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1 alling and are mutated in familial exudative vitreoretinopathy.
2 proliferative diseases such as proliferative vitreoretinopathy.
3 cations in the pathogenesis of proliferative vitreoretinopathy.
4 ated in Norrie disease or familial exudative vitreoretinopathy.
5 otic diseases, in RPE cells in proliferative vitreoretinopathy.
6  could be useful in preventing proliferative vitreoretinopathy.
7  RPE dedifferentiation such as proliferative vitreoretinopathy.
8  and a female infant with familial exudative vitreoretinopathy.
9 ch as diabetic retinopathy and proliferative vitreoretinopathy.
10 proliferative diseases such as proliferative vitreoretinopathy.
11 ogression in a rabbit model of proliferative vitreoretinopathy.
12 related human diseases such as proliferative vitreoretinopathy.
13 lmitis, retinal detachment and proliferative vitreoretinopathy.
14 nal detachment associated with proliferative vitreoretinopathy.
15 al hemangioblastomas, and familial exudative vitreoretinopathy.
16 ch as diabetic retinopathy and proliferative vitreoretinopathy.
17 ascular retinopathies, such as proliferative vitreoretinopathy.
18 the current approach to treatment of various vitreoretinopathies.
19 h vitreous hemorrhage (1), and proliferative vitreoretinopathy (1).
20  surgery is the development of proliferative vitreoretinopathy, accounting for the failure of 7% to 1
21  autosomal dominant neovascular inflammatory vitreoretinopathy (ADNIV, OMIM #193235).
22 istics of patients who develop proliferative vitreoretinopathy after retinoblastoma treatment.
23 s of such retinal disorders as proliferative vitreoretinopathy and age-related macular degeneration.
24 s reparative processes such as proliferative vitreoretinopathy and as a laboratory tool for RPE behav
25 inal membranes associated with proliferative vitreoretinopathy and idiopathic epiretinal membranes.
26 opathy autosomal dominant familial exudative vitreoretinopathy and suggest that it also may be a susc
27  be a useful strategy to treat proliferative vitreoretinopathy and uveitis, ocular diseases initiated
28 ailure to reattach the retina, proliferative vitreoretinopathy, and delayed reabsorption of subretina
29 tinal redetachment by limiting proliferative vitreoretinopathy, and protect against photoreceptor cel
30 imetabolites for modulation of proliferative vitreoretinopathy, antimicrobial agents for endophthalmi
31                                Proliferative vitreoretinopathy developed in response to subretinal or
32                                Proliferative vitreoretinopathy did not develop in saline-treated cont
33 crostructural features of familial exudative vitreoretinopathy (FEVR) have not been well described.
34 d with autosomal dominant familial exudative vitreoretinopathy (FEVR) in families linking to the EVR1
35  pathological features of familial exudative vitreoretinopathy (FEVR) in human patients.
36                           Familial exudative vitreoretinopathy (FEVR) is a genetically heterogeneous
37                           Familial exudative vitreoretinopathy (FEVR) is a hereditary eye disorder th
38                           Familial exudative vitreoretinopathy (FEVR) is an inherited blinding disord
39                           Familial exudative vitreoretinopathy (FEVR) is an inherited blinding disord
40                           Familial exudative vitreoretinopathy (FEVR) is an inherited blinding disord
41                           Familial exudative vitreoretinopathy (FEVR) is characterized by delayed ret
42 typically associated with familial exudative vitreoretinopathy (FEVR), can result from mutations in K
43 pants with a diagnosis of familial exudative vitreoretinopathy (FEVR), Norrie disease, Coats' disease
44 a known family history of familial exudative vitreoretinopathy (FEVR), were also excluded.
45 n both Norrie disease and familial exudative vitreoretinopathy (FEVR).
46 ed in the pathogenesis of familial exudative vitreoretinopathy (FEVR, MIM # 133780).
47 oxocariasis (n = 22; 4%), familial exudative vitreoretinopathy (FEVR; n = 18; 3%), rhegmatogenous ret
48 ent retinal detachments due to proliferative vitreoretinopathy, focusing on the most recent reports i
49                           Familial exudative vitreoretinopathy has a wide range of unrecognized or un
50 rent retinal detachment due to proliferative vitreoretinopathy has grown concomitantly with more expe
51 r cell involvement, similar to proliferative vitreoretinopathy in humans.
52 reoretinopathies, such as familial exudative vitreoretinopathies including Norrie disease.
53 he later contractile stages of proliferative vitreoretinopathy, interleukin-1 beta (IL-1 beta) and tr
54                                Proliferative vitreoretinopathy is caused by the contraction of fibrot
55 inal detachment complicated by proliferative vitreoretinopathy is now most frequently treated by pars
56     Complications consisted of proliferative vitreoretinopathy (n = 13), recurrent neovascularization
57 l vasculitis (n = 4, 6%), familial exudative vitreoretinopathy (n = 3, 4%), and others (n = 12, 18%).
58 amponading agent, preoperative proliferative vitreoretinopathy (PVR) and axial length (AL) of the eye
59 inal membranes associated with proliferative vitreoretinopathy (PVR) and idiopathic epiretinal membra
60 raction) that are intrinsic to proliferative vitreoretinopathy (PVR) and induce the disease itself.
61 n vivo models for experimental proliferative vitreoretinopathy (PVR) and provide a detailed protocol
62                                Proliferative vitreoretinopathy (PVR) exemplifies a disease that is di
63 th factor (PDGF) contribute to proliferative vitreoretinopathy (PVR) in experimental models of the di
64 ) is required for experimental proliferative vitreoretinopathy (PVR) in rabbits.
65                                Proliferative vitreoretinopathy (PVR) is a blinding disease associated
66                                Proliferative vitreoretinopathy (PVR) is a complication of retinal det
67                                Proliferative vitreoretinopathy (PVR) is a complication that develops
68                                Proliferative vitreoretinopathy (PVR) is a disorder characterized by t
69                                Proliferative vitreoretinopathy (PVR) is a nonneovascular blinding dis
70                                Proliferative vitreoretinopathy (PVR) is a recurring and problematic d
71                                Proliferative vitreoretinopathy (PVR) is a serious complication of ret
72                                Proliferative vitreoretinopathy (PVR) is a serious problem in vitreore
73                                Proliferative vitreoretinopathy (PVR) is characterized by the prolifer
74                                Proliferative vitreoretinopathy (PVR) is mediated by proliferation and
75                                Proliferative vitreoretinopathy (PVR) is the primary cause of failure
76 F-alpha is widely expressed in proliferative vitreoretinopathy (PVR) membranes and is present in the
77                                Proliferative vitreoretinopathy (PVR) occurs in approximately 10% of p
78 itreous cytokine expression of proliferative vitreoretinopathy (PVR) patients.
79 ts with retinal detachment and proliferative vitreoretinopathy (PVR) plus vitreous from eyes obtained
80                                Proliferative vitreoretinopathy (PVR) remains the most common cause of
81 ctive review of three cases of proliferative vitreoretinopathy (PVR) that developed after successful
82                                Proliferative vitreoretinopathy (PVR) thwarts the repair of rhegmatoge
83 ailure rates when grade 0 or B proliferative vitreoretinopathy (PVR) was present and higher level 2 f
84 vent or inhibit development of proliferative vitreoretinopathy (PVR) was tested in a dispase-induced
85 ) isoforms are associated with proliferative vitreoretinopathy (PVR), a sight-threatening complicatio
86 PE-19 is an in vitro model for proliferative vitreoretinopathy (PVR), an aberrant wound healing respo
87 e of NF-kappaB in experimental proliferative vitreoretinopathy (PVR), and may offer a novel approach
88                             In proliferative vitreoretinopathy (PVR), retinal pigment epithelial (RPE
89 t that drives RPE responses in proliferative vitreoretinopathy (PVR), suggesting that the IGF system
90 ith (n = 7) or without (n = 9) proliferative vitreoretinopathy (PVR), vitreous hemorrhage (n = 10), v
91 cular hole (n = 33), recurrent proliferative vitreoretinopathy (PVR)-related retinal detachment (n =
92 ution to the events leading to proliferative vitreoretinopathy (PVR).
93  retinal detachment related to proliferative vitreoretinopathy (PVR).
94 t in the context of high-grade proliferative vitreoretinopathy (PVR).
95 of a blinding condition called proliferative vitreoretinopathy (PVR).
96 e kinase inhibitor, to prevent proliferative vitreoretinopathy (PVR).
97 etinal surgery for established proliferative vitreoretinopathy (PVR).
98 macular degeneration (AMD) and proliferative vitreoretinopathy (PVR).
99 oblasts to induce experimental proliferative vitreoretinopathy (PVR).
100  context of in vitro models of proliferative vitreoretinopathy (PVR).
101  to establish its relevance to proliferative vitreoretinopathy (PVR).
102 macular degeneration (AMD) and proliferative vitreoretinopathy (PVR).
103 macular degeneration (AMD) and proliferative vitreoretinopathy (PVR).
104 breaks, mostly with associated proliferative vitreoretinopathy (PVR).
105 c diseases of the eye, such as proliferative vitreoretinopathy (PVR).
106  epiretinal membranes found in proliferative vitreoretinopathy (PVR).
107 nvestigation were: (1) grade B proliferative vitreoretinopathy (PVR; n = 917), (2) grade C-1 PVR (n =
108  CNV recurrence (OR, 2.6), and proliferative vitreoretinopathy (PVR; OR, 17.6) were statistically sig
109 intraocular fibrosis, known as proliferative vitreoretinopathy, results in a blinding tractional reti
110  retinal detachment (3.65) and proliferative vitreoretinopathy stages A, B, and C (2.06) were elevate
111 al study of patients entered into the French Vitreoretinopathy Study Group database.
112 typical of human hypovascularization-related vitreoretinopathies, such as familial exudative vitreore
113 an ophthalmic biobank (421 participants with vitreoretinopathies were included in this study).
114 ice for management of a variety of pediatric vitreoretinopathies were offered participation in an oph
115                        Patients with various vitreoretinopathies were prospectively enrolled in an op

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