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1 quinone, a late biosynthetic intermediate of vitamin K1.
2 re fed a diet supplemented with warfarin and vitamin K1.
3 of the oxidized primary donor and a reduced vitamin K1.
4 ticoagulation clinic examined the effects of vitamin K1 100 mug daily on INR variability and found no
8 vitamin K deficiency and minidose warfarin, vitamin K1-2,3-epoxide, PIVKA-II, and percentage underca
12 rew on brucella blood agar supplemented with vitamin K1 and hemin and on brucella laked blood agar su
14 idant, we report here the novel finding that vitamin K1 and K2 (menaquinone-4) potently inhibit gluta
19 CAC progression for subjects with low serum vitamin K1 compared with subjects without extreme CAC pr
22 nge was +5 +/- 20 Agatston units (AU) in the vitamin K1 group (n = 40) and +44 +/- 13 AU in the place
23 nd the direct carboxylase inhibitor 2-chloro-vitamin K1 have no effect on the protective function of
26 To examine the association between dietary vitamin K1 intake and the risk of incident cataracts in
27 rticipants in the highest tertile of dietary vitamin K1 intake had a lower risk of cataracts than tho
31 he ability to convert dietary phylloquinone (vitamin K1) into menaquinone-4 (vitamin K2) and store th
32 of warfarin and administering 2.5 mg of oral vitamin K1 is a reliable, safe, and inexpensive way to r
33 e of our primary analysis suggests low serum vitamin K1 is associated with greater CAC progression, t
36 es of warfarin, administering 2.5 mg of oral vitamin K1, measuring the INR after 24 to 48 hours, and
41 anar with the ring) conformations of UQ- and vitamin K1- radicals is estimated to be sufficiently lar
42 needed to determine whether improving serum vitamin K1 reduces CAC progression, especially in hypert
43 pective study and two prospective studies of vitamin K1 supplementation (with doses ranging from 100
45 as to determine the effect of phylloquinone (vitamin K1) supplementation on CAC progression in older
46 of the preferential distribution of dietary vitamin K1 to the liver to preserve coagulation function
47 s with SR-BI and CD36 significantly enhanced vitamin K1 uptake, which was subsequently decreased by t
49 roteins is dependent on formation of reduced vitamin K1 (Vit.K1H2) in the endoplasmic reticulum (ER),
50 approach for the simultaneous estimation of vitamin K1 (VK1) and heparin via cascaded channel multia
55 Hypertension medication users with low serum vitamin K1 were more likely to have extreme CAC progress
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