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1 urons (appearance of tetrodotoxin-sensitive, voltage-dependent sodium channels).
2 e sensors and the outer pore of a eukaryotic voltage-dependent sodium channel.
3 ve expression of distinct genes encoding the voltage-dependent sodium channel.
4 e for subsequent assembly of ankyrin and the voltage-dependent sodium channel.
5 ed at nodes of Ranvier with ankyrinG and the voltage-dependent sodium channel.
6 osamide is an antiseizure agent that targets voltage-dependent sodium channels.
7 perexcitability), due to a local response of voltage-dependent sodium channels.
8 te outgrowth and the increased expression of voltage-dependent sodium channels.
9  of Ranvier and are candidates to couple the voltage-dependent sodium channel and neurofascin to the
10 ansmitter receptors and ion channels such as voltage-dependent sodium channels and GABA-gated chlorid
11 oxins that bind to the extracellular face of voltage-dependent sodium channels and retard channel ina
12            Tetrodotoxin (TTX, 0.3 microM), a voltage-dependent sodium channel antagonist that blocks
13                                              Voltage-dependent sodium channels are believed to have e
14                                              Voltage-dependent sodium channels are the central player
15                                              Voltage-dependent sodium channels are uniformly distribu
16                                  Blockade of voltage-dependent sodium channels by TTX also reduced CS
17 e fractions that also contain high levels of voltage-dependent sodium channels, caspr, and neuron-gli
18                                              Voltage-dependent sodium channels cluster at high densit
19 mbrane proteins (neurofascin, NrCAM, and the voltage-dependent sodium channel) colocalize within a sp
20  LQT3, is caused by mutations in the cardiac voltage-dependent sodium channel gene, SCN5A.
21 h--as well as changes in the inactivation of voltage-dependent sodium channels in living cells.
22                                  The type II voltage-dependent sodium channel is present in neuronal
23 Although action potentials are not involved, voltage-dependent sodium channels may enhance subthresho
24                                              Voltage-dependent sodium channels mediate transient curr
25  discovery of a new class of neuroprotective voltage-dependent sodium channel modulators exemplified
26       Rat GP neurons express fast, transient voltage-dependent sodium channels (NaF channels) in thei
27                      Binding to the neuronal voltage-dependent sodium channel (NVSC) was evaluated fo
28  show that both glutamate NMDA receptors and voltage-dependent sodium channels play roles in CSD.
29 nes caused delayed inactivation of mammalian voltage-dependent sodium channels, resulting in a positi
30 distribution of ankyrinG 480/270 kDa and the voltage-dependent sodium channel, suggesting that the ad
31 evident when tetrodotoxin was added to block voltage-dependent sodium channels, suggesting that inter
32  recruitment of ankyrinG 480/270 kDa and the voltage-dependent sodium channel to cluster sites contai
33               One neuronal target is NaV1.2, voltage-dependent sodium channel type II, to which CaM b
34 evealed that the pore-lining (P) segments of voltage-dependent sodium channels undergo sizable motion
35                                              Voltage-dependent sodium channels were studied in dissoc

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