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1 na and we show that PtchlacZ+/- mice exhibit vitreoretinal abnormalities resembling those found in BC
2 ill be useful in investigating the effect of vitreoretinal adhesion on ocular hemorrhage caused by in
3 erlie vitreous liquefaction and weakening of vitreoretinal adhesion.
4 T is highly sensitive to visualize posterior vitreoretinal and choroidal structures into a single ful
5           The societal costs associated with vitreoretinal and other ophthalmic interventions include
6                                         Many vitreoretinal and other ophthalmologic interventions con
7 e following conditions: continuous posterior vitreoretinal attachment (PVA), vitreomacular adhesion (
8             Inclusion criteria were complete vitreoretinal attachment, no ocular pathology other than
9                                      Primary vitreoretinal B-cell lymphoma and uveitis might be chara
10  authors speculate that the integrity of the vitreoretinal border is an important factor in preventin
11 January 2009 to December 2014, at 4 tertiary vitreoretinal centers in Italy were enrolled.
12 halmic data and operative information from 3 vitreoretinal centers were entered prospectively into an
13 ied 144 eyes of 72 consecutive patients in a vitreoretinal clinical practice, reviewing multimodal im
14                                              Vitreoretinal clinical practice.
15      In 37 patients, 65 eyes with CSC from 2 vitreoretinal clinical practices were imaged using ultra
16 T transformed the clinical management of the vitreoretinal conditions, iOCT has the potential to be a
17                                    Snowflake vitreoretinal degeneration (SVD, MIM 193230) is a develo
18 och syndrome, characterized by age-dependent vitreoretinal degeneration and occipital encephalocele.
19 ings provide an explanation for high myopia, vitreoretinal degeneration and retinal detachment seen i
20 mutation can cause SVD and further show that vitreoretinal degeneration can arise through mutations i
21                                    Snowflake vitreoretinal degeneration should be considered in the d
22 characterized by occipital skull defects and vitreoretinal degeneration.
23 se-region, suggests a novel pathogenesis for vitreoretinal degeneration.
24                      However, distinguishing vitreoretinal diffuse large B-cell lymphoma from uveitis
25                                              Vitreoretinal diffuse large B-cell lymphoma is a rare di
26 trations of HGF/SF increase in proliferative vitreoretinal disease and increase in turn with increase
27                                 The field of vitreoretinal disease and surgery has seen tremendous gr
28                                   A panel of vitreoretinal disease experts was the foundation of the
29 omy allows patients who often have bilateral vitreoretinal disease to come to a stable postoperative
30          The role of HGF/SF in proliferative vitreoretinal disease was investigated.
31  plana vitrectomy surgery and the underlying vitreoretinal disease will allow the surgeon to address
32 y (OCT) has improved the care of adults with vitreoretinal disease, and OCT angiography (OCTA) is dem
33 control group (>50 years of age) without any vitreoretinal disease.
34 recting corneal astigmatism in patients with vitreoretinal diseases and cataract.
35 ent data on the economics of telemedicine in vitreoretinal diseases.
36 g the cost-effectiveness of telemedicine for vitreoretinal diseases.
37 emselves as specializing in the treatment of vitreoretinal diseases.
38 s review is to examine the macroeconomics of vitreoretinal diseases.
39 nvolved in the pathogenesis of PDR and other vitreoretinal diseases.
40  approach to the surgical management of this vitreoretinal disorder.
41 wngrowth, iridocorneal endothelial syndrome, vitreoretinal disorders, and penetrating keratoplasty.
42 wn to be present and active in proliferative vitreoretinal disorders, suggesting that Muller cells re
43 ity of Muller cells and its association with vitreoretinal disorders, we examined morphology, propaga
44 al tissue elastic modulus may have a role in vitreoretinal disorders.
45 ular developmental defects, including severe vitreoretinal dysplasia, optic nerve hypoplasia, persist
46  These results indicate that early bilateral vitreoretinal eye pathology coupled with skeletal fragil
47      In all cases, the primary surgeon was a vitreoretinal fellow.
48 tinal breaks and RD following primary PPV by vitreoretinal fellows is low and comparable to that of f
49 plana vitrectomy (PPV) is often performed by vitreoretinal fellows.
50 morbidities, presenting symptoms and vision, vitreoretinal findings, treatment regimens, culture data
51 tcomes were worse for patients who underwent vitreoretinal follow-up surgery, likely because of mecha
52 e also found in the ipsilateral vitreous and vitreoretinal interface (but not destructive chorioretin
53  of the major NO metabolite, nitrate, at the vitreoretinal interface (VRI) of normal and aged rat mod
54 hether an association between changes at the vitreoretinal interface (VRI), in particular traction (V
55 OCT or TD-OCT have similar ability to assess vitreoretinal interface abnormalities and outcomes of en
56 (OCT) protocol designed to evaluate formally vitreoretinal interface abnormalities on scans obtained
57                                    Eyes with vitreoretinal interface abnormalities or that had underg
58          There was no apparent damage of the vitreoretinal interface after unsuccessful pharmacologic
59 ght into the range of retinal defects at the vitreoretinal interface and fovea, which is not only use
60 use of rhegmatogenous retinal detachment and vitreoretinal interface disorders, as well as potential
61                 Quantitative and morphologic vitreoretinal interface features were assessed reproduci
62 image modification process that enhances the vitreoretinal interface first and then the choroid, whil
63 ides wide-field 3-dimensional information of vitreoretinal interface in diabetic eyes.
64 trials, and can facilitate identification of vitreoretinal interface pathology during care of individ
65                                          The vitreoretinal interface, which often consisted of lamell
66 t fibronectin and laminin, components of the vitreoretinal interface.
67 thin the retina and is accentuated along the vitreoretinal interface.
68  societal cost perspective associated with a vitreoretinal intervention.
69                                              Vitreoretinal interventions account for only a small por
70                                              Vitreoretinal interventions are generally cost-effective
71                                         Most vitreoretinal interventions are very cost effective usin
72 arative effectiveness and cost-effectiveness vitreoretinal interventions assessed in the US healthcar
73                                         Most vitreoretinal interventions confer considerable patient
74                              The majority of vitreoretinal interventions confer considerable value an
75                                              Vitreoretinal interventions have good to excellent compa
76            The average cost-utility of these vitreoretinal interventions is US$23 026/QALY (SD US$24
77 , and cost-utility analyses encompassing the vitreoretinal interventions of the following: (1) laser
78 est that ophthalmic interventions, including vitreoretinal interventions, are cost effective.
79 tive effectiveness and cost-effectiveness of vitreoretinal interventions, measured in quality-adjuste
80 ure was spent on ocular diseases and 0.3% on vitreoretinal interventions.
81 ngly used to study the cost-effectiveness of vitreoretinal interventions.
82 cine and its impact, or potential impact, on vitreoretinal interventions.
83                       Not all lymphomas with vitreoretinal involvement represent primary intraocular
84 extracellular proteins that characterize the vitreoretinal junction (fibronectin, laminin) and vitreo
85 ent comparative effectiveness studies in the vitreoretinal literature.
86  associated with overall survival in primary vitreoretinal lymphoma (PVRL) and ocular adnexal (OA)-uv
87        The best treatment option for primary vitreoretinal lymphoma (PVRL) without signs of central n
88 assessed the frequency of MYD88 mutations in vitreoretinal lymphoma (VRL) and their diagnostic potent
89 udy included 17 diffuse large B-cell primary vitreoretinal lymphoma and 12 uveitis patients.
90 maging and ultimately diagnosed with primary vitreoretinal lymphoma and/or primary central nervous sy
91                     The diagnosis of primary vitreoretinal lymphoma and/or primary central nervous sy
92 opathy, can precede the diagnosis of primary vitreoretinal lymphoma or primary central nervous system
93                                              Vitreoretinal lymphoma remains a clinical diagnostic cha
94 icroRNA-197, and microRNA-132 in the primary vitreoretinal lymphoma vitreous and higher microRNA-155,
95 had no history of lymphoma; the diagnosis of vitreoretinal lymphoma was followed by DLBCL after a lym
96 on in systemic lymphoma, mimicking a primary vitreoretinal lymphoma, is extremely rare.
97                    Selected samples (primary vitreoretinal lymphoma, n = 3; uveitis, n = 3) were arra
98 luation in all patients with newly diagnosed vitreoretinal lymphoma.
99 kines are promising diagnostic biomarkers of vitreoretinal lymphoma.
100 prognosis is particularly poor compared with vitreoretinal lymphomas even in response to chemotherapy
101                                          The vitreoretinal manifestations include anterior uveitis, v
102                 LRDI has not been applied to vitreoretinal ophthalmological problems previously.
103      Fifteen centers with both pediatric and vitreoretinal ophthalmologists participating in level 3
104 ed macular degeneration (AMD), without other vitreoretinal pathology.
105                                          The vitreoretinal pharmacokinetic profiles were similar betw
106 f culture-proven endophthalmitis in a single vitreoretinal practice over the course of 3 years and de
107                                              Vitreoretinal practice patterns changed significantly fr
108 ries at a single tertiary referral pediatric vitreoretinal practice.
109 ctive single-center case series at a private vitreoretinal practice.
110               CLINICAL RELEVANCE: To analyze vitreoretinal procedural trends, which may indicate stan
111                                   To analyze vitreoretinal procedural trends, which may indicate stan
112  identify the number of allowed services for vitreoretinal procedures and commonly used pharmacologic
113 his study was to identify changes in use for vitreoretinal procedures by measuring the number of allo
114                                              Vitreoretinal procedures grew 6-fold from 2000 to 2014.
115 itreal injections accounted for 0.55% of all vitreoretinal procedures in 2000 and increased to 87% in
116 e and 24 (37.5%) patients who had endoscopic vitreoretinal procedures initially before undergoing a c
117  been used successfully, temporarily, during vitreoretinal procedures.
118 cle highlights some of the current trends in vitreoretinal research that promise to be revolutionary
119          Based on recent advances in ongoing vitreoretinal research, the spectrum of treatable retina
120       OCT facilitates the detection of early vitreoretinal separation that indicates initial PVD.
121  negotiations between representatives of the Vitreoretinal Society of India (VRSI) and India's Centra
122 aterality of FH were recorded by 1 pediatric vitreoretinal specialist.
123 eolysis were reported in 15 patients by 7 US vitreoretinal specialists during the study period.
124                        Expedited referral to vitreoretinal specialists is recommended for management
125 inopathy, two conditions commonly treated by vitreoretinal specialists, are projected to affect more
126 er diabetic vitrectomy, and subsequently the vitreoretinal surgeon attempted to spare the lens.
127                                          The vitreoretinal surgeon may consider rendering an eye nonp
128 t could provide enhanced information for the vitreoretinal surgeon.
129 ickness macular holes (MHs) is important for vitreoretinal surgeons and their patients.
130             There remains no consensus among vitreoretinal surgeons regarding the optimal management
131                              This may enable vitreoretinal surgeons to benchmark their case-mix and o
132                       These results may help vitreoretinal surgeons to benchmark their intraoperative
133 ertaken by specialized ocular oncologists or vitreoretinal surgeons with experience in managing this
134  experts (3 pediatric ophthalmologists and 6 vitreoretinal surgeons) participated in the study.
135 reoretinopathy (PVR) is a serious problem in vitreoretinal surgeries.
136          Adverse events were associated with vitreoretinal surgery and immunosuppression.
137 e predictability in patients with a previous vitreoretinal surgery can be as good as in uncomplicated
138                          The role of iOCT in vitreoretinal surgery continues to be defined by active
139   A total of 20 of 739 eyes (2.7%) underwent vitreoretinal surgery for complications arising from cho
140 patients who subsequently required secondary vitreoretinal surgery for complications arising from suc
141 Inc, Irvine, CA) can improve the outcomes of vitreoretinal surgery for established proliferative vitr
142 review of consecutive patients who underwent vitreoretinal surgery for myopic traction maculopathy by
143                         Only 2 eyes required vitreoretinal surgery for nonclearing vitreous hemorrhag
144                       Patients who underwent vitreoretinal surgery had overall worse visual outcomes.
145 f 360 patients undergoing 27-gauge PPV for a vitreoretinal surgery indication.
146           Eyes undergoing 27 gauge PPV for a vitreoretinal surgery indication.
147             However, Cibis' contributions to vitreoretinal surgery only occupied the last 10 years of
148                              Eyes with prior vitreoretinal surgery or laser or anti-vascular endothel
149 erative sickle retinopathy were managed with vitreoretinal surgery over a 12-year period at a single
150 l of 565 eyes were included in this study of vitreoretinal surgery performed from April 2011 to June
151           Fifty-eyes of 50 adults undergoing vitreoretinal surgery were enrolled.
152                       Patients who underwent vitreoretinal surgery were randomized into 3 groups base
153 rom 13 sites where data on both cataract and vitreoretinal surgery were recorded on the same electron
154                          The indications for vitreoretinal surgery were recorded, as were the locatio
155 n the US and 96% completed a fellowship (25% vitreoretinal surgery, 22% cornea and external disease,
156 ta recorded included phakic status, previous vitreoretinal surgery, and anterior chamber (AC) cells a
157  adjunct for clinical decision making during vitreoretinal surgery, and OCT angiography (OCTA) has pr
158 ative optical coherence tomography (iOCT) in vitreoretinal surgery, assess the current state-of-the a
159 xpanding the scope and improving outcomes in vitreoretinal surgery.
160 nometry are in excellent agreement following vitreoretinal surgery.
161  and 0.5% bupivacaine in patients undergoing vitreoretinal surgery.
162  its role in the modern era of microincision vitreoretinal surgery.
163 issociated) light, is redefining its role in vitreoretinal surgery.
164 1911-1965) was one of the pioneers of modern vitreoretinal surgery.
165 was recovered from 47 individuals undergoing vitreoretinal surgery: 16 had nonproliferative diabetic
166                                     Numerous vitreoretinal surgical conditions and procedures have be
167 h removal of silicone oil without additional vitreoretinal surgical intervention at 6 months.
168                                              Vitreoretinal surgical repair for this condition is succ
169                    Continuous refinements in vitreoretinal surgical techniques and an increasing numb
170                                              Vitreoretinal surgical techniques for the management of
171                             With advances in vitreoretinal surgical techniques, however, the indicati
172                                  With proper vitreoretinal surgical techniques, posteriorly dislocate
173 ll further expand the horizon of iOCT in the vitreoretinal surgical theater.
174                                              Vitreoretinal symptoms of DLBCL in patients with systemi
175                                     Modified vitreoretinal techniques also have been developed, allow
176 s not unusual to require relatively advanced vitreoretinal techniques to achieve long-term surgical s
177 omitantly with more experience using various vitreoretinal techniques to manage these complicated cas
178 eral granulomas (57.1%), vasculitis (57.1%), vitreoretinal traction (57.1%), and chronic macular edem
179         Previous studies have suggested that vitreoretinal traction (VRT) may contribute to the progr
180 pithelium include typical findings of peaked vitreoretinal traction and retinal disorganization with
181 hes a new reproducible technique to quantify vitreoretinal traction during vitrectomy and demonstrate
182                                              Vitreoretinal traction was found in 39 eyes (40%).
183 d macular edema, central serous retinopathy, vitreoretinal traction, and age-related macular degenera
184 c traction maculopathy, epiretinal membrane, vitreoretinal traction, optic or scleral pit, or advance
185 ing into zone III (p = 0.023) and associated vitreoretinal trauma (p = 0.008).
186 posterior to rectus insertion and associated vitreoretinal trauma can adversely affect the outcome in
187 erative diabetic retinopathy at 16 different vitreoretinal units in the United Kingdom.
188   BRI or VEH was administered by gavage, and vitreoretinal vascular endothelial growth factor (VEGF)
189 findings, retinal ganglion cell (RGC) count, vitreoretinal vascular endothelial growth factor (VEGF)
190             BRI produced marked decreases in vitreoretinal VEGF and inhibition of BRB breakdown in di
191  of ischemia responsible for upregulation of vitreoretinal VEGF and thus reduce vascular leakage and
192 sure to high oxygen significantly attenuated vitreoretinal VEGF concentrations, retinal vascular leak
193       BRI treatment significantly attenuated vitreoretinal VEGF concentrations, retinal vascular leak
194 diabetic animals but significantly decreased vitreoretinal VEGF expression and BRB breakdown to level
195 tic rats demonstrated significantly elevated vitreoretinal VEGF expression, vitreal glutamate concent
196  for all), despite negligible differences in vitreoretinal VEGF levels at the time of evaluation (P >
197 atment also significantly decreased elevated vitreoretinal VEGF protein levels and retinal BRB leakag
198 retinopathy with neurodegeneration, elevated vitreoretinal VEGF protein levels, and increased BRB bre
199                                              Vitreoretinal VEGF protein, vitreal glutamate, and BRB b

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