ライフサイエンスコーパス: vmPFC

1 g, exploratory-like) behavioral repertoires (vmPFC).

2 AIC) and the ventromedial prefrontal cortex (vmPFC).

3 LA) or the ventral medial prefrontal cortex (vmPFC).

4 s and in the ventromedial prefrontal cortex (vmPFC).

5 activity in ventromedial prefrontal cortex (vmPFC).

6 al reward in ventromedial prefrontal cortex (vmPFC).

7 gnals in the ventromedial prefrontal cortex (vmPFC).

8 een NAcc and ventromedial prefrontal cortex (vmPFC).

9 value within ventromedial prefrontal cortex (vmPFC).

10 ation is the ventromedial prefrontal cortex (vmPFC).

11 atum and the ventromedial prefrontal cortex (vmPFC).

12 itatory connectivity between the IFG and the vmPFC.

13 a specific CeMA GABAergic projection to the vmPFC.

14 ess ratings, are specifically encoded in the VMPFC.

15 s stronger and started earlier in VS than in vmPFC.

16 ivity between the amygdala and IFG, OFC, and vmPFC.

17 be minimized via functional compensation in vmPFC.

18 esynaptic terminals in the dentate gyrus and vmPFC.

19 ce) were indiscriminately represented across vmPFC.

20 est (ROI) in ventromedial prefrontal cortex (VMPFC), a brain region selected for its association with

21 terns in the ventromedial prefrontal cortex (vmPFC), a key node in the brain's valuation and decision

22 Findings indicate that disrupted vmPFC/ACC function plays a role in jeopardizing recovery

23 Additionally, vmPFC/ACC hyperactivation during neutral trials and redu

24 This vmPFC/ACC hyperactivity significantly predicted subseque

25 ntrol subjects showed the reverse pattern of vmPFC/ACC responses to stress, alcohol cues, and relaxed

26 Increased vmPFC activation (for CS+ > CS-) during early conditioni

27 Furthermore, vmPFC activation also tracked normative valence ratings

28 The effects of vmPFC activation on cue-induced reinstatement depended o

29 ed by three a priori criteria: (1) increased vmPFC activation was observed in older versus younger ad

30 y initial hypoactivity followed by increased VmPFC activation, pointed to the VmPFC as a key locus of

31 Although LD participants showed attenuated vmPFC activity after being fed (t(13) = 4.11, P < 0.001)

32 ehavior, but the precise conditions by which vmPFC activity can be exploited to reduce cocaine relaps

33 , P < 0.001), HD participants showed greater vmPFC activity in the fed than in the fasted state (t(15

34 which stimulus attributes were reflected in vmPFC activity varied as a function of context.

35 In humans, prestimulus VMPFC activity was externally manipulated through change

36 creased retrieval demands; and (3) increased vmPFC activity was positively associated with performanc

37 n monkeys, the factor dominating prestimulus VMPFC activity was trial number, which likely indexed va

38 younger adults; (2) age-related increases in vmPFC activity were associated with increased retrieval

39 entral striatum and ventromedial prefrontal (VMPFC) activity was decreased in response to setbacks.

40 Intra-vmPFC administration of the glycine transport inhibitor

41 f glycine transport inhibitor ALX5407 in the vmPFC alleviated deficits in impulse control.

42 atings whereas lower dynamic activity in the VmPFC also predicted a higher level of maladaptive copin

43 is associated with abnormal structure of the vmPFC and age-related differences in the hippocampus, re

44 effects were also observed in patients with vmPFC and amygdala damage, but were absent in patients w

45 and olfactory (piriform) cortex and between vmPFC and amygdala revealed parallel pathways that suppo

46 the amygdala and insula and between the rACC/vmPFC and cognitive control network.

47 ation away from the long-term best option in vmPFC and dACC, respectively.

48 We find that vmPFC and dmPFC have distinguishable time courses and ti

49 Thus, our data show reward-related vmPFC and dmPFC responses have distinct time courses and

50 as associated with structural differences in vmPFC and hippocampus, regions implicated in emotional p

51 te DBS drove c-Fos expression locally in the vmPFC and in several distal monosynaptically connected r

52 Instead, changes in connectivity between vmPFC and OFC are correlated with learning-related chang

53 sory-specific) satiation effects in both the vmPFC and orbitofrontal cortex.

54 value, whereas connectivity changes between vmPFC and PC predict changes in perceived odour similari

55 nonsmokers showed activity decreases in the vmPFC and PCC, and increases in lateral frontoparietal r

56 larly predicted decreased dorsal striatal to vmPFC and presupplementary motor area connectivity, whic

57 odopsin (eNpHR3.0) in pyramidal cells of the vmPFC and studied the effect of activation or inhibition

58 nce for compensation in connectivity between vmPFC and the dorsolateral PFC during memory-dependent c

59 t activity evoked by visual cues between the VMPFC and the orbitofrontal cortex.

60 gen-level-dependent activity patterns of the vmPFC and the striatum that coexist with value signals.

61 idence on the causal mechanisms by which the vmPFC and ventral striatum interact during the anticipat

62 lts demonstrate that the correlation between VMPFC and VS activity and discounting occurs even in the

63 Nonetheless, the close match between vmPFC and VS suggests that cortex and its striatal targe

64 gene delivery of an activating Gq-DREADD to vmPFC and/or vmPFC projections to the nucleus accumbens

65 ons of human ventromedial prefrontal cortex (vmPFC) and dorsal anterior cingulate cortex (dACC) durin

66 sured in the ventromedial prefrontal cortex (vmPFC) and dorsal raphe nucleus (DRN).

67 ncluding the ventromedial prefrontal cortex (vmPFC) and hippocampus.

68 thicker cortex in the left ventromedial PFC (vmPFC) and left precentral gyrus.

69 activity in ventromedial prefrontal cortex (VMPFC) and ventral striatum (VS) when human subjects are

70 (ACC), and ventral medial prefrontal cortex (vmPFC)) and this stress diathesis, in maltreated youth w

71 ala (CeA), ventral medial prefrontal cortex (vmPFC), and orbitofrontal cortex (OFC), brain regions im

72 rally in the ventromedial prefrontal cortex (vmPFC), and right insula.

73 ation [the ventral medial prefrontal cortex (vmPFC)] and to sensory-driven regions associated with so

74 prefrontal-limbic model comprising the IFG, vmPFC, and amygdala.

75 r competition-based choice dynamics in human vmPFC, and introduce computational neurostimulation as a

76 l anterior cingulate cortex (rACC), anterior vmPFC, and subgenual cingulate significantly decreased f

77 n the right amygdala and bilateral IFG, OFC, vmPFC, anterior cingulate cortex, and frontopolar cortex

78 at functional interactions between dlPFC and vmPFC are a key aspect of context-dependent valuation an

79 As in macaques, human lOFC and mOFC/vmPFC are necessary for contingent learning and value-gu

80 striatum and ventromedial prefrontal cortex (vmPFC) are two central nodes of the "reward circuit" of

81 to those of ventromedial prefrontal cortex (vmPFC) Area 14 neurons, recorded in a risky choice task.

82 y increased VmPFC activation, pointed to the VmPFC as a key locus of the emotional and behavioral con

83 ychological results highlight a role for the vmPFC as part of a memory network including the medial t

84 entified the ventromedial prefrontal cortex (VMPFC) as a key node of the human brain valuation system

85 inhibitory functional connectivity with the vmPFC at the time of children's own choice.

86 e of context-specific attribute valuation in vmPFC at the time of choice.

87 hasticity of preferences might relate to the VMPFC automatically aggregating the values of contextual

88 gnitive processes along a posterior-anterior vmPFC axis.

89 V but no age-related differences in anterior vmPFC (BA 10/11, Z=4.5), which inversely correlated with

90 tional possibilities for clinical studies of vmPFC-based circuits, including neuropsychological asses

91 reduction in rearing were mimicked by intra-vmPFC blockade of AMPA-type but not NMDA-type glutamate

92 s with higher HRV showed both higher overall vmPFC blood-oxygen-level-dependent activity and attenuat

93 e signals in ventromedial prefrontal cortex (vmPFC) bore out this prediction.

94 nd 32 of the ventromedial prefrontal cortex (vmPFC), but a causal relationship between dysregulation

95 have shown that reward value signals in the vmPFC can be altered by emotion regulation processes; ho

96 probability and reward size, suggesting that vmPFC carries an integrated value signal; (2) anti-corre

97 optogenetic activation of the VGat-Cre(CeMA-vmPFC) circuit in awake, behaving animals produced a pos

98 lar for patients with lesions in DMF or mOFC/vmPFC, compared with Controls.

99 rs, it is essential to characterize amygdala-vmPFC connectivity changes during typical development.

100 rror responses in anterior insula and insula-vmPFC connectivity during self-esteem updates.

101 Enhanced insula-vmPFC connectivity during updating of those beliefs may

102 rthermore, age positively predicted amygdala-vmPFC connectivity in healthy youth, but negatively pred

103 is model indicated that the parental caudate-vmPFC connectivity in infancy predicted lower child exte

104 t decreases in dmPFC activation and amygdala-vmPFC connectivity may indicate abnormal developmental p

105 NAcc-vmPFC connectivity strength was negatively correlated wi

106 Amygdala-vmPFC connectivity varies with the role played by emotio

107 parental behavioral styles; stronger caudate-vmPFC connectivity was associated with more collaborativ

108 ns signaled gamble outcomes, suggesting that vmPFC contributes to both monitoring and choice processe

109 ase in the ventral medial prefrontal cortex (vmPFC) contributes to increased motor impulsivity during

110 striatum and ventromedial prefrontal cortex (vmPFC) (corrected P<0.05), which in turn correlated with

111 tters in the ventromedial prefrontal cortex (vmPFC) cortex in rats following long-term alcohol exposu

112 tofrontal (OFC) and ventromedial prefrontal (vmPFC) cortices, respectively.

113 oach to test whether participants with focal vmPFC damage (n = 6) would show a reduced influence of s

114 e most conspicuous memory disorder following vmPFC damage is confabulation; strategic retrieval model

115 observed in the vmPFC group, suggesting that vmPFC damage reduced the influence of schematic memory.

116 ngly opposite response pattern to those with vmPFC damage when making moral judgements.

117 To test this prediction, ten patients with vmPFC damage, four with present or prior confabulation,

118 In patients with vmPFC damage, visualization might also be reduced alongs

119 urbed in patients with either hippocampal or vmPFC damage.

120 neurosurgical patients with focal, bilateral vmPFC damage.

121 l, bilateral ventromedial prefrontal cortex (vmPFC) damage.

122 ockade in the LH-PeF nearly eliminated intra-vmPFC DAMGO-induced food intake without altering DAMGO-i

123 subtype in this structure) antagonized intra-vmPFC DAMGO-induced hyperlocomotion but enhanced food in

124 derlying the antidepressant-like activity of vmPFC DBS and identify dramatic circuit-mediated cellula

125 aluating the antidepressant-like activity of vmPFC DBS in the chronic social defeat stress (CSDS) mod

126 The effects of chronic vmPFC DBS on the physiology and morphology of geneticall

127 Furthermore, vmPFC DBS reversed CSDS-induced arborization of 5-HT den

128 Circuit-wide activation induced by vmPFC DBS was mapped with c-Fos immunolabeling.

129 e with the long-term therapeutic activity of vmPFC DBS.

130 ndirectly by ventromedial prefrontal cortex (vmPFC) DBS.

131 normal FPCN connectivity during WM, whereas vmPFC deactivation differences persisted regardless of W

132 cover that a value-anticipation mechanism in vmPFC declines in aging, and that this mechanism is asso

133 gnals in the ventromedial prefrontal cortex (vmPFC) differed between the groups (F(1,28) = 21.34, P <

134 muscimol+baclofen reversible inactivation of vmPFC, dmPFC, and OFC on 'incubated' cue-induced methamp

135 irmation condition produced more activity in VMPFC during exposure to health messages and went on to

136 g, greater neural flexibility signals in the VmPFC during stress correlated with active coping rating

137 neurotransmitters were monitored in the rat vmPFC during the performance of a challenging variant of

138 responding and glycine and serine levels in vmPFC during the performance of the standard 5-CSRTT.

139 ation of the ventromedial prefrontal cortex (vmPFC) during correct and error trials.

140 ation of the ventromedial prefrontal cortex (vmPFC) during extinction recall (etap2 = 0.178, P = .02)

141 resence of low-value third alternatives, and vmPFC effect sizes predicted individual variation in sub

142 ind that the ventromedial prefrontal cortex (vmPFC) encodes children's own preferences and the left d

143 e disorders propose that hypoactivity in the vmPFC engenders disinhibited activity of the amygdala an

144 The vmPFC exhibited inhibitory influence on the amygdala.

145 s are also linked to GAD, most likely via an vmPFC fear generalization.

146 Moreover, the reduced response in vmPFC for DG was predicted by higher rates of externaliz

147 diffusion MRI, we measured vmPFC thickness, vmPFC functional connectivity, and vmPFC structural conn

148 We derived an index reflecting vmPFC functioning from the BOLD reactivity on a continuu

149 fear extinction recall relates to changes in vmPFC gamma activity.

150 o show the otherwise observed alterations in vmPFC gamma power to extinguished CS(+).

151 s showed blunted task-related recruitment of vmPFC glycine and serine release, and the loss of an inv

152 data indicate that cue-elicited deficits in vmPFC group 1 mGluR function mediate resistance to extin

153 text on recognition were not observed in the vmPFC group, suggesting that vmPFC damage reduced the in

154 Since the vmPFC has strong anatomical and functional links with th

155 The ventromedial prefrontal cortex (vmPFC) has been implicated in a variety of social, cogni

156 The ventromedial prefrontal cortex (vmPFC) has been shown to negatively regulate cocaine-see

157 ofrontal cortex (OFC), and ventromedial PFC (vmPFC) have been linked to the regulation of anxiety dur

158 of the amygdala (CeMA) and ventromedial PFC (vmPFC) have critical roles for emotional regulation.

159 eys (a nonhuman primate that has far greater vmPFC homology to humans than rodents), areas 25 and 32

160 The vmPFC hypoactivation was associated with anxiety symptom

161 (dlPFC) and ventromedial prefrontal cortex (vmPFC) implicated in self-control choices would also und

162 e novel evidence for a critical role for the vmPFC in contributing to reward-related activity of the

163 These findings suggest a role for vmPFC in coordinating neural and physiological responses

164 ution that may be related to the role of the vmPFC in decision making.

165 We investigated the role of human vmPFC in false memory by combining a neuropsychological

166 of stimulus meaning, which then informs the vmPFC in inhibiting the amygdala.

167 surements implied greater myelination within vmPFC in misophonic individuals.

168 rack of research shows the importance of the vmPFC in multiple aspects of social cognition, such as f

169 ning task-related hyperconnectivity with the vmPFC in OCD, consistent with biased processing of the C

170 Consistent with the role of the vmPFC in providing access to details that are typical fo

171 unique evidence for the critical role of the vmPFC in regulating activity of the amygdala in humans a

172 oth nodes were functionally coupled with the vmPFC in support of the human coparental bond and this c

173 d-based choice and endorse the centrality of vmPFC in that process.

174 ement of the ventromedial prefrontal cortex (vmPFC) in conditioned drug seeking, but specific knowled

175 ted with the ventromedial prefrontal cortex (vmPFC) in humans, and damage to this region significantl

176 plicated the ventromedial prefrontal cortex (vmPFC) in memory schema, particularly in facilitating ne

177 Our findings suggest that vmPFC increases the influence of schematically congruent

178 ion within ventral medial prefrontal cortex (vmPFC) induces feeding and hyperactivity, resulting poss

179 e provide new evidence for bottom-up CeMA to vmPFC influence on reward-related behaviors.

180 The ventromedial prefrontal cortex (vmPFC), insula, amygdala, hypothalamus, and periaqueduct

181 sions to the ventromedial prefrontal cortex (vmPFC), insula, or amygdala ("target patients") against

182 sment of the action in question, whereas the vmPFC integrates that signal with a utilitarian assessme

183 However, the CeMA and ventromedial PFC (vmPFC) interaction in reward regulation remains poorly u

184 how that the ventromedial prefrontal cortex (vmPFC) inversely and ventral tegmental area directly tra

185 We argue that the vmPFC is a core element of a network that represents str

186 an and animal studies demonstrating that the vmPFC is a key node of cortical and subcortical networks

187 ole in schema-supported memory encoding, the vmPFC is also implicated in schema reinstatement itself.

188 The vmPFC is closely connected with another brain region-the

189 cond track of research demonstrates that the vmPFC is critical for the generation and regulation of n

190 One track of research indicates that the vmPFC is critical for the representation of reward- and

191 mPFC lesions to test the hypothesis that the vmPFC is necessary for enhancing ventral striatum respon

192 During such integrative moral judgments, the vmPFC is preferentially engaged relative to utilitarian

193 ts that representation of decision values in vmPFC is suborganized according to the underlying comput

194 ction in the ventromedial prefrontal cortex (vmPFC) is believed to play a pivotal role in the pathoge

195 The ventromedial prefrontal cortex (vmPFC) is closely associated with the ability to make co

196 amygdala and ventromedial prefrontal cortex (vmPFC) is compromised in multiple psychiatric disorders,

197 cortex/ventromedial prefrontal cortex (rACC/vmPFC) is the most consistent finding across studies, di

198 sion making, ventromedial prefrontal cortex (vmPFC) is thought to support choices by tracking the exp

199 abnormal connectivity among regions such as vmPFC, lateral orbitofrontal cortex, and parahippocampal

200 By contrast, the vmPFC lesion patients exhibited altered cue-related insu

201 Furthermore, we observed that the vmPFC-lesioned patients had decreased volumes of the acc

202 matched neurologically healthy subjects, the vmPFC-lesioned patients had reduced ventral striatal act

203 By contrast, mOFC/vmPFC-lesioned patients made more stochastic choices tha

204 osite approach to moral decision making than vmPFC-lesioned patients.

205 Patients with vmPFC lesions (n = 7) and healthy comparison participant

206 althy comparison subjects, the patients with vmPFC lesions exhibited potentiated amygdala responses t

207 If that is the case, vmPFC lesions should lead to impaired schema-based opera

208 RI in five neurosurgical patients with focal vmPFC lesions to test the hypothesis that the vmPFC is n

209 creased differential response with regard to vmPFC-localized gamma power.

210 ation of the ventromedial prefrontal cortex (VmPFC), marked by initial hypoactivity followed by incre

211 fy to what extent the deviations in amygdala-vmPFC maturation contribute to the onset of psychiatric

212 arable data in humans to address whether the vmPFC may be critical for the reward-related response pr

213 Alterations of the vmPFC may be one mechanism that mediates the pathway fro

214 ves, such as ventromedial prefrontal cortex (vmPFC), may also contribute to memory processes by weigh

215 erestingly, pharmacological manipulations of vmPFC mGluR1/5 produced no immediate effects on cue-indu

216 nd animal studies have demonstrated that the vmPFC modulates ventral striatum activity.

217 as ventromedial prefrontal cortex in humans; vmPFC/mOFC) is involved in constraining the decision to

218 These data indicate intra-vmPFC muOR signaling jointly modulates appetitive motiva

219 In contrast, intra-vmPFC muscimol infusions did not alter the overall intak

220 the stress response, and a specific role for VmPFC neuroflexibility in stress-resilient coping.

221 of designer receptors (DREADDs) to activate vmPFC neurons and examine the consequences on cocaine se

222 -DREADD approach to confine the Gq-DREADD to vmPFC neurons that project to the medial nucleus accumbe

223 We tested this idea by recording vmPFC neurons while macaques performed a gambling task w

224 Activation of vmPFC neurons with the Gq-DREADD reduced reinstatement o

225 separate analysis of the predictive power of vmPFC on behavior indicated that even after accounting f

226 lOFC (predominantly right hemisphere), mOFC/vmPFC, or dorsomedial prefrontal (DMF), and a comparison

227 Muscimol+baclofen injections into dmPFC, vmPFC, or OFC during late withdrawal had no effect on in

228 types in the ventromedial prefrontal cortex (vmPFC), orbitofrontal cortex, nucleus accumbens, hypotha

229 noncarriers, contributions of an additional VMPFC pathway best characterized deletion carriers.

230 In contrast, vmPFC patients showed consistently reduced false recall,

231 All four vmPFC patients with present or prior confabulation were

232 OFC patients compared with Controls and mOFC/vmPFC patients.

233 ecognition was also marginally reduced among vmPFC patients.

234 re associated with activity decreases in the vmPFC, PCC, and insula and increases in the lateral PFC,

235 AMC) and the ventromedial prefrontal cortex (vmPFC) play important roles in the expression and extinc

236 Our findings suggest that the vmPFC plays a key role in schematic memory processes by

237 are consistent with the proposition that the vmPFC plays an important role in integrating previous ex

238 The ventromedial prefrontal cortex (vmPFC) plays a critical role in a number of evaluative p

239 ncluding the ventromedial prefrontal cortex (vmPFC), posterior cingulate cortex (PCC), parahippocampu

240 s, including ventromedial prefrontal cortex (vmPFC), posteromedial cortex (PMC), hippocampus, and amy

241 value coding ventromedial prefrontal cortex (vmPFC) predicted choice behavior.

242 ue coding in ventromedial prefrontal cortex (vmPFC) predicted individual differences in satiety-relat

243 These results support the idea that vmPFC processes self-relevant information, and suggest t

244 cruitment of glutamate signaling in multiple vmPFC projection targets.

245 activation through functionally dissociable vmPFC projection targets.

246 escribed here suggest that the VGat-Cre(CeMA-vmPFC) projection acts to modulate existing reward-relat

247 The ventromedial prefrontal cortex (vmPFC) projection to the nucleus accumbens shell is impo

248 y of an activating Gq-DREADD to vmPFC and/or vmPFC projections to the nucleus accumbens shell allows

249 t specific knowledge of the temporal role of vmPFC pyramidal cells is lacking.

250 time-dependent switch in the contribution of vmPFC pyramidal cells to recall and extinction of cocain

251 to the human ventromedial prefrontal cortex (vmPFC) reduced the influence of existing knowledge on ne

252 valuation, and how functional connections to vmPFC reflect the current value of outcomes and guide go

253 such as the ventromedial Prefrontal Cortex (vmPFC), reflected the strength of prior preferences.

254 nal in human ventromedial prefrontal cortex (vmPFC) reflects both value comparison and confidence in

255 in humans, we demonstrate that damage to the vmPFC results in decreased ventral striatum activity dur

256 Our findings reveal an absence of vmPFC safety signaling in OCD, undermining flexible thre

257 ese results promote further investigation of vmPFC safety signaling in other anxiety disorders, with

258 To investigate the importance of vmPFC safety signaling, we used neuroimaging of Pavlovia

259 ion in OCD patients during reversal, whereas vmPFC safety signals were absent throughout learning in

260 g can occur to some extent in the absence of vmPFC safety signals, effective CS- signaling becomes cr

261 ss, with the ventromedial prefrontal cortex (VMPFC) showing greater modulation by EEV.

262 correlated with greater persistence, whereas VMPFC signal change to uncontrollable setbacks mediated

263 The vmPFC signal decreased in the presence of low-value thir

264 is expressed mainly in the alpha band for a vmPFC signal, which contrasts with a theta based dmPFC s

265 nce that the ventromedial prefrontal cortex (vmPFC) signals the satisfaction we expect from imminent

266 r a priori defined ROIs in the human AMC and vmPFC similarly modulate their oscillatory activity duri

267 hickness, vmPFC functional connectivity, and vmPFC structural connectivity within the corticolimbic s

268 erized developmental changes of the amygdala-vmPFC subregion functional and structural connectivity u

269 und that the ventromedial prefrontal cortex (vmPFC) supports compensation as defined by three a prior

270 ntations in the brain, located mainly in the vmPFC, temporal and parahippocampal cortices, thalamus,

271 ata revealing distinct subregions within the vmPFC that correspond to each of these three functions,

272 sting-state), and diffusion MRI, we measured vmPFC thickness, vmPFC functional connectivity, and vmPF

273 ee factors predict individual variability of vmPFC threat assessment in an independent fashion.

274 Our results strongly suggest that vmPFC threat processing is closely associated with broad

275 uctural and functional mechanisms underlying vmPFC threat-safety assessment in humans.

276 iors and emphasized top-down processing from vmPFC to CeMA.

277 and thereby alters general value signals in vmPFC to guide goal-directed behavior.

278 onses in the ventromedial prefrontal cortex (vmPFC) to food aromas compared with responses induced by

279 mory and the ventromedial prefrontal cortex (vmPFC) to generalized, schematic memory.

280 amygdala and ventromedial prefrontal cortex (vmPFC) to moral judgment.

281 (dmPFC) and ventromedial prefrontal cortex (vmPFC) to simultaneously measured EEG activity in human

282 tecture whereby interactions between ACC and vmPFC underpin an integration of immediate and delayed c

283 We show that vmPFC uniquely contributes a sustained activation profil

284 A and terminates in the vmPFC (VGat-Cre(CeMA-vmPFC)) using viral-vector-mediated, cell-type-specific

285 The findings anatomically decompose vmPFC value representations according to computational r

286 originates in the CeMA and terminates in the vmPFC (VGat-Cre(CeMA-vmPFC)) using viral-vector-mediated

287 Reduced anterior vmPFC volume may confer impaired recovery from illness,

288 youth with PTSD demonstrated decreased right vmPFC volumes compared with both maltreated youth withou

289 Finally, connectivity between striatum and vmPFC was associated with increased plasma interleukin (

290 tings and condition, increased signal in the vmPFC was associated with more positive valence ratings.

291 he functional connectivity between dlPFC and vmPFC was associated with the degree of context-specific

292 logical interaction analysis showed that the vmPFC was more highly connected to the dlPFC when indivi

293 D) signal in ventromedial prefrontal cortex (vmPFC) was parametrically modulated by the affective val

294 Five major response patterns observed in vmPFC were also observed in VS: (1) offer value encoding

295 e signals in ventromedial prefrontal cortex (vmPFC) were attenuated in older adults.

296 y coupled to ventromedial prefrontal cortex (vmPFC) when adaptive switches in choice were required.

297 for the first time that damage to the human vmPFC, when associated with confabulation, leads to defi

298 ted reward magnitude was only encoded in the VMPFC, which also reflected subjective cue values, as ex

299 nnectivity in centromedial amygdala-anterior vmPFC white matter was associated with greater anxiety/d

300 why they produce greater response in TPJ and vmPFC, without invoking competition between automatic an

301 ective loss or diminution of function of the vmPFC would result in heightened activity of the amygdal