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1  stroboscopy, showed immobility of the right vocal cord.
2 related spectral peaks, e.g., as produced by vocal cords.
3        The lowest densities were seen in the vocal cords.
4 sociated with brief partial adduction of the vocal cords.
5 thirds of laryngeal cancers originate at the vocal cords.
6                                              Vocal cord abnormalities occurred in 4/38 (11%) patients
7 , with multiple affected individuals in whom vocal cord and pharyngeal weakness may accompany the dis
8 typically involves abnormal adduction of the vocal cords during inspiration, mimics the symptoms of a
9                                              Vocal cord dysfunction (VCD) typically involves abnormal
10 ing those associated with severe obesity and vocal cord dysfunction (VCD).
11 hy with the added features of pharyngeal and vocal cord dysfunction (VCPDM) that has not been previou
12  of deficient oropharyngeal motor skills and vocal cord dysfunction is crucial to establish enteral n
13 cribes seven elite athletes with psychogenic vocal cord dysfunction who presented with acute dyspnea
14 esophageal reflux, obstructive sleep apnoea, vocal cord dysfunction, obesity, dysfunctional breathing
15 ons were limited to one transient unilateral vocal cord dysfunction.
16 variable extrathoracic airway obstruction of vocal cord dysfunction.
17        However, laryngoscopy revealed normal vocal cord function.
18  morbidity rate was 6% (wound separation and vocal cord hemiparesis, one each).
19  (n = 5), meningitis/cerebritis (n = 2), and vocal cord infection (n = 1).
20                                              Vocal cord motion was examined endoscopically in 11 of t
21                                     Abnormal vocal cord motion was observed in two of the 11 patients
22 o confirm ISLN anaesthesia, and to visualise vocal cord movement and laryngeal closure.
23                                     The left vocal cord movement was impaired.
24                            Optic atrophy and vocal cord palsy were observed in patients with severe d
25                               Optic atrophy, vocal cord palsy, and auditory impairment were observed
26  and respiratory muscle weakness but without vocal cord palsy.
27 complications such as hypoparathyroidism and vocal cord paralysis in a small proportion of patients.
28 ion, while 1 minor skin burn and 1 permanent vocal cord paralysis occurred in the RFA treatment group
29 roup, tracheal surgery, hypotonic airway, or vocal cord paralysis or if they died before extubation.
30 ultiple organ system failure, age of <6 mos, vocal cord paralysis, and noncooperation with nasal mask
31 l dysmotility (mid esophageal hematoma), and vocal cord paralysis, resulting in hoarse voice (upper e
32 ntellectual disabilities and, in some cases, vocal cord paralysis, tracheomalacia and cyclic vomiting
33 rotid glands (PRP, 51%; mean SUV, 1.90), and vocal cords (PRP, 19%; mean SUV, 1.77).
34 er radiotherapy as a function of whether the vocal cord regained mobility or remained fixed during or
35                The proper development of the vocal cords requires embryos to contain a certain number
36 ity of the speaker is carried largely in the vocal cord source and the message is shaped by the ever-
37                   Of the 12 patients, 10 had vocal cord tumors, one had a hypopharyngeal tumor, and o
38 nt were assessed from the hard palate to the vocal cords using T1-weighted images.
39 he removal of enteral content from below the vocal cords, usually during endotracheal tube placement.
40 ected involving the whole length of the left vocal cord, with abnormal mucosa also seen in the right
41 ance of liquid or puree bolus below the true vocal cords without coughing during a FEES examination.

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