ライフサイエンスコーパス: voltage-dependent channel

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1 KCNE1 (MinK), producing a slowly activating voltage-dependent channel.

2 everely distorted measurements and recruited voltage-dependent channels.

3 micron) representing a high-density array of voltage-dependent channels.

4 E3) is essential for the specific control of voltage-dependent channel activation characteristics of

5 Corresponding to voltage-dependent channel activation, we observed a robu

6 ation and is not observed during ligand- and voltage-dependent channel activation.

7 trated sources of calcium are influx through voltage-dependent channels, and release of calcium from

8 odgkin-Katz (GHK) configuration that employs voltage-dependent channel binding functions for external

9 CNS physiology depend critically on powerful voltage-dependent channel block by external Mg(2+) (Mg(2

10 Voltage-dependent channel block by external Mg2+ (Mg2+(o

11 With the Woodhull model of voltage-dependent channel block, the values of Kd(0) wer

12 ty or at concentrations high enough to cause voltage-dependent channel block.

13 wn NMDAR antagonists are positively charged, voltage-dependent channel blockers.

14 specialized inactivation gate, as for other voltage-dependent channels, but by reclosure of the same

15 astrocytes occurs, at least in part, through voltage-dependent channels, but the exact mechanisms inv

16 mitochondrial burden can be traced to L-type voltage-dependent channels (channels for which there are

17 452A nearly eliminated the effects of Rg3 on voltage-dependent channel gating but did not prevent the

18 ut not generally different from wild type in voltage-dependent channel gating.

19 of putative mechanisms underlying Ca2+- and voltage-dependent channel inactivation, and (b) close fi

20 currents by causing nearly complete loss of voltage-dependent channel inactivation.

21 lving cytosolic calcium entry through L-type voltage-dependent channels is required for PACAP to evok

22 elated potassium channel (hERG, Kv11.1) is a voltage-dependent channel known for its role in repolari

23 Priming the stores with Ca2+ entry through voltage dependent channels modulated wave properties but

24 dependence on L-type Ca2+ channels and other voltage-dependent channels (Na+ and K+ channels) in the

25 ons remained negative to potentials at which voltage-dependent channels open.

26 ]i and tension are the entry of Ca2+ through voltage-dependent channels opened by depolarization or d

27 te how sensor coupling to the pore generates voltage-dependent channel opening, we solved the crystal

28 k calcium release from ER and influx through voltage-dependent channels prevented mitochondrial ROS a

29 ubthreshold synaptic inputs are modulated by voltage-dependent channels, raising the possibility that

30 results establish the general principles of voltage-dependent channel structure in a biological memb

31 e common structural motif for members of the voltage-dependent channel superfamily.

32 tion, part of which remained after all major voltage-dependent channels were blocked.

33 or changes in driving force occurred and no voltage-dependent channels were opened.

34 5.4 mmol/L K+; pipette, 140 mmol/L K+), two voltage-dependent channels were recorded with conductanc

35 nese hamster ovary cells produced Ca2+ - and voltage-dependent channels with macroscopic and unitary

36 wo structures provide atomic descriptions of voltage-dependent channels with unprecedented clarity.

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