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1 na and we show that PtchlacZ+/- mice exhibit vitreoretinal abnormalities resembling those found in BC
2 ill be useful in investigating the effect of vitreoretinal adhesion on ocular hemorrhage caused by in
4 T is highly sensitive to visualize posterior vitreoretinal and choroidal structures into a single ful
7 e following conditions: continuous posterior vitreoretinal attachment (PVA), vitreomacular adhesion (
10 authors speculate that the integrity of the vitreoretinal border is an important factor in preventin
12 halmic data and operative information from 3 vitreoretinal centers were entered prospectively into an
13 ied 144 eyes of 72 consecutive patients in a vitreoretinal clinical practice, reviewing multimodal im
16 T transformed the clinical management of the vitreoretinal conditions, iOCT has the potential to be a
18 och syndrome, characterized by age-dependent vitreoretinal degeneration and occipital encephalocele.
19 ings provide an explanation for high myopia, vitreoretinal degeneration and retinal detachment seen i
20 mutation can cause SVD and further show that vitreoretinal degeneration can arise through mutations i
26 trations of HGF/SF increase in proliferative vitreoretinal disease and increase in turn with increase
29 omy allows patients who often have bilateral vitreoretinal disease to come to a stable postoperative
31 plana vitrectomy surgery and the underlying vitreoretinal disease will allow the surgeon to address
32 y (OCT) has improved the care of adults with vitreoretinal disease, and OCT angiography (OCTA) is dem
41 wngrowth, iridocorneal endothelial syndrome, vitreoretinal disorders, and penetrating keratoplasty.
42 wn to be present and active in proliferative vitreoretinal disorders, suggesting that Muller cells re
43 ity of Muller cells and its association with vitreoretinal disorders, we examined morphology, propaga
45 ular developmental defects, including severe vitreoretinal dysplasia, optic nerve hypoplasia, persist
46 These results indicate that early bilateral vitreoretinal eye pathology coupled with skeletal fragil
48 tinal breaks and RD following primary PPV by vitreoretinal fellows is low and comparable to that of f
50 morbidities, presenting symptoms and vision, vitreoretinal findings, treatment regimens, culture data
51 tcomes were worse for patients who underwent vitreoretinal follow-up surgery, likely because of mecha
52 e also found in the ipsilateral vitreous and vitreoretinal interface (but not destructive chorioretin
53 of the major NO metabolite, nitrate, at the vitreoretinal interface (VRI) of normal and aged rat mod
54 hether an association between changes at the vitreoretinal interface (VRI), in particular traction (V
55 OCT or TD-OCT have similar ability to assess vitreoretinal interface abnormalities and outcomes of en
56 (OCT) protocol designed to evaluate formally vitreoretinal interface abnormalities on scans obtained
59 ght into the range of retinal defects at the vitreoretinal interface and fovea, which is not only use
60 use of rhegmatogenous retinal detachment and vitreoretinal interface disorders, as well as potential
62 image modification process that enhances the vitreoretinal interface first and then the choroid, whil
64 trials, and can facilitate identification of vitreoretinal interface pathology during care of individ
72 arative effectiveness and cost-effectiveness vitreoretinal interventions assessed in the US healthcar
77 , and cost-utility analyses encompassing the vitreoretinal interventions of the following: (1) laser
79 tive effectiveness and cost-effectiveness of vitreoretinal interventions, measured in quality-adjuste
84 extracellular proteins that characterize the vitreoretinal junction (fibronectin, laminin) and vitreo
86 associated with overall survival in primary vitreoretinal lymphoma (PVRL) and ocular adnexal (OA)-uv
88 assessed the frequency of MYD88 mutations in vitreoretinal lymphoma (VRL) and their diagnostic potent
90 maging and ultimately diagnosed with primary vitreoretinal lymphoma and/or primary central nervous sy
92 opathy, can precede the diagnosis of primary vitreoretinal lymphoma or primary central nervous system
94 icroRNA-197, and microRNA-132 in the primary vitreoretinal lymphoma vitreous and higher microRNA-155,
95 had no history of lymphoma; the diagnosis of vitreoretinal lymphoma was followed by DLBCL after a lym
100 prognosis is particularly poor compared with vitreoretinal lymphomas even in response to chemotherapy
106 f culture-proven endophthalmitis in a single vitreoretinal practice over the course of 3 years and de
112 identify the number of allowed services for vitreoretinal procedures and commonly used pharmacologic
113 his study was to identify changes in use for vitreoretinal procedures by measuring the number of allo
115 itreal injections accounted for 0.55% of all vitreoretinal procedures in 2000 and increased to 87% in
116 e and 24 (37.5%) patients who had endoscopic vitreoretinal procedures initially before undergoing a c
118 cle highlights some of the current trends in vitreoretinal research that promise to be revolutionary
121 negotiations between representatives of the Vitreoretinal Society of India (VRSI) and India's Centra
125 inopathy, two conditions commonly treated by vitreoretinal specialists, are projected to affect more
133 ertaken by specialized ocular oncologists or vitreoretinal surgeons with experience in managing this
137 e predictability in patients with a previous vitreoretinal surgery can be as good as in uncomplicated
139 A total of 20 of 739 eyes (2.7%) underwent vitreoretinal surgery for complications arising from cho
140 patients who subsequently required secondary vitreoretinal surgery for complications arising from suc
141 Inc, Irvine, CA) can improve the outcomes of vitreoretinal surgery for established proliferative vitr
142 review of consecutive patients who underwent vitreoretinal surgery for myopic traction maculopathy by
149 erative sickle retinopathy were managed with vitreoretinal surgery over a 12-year period at a single
150 l of 565 eyes were included in this study of vitreoretinal surgery performed from April 2011 to June
153 rom 13 sites where data on both cataract and vitreoretinal surgery were recorded on the same electron
155 n the US and 96% completed a fellowship (25% vitreoretinal surgery, 22% cornea and external disease,
156 ta recorded included phakic status, previous vitreoretinal surgery, and anterior chamber (AC) cells a
157 adjunct for clinical decision making during vitreoretinal surgery, and OCT angiography (OCTA) has pr
158 ative optical coherence tomography (iOCT) in vitreoretinal surgery, assess the current state-of-the a
165 was recovered from 47 individuals undergoing vitreoretinal surgery: 16 had nonproliferative diabetic
176 s not unusual to require relatively advanced vitreoretinal techniques to achieve long-term surgical s
177 omitantly with more experience using various vitreoretinal techniques to manage these complicated cas
178 eral granulomas (57.1%), vasculitis (57.1%), vitreoretinal traction (57.1%), and chronic macular edem
180 pithelium include typical findings of peaked vitreoretinal traction and retinal disorganization with
181 hes a new reproducible technique to quantify vitreoretinal traction during vitrectomy and demonstrate
183 d macular edema, central serous retinopathy, vitreoretinal traction, and age-related macular degenera
184 c traction maculopathy, epiretinal membrane, vitreoretinal traction, optic or scleral pit, or advance
186 posterior to rectus insertion and associated vitreoretinal trauma can adversely affect the outcome in
188 BRI or VEH was administered by gavage, and vitreoretinal vascular endothelial growth factor (VEGF)
189 findings, retinal ganglion cell (RGC) count, vitreoretinal vascular endothelial growth factor (VEGF)
191 of ischemia responsible for upregulation of vitreoretinal VEGF and thus reduce vascular leakage and
192 sure to high oxygen significantly attenuated vitreoretinal VEGF concentrations, retinal vascular leak
194 diabetic animals but significantly decreased vitreoretinal VEGF expression and BRB breakdown to level
195 tic rats demonstrated significantly elevated vitreoretinal VEGF expression, vitreal glutamate concent
196 for all), despite negligible differences in vitreoretinal VEGF levels at the time of evaluation (P >
197 atment also significantly decreased elevated vitreoretinal VEGF protein levels and retinal BRB leakag
198 retinopathy with neurodegeneration, elevated vitreoretinal VEGF protein levels, and increased BRB bre
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