ライフサイエンスコーパス: voltage-dependent

1 nucleotides of AtALMT9-mediated currents was voltage dependent.

2 rge unitary conductance, and are only weakly voltage dependent.

3 rvation that outer hair cell surface area is voltage-dependent.

4 e Na(+) gradient, rendering it intrinsically voltage-dependent.

5 nal properties of Cav3.2 channels, including voltage-dependent activation and inactivation and kineti

6 ion of peak transient Na(+) currents and the voltage-dependent activation and steady-state inactivati

7 -function) due to a hyperpolarizing shift of voltage-dependent activation combined with either decrea

8 es the peak current density and improves the voltage-dependent activation gating of CaV1.2 channels w

9 1 and the CaValpha2delta1-dependent shift in voltage-dependent activation gating.

10 In some K2P channels, strong voltage-dependent activation has been observed, but the

11 of Cav1.4FL, Cav1.4Deltaex 47 shows similar voltage-dependent activation in the presence and absence

12 tructural determinants that regulate CDI and voltage-dependent activation of Cav1.4, and is necessary

13 2.1 channels fluxing Li(+) currents, so that voltage-dependent activation of channel gating is no lon

14 surface proteolytic cleavage of alpha2delta, voltage-dependent activation of channels is promoted, in

15 concentration, Rg3 shifted the half-point of voltage-dependent activation of currents by -14 mV for E

16 , we observed a hyperpolarizing shift in the voltage-dependent activation of Ih in SNI neurons, where

17 P abolished the hyperpolarizing shift in the voltage-dependent activation of Ih observed in SNI neuro

18 s processing is an essential step permitting voltage-dependent activation of plasma membrane N-type (

19 lytic processing of alpha2delta then permits voltage-dependent activation of the channels, acting as

20 i's organ drives cochlear amplification by a voltage-dependent activation of the molecular motor, pre

21 n effects, including a depolarizing shift of voltage-dependent activation or a hyperpolarizing shift

22 n R1389H channels including ion selectivity, voltage-dependent activation or voltage-dependent inacti

23 and visualize discrete transitions along the voltage-dependent activation pathway.

24 13) containing alpha2delta4 exhibited weaker voltage-dependent activation than those with alpha2delta

25 BHQ slows deactivation, inhibits voltage-dependent activation, and potentiates Ca(2)(+)-d

26 f-function effects (hyperpolarizing shift of voltage-dependent activation, increased amplitude) in ni

27 increase in L-type current without altering voltage-dependent activation, thus reflecting an increas

28 had similar single-channel conductances and voltage-dependent activation.

29 yclic nucleotide-gated channels that display voltage-dependent activity that is also modulated by cAM

30 Voltage-dependent and calcium-activated K(+) (MaxiK, BK)

31 Moreover, we show that the inhibition is voltage-dependent and competes with that by amiloride, a

32 nity of M2R for distinct ligands varies in a voltage-dependent and ligand-specific manner.

33 eraction, modulation of Cav2.2 was primarily voltage-dependent and transiently relieved by depolarizi

34 The photothermal heat shock current was voltage-dependent and was from thermally driven displace

35 ngly enhanced by preincubation, was slightly voltage-dependent, and its washout was accelerated by bo

36 Moreover, the inhibition was voltage-dependent, and strong depolarizing prepulses att

37 (3) Glycine directly bounded to voltage dependent anion channel 1 (VDAC1) on the mitocho

38 ation of a peptide that resulted in block of voltage-dependent anion channel (VDAC) "rescued" mitocho

39 ith the mitochondrial outer membrane protein voltage-dependent anion channel (VDAC) blocks traffic th

40 1-ATP synthase, and the mitochondrial marker voltage-dependent anion channel (VDAC) have various expr

41 The voltage-dependent anion channel (VDAC) is the major path

42 The voltage-dependent anion channel (VDAC) is the most abund

43 The voltage-dependent anion channel (VDAC) mediates and gate

44 d the underlying mechanism was identified as voltage-dependent anion channel (VDAC) oligomerization.

45 Voltage-dependent anion channel (VDAC) proteins are majo

46 scription factor A (TFAM), citrate synthase, voltage-dependent anion channel (VDAC), and cytochrome c

47 centrations, alpha-syn reversibly blocks the voltage-dependent anion channel (VDAC), the major channe

48 ns represented by the metabolite transporter voltage-dependent anion channel (VDAC), the protein tran

49 N-SH cells where it is co-localized with the voltage-dependent anion channel (VDAC), which is also a

50 les involved in Ca(2+) exchange, through the voltage-dependent anion channel (VDAC)-1/glucose-regulat

51 The voltage-dependent anion channel 1 (VDAC-1) is an importa

52 rated that mitochondrial calcium released by voltage-dependent anion channel 1 (VDAC1) after sciatic

53 ,4,5-trisphosphate receptors (IP3Rs) and the voltage-dependent anion channel 1 (VDAC1) at the outer m

54 hemical mitophagy inducer, overexpression of voltage-dependent anion channel 1 (VDAC1) induced Parkin

55 The outer mitochondrial membrane protein voltage-dependent anion channel 1 (VDAC1) is a convergen

56 by gene ontology analysis, the expression of voltage-dependent anion channel 1 (VDAC1), a constituent

57 The voltage-dependent anion channel 1 (VDAC1), found in the

58 acromolecular complex composed of the VDAC1 (voltage-dependent anion channel 1), the GRP75 (chaperone

59 We further discover the mitochondrial porin voltage-dependent anion channel 2 (VDAC2) as essential c

60 for the first time that StAR interacts with voltage-dependent anion channel 2 (VDAC2) at the mitocho

61 TPR3 binds to voltage-dependent anion channel 3, but although this is

62 el membrane proteins, including the abundant voltage-dependent anion channel and the cation-preferrin

63 Moreover, the voltage-dependent anion channel antagonist TRO19622 had

64 ability and reduced epithelial mitochondrial voltage-dependent anion channel expression were observed

65 structurally different beta-barrel channels: voltage-dependent anion channel from outer mitochondrial

66 As expected, TcMCU was co-localized with the voltage-dependent anion channel to the mitochondria.

67 of our neurosteroid ligand in the IMP, mouse voltage-dependent anion channel-1 (mVDAC1), and top-down

68 Voltage-dependent anion channel-1 (VDAC1) is a highly re

69 Human voltage-dependent anion channel-2 (hVDAC-2) functions pr

70 The voltage-dependent anion channels (VDAC) were identified

71 Voltage-dependent anion channels (VDACs) are the most ab

72 rential mitochondrial localization of HK2 at voltage-dependent anion channels provides access to ATP

73 bitory factor, antioxidant SOD3, ion channel voltage-dependent anion-selective channel protein 3 (VDA

74 We observed voltage-dependent asymmetric distortions of the VDAC-1 b

75 hesion molecule (NRCAM) and calcium channel, voltage-dependent, beta 2 subunit (CACNB2), as well as g

76 l(-) channel (CaCC), is activated by direct, voltage-dependent, binding of intracellular Ca(2+) .

77 sing calcium permeability, and relieving the voltage-dependent block by endogenous intracellular poly

78 We use a detailed model to account for voltage dependent Ca(2+) release and inactivation.

79 localize and form ion channel complexes with voltage-dependent Ca(2+) (CaV) channels.

80 ctional beta-cell mass determination through voltage-dependent Ca(2+) channel (VDCC)-mediated interna

81 e thought to act as allosteric modulators of voltage-dependent Ca(2+) channel activation, whereas phe

82 rticosterone (11-DHC) and cortisone suppress voltage-dependent Ca(2+) channel function and Ca(2+) flu

83 -gated Na(+) channels (Nav) and subsequently voltage-dependent Ca(2+) channels (Cav).

84 Voltage-dependent Ca(2+) channels (CaVs) represent the p

85 pecific SM tracking of transmembrane CD4 and voltage-dependent Ca(2+) channels (VDCC) was achieved wi

86 [Ca(2+)] spikes, which depend on the L-type voltage-dependent Ca(2+) channels (VDCCs) and require ac

87 hibitors of BDNF-TrkB signaling or of L-type voltage-dependent Ca(2+) channels (VDCCs) block the anti

88 lation of ryanodine receptors (RyRs), L-type voltage-dependent Ca(2+) channels (VDCCs) or TMEM16A Ca(

89 ) action potentials due to the activation of voltage-dependent Ca(2+) channels (VDCCs), which leads t

90 auses cell depolarization, Ca(2+) influx via voltage-dependent Ca(2+) channels and a rise in intracel

91 strointestinal muscles are important because voltage-dependent Ca(2+) channels in smooth muscle cells

92 However, glucose-stimulated Ca(2+) entry via voltage-dependent Ca(2+) channels is reduced in islet be

93 t calcium channel-forming subunits of L-type Voltage-dependent Ca(2+) channels, expressed in many typ

94 structure dissimilarity to known ligands for voltage-dependent Ca(2+) channels, selective binding aff

95 tic beta-cells is caused by Ca(2+) entry via voltage-dependent Ca(2+) channels.

96 During a triggered AP, a voltage-dependent Ca(2+) conductance was fractionally ac

97 1 reduced K(+)-induced insulin secretion and voltage-dependent Ca(2+) currents.

98 TOCs) that hyperpolarize the cell and reduce voltage-dependent Ca(2+) entry.

99 t hyperpolarize the cell membrane and reduce voltage-dependent Ca(2+) influx.

100 annels, depolarizing beta cells, and opening voltage-dependent Ca2+ channels to elicit insulin exocyt

101 ell depolarization and subsequent opening of voltage-dependent Ca2+ channels to elicit insulin granul

102 del is robust and reproduces many aspects of voltage dependent calcium signaling in frog skeletal mus

103 cally reduces calcium influx by inactivating voltage-dependent calcium and sodium channels and decrea

104 TMEM16A is opened by voltage-dependent calcium binding and regulated by perme

105 CAM(flx) sprouts were associated with L-type voltage-dependent calcium channel (L-VDCC) immunoreactiv

106 el invokes the modulation of CaV2.3 (R-type) voltage-dependent calcium channel (VDCC) currents observ

107 NNK induces voltage-dependent calcium channel (VDCC)-intervened calc

108 n, the metabotropic glutamate receptor 6 and voltage-dependent calcium channel alpha1.4, are not dete

109 CACNA1C encoding a subunit of a voltage-dependent calcium channel was upregulated in cal

110 Voltage-dependent calcium channels (Cav) of the T-type f

111 ated inhibitory peptide; or the blocker of L-voltage-dependent calcium channels (L-VDCCs), nifedipine

112 Here, we describe how surface mobility of voltage-dependent calcium channels (VDCCs) modulates rel

113 This effect was occluded by block of R-type voltage-dependent calcium channels (VDCCs), but not by i

114 This limits calcium influx through voltage-dependent calcium channels and dampens adrenergi

115 The CaV2.2 (N-type) and CaV2.1 (P/Q-type) voltage-dependent calcium channels are prevalent through

116 he emerging model is that calcium influx via voltage-dependent calcium channels at the calcium microd

117 eviously unknown yet critical role of L-type voltage-dependent calcium channels in the expression and

118 ifs is inducible by influx of Ca(2+) through voltage-dependent calcium channels upon beta-adrenergic

119 Moreover, blockade of L-type voltage-dependent calcium channels with diltiazem or nif

120 hippocalcin might play a role in regulating voltage-dependent calcium channels.

121 and/or S1P2 receptors, partially via L-type voltage-dependent calcium channels.

122 these effects are dependent on activation of voltage-dependent calcium channels.

123 with strong similarity to canonical VSDs in voltage-dependent cation channels and enzymes.

124 Gating of voltage-dependent cation channels involves three general

125 Voltage-dependent CaV1.2 L-type Ca(2+) channels (LTCC) a

126 452A nearly eliminated the effects of Rg3 on voltage-dependent channel gating but did not prevent the

127 elated potassium channel (hERG, Kv11.1) is a voltage-dependent channel known for its role in repolari

128 everely distorted measurements and recruited voltage-dependent channels.

129 More importantly, the voltage-dependent characteristics of Ln(3+)'s action led

130 We also note that the occurrence of voltage-dependent charge movements in other SLC26 family

131 This voltage-dependent closure is not dependent on the presen

132 The objective of this study was to develop a voltage dependent compartment model of Ca(2+) dynamics i

133 dendrites maintain the independence of their voltage-dependent computations, despite these repeated v

134 udied the effects of non-Markovian power-law voltage dependent conductances on the generation of acti

135 Following developmental changes in voltage-dependent conductances, these same neurons becom

136 gnal via the influx of calcium ion (Ca(2+)), voltage-dependent conformational change (VDeltaC), or a

137 nal [Cl(-)] suggest that CLC gating involves voltage-dependent conformational changes as well as coor

138 ants were blocked, but Na(+) binding and the voltage-dependent conformational transitions were unaffe

139 time course and magnitude of this secondary, voltage-dependent contribution to ACh-evoked potassium c

140 We also observe a voltage-dependent contribution whose origin is only part

141 his phenomenon depends on the recruitment of voltage-dependent currents (e.g., NMDAR-mediated Ca(2+)

142 Neuronal subthreshold voltage-dependent currents determine membrane properties

143 PICs are intrinsic, voltage-dependent currents that activate strongly when m

144 L-type Ca(2+) channels terminates because of voltage-dependent deactivation and not by Ca(2+)-depende

145 synapses between Cx36-KO neurons had faster voltage-dependent decay kinetics and conductance asymmet

146 ound to exhibit optical properties including voltage-dependent electroluminescence and wide-range exc

147 was observed, there was a mild impairment of voltage-dependent electromotility of outer hair cells.

148 ion acts to refill those stores by promoting voltage-dependent entry of extracellular calcium.

149 nonfluorescent states, D1 and D2, exist in a voltage-dependent equilibrium.

150 sium ('SK') channels, and longer-lasting and voltage-dependent excitation involving non-specific cati

151 sium ('SK') channels, and longer-lasting and voltage-dependent excitation involving non-specific cati

152 rd site (site III) exclusively binds Na in a voltage-dependent fashion.

153 acidification-induced uncoupling, absence of voltage-dependent fast inactivation, longer channel open

154 ne such gene is Shaker (Sh), which encodes a voltage-dependent fast K(+) channel (Kv1.1).

155 uorometry measurements were performed with a voltage-dependent fluorophore/quencher pair.

156 d current decrease to two main mechanisms: a voltage-dependent "foot-in-the-door" pore block and an a

157 PhTX-343 inhibition was strongly voltage-dependent for all subunit combinations except al

158 region, which likely underlie differences in voltage-dependent gating between these channels.

159 Here, we show voltage-dependent gating is common to most K2P channels

160 llular ATP, respectively, as cofactors, thus voltage-dependent gating is dependent on multiple stimul

161 t results from a unique combination of steep voltage-dependent gating kinetics and ultra-fast voltage

162 y studied, the detailed structural basis for voltage-dependent gating mechanisms remain obscure.

163 Connexin-based channels exhibit two distinct voltage-dependent gating mechanisms termed slow and fast

164 n peak current density and modulation of the voltage-dependent gating of CaV1.2.

165 ted increase in the peak current density and voltage-dependent gating of CaV1.2.

166 a viral potassium channel, was able to drive voltage-dependent gating of the channel pore.

167 Voltage-dependent gating of the channel was regulated by

168 ding pH-dependent Zn(2+) inhibition, lack of voltage-dependent gating, and activation at modest pH va

169 novel insights on fundamental principles of voltage-dependent gating.

170 d, which suggest an alternative mechanism of voltage-dependent gating.

171 act to modulate Ca(2+)-calmodulin as well as voltage-dependent gating.

172 in and exhibits prominent, barttin-regulated voltage-dependent gating.

173 that acidification reversibly increases its voltage-dependent gating.

174 porters in the CLC family display pronounced voltage-dependent gating.

175 etermine the effects of mutations on channel voltage-dependent gating.

176 ce reports of natural full agonists at other voltage-dependent GPCRs only show alterations in affinit

177 ylthiophene) electrochemical devices exhibit voltage-dependent heterogeneous swelling consistent with

178 was found to be a K(+)-selective channel of voltage-dependent high capacity and low affinity, wherea

179 mutation also caused an opposite increase in voltage-dependent inactivation (VDI), resulting in a mul

180 alent, as they produce comparable defects in voltage-dependent inactivation and cause similar manifes

181 pendent, and involved enhanced steady-state, voltage-dependent inactivation of IA.

182 rectify during depolarization due to rapid, voltage-dependent inactivation, but rebound during repol

183 ta2X13-containing channels exhibited greater voltage-dependent inactivation.

184 selectivity, voltage-dependent activation or voltage-dependent inactivation.

185 In stellate cells, a voltage-dependent increase in membrane resistance at sub

186 ion channel and act as blockers are use- and voltage-dependent inhibitors of NMDAR activity and have

187 f the oxytocin receptor and is mediated by a voltage-dependent inward current.

188 Results Experiments revealed tube voltage-dependent iodine attenuation curves, which were

189 canonical VSD-pore coupling, are at work in voltage-dependent ion channel activation.

190 hmogenic electrical remodelling, we examined voltage-dependent ion channels in cardiomyocytes.

191 losing of the central ion-conducting pore in voltage-dependent ion channels is gated by changes in me

192 tivation is an intrinsic property of several voltage-dependent ion channels, closing the conduction p

193 largely shaped by different combinations of voltage-dependent ion channels.

194 uit of Xenopus tadpoles, I study how certain voltage-dependent ionic currents affect firing threshold

195 and large-conductance, Ca(2+)-activated, and voltage dependent K(+) (BK) channels.

196 4-aminopyridine or related voltage-dependent K channel blockers could be a useful a

197 actory information by decreasing activity of voltage-dependent K channels (Kv1.3).

198 ly expressed, large-conductance, Ca(2+)- and voltage-dependent K(+) (BK) channels play an important r

199 Ca(2+)- and voltage-dependent K(+) (BK) currents and the expression

200 KEY POINTS: Several different voltage-dependent K(+) (KV ) channel isoforms are expres

201 ance-size arteries express several different voltage-dependent K(+) (KV ) channels, including KV 1.5

202 study, we examine the mechanism by which the voltage-dependent K(+) (Kv) channel Kv2.1 (KCNB1) facili

203 d E321 much closer together than observed in voltage-dependent K(+) (Kv) channel structures, requirin

204 ontrols the membrane expression of the human voltage-dependent K(+) channel human ether-a-go-go-relat

205 STRACT: Large-conductance KCa (BK) and other voltage-dependent K(+) channels (Kv) are highly expresse

206 ance Ca(2+) -activated K(+) channel (BK) and voltage-dependent K(+) channels (Kv) on [Ca(2+) ]i respo

207 on with peptide toxins that target different voltage-dependent K(+) channels.

208 amplitude and activation rate of whole-cell voltage-dependent K(+) currents sensitive to the Kv7 blo

209 At 5 weeks, only voltage-dependent K(+) currents were reduced, possibly r

210 Our data suggest that voltage-dependent K+ channel inhibition with 4-aminopyri

211 The Kv-like (potassium voltage-dependent) K(+) channels at the plasma membrane,

212 NaV1.5 constructs were created to track the voltage-dependent kinetics of conformational changes wit

213 These currents saturated with a voltage-dependent Km = 90 microM at -80 mV.

214 s-nitro-phenyl ROMK inhibitor VU591 exhibits voltage-dependent knock-off at hyperpolarizing potential

215 Voltage-dependent Kv3 channels play an important role in

216 nerative disease induced by mutations in the voltage-dependent Kv3.3 potassium channel.

217 a hormone known to modulate the activity of voltage-dependent L- and T-type Ca(2+) channels that are

218 The voltage-dependent L-type Ca(2+) channel was identified a

219 d Cl(-) channels (ANO1, encoded by Ano1) and voltage-dependent L-type Ca(2+) channels (CavL , encoded

220 tivated Cl(-) channels (encoded by Ano1) and voltage-dependent L-type Ca(2+) channels (encoded by Cac

221 Calcium influx through the voltage-dependent L-type calcium channel (CaV1.2) rapidl

222 dated the BD risk genes ankyrin-3 (ANK3) and voltage-dependent L-type calcium channel subunit beta-3

223 CCs, including mutations in calcium channel, voltage-dependent, L-type, alpha1D-subunit (CACNA1D; 6 o

224 We then characterized the voltage-dependent lifetime of ASAP1, CAESR, and ArcLight

225 ASAP1 and CAESR showed voltage-dependent lifetimes, whereas ArcLight did not.

226 rovides an alternative view, suggesting that voltage-dependent linear capacitance changes are not rea

227 One component is gate-voltage dependent, linear in power at room temperature a

228 Blocking BK channels relieves voltage-dependent magnesium block of NMDA receptors, the

229 glutamate in combination with the relief of voltage-dependent magnesium block to open an ion conduct

230 ated by increased intracellular calcium in a voltage-dependent manner but, unlike many other TRP chan

231 Ca(2+) sparks are generated in a voltage-dependent manner to initiate spontaneous transie

232 ABSTRACT: Ca(2+) sparks are generated in a voltage-dependent manner to initiate spontaneous transie

233 bits the whole cell NMDA-evoked current in a voltage-dependent manner with IC50 values of 20.9 mum, 5

234 pHo revealed that Ho both activates IH (in a voltage-dependent manner) and inhibits it (in a voltage-

235 presequence peptide in a concentration- and voltage-dependent manner.

236 teroids inhibit mutated NMDAR responses in a voltage-dependent manner.

237 te Ca(2+)-induced Ca(2+) release events in a voltage-dependent manner.

238 omoting a rise in intracellular pH through a voltage-dependent mechanism.

239 at thalamocortical neurons have postsynaptic voltage-dependent mechanisms that can amplify integrated

240 Outer hair cells (OHC) possess voltage-dependent membrane bound molecular motors, ident

241 consideration for the role of non-linear and voltage-dependent membrane properties.

242 meric GluN1-GluN2B-GluN2D NMDARs have weaker voltage-dependent Mg(2+) block (delta = 0.56) than GluN1

243 rats as a model system, we characterize the voltage-dependent Mg(2+) block properties of triheterome

244 hree-state model implicitly avoids measuring voltage-dependent motor capacitance, it registers deltaC

245 -type inactivation in hERG1 K(+) channels is voltage-dependent, much faster in onset and greatly atte

246 Ketamine, a non-competitive, voltage-dependent N-Methyl-D-aspartate receptor (NMDAR)

247 SNAP caused a depolarizing shift in voltage-dependent N-type channel activation, but it had

248 Although voltage-dependent Na(+) channels are abundantly expresse

249 In this study, we showed that voltage-dependent Na(+) currents are regulated in a slow

250 s of AP-related costs are typically based on voltage-dependent Na(+) currents that drive active trans

251 ll explained by a tetrodotoxin-sensitive and voltage-dependent Na(+) persistent inward current (NaPIC

252 n of the myocardium, which causes a block of voltage-dependent Na+ channels throughout the myocardial

253 The voltage-dependent nature of this process leads to the tr

254 that decay in approximately 1 ms and mildly voltage-dependent NMDA receptor EPSCs of approximately 0

255 absorption spectrum of the rhodopsin lead to voltage-dependent nonradiative quenching of the appended

256 activates KCNQ2-5 channels by shifting their voltage-dependent opening to more negative voltages, is

257 al changes that likely prime the channel for voltage-dependent opening.

258 ing has to be considered together with other voltage-dependent partial reactions to cooperatively det

259 ents nerve injury-induced methylation of the voltage-dependent potassium (Kv) channel subunit Kcna2 p

260 ood flow responses, and identify upregulated voltage-dependent potassium channel (KV) number in cereb

261 The voltage-dependent potassium channel Kv1.3 plays essentia

262 nd that reduced functional expression of the voltage-dependent potassium channel subunit Kv1.1 substa

263 The voltage-dependent potassium channel subunit Kv3.3 is exp

264 s inhibits neuronal excitability through the voltage-dependent potassium channel, promotes white adip

265 Changes in voltage-dependent potassium channels (Kv channels) assoc

266 elopmental transcriptional regulation of Kv1 voltage-dependent potassium channels and the resulting p

267 that METH exposure affected the activity of voltage-dependent potassium channels in these neurons.

268 Voltage-gated Kv7 (KCNQ) channels are voltage-dependent potassium channels that are activated

269 nit KCNQ1 to generate the slowly activating, voltage-dependent potassium current (IKs) in the heart t

270 Little is known about the voltage-dependent potassium currents underlying spike re

271 These channels exhibit a behavior called voltage-dependent potentiation (VDP), which appears to b

272 deactivation seemingly occurs as a strictly voltage dependent process, implying that the kinetic eve

273 atch-clamping allows ion transport and other voltage-dependent processes to be studied while controll

274 erized by a unique time- and transjunctional voltage-dependent profile, we investigated whether the e

275 These findings aligned with the voltage-dependent profiles of L- and T-type Ca(2+) chann

276 poral-coding neurons but were transformed by voltage-dependent properties and push-pull excitatory-in

277 These voltage-dependent protein changes include asymmetric bar

278 and validate the method on two prototypical voltage-dependent proteins, the Kv1.2 K(+) channel and t

279 detection in all-silicon devices, exhibiting voltage-dependent quantum efficiencies in the range of a

280 hus, the alanine residue Ala(254) determines voltage-dependent rectification upon receptor desensitiz

281 optical stimulation due to ChR2 kinetics and voltage-dependent rectification.

282 ellular stores, mAChR activation facilitates voltage-dependent refilling of calcium stores, thereby m

283 Extending our study into the nonlinear, voltage-dependent regime, we increased stimulus amplitud

284 ic reticulum luminal Ca(2+), and Ca(2+)- and voltage-dependent regulation of RyR1-G4941K mutant chann

285 crease resurgent currents, which involve the voltage-dependent release of an open channel blocker.

286 From the gate voltage-dependent response, we extract the binding const

287 has no charge in this region, still presents voltage-dependent slow gating suggests that charges stil

288 carboxylic acid) (RPR) induces a pronounced, voltage-dependent slowing of hERG1 deactivation.

289 ated by a nonlinear dynamical interaction of voltage-dependent sodium and fast-inactivating potassium

290 osamide is an antiseizure agent that targets voltage-dependent sodium channels.

291 rrent (INaP), a non-inactivating mode of the voltage-dependent sodium current, paradoxically increase

292 We show that this discrepancy is due to a voltage-dependent spike-synchronization mechanism inhere

293 In rare cases, rapid, voltage-dependent stochastic switching is observed, cons

294 Current amplitudes were voltage dependent, strongly potentiated in oocytes prein

295 of Nav1.4 with the bound toxins, and reveal voltage-dependent structural changes related to channel

296 age sensitivity of exchange is caused by the voltage-dependent third Na(+) binding.

297 ion potential can exhibit distinct time- and voltage-dependent thresholds, and also demonstrating tha

298 gical model of repolarization similar to the voltage dependent, time-independent rectifying outward p

299 Moreover, Kv1.3 voltage-dependent transitions from closed to open confor

300 In summary, our work highlights that the voltage-dependent triggering of Ca(2+) sparks/STOCs is n