ライフサイエンスコーパス: voltage-gated @1 channel

1 nificantly altered due to changes in several voltage-gated ion channels.

2 hloride ions, and are blocked by blockers of voltage-gated ion channels.

3 ey intermediate position in the evolution of voltage-gated ion channels.

4 ur gating charges commonly found in those of voltage-gated ion channels.

5 ivated state observed in structures of other voltage-gated ion channels.

6 dependent on nonlinearities associated with voltage-gated ion channels.

7 a; however, new studies have also implicated voltage-gated ion channels.

8 n channel function, with a major emphasis on voltage-gated ion channels.

9 revious studies concluded they are devoid of voltage-gated ion channels.

10 urrent is reminiscent of an omega current in voltage-gated ion channels.

11 assemble with and modulate the properties of voltage-gated K(+) channels.

12 transmembrane domain that closely resembles voltage-gated K(+) channels.

13 assemble with and modulate the properties of voltage-gated K(+) channels.

14 n (c.4447G>A; p.E1483K) in SCN8A, encoding a voltage-gated sodium channel.

15 gnal from the RyR1 facilitates gating of the voltage-gated calcium channel.

16 ibution from entry through NMDA receptors or voltage-gated sodium channels.

17 rom wild-type and epilepsy-associated mutant voltage-gated sodium channels.

18 latus that delays inactivation of vertebrate voltage-gated sodium channels.

19 seases, both genetic and acquired, involving voltage-gated calcium channels.

20 lta2 modulates the abundance and function of voltage-gated calcium channels.

21 s in a calcium-dependent manner and binds to voltage-gated calcium channels.

22 mechanisms of direct G protein inhibition of voltage-gated calcium channels.

23 the transient opening of different types of voltage-gated calcium channels.

24 s, including subunits of GABAA receptors and voltage-gated calcium channels.

25 mutated hippocalcin, mostly driven by N-type voltage-gated calcium channels.

26 This is the case for voltage-gated calcium channels.

27 e proteins that can bind and modulate L-type voltage-gated calcium channels.

28 L-type Ca(2+) currents conducted by voltage-gated calcium channel 1.2 (CaV1.2) initiate exci

29 om tarantula venom able to inhibit the human voltage-gated sodium channel 1.7 (hNaV1.7), a channel re

30 The prominent role of voltage-gated sodium channel 1.7 (Nav1.7) in nociception

31 The voltage-gated sodium channel 1.7 (Nav1.7) plays an impor

32 , in mice, this effect is mediated solely by voltage-gated sodium channel 1.8 (NaV1.8).

33 ting that ipRGC-to-DAC transmission requires voltage-gated Na(+) channels; (2) this transmission is p

34 Cone snail toxins are well known blockers of voltage-gated sodium channels, a property that is of bro

35 -activated potassium (BK) channels and Kv3.3 voltage-gated potassium channels accompanies the inabili

36 gulate TEP and JI Epistasis assays show that voltage-gated Na(+) channels act downstream of H2O2 to m

37 maging in rat slices, we find that dendritic voltage-gated sodium channels allow somatic action poten

38 presynaptic action potentials (APs) activate voltage-gated calcium channels, allowing calcium to ente

39 Inborn errors of Cacna1a, the P/Q-type voltage-gated calcium channel alpha subunit gene, expres

40 The mutations in the coding region of voltage-gated sodium channel alpha 1 subunit gene, SCN1A

41 However, expression analysis of voltage-gated sodium channel alpha subunits revealed NaV

42 a2-subunits in the ventricle is to chaperone voltage-gated sodium channel alpha-subunits to the plasm

43 GABAB receptors (GABAB Rs) suppress voltage-gated calcium channels and activate G-protein co

44 In voltage-gated K(+) channels and the prokaryotic KcsA cha

45 BTX targets voltage-gated Na(+) channels and enables them to open pe

46 inactivated conformational cycle in a single voltage-gated sodium channel and give insight into the s

47 enous cannabinoids have been shown to target voltage-gated sodium channels and cannabidiol has recent

48 s required for the interaction of FGF14 with voltage-gated sodium channels and neuronal excitability.

49 rs and not through NMDA receptors or through voltage-gated sodium channels and that the spine neck is

50 MDA, GABA-A, mGluR2/3 receptors and Nav, Cav voltage-gated ion channels) and demonstrated the ability

51 led the peptide's putative molecular target (voltage-gated sodium channels) and mechanism of action (

52 ragile X mental retardation protein complex, voltage-gated calcium channels, and genes implicated in

53 pha2delta proteins are auxiliary subunits of voltage-gated calcium channels, and influence their traf

54 ts, at least in part, through its actions on voltage-gated sodium channels, and resurgent current may

55 Multi-domain voltage-gated ion channels appear to have evolved throug

56 Voltage-gated calcium channels are essential players in

57 The auxiliary alpha2delta subunits of voltage-gated calcium channels are extracellular membran

58 EAG-like (ELK) voltage-gated potassium channels are abundantly expresse

59 Kv3.1 and Kv3.2 voltage-gated potassium channels are expressed on parval

60 ABSTRACT: Voltage-gated sodium channels are critical for neuronal

61 Activation and inactivation of voltage-gated sodium channels are critical for proper el

62 Voltage-gated sodium channels are crucial determinants o

63 Fast opening and closing of voltage-gated sodium channels are crucial for proper pro

64 Voltage-gated sodium channels are essential for electric

65 Voltage-gated sodium channels are responsible for action

66 However, when voltage-gated sodium channels are temporarily blocked, c

67 Voltage-gated sodium channels are the primary target of

68 ich includes oscillatory calcium signals via voltage-gated calcium channels as a key component.

69 enes and their implications, with a focus on voltage-gated calcium channels as part of the disease pr

70 Oligomers of homomeric voltage-gated potassium channels associate early in biog

71 myelinated nerves requires the clustering of voltage-gated sodium channels at nodes of Ranvier.

72 myelinating Schwann cells, such as clustered voltage-gated sodium channels at the node of Ranvier and

73 deficient for exon 1b, PV interneurons lack voltage-gated sodium channels at their axonal initial se

74 tors for thymoma (as recognized for neuronal voltage-gated calcium channel autoantibodies).

75 GIC behavior, we addressed how the bacterial voltage-gated sodium channel (BacNa(V)) C-terminal cytop

76 Bacterial voltage-gated sodium channels (BacNavs) serve as models

77 Mutations in SCN2B, encoding voltage-gated sodium channel beta2-subunits, are associa

78 nts revealed that Ae1a potently inhibits the voltage-gated sodium channel BgNaV1 from the German cock

79 Voltage-gated calcium-channel blockade prevents iron ent

80 Consistent with this idea, administration of voltage-gated potassium channel blockers restores conduc

81 nels share a general architecture with other voltage-gated ion channels but are distinguished by the

82 rsed by restoring the expression levels of a voltage-gated calcium channel, cacophony.

83 , single-channel current amplitude of native voltage-gated calcium channels can be resolved accuratel

84 Voltage-gated calcium channels can coassemble with auxil

85 ction mutations in hERG (encoding the Kv11.1 voltage-gated potassium channel) cause long-QT syndrome

86 lease-activated calcium modulator 1 but also voltage-gated calcium channel (Cav) 1 channels.

87 FMRP was shown to directly interact with the voltage-gated calcium channel, Cav 2.2, and reduce its t

88 e pore-forming alpha1 subunit of the cardiac voltage-gated calcium channel Cav1.2 at Ser1928, suggest

89 Here we show that the voltage-gated calcium channel CaV1.3 and the big conduct

90 ional and mechanical coupling between L-type voltage-gated calcium channels (CaV1.1) and the ryanodin

91 des the pore-forming subunit of the neuronal voltage-gated calcium channel Cav2.1.

92 encoding the alpha1A subunit of the P/Q-type voltage-gated calcium channel Cav2.1.

93 CaVbeta subunits interact with the voltage-gated calcium channel CaV2.2 on a site in the in

94 CACNA1H encodes a voltage-gated calcium channel (CaV3.2) expressed in adre

95 thesis that TSP4 activates its receptor, the voltage-gated calcium channel Cavalpha2delta1 subunit (C

96 ell as cacophony (cac) and paralytic (para), voltage-gated ion channels central to neuronal excitabil

97 tracellular pH via HCO3 (-) flux through the voltage-gated ClC channel CLH-1.

98 rated function of a molecular aggregate (the voltage-gated Na(+) channel complex) that includes the b

99 n 7 patients (6.3%): 3 (2.7%) had TPO-Ab and voltage-gated potassium channel complex (VGKCc) Ab, 2 (1

100 a inactivated 1 (LGI1) is a component of the voltage-gated potassium channel complex.

101 sferring the NOMPC ARs to mechanoinsensitive voltage-gated potassium channels confers mechanosensitiv

102 Here, we recorded the voltage-gated calcium channel current in nucleated patch

103 regulated by a number of factors, including voltage-gated ion channels, D2-autoreceptors, and nAChRs

104 requires a physical interaction between the voltage-gated calcium channel dihydropyridine receptor (

105 al muscle excitation-contraction coupling, a voltage-gated calcium channel directly activates opening

106 Most small-molecule inhibitors of voltage-gated ion channels display poor subtype specific

107 in a reduction in the fraction of available voltage-gated sodium channels due to insufficient recove

108 tion via GABA type A receptor activation and voltage-gated calcium channels during early postnatal de

109 KNIR-1 treatment of cells expressing voltage-gated K(+) channels enabled the visualization of

110 witches into RGCs, where they associate with voltage-gated ion channels, enabling light to control ac

111 mutations in previously unreported HVCN1, a voltage-gated proton channel-encoding gene and B-cell re

112 Kv1.3 is a voltage-gated potassium channel expressed on myelin-reac

113 KV10.1 is a voltage-gated potassium channel expressed selectively in

114 H caused a reduction in cacophony, a Type II voltage-gated calcium channel, expression and that genet

115 KCNQ (voltage-gated K(+) channel family 7 (Kv7)) channels cont

116 a gene that encodes an auxiliary protein of voltage-gated Na(+) channels, fibroblast growth factor 1

117 o acids in the S4 transmembrane segment of a voltage-gated ion channel form ion-conducting pathways t

118 Voltage-gated potassium channels formed by KCNQ2 and KCN

119 rom the symposium on the structural basis of voltage-gated K(+) channel function, as well as the mech

120 n Culex quinquefasciatus display CNV for the voltage-gated sodium channel gene (Vgsc), target-site of

121 gous loss-of-function mutations in the brain voltage-gated sodium channel gene SCN1A.

122 caused by de novo missense mutations in the voltage-gated sodium channel gene SCN8A Here, we investi

123 erity have been associated with mutations in voltage-gated sodium channel genes.

124 Here we introduce roNaV2, an engineered voltage-gated Na(+) channel harboring a selenocysteine i

125 Although a gate residue in the eukaryotic voltage-gated sodium channel has been identified, the mi

126 Slow inactivation of voltage-gated sodium channels has been discussed to be t

127 Neuronal voltage-gated calcium channels have evolved as one of th

128 Kv1.2 and related voltage-gated potassium channels have a highly conserved

129 Mutations in brain isoforms of voltage-gated sodium channels have been identified in pa

130 ate reduced form (NADPH) oxidase complex and voltage-gated proton channels (HV 1).

131 ed release of protons through either Arch or voltage-gated proton channel Hv1 activated neighbouring

132 The voltage-gated proton channel Hv1 plays a critical role i

133 we address the potentiality of targeting the voltage-gated proton channel, Hv1, as a novel strategy t

134 LGI1-IgG-positive specimens had higher voltage-gated potassium channel-IgG immunoprecipitation

135 knowledge, in vivo phosphorylation map of a voltage-gated calcium channel in a mammalian brain.

136 receptor 2 (mGluR2) signaling, which acts on voltage-gated calcium channels in SACs, selectively rest

137 sential for estimating numbers of functional voltage-gated calcium channels in the membrane and the s

138 enetic validation for the role of the Nav1.7 voltage-gated ion channel in pain signaling pathways mak

139 sion patterns and native axonal locations of voltage-gated ion channels in ANFs are unknown.

140 but difficulties in isolating the effects of voltage-gated ion channels in the AIS from those of the

141 Rather, it is mediated by voltage-gated ion channels in the cone membrane and acts

142 ons in the ryanodine receptor but not in the voltage-gated calcium channel, indicating that these phe

143 l glucose (20%) secretion was blocked by the voltage-gated Ca channel inhibitor, nifedipine, or by hy

144 Voltage-gated sodium channels initiate action potentials

145 How KCNE3 subunits turn voltage-gated KCNQ1 channels into apparent voltage-indep

146 The ether a go-go family of voltage-gated potassium channels is structurally distinc

147 The Hv1 voltage-gated proton channel is a dimeric complex consis

148 The Nav1.1 voltage-gated sodium channel is a critical contributor t

149 The NaV1.7 voltage-gated sodium channel is implicated in human pain

150 Inhibition of voltage-gated sodium channels is neuroprotective in prec

151 gia, the first human pain syndrome linked to voltage-gated sodium channels, is widely regarded as a g

152 Voltage gated Ca2+ channels, K(+)ATP channels and the al

153 The voltage-gated potassium channel Kcnh4a, which is express

154 tifier current (IKs), through the tetrameric voltage-gated ion channel, KCNQ1, and its beta-subunit,

155 Voltage-gated K(+) channels (Kv) are responsible for rep

156 hought to be due, in part, to suppression of voltage-gated potassium channels (Kv ) in pulmonary arte

157 hought to be due, in part, to suppression of voltage-gated potassium channels (Kv ) in pulmonary arte

158 FMRP has been confirmed to bind voltage-gated potassium channels (Kv 3.1 and Kv 4.2) mRN

159 Voltage-gated potassium channels (Kv), which play a role

160 n in KCNA2 (c.881G>A, p.R294H), encoding the voltage-gated K(+) -channel, KV 1.2, in two unrelated fa

161 oss-of-function or a gain-of-function of the voltage-gated K+ channel Kv1.2, were described to cause

162 sine RNA editing in transcripts encoding the voltage-gated potassium channel Kv1.1 converts an isoleu

163 4A] is a potent and selective blocker of the voltage-gated potassium channel Kv1.3, which is a highly

164 for conditions treatable by blockade of the voltage-gated potassium channel Kv1.3.

165 e the second transmembrane segment, S2, of a voltage-gated potassium channel, Kv1.3, as a model to pr

166 The voltage-gated K(+) channel Kv2.1 has been intimately lin

167 The voltage-gated K(+) channels Kv7.2 and Kv7.3 are located

168 The voltage-gated K(+) channels Kv7.2 and Kv7.3 exert strong

169 To determine whether L-type voltage-gated calcium channels (L-VGCCs) are required fo

170 The mechanism by which mutations in voltage-gated potassium channels lead to EA is still unk

171 utations in Nav.1.7, the main pain signaling voltage-gated sodium channel, lead to its truncations an

172 onin norepinephrine reuptake inhibitors, and voltage-gated calcium channel ligands in the treatment o

173 L-type voltage-gated calcium channels (LTCCs) are implicated in

174 hether manipulation of splicing of mammalian voltage-gated sodium channels may be exploitable to prov

175 Voltage-gated ion channels mediate electrical dynamics i

176 at share a subunit structure consisting of a voltage-gated K(+) channel motif coupled to a cytoplasmi

177 Axonal voltage-gated sodium channel mRNA and local trafficking

178 The tremendous therapeutic potential of voltage-gated sodium channels (Na(v)s) has been the subj

179 Voltage-gated sodium channels (Na(V)s) provide the initi

180 quantify isoflurane binding to the bacterial voltage-gated sodium channel NaChBac.

181 ABSTRACT: Voltage-gated sodium channel NaV 1.7 is required for acu

182 Thus, influx of Na(+) via voltage-gated Na(+) channels (NaV ) has emerged as an im

183 Voltage-gated Na(+) channels (Nav ) modulate neuronal ex

184 Depolarization leads to the opening of voltage-gated Na(+) channels (Nav) and subsequently volt

185 Voltage-gated sodium channel (NaV) mutations cause genet

186 metabotropic glutamate receptor-1 (mGluR1), voltage-gated sodium channels (Nav ) and glutamate trans

187 Ts3 binds to the domain IV voltage sensor of voltage-gated sodium channels (Nav ) and slows down thei

188 phibian prey, which select for TTX-resistant voltage-gated sodium channels (Nav) [12-16].

189 They are rich in voltage-gated sodium channels (Nav) and thus underpin ra

190 heless, Nfasc140, like Nfasc186, can cluster voltage-gated sodium channels (Nav) at the developing no

191 We examined the repertoire of voltage-gated sodium channels (NaV) in fluorescence-sort

192 mans and other vertebrates, target conserved voltage-gated sodium channels (NaV) of nerve and muscle,

193 Voltage-gated sodium channels (NaV) play an important ro

194 ic tension, the thermal random motion of the voltage-gated sodium channels (Nav), which are bound to

195 by de novo gain-of-function mutations in the voltage-gated Na channel Nav1.6.

196 nction mutations in the human SCN11A-encoded voltage-gated Na(+) channel NaV1.9 cause severe pain dis

197 d by loss-of-function mutations in the brain voltage-gated sodium channel NaV1.1.

198 Mutations in SCN2A, a gene encoding the voltage-gated sodium channel Nav1.2, have been associate

199 Therefore, EC cells use Scn3a-encoded voltage-gated sodium channel NaV1.3 for electrical excit

200 ed a critical role for the regulation of the voltage-gated sodium channel NaV1.5 in the heart by the

201 This is due to the voltage-gated sodium channel Nav1.7 (Scn9a), previously

202 ates that the expression and function of the voltage-gated sodium channel Nav1.7 are increased in a p

203 Mutations in the voltage-gated sodium channel Nav1.7 are linked to inheri

204 Human genetic studies have implicated the voltage-gated sodium channel NaV1.7 as a therapeutic tar

205 Voltage-gated sodium channel Nav1.7 is a central player

206 Trafficking of the voltage-gated sodium channel NaV1.7 is dysregulated in n

207 iant in the second intracellular loop of the voltage-gated sodium channel NaV1.7, encoded by the SCN9

208 Under physiological conditions, the voltage-gated sodium channel Nav1.8 is expressed almost

209 notoxins MrVIA, MrVIB, and MfVIA inhibit the voltage-gated sodium channel NaV1.8, a well described ta

210 nsfer (LRET) between the rat skeletal muscle voltage-gated sodium channel (Nav1.4) and fluorescently

211 des the alpha-subunit of the skeletal muscle voltage-gated sodium channel (Nav1.4).

212 Gain-of-function mutations in the voltage-gated sodium channel (Nav1.5) are associated wit

213 Cardiac voltage-gated sodium channels (Nav1.5) play an essential

214 ge-gated sodium channel (SCN5A gene encoding voltage-gated sodium channel [NaV1.5]) cause congenital

215 c evidence has clearly demonstrated that the voltage-gated sodium channel, Nav1.7, is critical to pai

216 Neurotransmitter release depends on voltage-gated Na(+) channels (Navs) to propagate an acti

217 Voltage-gated sodium channels (NaVs) are activated by tr

218 Voltage-gated sodium channels (Navs) play crucial roles

219 Voltage-gated sodium channels (Navs) play essential role

220 Improper function of voltage-gated sodium channels (NaVs), obligatory membran

221 results show that a major diversification of voltage-gated K(+) channels occurred in ancestral paraho

222 VopQ forms a nonspecific, voltage-gated membrane channel of 18 A resulting in neut

223 Voltage-gated potassium channels of the KCNQ (Kv7) subfa

224 Tp1a had no effect on human voltage-gated calcium channels or nicotinic acetylcholin

225 KEY POINTS: Voltage-gated sodium channels play a fundamental role in

226 this was attributable to interdependence of voltage-gated potassium channel properties.

227 as used to quantify postsynaptic density and voltage-gated calcium channel protein expression.

228 Hundreds of mutations in genes encoding voltage-gated ion channels responsible for action potent

229 molecular methods to determine how Kv3.4, a voltage-gated K(+) channel robustly expressed in dorsal

230 Mutations of the cardiac voltage-gated sodium channel (SCN5A gene encoding voltag

231 tiated and propagated by a single isoform of voltage gated sodium channels - SCN5A.

232 euritis at concentrations at which it blocks voltage-gated sodium channels selectively.

233 CaV1.1e is the voltage-gated calcium channel splice variant of embryoni

234 The voltage-gated potassium channel subfamily A member 3 (Kv

235 Drug block of voltage-gated potassium channel subtype 11.1 human ether

236 Specifically, voltage-gated sodium channel subtype NaV 1.7 is required

237 Neuron, Tedeschi et al. (2016) describe the voltage-gated calcium channel subunit alpha2delta2 as a

238 ched comparison subjects as well as aberrant voltage-gated calcium channel subunit protein expression

239 The effect of increased voltage-gated calcium channel subunit protein expression

240 Nerve injury-induced downregulation of voltage-gated potassium channel subunit Kcna2 in the dor

241 xpression of a human-specific isoform of the voltage-gated sodium channel subunit SCN4B was significa

242 mechanical allodynia by regulating auxiliary voltage-gated calcium channel subunits alpha2delta-1 and

243 KCNQ2) and Kv7.3 (KCNQ3) genes, encoding for voltage-gated K(+) channel subunits underlying the neuro

244 They belong to the voltage-gated ion channel superfamily but their activiti

245 The channel domain exhibits the voltage-gated ion channel superfamily fold with distinct

246 ng human CaV3.3 alpha1 subunit, a subtype of voltage-gated calcium channel that contributes to T-type

247 he mechanism of dopamine activation requires voltage-gated calcium channels that are also present at

248 sights into the function and pharmacology of voltage-gated ion channels that have emerged from unnatu

249 is composed of diverse animal-specific, non-voltage-gated ion channels that play important roles in

250 ), studied here, is caused by mutations in a voltage-gated potassium channel that contributes to the

251 KCNQ1 is a voltage-gated potassium channel that is modulated by the

252 ein called ether a go-go, EAG1 or KV10.1), a voltage-gated potassium channel that is predominantly ex

253 Kv11.1 (hERG) is a voltage-gated potassium channel that shows very slow ion

254 ed and likely to have evolved from ancestral voltage-gated sodium channels that are widely expressed

255 In the modular voltage-gated ion channels the central ion-conducting po

256 that heterologously expressed human cardiac voltage-gated sodium channel, the principle cardiac sodi

257 For all the subtypes of voltage-gated calcium channel, their gating properties a

258 y fiber terminals leverage distinct types of voltage-gated calcium channels to mediate short-term fac

259 knockdown, alter splicing of the Drosophila voltage-gated sodium channel to favour inclusion of exon

260 ature of the paddle motif in all three major voltage-gated ion channel types (Kv, Nav, and Cav).

261 les, and reduced fusion triggered by opening voltage-gated calcium channels under voltage clamp, with

262 mal motile cilia is not tightly regulated by voltage-gated calcium channels, unlike that of well-stud

263 depend on NMDA receptors (nmdaLTP) or L-type voltage-gated calcium channels (vdccLTP).

264 increase caused by NMDA-receptor (NMDAR) and voltage-gated Ca2+ -channel (VGCC) activation is thought

265 al for exocytosis, also controls presynaptic voltage-gated calcium channel (VGCC) function dictating

266 he final coding exon (exon 47) of the Cav2.1 voltage-gated calcium channel (VGCC) gene produces two m

267 P/Q-type voltage-gated calcium channel (VGCC) puncta colocalized

268 located in an intronic region of the L-type voltage-gated calcium channel (VGCC) subunit gene CACNA1

269 emphasized the importance of the T-type low-voltage-gated calcium channels (VGCC) in different cance

270 ddition to Gbetagamma-mediated modulation of voltage-gated calcium channels (VGCC), inhibition can al

271 t for gating currents is well documented for voltage-gated cation channels (VGCC), and it is consider

272 allow somatic action potentials to activate voltage-gated calcium channels (VGCCs) along the entire

273 fied their ability to activate L- and N-type voltage-gated calcium channels (VGCCs) and delineated th

274 tivity-dependent potentiation of presynaptic voltage-gated calcium channels (VGCCs) underlies 3,4-dia

275 erties, density, and the spatial location of voltage-gated calcium channels (VGCCs).

276 These dimeric voltage-gated ion channel (VGIC) superfamily members hav

277 Voltage-gated ion channels (VGICs) are outfitted with di

278 agnetic resonance imaging has linked chronic voltage-gated potassium channel (VGKC) complex antibody-

279 ies against the extracellular domains of the voltage-gated potassium channel (VGKC) complex proteins,

280 ped redefine antigenic components within the voltage-gated potassium channel (VGKC) complex.

281 Antibodies against the voltage-gated potassium channel (VGKC) were first recogn

282 ceptor (NMDAR), the glycine receptor (GlyR), voltage-gated potassium channel (VGKC)-complex and the a

283 otein-like 2 in 11 patients, uncharacterised voltage-gated potassium channel (VGKC)-complex antigens

284 osequencing genotyping and sequencing of the voltage gated sodium channel (VGSC) gene did not detect

285 Voltage-gated sodium channel (VGSC) beta subunits signal

286 Veratridine (VTD) is a voltage-gated sodium channel (VGSC) modifier that is use

287 Voltage-gated sodium channel (VGSC) mutations cause seve

288 agnitude shorter than the activation time of voltage-gated sodium channels (VGSC) would evoke action

289 s to efficiently overexpress large mammalian voltage-gated sodium channels (VGSC).

290 f structural and functional relationships of voltage-gated sodium channels (VGSC).

291 Functional expression of voltage-gated Na(+) channels (VGSCs) has been demonstrat

292 ies of myelinated fibres, however, show that voltage-gated sodium channels (VGSCs) aggregate with cel

293 KCNQ2/3 (Kv7.2/7.3) channels and voltage-gated sodium channels (VGSCs) are enriched in th

294 Voltage-gated sodium channels (VGSCs) are responsible fo

295 We have recently demonstrated that voltage-gated sodium channels (VGSCs) in dorsal root gan

296 Voltage-gated sodium channels (VGSCs) regulate invasion

297 Clustering of voltage-gated sodium channels (VGSCs) within the neurona

298 Kidins220 association with brain voltage-gated sodium channels was shown by co-immunoprec

299 Subcellular positioning of voltage-gated ion channels will enable multicompartmenta

300 KCNE3 (MiRP2) forms heteromeric voltage-gated K(+) channels with the skeletal muscle-exp