ライフサイエンスコーパス: volume index

1 ardiac performance (cardiac index and stroke volume index).

2 study provides reference values of normal LA volume index.

3 itus and smoking were not associated with LA volume index.

4 y resistance index and an increase in stroke volume index.

5 istance, while increasing cardiac and stroke volume index.

6 of arterial afterload were related to stroke volume index.

7 ncrease in the left ventricular end-systolic volume index.

8 ing was defined as change in LV end-systolic volume index.

9 e, and larger left ventricular end-diastolic volume index.

10 -diastolic volume index, and LV end-systolic volume index.

11 P<0.05) ethnicities were associated with LA volume index.

12 x, and larger left ventricular end-diastolic volume index.

13 stolic ventricular volume indexes and stroke volume index.

14 in cardiac performance as measured by stroke volume index.

15 oint was change in left ventricular systolic volume index.

16 ic dysfunction (E/e') and 5 with left atrial volume index.

17 every 1 mL/m(2) increase in LV end systolic volume index.

18 riation monitoring, and global end-diastolic volume index.

19 ments in both LV end-systolic and -diastolic volume indexes.

20 end-diastolic pressure and left ventricular volume indexes.

21 ; 95% CI, -0.008-0.014; p = .571) and stroke volume index (0.17 mL/m(2); 95% CI, -0.03-0.37; p = .094

22 iated with higher mean pulse pressure/stroke volume index (1.24 and 1.15 versus 1.02 mm Hg/mL x m2) a

23 54 +/- 9%, p < 0.0001) and LV end-diastolic volume index (108 +/- 28 ml/m(2) to 78 +/- 24 ml/m(2), p

24 ection fraction (11% versus 3%), left atrial volume index (-11.9 versus -4.7 mL/m(2)), and right vent

25 e (79 pg/mL), left ventricular end-diastolic volume index (110 mL/m2), and left ventricular ejection

26 jection fraction, 23+/-9%; mean end-systolic volume index, 113+/-48 mL; mean total myocardial scar %,

27 The decrease in end-diastolic volume index (14+/-3) was different from that seen at ba

28 mendan increased cardiac index (22%), stroke volume index (15%), and heart rate (7%) and decreased sy

29 6.7+/-6.9 mL/m(2); P<0.001) and right atrial volume index (15.66+/-3.09 versus 20.47+/-4.82 mL/m(2);

30 .21 +/- 0.07, left ventricular end-diastolic volume index 180 +/- 64 ml/m2.

31 Left atrial volume index (24.0+/-3.6 versus 26.7+/-6.9 mL/m(2); P<0.

32 mprovement in left ventricular end-diastolic volume index (-26.2 versus -7.4 mL/m(2)), left ventricul

33 mm Hg vs. 10 +/- 5 mm Hg, p < 0.001) and LA volume index (28 +/- 12 ml/m(2) vs. 21 +/- 14 ml/m(2), p

34 -7.4 mL/m(2)), left ventricular end-systolic volume index (-28.7 versus -9.1 mL/m(2)), left ventricul

35 MR patients had larger left atria (volume index: 32 cm(3)/m(2) vs. 26 cm(3)/m(2), p = 0.008

36 itus (25% versus 41%, P=0.009), lower stroke volume index (36.4+/-8.4 versus 34.4+/-8.7 mL/m2, P=0.02

37 lar velocity greater than 10 and left atrial volume index 40 mL/m2 or more were associated with a 3.4

38 ; P<0.001) and had increased RV end-systolic volume index (43 versus 35 mL/m2; P=0.03), decreased RV

39 The median E/e' (16), left atrial volume index (44 mL/m2), and pulmonary artery systolic p

40 d dilated right-sided chambers (right atrial volume index, 44 +/- 19 mL/m(2); RV end-diastolic area,

41 th LF compared with those with normal stroke volume index (47% versus 34%; hazard ratio, 1.5; 95% con

42 minute; P<0.001) and to reduced peak stroke volume index (47+/-10 mL/min per m(2) versus 54+/-15 mL/

43 2), as did the left ventricular end-systolic volume index (48.4 +/- 19.7 ml/m(2) vs. 43.1 +/- 16.2 ml

44 -62 ms; P<0.0001), and increased left atrial volume index (49+/-18 versus 42+/-15 mL/m2; P=0.02).

45 ence interval, 0.2-0.5; P=0.0001) and stroke volume index (5.2 mL.m(-2); 95% confidence interval, 2.0

46 icant reduction of left ventricular systolic volume index (-5.8%, P=0.017), and noninfarct myocardial

47 9), HFrEF patients had larger LA volumes (LA volume index 50 versus 41 mL/m(2); P<0.001), whereas HFp

48 ose with persistent AF had larger maximum LA volume index (56+/-17 versus 49+/-13 mL/m(2); P=0.036),

49 nd significant reductions in LV end systolic volume index (-6.7 +/- 21.1 versus 2.1 +/- 17.6 mL/m(2);

50 mposite score, left ventricular end systolic volume index, 6-minute walk time, and quality of life in

51 59% specificity), change in pulmonary blood volume index (77% sensitivity, 82% specificity), and cha

52 45%; P<0.001) vascular compliance and stroke volume index (8%; P=0.01).

53 +/- 33 g/m2; P < 0.001) and LV end-diastolic volume index (82 +/- 35 versus 34 +/- 16 ml/m2; P < 0.00

54 tivity, 98% specificity) and pulmonary blood volume index (92% sensitivity, 68% specificity), and by

55 ients with a right ventricular end-diastolic volume index above 150 ml/m(2) and peak exercise systoli

56 ients with a right ventricular end-diastolic volume index above 150 ml/m(2) and peak exercise systoli

57 h (P < 0.01): right ventricular end-systolic volume index adjusted for age and sex, and the relative

58 essure, and left ventricular and left atrial volume index (all P<0.001) were more benign in mid-late

59 aller baseline left ventricular end systolic volume index also were also associated with better survi

60 in the MMF-withdrawal group, the left atrial volume index (an indicator of chronic LV diastolic dysfu

61 alysis showed that left ventricular systolic volume index and %MDE(periphery) were the strongest pred

62 rom baseline right ventricular end-diastolic volume index and a 429 ml (P < 0.001) reduction in resid

63 ase (least square mean+/-SE) in end-systolic volume index and a 6+/-1% increase in left ventricular e

64 and left ventricular function, end-systolic volume index and B-type natriuretic peptide were most st

65 t the patients with LGSAS had reduced stroke volume index and cardiac index (P=0.003 for both).

66 renal dysfunction, yet smaller end-diastolic volume index and cardiac output and increased EDP compar

67 loading correlated well with initial stroke volume index and changes in stroke volume index, respect

68 We measured stroke volume index and collapsibility index of the inferior ve

69 LA volume index and E/e' predicted exercise PCWP>30 mm Hg w

70 Left ventricle end-diastolic volume index and end-systolic volume index were reduced

71 After adjustment for LV volume index and functional class, patients with LGE had

72 ression model, left ventricular end-systolic volume index and left atrial volume index were independe

73 Right ventricular end-systolic volume index and left ventricular strain-rate e'-wave pr

74 is associated with reduced LV end-diastolic volume index and LV end-diastolic mass index in a large

75 eater reduction of LV systolic and diastolic volume index and LV mass index as compared with placebo.

76 LV end-diastolic volume index, end-systolic volume index and LVEF between diabetic patients and heal

77 In the proximal stent segment, stent volume index and minimum stent area were larger after PO

78 combined with right ventricular end-systolic volume index and strain-rate e'-wave in the multivariate

79 nship between left ventricular end-diastolic volume index and stroke volume index was confirmed in gr

80 , and waveform analysis of changes in stroke volume index and systemic vascular resistance index were

81 . 37.7 +/- 15.4; p = .03), plus lower stroke volume index and worse cardiac function with higher left

82 duction in LV end-diastolic and end-systolic volume indexes and a 3-fold greater increase in LV eject

83 contrast, initial end-diastolic ventricular volume indexes and changes in end-diastolic ventricular

84 contrast, initial end-diastolic ventricular volume indexes and changes in these ventricular volume i

85 dial function, as suggested by higher stroke volume indexes and left ventricular stroke work indexes

86 pressure and both end-diastolic ventricular volume indexes and stroke volume index.

87 ssure, heart rate, cardiac index, and stroke volume index) and metabolic data (serum blood urea nitro

88 pressure, 0.56 for the global end-diastolic volume index, and 0.64 for the left ventricular end-dias

89 ociated with lower ejection fraction, stroke volume index, and aortic valve mean gradient up to 3 yea

90 in cardiac index, change in pulmonary blood volume index, and change in PaO2/FIO2 ratio individually

91 in cardiac index, change in pulmonary blood volume index, and change in PaO2/FIO2 ratio were lower i

92 index, relative wall thickness, left atrial volume index, and deceleration time were still associate

93 ution such as cardiac index, pulmonary blood volume index, and extravascular lung water.

94 vements in left ventricular (LV) mass index, volume index, and fractional shortening were seen in 48,

95 on, including severe tachycardia, low stroke volume index, and high inferior vena cava collapsibility

96 id therapy was directed by maximizing stroke volume index, and in the study group, stroke volume inde

97 age, elevated creatinine, larger left atrial volume index, and larger left ventricular end-diastolic

98 entral venous pressure, global end-diastolic volume index, and left ventricular end-diastolic area in

99 lume, LV ejection fraction, LV end-diastolic volume index, and LV end-systolic volume index.

100 e reductions in cardiac output/index, stroke volume index, and oxygen delivery index and increases in

101 ntricular mechanical delay, the end-systolic volume index, and the area of the mitral regurgitant jet

102 s of E/e' ratio, and decrease in left atrial volume index as compared with placebo.

103 volume index, and in the study group, stroke volume index assessed prior to severe acute pancreatitis

104 h a regime using individual values of stroke volume index assessed prior to severe acute pancreatitis

105 natriuretic peptide, and larger left atrial volume index at HFpEF diagnosis compared with sinus rhyt

106 sus 7.0 +/- 3.7 mm, P=0.002), a larger lipid volume index [averaged lipid arcxlipid length] (1605.5 +

107 g (B=-0.1; P=0.0001), lower LV end-diastolic volume index (B=0.6; P=0.0001), and lower LV end-diastol

108 ty (beta=1.3 mL/m(2), P<0.01), end-diastolic volume index (beta=0.4 mL/m(2), P<0.0001), Chinese Ameri

109 ular reconstruction reduced the end-systolic volume index by 19%, as compared with a reduction of 6%

110 e cardiac index by 24 +/- 11% and the stroke volume index by 23 +/- 12% (p < 0.05).

111 3DE LA volumes indexed by body surface area were similar in men

112 , mean arterial pressure, heart rate, stroke volume index, cardiac output/index, left ventricular str

113 central venous pressure and change in stroke volume index/cardiac index and the percentage of fluid r

114 central venous pressure and change in stroke volume index/cardiac index was 0.18 (95% CI, 0.1-0.25),

115 This new total right/left-volume index correlated with almost all clinically used

116 denervation, left ventricular end-diastolic volume index, creatinine, and no angiotensin inhibition.

117 1 to 12.0+/-3.3 mm/mm, P=0.01), as did lumen volume index (DA+, 13.2+/-3.1 to 10.5+/-2.7 mm/mm, P<0.0

118 a cross-sectional analysis of Google search volume index data and US cancer incidences and mortaliti

119 The left ventricular end-diastolic volume index decreased (90.1 +/- 28.2 ml/m(2) at baselin

120 Stroke volume index decreased by 7.0 ml/m2 (p < 0.0005) with MT

121 +/- 12.4%, and left ventricular end systolic volume index decreased from 109 +/- 71 to 69 +/- 42 ml/m

122 Left ventricular mass index and left atrial volume index decreased from 122.6 +/- 42.6 to 98.5 +/- 3

123 In both groups vessel volume index decreased significantly (DA+, 16.3+/-3.6 to

124 LV end-diastolic and end-systolic volume indexes decreased in patients with CRT turned on

125 50-mg group, end-systolic and end-diastolic volume indexes decreased relative to baseline but were n

126 triuretic peptide, ejection fraction, stroke volume index, E/E', and left ventricular mass index (haz

127 the diastolic filling period, end-diastolic volume index (EDVI) fell substantially more in the older

128 ratio and preceding changes in end-diastolic volume indexed (EDVi) to body surface area and the eject

129 iovascular responses (cardiac output, stroke volume index, ejection fraction, peak systolic pressure/

130 ificant differences in mean LV end-diastolic volume index, end-systolic volume index and LVEF between

131 xide was quantitatively predictive of stroke volume index estimated by transesophageal echocardiograp

132 73 +/- 8 ml/m(2), p = 0.03) and end-systolic volume index (ESVI) 20 +/- 6 ml/m(2) vs. 17 +/- 4 ml/m(2

133 EF below the median (27%), and end-systolic volume index (ESVI) above the median (79 ml/m(2)).

134 ricular ejection fraction (EF), end-systolic volume index (ESVI) and infarct size (IS), as measured i

135 3D wall motion tracking for RV end-systolic volume index (ESVi), RV ejection fraction (EF), and RV g

136 outcomes could be modulated by end-systolic volume index (ESVi).

137 vely characterized EaI/E(LV)I = end-systolic volume index (ESVI)/stroke volume index (SVI) and its tw

138 etween examinations 1 and 5) in end-systolic volume indexed (ESVi) to body surface area.

139 pressure, and left ventricular end-diastolic volume index failed to correlate significantly with chan

140 ed LV strain rates and LVES wall stress/LVES volume index following MV repair indicate contractile dy

141 difference in the change in LV end-diastolic volume index from days 0 to 90 with PG-116800 versus pla

142 in/m(2); P = .001), and global end-diastolic volume index (GEDVI) (726 vs 775 mL/m(2); P = .003) incr

143 measurements included cardiac index, stroke volume index, global and regional contractile function (

144 rest and during exercise in patients with LA volume index >/=35 mL/m(2) (P<0.01), despite similar car

145 into 2 groups according to left atrial (LA) volume index >/=35 mL/m(2).

146 , RAP >10 mm Hg, sPAP >40 mm Hg, left atrial volume index >33 ml/m(2), ratio of mitral inflow early d

147 vided into those with DD (MI+DD; left atrial volume index >34 mL/m(2) and diastolic E/e' ratio>8; n=3

148 lar in 125 patients with normal flow (stroke volume index >35 mL/m2; P=0.22).

149 re defined in CG-AS with normal flow (stroke-volume-index >35 ml/m(2)).

150 aortic valve area (<1 cm(2)), normal stroke volume index (>/=35 mL/m(2)), and either high mean gradi

151 on (increased right ventricular end-systolic volume index), high Acute Physiology and Chronic Health

152 a 13% reduction in left ventricular systolic volume index in comparison with the lowest quartile.

153 ic blood pressure, cardiac index, and stroke volume index in this pediatric porcine model of hemorrha

154 ume indexes and changes in these ventricular volume indexes in response to 3 L of normal saline loadi

155 xes and changes in end-diastolic ventricular volume indexes in response to saline loading correlate s

156 olume expansion, a relevant (>/= 10%) stroke volume index increase was recorded in 56% patients.

157 Resting left ventricular end-diastolic volume index increased (P=0.001) within the sildenafil g

158 Stroke volume index increased after first fluid bolus in 80% of

159 Left atrium volume index increased during CNI (46.73+/-16.3 5-54.20+

160 During the first year after Tx, plaque volume index increased significantly in DA+ group, but d

161 Increased LA volume index is a powerful predictor of mortality after

162 LV end systolic volume index is a significant predictor of mortality/mor

163 sex (both P< or =0.01), were LV end-systolic volume index, LA volume, atrial fibrillation, and sympto

164 tients were divided according to left atrial volume index (LAVi) >/=32 ml/m(2).

165 al longitudinal strain (GLS) and left atrial volume index (LAVI) have been recently proposed as novel

166 e was to evaluate whether normal left atrial volume index (LAVI) is a predictor of a normal stress ec

167 al annular velocity (S, E'), and left atrial volume index (LAVI).

168 .04-1.43; P=0.015) and increased left atrial volume index (LAVi; adjusted hazard ratio/unit increase,

169 es on ECG, and left ventricular end-systolic volume index, LGE maintained a >4-fold hazards increase

170 body mass index, diabetes, LV end-diastolic volume index, LGE, EF) (hazard ratio = 2.051 per mm decr

171 mportant subsets of patients with low stroke volume index (low flow) and low-gradient with reduced (c

172 evaluable echocardiograms (92%), LF (stroke volume index </=35 mL/m(2)) was observed in 530 (55%); L

173 d gradient: low flow was defined as a stroke volume index </=35 mL/m(2), low gradient as a mean gradi

174 ears, 90% for left ventricular end-diastolic volume index <100 mL/m(2) versus 48% for >/=100 mL/m(2))

175 35) and those without DD (MI-DD; left atrial volume index <34 mL/m(2) and E/e' ratio<8; n=11).

176 including paradoxical low-flow (i.e., stroke volume index <35 ml/m(2)), low-gradient (LF-LG) and norm

177 r ejection fraction [LVEF] >/=50% but stroke volume index <35 ml/m(2)), low-gradient (mean gradient [

178 hort identified preoperative RV end-systolic volume index <90 mL/m(2) and QRS duration <140 ms to be

179 Patients were stratified by stroke volume index (<35 mL/m(2) [low flow, LF] versus >/=35 mL

180 g water (<10 mL/kg) and global end-diastolic volume index (<850 mL/m) in the transpulmonary thermodil

181 ients were hypovolemic (global end-diastolic volume index<680 mL/m) on enrollment.

182 ean change from baseline in LV end-diastolic volume index (LVEDVI) at 6 months.

183 LV mass index, aortic valve area, and stroke volume index, LVEF was independently predictive of morta

184 point was the left ventricular end-systolic volume index (LVESVI) at 12 months, as assessed with the

185 The left ventricular end-systolic volume index (LVESVI) decreased from 80.4 +/- 51.4 ml/m(

186 crease in left ventricular (LV) end-systolic volume index (LVESVI) of more than 10% from 1 week to 6

187 ference in the left ventricular end-systolic volume index (LVESVI) or survival after 1 year.

188 atients with a left ventricular end-systolic volume index (LVESVI) reduction of at least 15% were con

189 ta analysis of left ventricular end-systolic volume index (LVESVi) was performed to adjust for the co

190 l changes in MR severity and LV end-systolic volume index (LVESVi) were evaluated by linear mixed-mod

191 point was the left ventricular end-systolic volume index (LVESVI), a measure of left ventricular rem

192 ference in the left ventricular end-systolic volume index (LVESVI), survival, or adverse events at 1

193 In placebo patients, postintervention lumen volume index (LVI) was significantly greater in re-stent

194 Lung weight index (LWI), lung volume index (LVI), and alveolar cell proliferation inde

195 Right ventricular end-diastolic volume index measured by means of cardiovascular magneti

196 Left atrial volume index might be a simple means of identifying pati

197 ting for clinical risk factors, end-systolic volume index, mitral regurgitation, incomplete revascula

198 carotid intima-media thickness, left atrial volume index, monocyte count and serum YKL-40 levels.

199 he CABG group: left ventricular end-systolic volume index, MR volume, and plasma B-type natriuretic p

200 d the population was divided according to LA volume index of 32 mL/m2 (2 SDs above normal).

201 stemic vascular resistance index, and stroke volume index on days 3-7.

202 nts with increased preoperative end-systolic volume index or B-type natriuretic peptide.

203 ostic value beyond left ventricular systolic volume index or ejection fraction.

204 antly with initial end-diastolic ventricular volume indexes or cardiac performance (cardiac index and

205 te with changes in end-diastolic ventricular volume indexes or cardiac performance.

206 1, P=0.31) or end-diastolic (r=0.10, P=0.38) volume indexes or LV ejection fraction (r=0.07, P=0.72).

207 p = 0.035), and smaller baseline left atrial volume index (OR: 1.47; p < 0.001).

208 (p < .01), and a 33% decrease in the stroke volume index (P < .01).

209 x (p < 0.001; 95% CI, 0.97-0.99), and stroke volume index (p < 0.01; 95% CI, 0.96-0.99) in predicting

210 ce index (P < .01), right ventricular stroke volume index (P </= .01), and pulmonary artery capacitan

211 th changes in left ventricular end-diastolic volume index (p = 0.26), LVESVI (p = 0.41), or left vent

212 volume index (P=0.007), and LV end-systolic volume index (P< or =0.0001).

213 0.001 and P=0.0007, respectively), RV stroke volume index (P<0.0001), and left ventricular end-diasto

214 n reduction in left ventricular end-systolic volume index (P<0.0001), whereas non-LBBB patients had s

215 reduction in left ventricular end-diastolic volume index (P<0.001), with a concomitant small stable

216 rease in the right ventricular end-diastolic volume index (P<0.001).

217 ween noninvasive and invasive AVA was stroke volume index (P<0.01).

218 eased heart rate (P(group)=0.01), and stroke volume index (P(group)=0.004) compared with TTM36.

219 ejection fraction (P=0.001), LV end-systolic volume index (P=0.0006), or segmental WMA (P=0.002).

220 oke volume index (P=0.015) and end-diastolic volume index (P=0.001) decreased during exercise.

221 <0.0001), and left ventricular end-diastolic volume index (P=0.0015).

222 ncreased cardiac index (P<0.0001) and stroke volume index (P=0.003), especially at high-intensity exe

223 ll motion score (P=0.0007), LV end-diastolic volume index (P=0.007), and LV end-systolic volume index

224 Stroke volume index (P=0.015) and end-diastolic volume index (P

225 lar velocity (P=0.02), increased left atrial volume index (P=0.05), and lower mean arterial pressure

226 7 mL/m(2) at 3-year follow-up (end-diastolic volume index, P=0.0056; end-systolic volume index, P=0.4

227 astolic volume index, P=0.0056; end-systolic volume index, P=0.4719).

228 ly, arterial pressure, cardiac index, stroke volume index, pH, and creatinine were all significantly

229 The stroke volume index produced by precordial compression averaged

230 us pressure, right ventricular end-diastolic volume index, pulmonary artery occlusion pressure, and l

231 The coronary plaque volume index (PVI) was determined by dividing the wall v

232 = 0.17; p = .001), baseline pulmonary blood volume index (r = 0.15; p = .001), change in pulmonary b

233 = 0.15; p = .001), change in pulmonary blood volume index (r = 0.16; p < .001), and change in PaO2/FI

234 rbon dioxide was highly predictive of stroke volume index (r =.88, p <.001) with a mean bias of 0.003

235 c volume index (r=0.62, P<0.01), left atrial volume index (r=0.41, P<0.05) but lower left ventricular

236 with increased left ventricular end-systolic volume index (r=0.62, P<0.01), left atrial volume index

237 ocardial 3-nitrotyrosine content with stroke volume indexes (r = -.701; p = .004).

238 ndex: r = 0.35, p < 0.0001; LV end-diastolic volume index: r = 0.43, p < 0.0001) and LVEF (r = -0.66,

239 volume measurements in CMR: total right/left-volume index=(RA+aRV+fRV)/(LA+LV).

240 R interval, p < 0.0001), as LVES stress/LVES volume index ratio was depressed at baseline and followi

241 End-systolic volume index remained unchanged (P=0.8).

242 Automated vessel wall volume index remained unchanged from baseline to follow-

243 al stroke volume index and changes in stroke volume index, respectively.

244 ac index and heart rate and a reduced stroke volume index response.

245 he MRI-derived left ventricular end-systolic volume index, RV, and OMR category (severe versus modera

246 Our data suggest that the total right/left-volume index should be used as a new and simplified CMR

247 A measure of flow (stroke volume index) should be included in the evaluation and t

248 CMR-derived end-diastolic volume index showed a weaker association with outcome (p

249 on, both groups had similar LV end-diastolic volume indexes, stroke volumes, FS, circumferential ESS,

250 ter to 116.7 +/- 2.7% of B, P = 0.02; stroke volume index (SV(index)) to 173.1 +/- 40.1% of B, P = 0.

251 )I = end-systolic volume index (ESVI)/stroke volume index (SVI) and its two determinants EaI = end-sy

252 tional class, 6-minute walk distance, stroke volume index (SVI), and right atrial pressure were indep

253 -one subjects (25 with low flow [LF], stroke volume index [SVI] </=35 ml/m(2), 16 with normal flow [N

254 Low flow (i.e., reduced stroke volume index [SVi]) can occur with both reduced and pres

255 total peripheral resistance index and stroke volume index tended to decrease and increase, respective

256 extent of LGE and a higher LV end-diastolic volume index than other groups, but levels of inflammato

257 line cardiac index, baseline pulmonary blood volume index, the change in cardiac index, change in pul

258 For stroke volume index, the median AUC for esmolol was 4 mL/m2 (IQ

259 Left atrial volume indexed to body surface area (LA index) was 55 +/

260 n between diastolic function and left atrial volume indexed to body surface area (LAVi) in a populati

261 PLF as defined by LVEF of >/=50% but stroke volume indexed to body surface area (SVi) of </=35 ml m(

262 METHODS AND LA volume indexed to body surface area was measured by card

263 Left ventricular EDV and end-systolic volume indexed to body surface area were smaller in wome

264 oxygenation index, dynamic compliance, tidal volume indexed to body weight of a spontaneous breath, f

265 mal negative inspiratory pressure, and tidal volume indexed to body weight of a spontaneous breath.

266 r left ventricular (LV) mass and left atrial volume indexed to height(2.7) in both men and women (P<0

267 reduced aortic valve area and normal stroke volume index undergoing AVR underwent echocardiography,

268 baseline in right ventricular end-diastolic volume index versus placebo.

269 lumetric data were standardized by length as volume index (VI).

270 In 25 patients (64%) LA volume index was >/=35 mL/m(2).

271 LA volume index was >32 mL/m2 in 142 (45%).

272 Mean total right/left-volume index was 2.6+/-1.7 (normal values: 1.1+/-0.1).

273 The mean LA volume index was 36.5+/-11.4 mL/m(2) in the entire cohor

274 whereas left ventricular end-systolic (LVES) volume index was 60% above normal pre- and post-MV repai

275 nflow velocities, RAP, sPAP, and left atrial volume index was 90% accurate in distinguishing normal f

276 LA volume index was a powerful predictor of mortality and r

277 LV end systolic volume index was also evaluated as a predictor of mortal

278 Left atrial volume index was categorized as < or =28 ml/m2 (normal),

279 icular end-diastolic volume index and stroke volume index was confirmed in group 2 subjects using mat

280 A total right/left-volume index was defined from end-diastolic volume measu

281 End-diastolic volume index was determined by echo Doppler.

282 Left atrial volume index was measured retrospectively with the bipla

283 p < 0.05), but left ventricular end-systolic volume index was not different (p = 0.49).

284 LV end systolic volume index was predictive of mortality/morbidity; the

285 The difference in left atrium (Delta LA) volume index was significant (P=0.002) and was not assoc

286 argest increases in stroke volume and stroke volume index were during zero-resistance cycling.

287 Baseline cardiac index and pulmonary blood volume index were higher, whereas change in cardiac inde

288 ar end-systolic volume index and left atrial volume index were independent predictors of extracellula

289 Cardiac index and stroke volume index were lower in the untreated MI group than i

290 jection fraction and left ventricular stroke volume index were most strongly predictive of survival o

291 end-diastolic volume index and end-systolic volume index were reduced from 128.4+/-22.1 and 94.9+/-2

292 Changes in the left atrial volume index were significantly associated with treatmen

293 ic blood pressure, cardiac index, and stroke volume index were significantly greater during ITD-assis

294 Plaque, lumen, and vessel volume indexes were calculated by volume/measured length

295 During Ex plus expiratory load, stroke volume indexes were reduced to baseline, non-exercise le

296 function (based on e', E/e', and left atrial volume index) were each independently and additively ass

297 ic blood pressure, cardiac index, and stroke volume index, when the ITD alone and ITD plus positive e

298 ts of such consequent maximization of stroke volume index with a regime using individual values of st

299 r of change in left ventricular end-systolic volume index with monotonic increases as QRS duration pr

300 Doxycycline improved cardiac and stroke volume index with no chronotropic effect in doxycycline-