ライフサイエンスコーパス: voiding

1 d urinary retention (requiring early morning voiding).

2 r to be important for coordination of proper voiding.

3 y and inhibitory effects on the frequency of voiding.

4 intaining continence and producing efficient voiding.

5 ynamic acquisition without disruption due to voiding.

6 , whereas withdrawals were suppressed during voiding.

7 not leak, and empties completely by natural voiding.

8 stimulation, bladder training, and prompted voiding.

9 ating detrusor pressure and flow rate during voiding.

10 aks, or does not empty completely by natural voiding.

11 ested that several transmitters may modulate voiding.

12 ollowed by place and position preference for voiding.

13 s in urine volume during bladder filling and voiding.

14 quency and was markedly decreased with early voiding.

15 metabolites and increasing the frequency of voiding.

16 al ureters and on radiographs obtained after voiding.

17 the uptake phase as well as before and after voiding.

18 and anorectal junction during liquid medium voiding.

19 nonsecretor phenotype, or delayed postcoital voiding.

20 ling, which can be manually compressed after voiding.

21 ack from maintaining continence to producing voiding.

22 xes to either maintain continence or promote voiding.

23 te that urothelium itself directly modulates voiding.

24 hat close the ureterovesical junction during voiding.

25 the bladder dose can be reduced by frequent voiding.

26 bladder dose can be reduced by more frequent voiding.

27 ow rate suggested poor meatal opening during voiding.

28 lated using OLINDA 1.1 software, assuming no voiding.

29 MBq, depending on the assumptions on bladder voiding.

30 DFV by internalizing apical membrane during voiding.

31 es implicated in facial expression and urine voiding.

32 /- 2.4, and 20.9 +/- 5.2 muSv/MBq for the no-voiding, 2.5-h-voiding, and 1-h-voiding models, respecti

33 2; dysuria without infection, 6; difficulty voiding, 3; and silent stones, 2.

34 ients, renography was interrupted because of voiding (30%), whereas this occurred in only 3 of the F

35 uring the first cycle of bladder filling and voiding, a second cycle was performed.

36 expert opinion, women with diabetes without voiding abnormalities presenting with acute cystitis sho

37 ent strategies were devised, including timed voiding alone (n=6), clean intermittent catheterization

38 a maximum of 10) were normal, showing normal voiding and erectile function, respectively.

39 commodate large changes in cell shape during voiding and filling cycles.

40 phrine (3000 nmol) completely blocked reflex voiding and induced overflow incontinence at a high base

41 the micturition control circuitry, to defer voiding and maintain urinary continence, even when the b

42 Urinary voiding and nocturia were worse in the radiotherapy grou

43 n, with ON cells promoting the initiation of voiding and OFF cells promoting urine storage.

44 inistration reduces the frequency of bladder voiding and restores the voided volume of CYP-treated mi

45 on, catheter size, catheter in or out during voiding and sex on flow rate, flow pattern, voiding pres

46 al cord injury disrupts voluntary control of voiding and the normal reflex pathways that coordinate b

47 ) is thought to contain neurons that trigger voiding and thereby function as the "pontine micturition

48 nabled bacteria to escape elimination during voiding and to re-emerge in the urine as the bladder dis

49 9 +/- 5.2 muSv/MBq for the no-voiding, 2.5-h-voiding, and 1-h-voiding models, respectively.

50 in controlling facial expression and urinary voiding, and also in bladder smooth muscle, consistent w

51 ctions, vesicoureteral reflux, dysfunctional voiding, and appropriate imaging studies.

52 ure had urinary tract infections, difficulty voiding, and postoperative urge incontinence.

53 eting behaviours (premature voiding, delayed voiding, and straining to void) were positively associat

54 iours (especially premature voiding, delayed voiding, and straining to void), as these unhealthy toil

55 the dynamic processes of bladder filling and voiding apical membrane dynamics depend on sequential an

56 Assuming bladder voiding at 2- or 4-hr intervals, the critical organ that

57 For a single bladder voiding at 6 h after (18)F-FLT injection, the (18)F-FLT

58 Cystometrogram study and tracing analysis of voiding behavior revealed that the ketamine-treated rats

59 bladder (megacystis) phenotype and abnormal voiding behavior similar to that found in patients.

60 Voiding behavior was monitored using specially designed

61 related to micturition and initiate specific voiding behaviors so that micturition occurs in environm

62 In vivo natural voiding behaviour, sensory nerve activity, urothelial ce

63 common among the female nurses, with delayed voiding being the unhealthiest toileting behaviour, whic

64 had a higher frequency of low-amplitude, non-voiding bladder contractions.

65 feedback programs for treating dysfunctional voiding, Botox injections for overactive bladder and an

66 ears completely with manual compression, and voiding can be maintained in this way.

67 fort or pain during or after ejaculation and voiding complaints such as irritative and obstructive vo

68 inary retention, return of reflexive bladder voiding contractions coincident with EUS EMG activation,

69 ave a significant role in the control of non-voiding contractions in vivo.

70 a therapeutic approach for management of non-voiding contractions, a condition which characterizes ma

71 oduction and caused a marked increase in non-voiding contractions.

72 y decreased the frequency of spontaneous non-voiding contractions.

73 erfering RNA, increased the interval between voiding contractions.

74 ed clearance of infection compared to normal-voiding controls.

75 s dramatic volume changes during filling and voiding cycles.

76 e seen on Retrograde Urethrography (RGU) and Voiding Cysto-Urethrography (VCUG), i.e. linear incomple

77 Intravenous urography, voiding cysto-urethrography and MRI confirmed the ultras

78 ation was followed by intravenous urography, voiding cysto-urethrography and MRI of the abdomen.

79 Academy of Pediatrics recommend obtaining a voiding cystourethrogram and a renal ultrasonogram for y

80 coureteral reflux that is missed by standard voiding cystourethrogram but detectable during positiona

81 ammatory markers (model 2); and model 2 plus voiding cystourethrogram findings (model 3).

82 A voiding cystourethrogram for the identification of reflu

83 enatal hydronephrosis with an ultrasound and voiding cystourethrogram is reasonable and may reduce th

84 the determination of the degree of reflux by voiding cystourethrogram is to guide the institution of

85 a history of prenatal hydronephrosis with a voiding cystourethrogram may reduce this risk.

86 patients require postnatal evaluation with a voiding cystourethrogram to investigate for vesicoureter

87 ty of models requiring a blood draw and/or a voiding cystourethrogram.

88 Only 13 voiding cystourethrograms (VCUGs) (2.5%) of 517 were fou

89 diagnosis of vesicoureteral reflux involves voiding cystourethrograms, which are invasive and costly

90 Retrograde urethrography (RU) and voiding cystourethrography (VCU) are traditional diagnos

91 Retrograde Urethrography - Voiding Cystourethrography evaluation of the urethra and

92 Magnetic resonance voiding cystourethrography has been used to rule out ves

93 In many centers, voiding cystourethrography is a routine part of pretrans

94 Voiding cystourethrography should be performed routinely

95 ed within 72 hours after diagnosis, contrast voiding cystourethrography was performed one month later

96 n included US, MR imaging, autopsy, surgery, voiding cystourethrography, computed tomography, angiogr

97 ltrasonography (US), renal scintigraphy, and voiding cystourethrography.

98 Unhealthy toileting behaviours (premature voiding, delayed voiding, and straining to void) were po

99 y toileting behaviours (especially premature voiding, delayed voiding, and straining to void), as the

100 mpted to correlate common clinical measures (voiding diaries, pad testing, urodynamics) with quality-

101 Voiding difficulty and acute urinary retention are infre

102 l pressure, large residual urine volume, and voiding difficulty.

103 tion of the pelvic floor and contributing to voiding difficulty.

104 balance symptom improvement and incidence of voiding difficulty.

105 ncreased interest in pediatric nonneurogenic voiding disorders (NNVDs), urodynamic testing and therap

106 Idiopathic voiding disorders affect up to 10-15% of men and women.

107 athway may play an important role in urinary voiding disorders characterized by abnormal bladder moti

108 y demonstrating the inaccuracy of predicting voiding disorders on the basis of uroflow alone or the m

109 ons in both spinal cord signaling and reflex voiding during bladder filling (under anesthesia).

110 Voiding dysfunction after anti-incontinence procedures i

111 le of preoperative urodynamics in predicting voiding dysfunction after anti-incontinence surgery is r

112 of the urothelial barrier, a major cause of voiding dysfunction and bladder pain syndrome.

113 eye movement behaviour disorder and urinary voiding dysfunction appear to precede the development of

114 5-HT3A mutant mice had marked voiding dysfunction characterized by a loss of micturiti

115 of women who appear to be at higher risk of voiding dysfunction following incontinence surgery, and

116 Urinary voiding dysfunction in childhood, manifesting as inconti

117 Voiding dysfunction in children encompasses a wide spect

118 Over the last several decades voiding dysfunction in children has primarily been assoc

119 This overview of voiding dysfunction in children outlines the established

120 ill focus on the diagnosis and management of voiding dysfunction in neurologically and anatomically n

121 tive urodynamics in predicting postoperative voiding dysfunction in patients undergoing anti-incontin

122 Several aspects of voiding dysfunction in women remain under investigation,

123 hough not useful in the primary treatment of voiding dysfunction is equivalent in potency to biofeedb

124 Voiding dysfunction may play an etiological role in cong

125 Voiding dysfunction may spontaneously improve or require

126 prognostic information regarding the risk of voiding dysfunction postoperatively and the possibility

127 The rates of voiding dysfunction requiring surgery were 2.7% in those

128 afferent neurons may therefore contribute to voiding dysfunction seen in diabetes mellitus.

129 Voiding dysfunction typically presents after toilet trai

130 igher rate of lower urinary tract injury and voiding dysfunction when compared with transobturator ta

131 so assessed postoperative urge incontinence, voiding dysfunction, and adverse events.

132 lications (urinary tract infection, urgency, voiding dysfunction, and mesh erosion) were more common

133 of clinical presentations such as hematuria, voiding dysfunction, flank pain, abdominal pain, nephrol

134 se medications in the treatment of pediatric voiding dysfunction, neurogenic bladder, chronic lower u

135 rates of positive provocative stress tests, voiding dysfunction, or adverse events.

136 complications include but are not limited to voiding dysfunction, urinary retention, vaginal extrusio

137 th overactivity syndromes and nonobstructive voiding dysfunction.

138 ck therapy for the treatment of recalcitrant voiding dysfunction.

139 reatment of benign prostatic hyperplasia and voiding dysfunction.

140 rned voiding patterns that contribute to the voiding dysfunction.

141 offers promise for managing both storage and voiding dysfunction.

142 basis of lower renal tract malformations and voiding dysfunction.

143 urinary-tract infection, hydronephrosis, and voiding dysfunctions as a result of neurogenic bladders.

144 orrelation between motoneuronal survival and voiding efficiency was observed in the implanted group.

145 duction of bladder activity by more frequent voiding facilitated by increased urine volume in hydrate

146 tivity (-38%, n = 6) or completely inhibited voiding (four sites).

147 unced autonomic arousal and increased reflex voiding frequency (+237%, n = 34).

148 o reduce ongoing GABA tone, increased reflex voiding frequency (+467%, n = 16) and tonic activity in

149 er sites in the PAG, either depressed reflex voiding frequency (-60%, n = 7) and tonic EUS EMG activi

150 th factor (NGF) in contributing to increased voiding frequency and altered sensation from the urinary

151 GF-beta) signalling contributes to increased voiding frequency and decreased bladder capacity with cy

152 In aged mice, increased voiding frequency and enhanced low threshold afferent ne

153 der hyporeflexia, characterized by decreased voiding frequency and increased bladder capacity, but no

154 Bladder tau decreased with voiding frequency and was markedly decreased with early

155 for 1 h reduced the effect of acetic acid on voiding frequency as reflected by an increase in the int

156 concentrations of OxoM (5 microM) decreased voiding frequency by approximately 30%, an effect blunte

157 concentrations of OxoM (40 microM) increased voiding frequency by approximately 45%, an effect blunte

158 g) did not mimic the intravesical effects on voiding frequency.

159 ion that may ultimately facilitate increased voiding frequency.

160 in the bladder perfusate, and also increased voiding frequency; these effects were suppressed by BB-F

161 technique, short-term and long-term results, voiding function after feminizing genitoplasty, and the

162 e control vector (HSV-lacZ), indicating that voiding function was improved after HSV vector-mediated

163 rent nerve interactions can influence reflex voiding function.

164 ntraspinal circuit that regulates sexual and voiding function.

165 in inappropriate places without a change in voiding function.

166 ladder and that luminal release can modulate voiding function.

167 r the luminal surface of the bladder affects voiding functions via mechanisms involving ATP and NO re

168 nt patient's behavior, especially his or her voiding habits, and by teaching skills for preventing ur

169 usly hypertensive rats (SHR) and hyperactive voiding in rats with urethral obstruction are characteri

170 hyperinnervation of bladder and hyperactive voiding in SHRs.

171 brainstem neurons involved in continence and voiding in the female rat.

172 ion, and a treatment option that can restore voiding in this group of patients - sacral nerve electri

173 at 536 +/- 61 muGy/MBq using a 4.8-h bladder-voiding interval for the male phantom.

174 .5 +/- 0.30 mSv (at 300 MBq), with a bladder-voiding interval of 0.75 h.

175 Reducing the urinary voiding interval to 60 or 90 min lowered the urinary bla

176 Simulations in which the urinary voiding interval was decreased from 4.8 to 0.6 h produce

177 jected dose of 10 mCi (370 MBq) and a 1-hour voiding interval, a patient would be exposed to an effec

178 For a 0.75-h voiding interval, the bladder dose was reduced to 0.10 +

179 With a 2.4-h voiding interval, the calculated effective dose was 6.98

180 Assuming a 2.4-h urine voiding interval, the calculated effective doses from th

181 With a 2.4-h voiding interval, the effective dose was 5.7 microSv/MBq

182 For a 1-h voiding interval, these doses were reduced to 15 +/- 2 m

183 the dynamic bladder model with a 1-h bladder-voiding interval.

184 rinary bladder wall (0.021 mGy/MBq with 2-hr voiding intervals or 0.029 mGy/MBq with 4-hr voiding int

185 , avoidance of excessive fluids, and regular voiding intervals that reduce urgency incontinence episo

186 y bladder wall (with hydration and 1- to 2-h voiding intervals).

187 voiding intervals or 0.029 mGy/MBq with 4-hr voiding intervals).

188 00868 +/- 0.00481 cGy/MBq (to bladder wall) (voiding intervals, 1-2 h), and the effective dose equiva

189 d 0.067 +/- 0.007 mGy/MBq for the 2- and 4-h voiding intervals, respectively.

190 be reduced significantly by frequent bladder-voiding intervals.

191 either restricted in this age group nor does voiding into a diaper cause disruption.

192 Normal voiding is dependent on mechanoceptive Adelta-fiber blad

193 age were excluded from consideration because voiding is neither restricted in this age group nor does

194 in the physiological control of storage and voiding is unclear.

195 a cells functions to recover membrane during voiding, is integrin regulated, occurs by a RhoA- and dy

196 om dysfunctional voiding - unfavorable pouch voiding mechanics, insufficient pouch pressure generatio

197 re inspected for evidence of interruption or voiding midstudy.

198 rem/mCi +/- 0.436 [0.233 mSv/MBq +/- 0.118], voiding model) and uptake in the spleen (0.250 rem/mCi +

199 With the 1-h voiding model, these doses reduced to 15 +/- 1 muSv/MBq

200 376 +/- 19 muGy/MBq using the 4.8-h bladder-voiding model.

201 q for the no-voiding, 2.5-h-voiding, and 1-h-voiding models, respectively.

202 th arousal and attentional processes so that voiding occurs under appropriate conditions.

203 ange or a reduction in urethral pressure and voiding of small amounts of fluid.

204 dramatic cell deformation during filling and voiding of the bladder.

205 dder is locked in storage mode, switching to voiding only when it is judged safe and/or socially appr

206 well as behavioral therapy to modify learned voiding patterns that contribute to the voiding dysfunct

207 discussing voiding physiology, dysfunctional voiding patterns, acute urinary retention, urine collect

208 er and more likely to receive a diagnosis of voiding-phase dysfunction, but these changes did not lea

209 earch on urinary tract infection, discussing voiding physiology, dysfunctional voiding patterns, acut

210 ptoms were not significantly associated with voiding place or position preference.

211 voiding and sex on flow rate, flow pattern, voiding pressures, presence of overactivity and interpre

212 with urinary incontinence and dysfunctional voiding problems can be very difficult at times.

213 play a major role in the causation of their voiding problems, whereas delayed arousal from sleep in

214 prostate size, residual urinary volume after voiding, quality of life, laboratory values, and the rat

215 1.38), prostate size, residual volume after voiding, quality of life, or serum prostate-specific ant

216 An earlier bladder voiding reduced these doses by 25%-45%.

217 helium functions as a sensor to initiate the voiding reflex, during which it releases ATP via multipl

218 ladder of the rat increased the frequency of voiding reflexes by 8 fold and increased c-fos expressio

219 ections to Barrington's nucleus may regulate voiding reflexes during sexual behavior.

220 : (1) inhibitory control of the frequency of voiding reflexes presumably by regulating afferent proce

221 The voiding schedule includes an extended nighttime gap duri

222 .004 mSv/MBq, respectively, depending on the voiding schedule.

223 Urinary voiding should be performed during (18)F-FPEB studies to

224 erform a second cycle of bladder filling and voiding should take into account the pretest probability

225 We found that voiding-stimulated CE, which depended on beta(1) integri

226 treatment of disorders of urine storage and voiding such as overactive bladder.

227 rnal enuresis, while others manifest diurnal voiding symptoms (DVS) as well, including urinary freque

228 creased risk of a rapid change in irritative voiding symptoms and decreased urinary flow but not obst

229 d decreased urinary flow but not obstructive voiding symptoms or prostate size.

230 ate volume, serum prostate-specific antigen, voiding symptoms, and urinary flow were minor.

231 ompared with no NSAID use) and overall LUTS, voiding symptoms, or nocturia in men or women.

232 us treatments for BPH with questionnaires on voiding symptoms, related complications, and sexual func

233 atios and 95% confidence intervals for LUTS, voiding symptoms, storage symptoms, and nocturia.

234 n of novel therapies for pain and irritative voiding symptoms.

235 ptoms, 40% had filling symptoms, and 18% had voiding symptoms.

236 omplaints such as irritative and obstructive voiding symptoms: urinary frequency, urgency, and dysuri

237 ms have a combination of both 'storage' and 'voiding' symptoms, suggesting possible coexisting bladde

238 ficantly lower rate of disruption because of voiding than the F-15 protocol, likely due to the shorte

239 The optimal first voiding time is from 40 min to 3 hr postadministration,

240 re must be differentiated from dysfunctional voiding - unfavorable pouch voiding mechanics, insuffici

241 ewing the circumcision debate, dysfunctional voiding, vesicoureteral reflux, and the diagnosis and fo

242 The mean time of voiding was 18.3 min (range, 12-25 min) for F-15 patient

243 nce episodes as recorded in a 7-day diary of voiding were similar in the intervention group and the c

244 orders includes urge syndrome, dysfunctional voiding with an uncoordination between the detrusor and

245 includes (1) uroflowmetry, an assessment of voiding without catheters in place; (2) cystometry, whic