high stimulation frequencies, r
10 ene, encoding for TTP, result in ataxia with vitamin E deficiency (AVED) syndrome, typified by low vi
13 -susceptible apolipoprotein E knockout mice, vitamin E deficiency caused by disruption of the alpha-t
15 alpha-tocopherol transfer protein also cause vitamin E deficiency-defects in this protein cause an im
16 active oxygen species in two mouse models of vitamin E deficiency: dietary deficiency and a genetic m
17 at cause the hereditary disorder ataxia with vitamin E deficiency diminish the effect of tocopherol o
18 sophisticated genetic studies of humans with vitamin E deficiency have led to an understanding of thi
21 y oxidative stress due to either selenium or vitamin E deficiency increases cardiac damage in mice in
25 cerebellar Purkinje cells in patients having vitamin E deficiency states or diseases associated with
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