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1 urons (appearance of tetrodotoxin-sensitive, voltage-dependent sodium channels).
2 e sensors and the outer pore of a eukaryotic voltage-dependent sodium channel.
3 ve expression of distinct genes encoding the voltage-dependent sodium channel.
4 e for subsequent assembly of ankyrin and the voltage-dependent sodium channel.
5 ed at nodes of Ranvier with ankyrinG and the voltage-dependent sodium channel.
6 osamide is an antiseizure agent that targets voltage-dependent sodium channels.
7 perexcitability), due to a local response of voltage-dependent sodium channels.
8 te outgrowth and the increased expression of voltage-dependent sodium channels.
9 of Ranvier and are candidates to couple the voltage-dependent sodium channel and neurofascin to the
10 ansmitter receptors and ion channels such as voltage-dependent sodium channels and GABA-gated chlorid
11 oxins that bind to the extracellular face of voltage-dependent sodium channels and retard channel ina
17 e fractions that also contain high levels of voltage-dependent sodium channels, caspr, and neuron-gli
19 mbrane proteins (neurofascin, NrCAM, and the voltage-dependent sodium channel) colocalize within a sp
23 Although action potentials are not involved, voltage-dependent sodium channels may enhance subthresho
25 discovery of a new class of neuroprotective voltage-dependent sodium channel modulators exemplified
29 nes caused delayed inactivation of mammalian voltage-dependent sodium channels, resulting in a positi
30 distribution of ankyrinG 480/270 kDa and the voltage-dependent sodium channel, suggesting that the ad
31 evident when tetrodotoxin was added to block voltage-dependent sodium channels, suggesting that inter
32 recruitment of ankyrinG 480/270 kDa and the voltage-dependent sodium channel to cluster sites contai
34 evealed that the pore-lining (P) segments of voltage-dependent sodium channels undergo sizable motion
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